Search results for " vivo"

showing 10 items of 1661 documents

Comparative toxicity and cell-tissue distribution study on nanoparticular iron complexes using avian embryos and HepG2-cells

2007

In this study the toxicity and intracellular availability of iron from iron dextran (FeD), iron sucrose (FeS), and iron gluconate (FeG) was compared in organs of avian (turkey) embryos and in isolated cells (HepG2) in cell culture. Iron uptake was more pronounced in embryonic liver than in renal tissue. Cellular iron uptake in liver and kidney was more or less similar for the different compounds. Only some experiments showed slightly greater iron concentrations in liver and kidney with FeG compared with FeD and FeS. Significant differences were found in the survival ratios of the eggs and the embryo weights depending on the type of iron complex administered. The rank order of toxicities was…

TurkeysCarcinoma HepatocellularEmbryo NonmammalianLiver cytologyChick EmbryoBiologyKidneyIron sucroseIn ovoFerric CompoundsGluconatesAndrologyGlucaric AcidIn vivoCell Line TumorPhysiology (medical)medicineAnimalsHumansFerric Oxide SaccharatedBody WeightLiver NeoplasmsBiochemistry (medical)Public Health Environmental and Occupational HealthKidney metabolismGeneral MedicineIn vitroSurvival RateLiverBiochemistryCell cultureToxicityHematinicsNanoparticlesIron-Dextran Complexmedicine.drugTranslational Research
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Indirect oxidation of the antitumor agent procarbazine by tyrosinase—Possible application in designing anti-melanoma prodrugs

2008

The interaction of tyrosinase with the anticancer drug procarbazine has been investigated. In the presence of the enzyme alone no oxidation of this dialkylhydrazine above the background level was observed. However, when phenolic substrates (4-tert-butylcatechol or N-acetyl-l-tyrosine) were included in the reaction mixture, procarbazine was rapidly degraded. Oxygen consumption measurements showed that in a mixture both the phenolic substrate and the drug were oxidized. The major product of procarbazine degradation was isolated and identified as azoprocarbazine, the first active metabolite of this drug detected in previous in vivo and in vitro studies. This indirect oxidation of the hydrazine…

TyrosinaseClinical BiochemistryPharmaceutical ScienceAntineoplastic AgentstyrosinaseProcarbazineBiochemistryStructure-Activity Relationshipchemistry.chemical_compoundOxygen ConsumptionIn vivoDrug DiscoverymelanomamedicineOrganic chemistryProdrugsHydrazine (antidepressant)PhenolsMolecular BiologyActive metaboliteMolecular StructureMonophenol MonooxygenaseOrganic ChemistrySubstrate (chemistry)hydrazineProdrugHydrazineschemistryProcarbazineMolecular Medicineredox exchangeprodrugAgaricalesOxidation-Reductionmedicine.drugBioorganic & Medicinal Chemistry Letters
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Estudio sobre el estado de la música en directo en la ciudad de Valencia.

2010

Análisis de la situación en qué se encuentra actualmente el sector de la música en directo en la ciudad de Valencia. Se aborda desde una problemática actual y se enumeran las posibles soluciones, para a posteriori reflexionar sobre el futuro que auguran para este sector sus propios protagonistas.

UNESCO::CIENCIAS DE LAS ARTES Y LAS LETRASmúsica; Valencia; en vivo; directo; festivales; conciertos
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An inter-laboratory validation of methods of lipid peroxidation measurement in UVA-treated human plasma samples

2010

Lipid peroxidation products like malondialdehyde, 4-hydroxynonenal and F2-isoprostanes are widely used as markers of oxidative stress in vitro and in vivo. This study reports the results of a multi-laboratory validation study by COST Action B35 to assess inter-laboratory and intra-laboratory variation in the measurement of lipid peroxidation. Human plasma samples were exposed to UVA irradiation at different doses (0, 15 J, 20 J), encoded and shipped to 15 laboratories, where analyses of malondialdehyde, 4-hydroxynonenal and isoprostanes were conducted. The results demonstrate a low within-day-variation and a good correlation of results observed on two different days. However, high coefficie…

Ultraviolet RaysClinical Chemistry TestsEnzyme-Linked Immunosorbent AssayIsoprostanesmedicine.disease_causeF2-isoprostanesSensitivity and SpecificityBiochemistryHigh-performance liquid chromatographyMass Spectrometry4-HydroxynonenalLipid peroxidationPlasmachemistry.chemical_compoundIn vivoMalondialdehydemedicineHumansChromatography High Pressure LiquidAldehydesChromatographyChemistryReproducibility of Resultsoxidative stress; F2-Isoprostanes; 4.-hydroxynonenal; malondialdehydeGeneral MedicineOxidative stress; F2-isoprostanes; 4-hydroxynonenal; malondialdehydeMalondialdehydeIsoprostanes4-hydroxynonenalF2-IsoprostanesBiochemistryOxidative stressLipid PeroxidationOxidative stressChromatography Liquid
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Inflammation Determines the Pro-Adhesive Properties of High Extracellular D-Glucose in Human Endothelial Cells In Vitro and Rat Microvessels In Vivo

2010

BACKGROUND: Hyperglycemia is acknowledged as an independent risk factor for developing diabetes-associated atherosclerosis. At present, most therapeutic approaches are targeted at a tight glycemic control in diabetic patients, although this fails to prevent macrovascular complications of the disease. Indeed, it remains highly controversial whether or not the mere elevation of extracellular D-glucose can directly promote vascular inflammation, which favors early pro-atherosclerotic events. METHODS AND FINDINGS: In the present work, increasing extracellular D-glucose from 5.5 to 22 mmol/L was neither sufficient to induce intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion mo…

Umbilical VeinsEndotheliumCardiovascular Disorders/Coronary Artery Diseaselcsh:MedicineInflammationIn vivoDiabetes mellitusCell AdhesionmedicineExtracellularAnimalsHumansLeukocyte RollingCardiovascular Disorders/Vascular BiologyUNESCO::CIENCIAS DE LA VIDA::Microbiología ::Otraslcsh:ScienceCell adhesionInflammationMultidisciplinaryInflammation; Pro-adhesive properties; High extracellular D-glucose; Human endothelial cells In Vitro; Rat microvessels In VivoDose-Response Relationship DrugChemistrylcsh:RCardiovascular Disorders/Peripheral Vascular DiseaseAdhesivenessEndothelial CellsChemotaxismedicine.diseaseIn vitroRatsChemotaxis LeukocyteDiabetes and EndocrinologyCell Biology/Cell AdhesionGlucosemedicine.anatomical_structureHyperglycemiaMicrovesselsImmunologyCancer researchlcsh:QEndothelium Vascularmedicine.symptomResearch Article
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Negative feedback regulation of the yeast CTH1 and CTH2 mRNA binding proteins is required for adaptation to iron deficiency and iron supplementation.

2013

Iron (Fe) is an essential element for all eukaryotic organisms because it functions as a cofactor in a wide range of biochemical processes. Cells have developed sophisticated mechanisms to tightly control Fe utilization in response to alterations in cellular demands and bioavailability. In response to Fe deficiency, the yeast Saccharomyces cerevisiae activates transcription of the CTH1 and CTH2 genes, which encode proteins that bind to AU-rich elements (AREs) within the 3′ untranslated regions (3′UTRs) of many mRNAs, leading to metabolic reprogramming of Fe-dependent pathways and decreased Fe storage. The precise mechanisms underlying Cth1 and Cth2 function and regulation are incompletely u…

Untranslated regionSaccharomyces cerevisiae ProteinsIronRNA StabilitySaccharomyces cerevisiaeMolecular Sequence DataSaccharomyces cerevisiaeBiologyCofactorTristetraprolinIn vivoTranscription (biology)Gene Expression Regulation FungalAutoregulationRNA MessengerMolecular BiologyGene3' Untranslated RegionsAU Rich ElementsBase SequenceCell BiologyArticlesbiology.organism_classificationMolecular biologyAdaptation PhysiologicalYeastCell biologyDNA-Binding Proteinsbiology.proteinTranscription FactorsMolecular and cellular biology
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miR-15a-3p Protects Against Isoniazid-Induced Liver Injury via Suppressing N-Acetyltransferase 2 Expression

2021

Isoniazid (INH), an effective first-line drug for tuberculosis treatment, has been reported to be associated with hepatotoxicity for decades, but the underlying mechanisms are poorly understood. N-acetyltransferase 2 (NAT2) is a Phase II enzyme that specifically catalyzes the acetylation of INH, and NAT2 expression/activity play pivotal roles in INH metabolism, drug efficacy, and toxicity. In this study, we systematically investigated the regulatory roles of microRNA (miRNA) in NAT2 expression and INH-induced liver injury via a series of in silico, in vitro, and in vivo analyses. Four mature miRNAs, including hsa-miR-15a-3p, hsa-miR-628-5p, hsa-miR-1262, and hsa-miR-3132, were predicted to …

Untranslated regionisoniazidQH301-705.5In silicoBiologyhsa-miR-15a-3pBiochemistry Genetics and Molecular Biology (miscellaneous)BiochemistryN-acetyltransferase 2In vivomicroRNAmedicineMolecular BiosciencesEpigeneticsBiology (General)Molecular BiologyOriginal ResearchLiver injuryIsoniazidregulationmedicine.diseasebody regionsToxicityCancer researchdrug-induced liver injurymedicine.drugFrontiers in Molecular Biosciences
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Ex vivo study of bacterial coronal leakage in indirect pulp treatment

2012

Objective: The aim of this study was to evaluate, ex vivo, bacterial coronal leakage with different antimicrobial agents applied to the dentine for indirect pulp treatment (IPT). Study Design: Sixty extracted teeth were prepared and randomly distributed into 5 groups (n=10): Group 1: no antimicrobial dentine treatment; group 2: 1% chlorhexidine (CHX)+1% thymol varnish (Cervitec®); group 3: 2 % CHX solution; group 4: 40% CHX varnish (EC40™) and group 5: Clearfil™ Protect Bond (CPB). Ten teeth served as controls. The teeth were restored using a resin-modified glass ionomer cement (GIC) and then mounted in a two-chamber device. The coronal access was exposed to Streptococcus mutans for 45 days…

VarnishGlass ionomer cementDentistryOdontologíaIn Vitro TechniquesStreptococcus mutansAnti-Infective Agentsstomatognathic systemClinical and Experimental DentistrymedicineHumansGeneral DentistryDental PulpDental LeakagePulp treatmentbiologyChemistrybusiness.industryChlorhexidine:CIENCIAS MÉDICAS [UNESCO]Antimicrobialbiology.organism_classificationCiencias de la saludStreptococcus mutansstomatognathic diseasesOtorhinolaryngologyvisual_artCoronal planeDentinUNESCO::CIENCIAS MÉDICASvisual_art.visual_art_mediumResearch-ArticleSurgerybusinessEx vivomedicine.drugMedicina Oral Patología Oral y Cirugia Bucal
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In Vivo Modulation of Angiogenesis and Immune Response on a Collagen Matrix via Extracorporeal Shockwaves

2020

The effective management of tissue integration and immunological responses to transplants decisively co-determines the success of soft and hard tissue reconstruction. The aim of this in vivo study was to evaluate the eligibility of extracorporeal shock wave therapy (ESWT) with respect to its ability to modulate angiogenesis and immune response to a collagen matrix (CM) for tissue engineering in the chorioallantoic membrane (CAM) assay, which is performed with fertilized chicken eggs. CM were placed on the CAM on embryonic development day (EDD) 7

Vascular Endothelial Growth Factor A0301 basic medicineAngiogenesismedicine.medical_treatmentNitric Oxide Synthase Type IIChick EmbryoChorioallantoic Membranelcsh:ChemistryNeovascularizationangiogenesischemistry.chemical_compoundmacrophage response0302 clinical medicineTissue engineeringlcsh:QH301-705.5Spectroscopyoral inflammationTissue Scaffoldsvascular endothelial growth factorGeneral MedicineComputer Science ApplicationsVascular endothelial growth factorChorioallantoic membraneExtracorporeal shockwave therapyCollagenmedicine.symptomchorioallantoic membrane assayNeovascularization PhysiologicArticleCatalysisAvian ProteinsInorganic ChemistryAndrology03 medical and health sciencesImmune systemIn vivomatrix metalloproteasesmucoderm®medicineAnimalsddc:610Physical and Theoretical Chemistrymucoderm<sup>®</sup>Molecular BiologyTissue Engineeringbusiness.industryOrganic Chemistrycollagen matrix030206 dentistryextracorporeal shockwave therapyHypoxia-Inducible Factor 1 alpha SubunitMatrix Metalloproteinases030104 developmental biologylcsh:Biology (General)lcsh:QD1-999chemistrybusiness
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Endothelin-1-Mediated Drug Resistance in EGFR-Mutant Non-Small Cell Lung Carcinoma.

2020

Abstract Progression on therapy in non-small cell lung carcinoma (NSCLC) is often evaluated radiographically, however, image-based evaluation of said therapies may not distinguish disease progression due to intrinsic tumor drug resistance or inefficient tumor penetration of the drugs. Here we report that the inhibition of mutated EGFR promotes the secretion of a potent vasoconstrictor, endothelin-1 (EDN1), which continues to increase as the cells become resistant with a mesenchymal phenotype. As EDN1 and its receptor (EDNR) is linked to cancer progression, EDNR-antagonists have been evaluated in several clinical trials with disappointing results. These trials were based on a hypothesis that…

Vascular Endothelial Growth Factor ACancer ResearchLung NeoplasmsAmbrisentanOncology and CarcinogenesisDrug ResistanceBiological AvailabilityAntineoplastic AgentsDrug resistanceCell LineMiceErlotinib HydrochlorideGefitinibIn vivomedicineAnimalsHumansOncology & CarcinogenesisNon-Small-Cell LungProtein Kinase InhibitorsLungCancerTumor microenvironmentTumorEndothelin-1business.industryCarcinomaLung CancerCancerEvaluation of treatments and therapeutic interventionsGefitinibmedicine.diseaseEndothelin 1Xenograft Model Antitumor AssaysErbB ReceptorsOncologyVasoconstriction5.1 Pharmaceuticals6.1 PharmaceuticalsCancer cellMutationCancer researchNeoplasmDevelopment of treatments and therapeutic interventionsbusinessmedicine.drug
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