Search results for " inhibition"

showing 10 items of 435 documents

Characterization of somatostatin- and cholecystokinin-immunoreactive periglomerular cells in the rat olfactory bulb.

2005

Periglomerular cells (PG) are interneurons of the olfactory bulb (OB) that modulate the first synaptic relay of the olfactory information from the olfactory nerve to the dendrites of the bulbar principal cells. Previous investigations have pointed to the heterogeneity of these interneurons and have demonstrated the presence of two different types of PG. In the rat OB, type 1 PG receive synaptic contacts from the olfactory axons and are γ-aminobutyric acid (GABA)-ergic, whereas type 2 PG do not receive synaptic contacts from the olfactory axons and are GABA immunonegative. In this study, we analyze and characterize neurochemically a group of PG that has not been previously classified either …

Olfactory systemCalbindinsNeuropilOlfactory NervePresynaptic TerminalsSynaptic MembranesNeuropeptideOlfactionBiologyCalbindinSynaptic TransmissionS100 Calcium Binding Protein GOlfactory nerveMicroscopy Electron TransmissionInterneuronsNeural PathwaysNeuropilmedicineAnimalsRats Wistargamma-Aminobutyric AcidGeneral NeuroscienceNeural InhibitionImmunohistochemistryOlfactory BulbOlfactory bulbRatsSmellmedicine.anatomical_structurenervous systemFemaleCalretininCholecystokininSomatostatinNeuroscienceThe Journal of comparative neurology
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Nitric oxide synthase containing periglomerular cells are GABAergic in the rat olfactory bulb.

2003

In the olfactory glomeruli of the rat olfactory bulb, there is a population of periglomerular cells (PG) that contains the neuronal isoform of the nitric oxide synthase (nNOS). To date, these PG have not been characterized neurochemically and it has not been determined whether they are type 1 (GABAergic PG that receive synaptic contacts from the olfactory axons) or type 2 PG (non-GABAergic PG that do not receive synapses from the olfactory axons). Combining pre-embedding NADPH-diaphorase histochemistry and post-embedding immunoperoxidase detection of GABA, we demonstrate that nNOS-containing PG are GABAergic and therefore, belong to the type 1 PG. The possible actions of nitric oxide in the…

Olfactory systemmedicine.medical_specialtyOlfactory NervePopulationCentral nervous systemPresynaptic TerminalsOlfactionNitric OxideSynaptic TransmissionOlfactory nerveInternal medicinemedicineAnimalsRats Wistareducationgamma-Aminobutyric AcidNeuronseducation.field_of_studybiologyChemistryGeneral NeuroscienceNeural InhibitionImmunohistochemistryOlfactory BulbCell biologyOlfactory bulbRatsNitric oxide synthaseEndocrinologymedicine.anatomical_structurenervous systemSynapsesbiology.proteinGABAergicNitric Oxide SynthaseNeuroscience letters
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Mechanisms of Raf-1 Kinase Inhibitor Protein Dysregulation in Triple-Negative Breast Cancers and Identification of Possible Novel Therapeutic Approac…

2014

Triple-negative breast cancers (TNBCs) are a heterogenous group of breast cancers characterized by poor prognosis because they are not amenable to targeted therapies. We have taken into account that altered expression of Raf-1 kinase inhibitor protein (RKIP), a tumor and metastasis suppressor and a promoter of drug-induced apoptosis, is frequent in TNBCs and may be involved in their aggressive biology. Interestingly, the analysis of the possible mechanisms of RKIP downregulation in TNBCs permits the identification and recapitulation of different possible approaches, including epigenetic modulation, e.g., by DNA demethylating agents or histone deacetylase inhibition, and NF-κB inhibition. Th…

Oncologymedicine.medical_specialtybiologybusiness.industryCyclin-dependent kinase 4Settore MED/06 - Oncologia MedicaInhibitor proteinBiochemistryEpigenetic therapy NF-κB inhibition Raf-1 kinase inhibitor protein triple-negative breast cancersInternal medicineRaf 1 kinaseGeneticsbiology.proteinCancer researchSettore BIO/14 - FarmacologiaMolecular MedicineMedicineIdentification (biology)businessTriple negativeEpigenetic therapyBiotechnology
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Job skill discretion and emotion control strategies as antecedents of recovery from work

2014

Recovery from work protects employees’ health and well-being, and therefore it is important to understand its antecedents. The aim of this study conducted among 183 middle-aged participants drawn from the Finnish Jyvaskyla Longitudinal Study of Personality and Social Development was to examine whether job skill discretion and emotion control strategies (emotional rumination and emotional inhibition) are related to psychological aspects of recovery from work (subjective recovery evaluation, psychological detachment and relaxation). The results of hierarchical general linear models confirmed the hypothesis that job skill discretion is positively associated with subjective recovery evaluation …

Organizational Behavior and Human Resource ManagementLongitudinal studyjob skill discretionRelaxation (psychology)media_common.quotation_subjectControl (management)Social changeDiscretionemotional inhibitionemotional ruminationrecoveryjob resourcesRuminationwork-related stressmedicinePersonalitymedicine.symptomPsychologyEmotional exhaustionSocial psychologyApplied Psychologyta515media_commonEuropean Journal of Work and Organizational Psychology
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MCC1019, a selective inhibitor of the Polo-box domain of Polo-like kinase 1 as novel, potent anticancer candidate

2019

Polo-like kinase (PLK1) has been identified as a potential target for cancer treatment. Although a number of small molecules have been investigated as PLK1 inhibitors, many of which showed limited selectivity. PLK1 harbors a regulatory domain, the Polo box domain (PBD), which has a key regulatory function for kinase activity and substrate recognition. We report on 3-bromomethyl-benzofuran-2-carboxylic acid ethyl ester (designated: MCC1019) as selective PLK1 inhibitor targeting PLK1 PBD. Cytotoxicity and fluorescence polarization-based screening were applied to a library of 1162 drug-like compounds to identify potential inhibitors of PLK1 PBD. The activity of compound MC1019 against the PLK1…

PBD Polo box domainMTD maximal tolerance doseCDC25 cell division cycle 25HIF-1α hypoxia-inducible factor 1 αMST microscale thermophoresisIC50 50% inhibition concentrationMFP M phase promoting factorPARP-1 poly(ADP-ribose) polymerase-10302 clinical medicineFOXO forkhead box ONec-1 necrostatin 1CDC2 cell division cycle protein 2 homologGeneral Pharmacology Toxicology and PharmaceuticsMitotic catastropheCDK cyclin-dependent kinase0303 health sciencesChemistryPolo-like kinaseMono-targeted therapyCell cycleBUBR1 budding uninhibited by benzimidazole-related 1Polo box domain030220 oncology & carcinogenesisPLK1 Polo-like kinaseNecroptosisSpindle damagePLK1IHC immunohistochemistryOriginal articleNecroptosisCell cyclePLK1APC/C anaphase-promoting complex/cyclosomePLK3ABC avidin-biotin complexPI propidium iodide03 medical and health sciencesFBS fetal bovine serumPDB Protein Data BankKd the dissociation constantKinase activity030304 developmental biologyAkt/PKB signaling pathwayCell growthlcsh:RM1-950LC3 light chain 3lcsh:Therapeutics. PharmacologyCancer researchDAPKs death-associated protein kinase3-MA 3-methyladenineDAPI 4′6-diamidino-2-phenylindoleSAC spindle assembly checkpointActa Pharmaceutica Sinica B
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Endoplasmic Reticulum Stress Inhibition Protects against Excitotoxic Neuronal Injury in the Rat Brain

2007

Elevated brain glutamate with activation of neuronal glutamate receptors accompanies neurological disorders, such as epilepsy and brain trauma. However, the mechanisms by which excitotoxicity triggers neuronal injury are not fully understood. We have studied the glutamate receptor agonist kainic acid (KA) inducing seizures and excitotoxic cell death. KA caused the disintegration of the endoplasmic reticulum (ER) membrane in hippocampal neurons and ER stress with the activation of the ER proteins Bip, Chop, and caspase-12. Salubrinal, inhibiting eIF2α (eukaryotic translation initiation factor 2 subunit α) dephosphorylation, significantly reduced KA-induced ER stress and neuronal deathin vivo…

PERKMaleKainic acidProgrammed cell deathcaspase-12ExcitotoxicityBiologymedicine.disease_causeEndoplasmic ReticulumHippocampusCalcium in biologyeIF2 alphaSalubrinalchemistry.chemical_compoundsalubrinalmedicineExcitatory Amino Acid AgonistsAnimalsRats WistarNeuronsKainic AcidhippocampuGeneral NeuroscienceEndoplasmic reticulumGlutamate receptorBrainNeural InhibitionArticlesCell biologyRatsOxidative StresschemistryUnfolded protein responseNeuroscience
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Development of natural and synthetic compounds as kinase inhibitors targeting cancer cells and cancer stem cells

2023

Pancreatic ductal adenocarcinomaInhibition of migrationantibiotic resistanceImidazo[21-b][134]thiadiazole derivativeskinase inhibitorsanti-biofilm agentsAntiproliferative activityFAK inhibitionSettore CHIM/08 - Chimica Farmaceutica
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Modelling the spatial and temporal constrains of the GABAergic influence on neuronal excitability

2021

GABA (γ-amino butyric acid) is an inhibitory neurotransmitter in the adult brain that can mediate depolarizing responses during development or after neuropathological insults. Under which conditions GABAergic membrane depolarizations are sufficient to impose excitatory effects is hard to predict, as shunting inhibition and GABAergic effects on spatiotemporal filtering of excitatory inputs must be considered. To evaluate at which reversal potential a net excitatory effect was imposed by GABA (EGABAThr), we performed a detailed in-silico study using simple neuronal topologies and distinct spatiotemporal relations between GABAergic and glutamatergic inputs. These simulations revealed for GABAe…

Patch-Clamp TechniquesAction potentialPhysiologyAction PotentialsSynaptic TransmissionNervous SystemBiochemistryMiceNerve FibersAnimal CellsMedicine and Health SciencesGABAergic NeuronsBiology (General)gamma-Aminobutyric AcidNeuronsMembrane potentialEcologyChemistryPyramidal CellsDepolarizationNeurochemistryNeurotransmittersCA3 Region HippocampalElectrophysiologyReceptors GlutamateComputational Theory and MathematicsModeling and SimulationExcitatory postsynaptic potentialGABAergicAnatomyCellular TypesShunting inhibitionResearch Articlemedicine.drugQH301-705.5Models NeurologicalNeurophysiologyAMPA receptorMembrane Potentialgamma-Aminobutyric acidCellular and Molecular NeuroscienceGlutamatergicSpatio-Temporal AnalysisGeneticsmedicineAnimalsComputer SimulationReceptors AMPAReversal potentialMolecular BiologyEcology Evolution Behavior and SystematicsComputational BiologyBiology and Life SciencesNeural InhibitionDendritesCell BiologyNeuronal DendritesAxonsMice Inbred C57BLAnimals Newbornnervous systemCellular NeuroscienceSynapsesDepolarizationNeuroscienceNeurosciencePLOS Computational Biology
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Effect of depolarizing GABAA-mediated membrane responses on excitability of Cajal-Retzius cells in the immature rat neocortex

2011

In immature neurons activation of ionotropic GABA receptors induces depolarizing membrane responses due to a high intracellular Cl− concentration ([Cl−]i). However, it is difficult to draw conclusions about the functional consequences of subthreshold GABAergic depolarizations, since GABAergic membrane shunting and additional effects on voltage-dependent ion channels or action potential threshold must be considered. To systematically investigate factors that determine the GABAergic effect on neuronal excitability we performed whole cell patch-clamp recordings from Cajal-Retzius cells in immature rat neocortex, using [Cl−]i between 10 and 50 mM. The effect of focal GABA application was quant…

Patch-Clamp TechniquesPhysiologyModels NeurologicalAction PotentialsDifferential ThresholdNeocortexMembrane PotentialsGABA AntagonistsChloridesInterneuronsmedicineAnimalsPatch clampGABAergic NeuronsRats WistarReceptorgamma-Aminobutyric AcidNeocortexGABAA receptorChemistryGeneral NeuroscienceReceptors GABA-ARatsPyridazinesRheobasemedicine.anatomical_structureAnimals NewbornIon Channel GatingNeuroscienceShunting inhibitionIntracellularIonotropic effectJournal of Neurophysiology
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Pursuit of the emerging dialogue between psychoanalysis and neuroscience: clinical and research perspectives.

2005

Patient Care TeamBrain MappingPsychoanalysisInterprofessional RelationsEmotionsStatistics as TopicNeurosciencesBrainNeural InhibitionMagnetic Resonance ImagingPsychoanalysisDreamsPsychiatry and Mental healthClinical PsychologyBorderline Personality DisorderPsychoanalytic TheoryHumansNerve NetPsychologyPsychomotor PerformanceDefense MechanismsForecastingThe International journal of psycho-analysis
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