Search results for "Animal"

showing 10 items of 22159 documents

Effects of indomethacin on muscarinic inhibition of endogenous noradrenaline release from rat isolated trachea

1993

The release of endogenous noradrenaline from rat isolated tracheae was evoked by electrical field stimulation (3 Hz, 540 pulses) in the presence of yohimbine, desipramine and tyrosine. The muscarine receptor agonist oxotremorine concentration-dependently inhibited the evoked release of noradrenaline by 95% at 1 μmol/l, EC50 values in two series of experiments 41 and 57 nmol/l, respectively. The effect of oxotremorine was antagonized by the non-selective muscarine receptor antagonist scopolamine (10–1000 nmol/l) in a manner suggesting a simple competitive interaction (slope of Schild plot −0.94; pA2 value 8.88). However, the M2 selective muscarine receptor antagonist methoctramine (0.1–10 μm…

medicine.medical_specialtymedicine.drug_classIndomethacinDiaminesIn Vitro TechniquesRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineMethoctramineAnimalsPharmacologyMuscarineOxotremorineGeneral MedicineMuscarinic acetylcholine receptor M1Receptor antagonistReceptors MuscarinicPirenzepineRatsTracheaSchild regressionEndocrinologychemistryProstaglandinsFemalemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct

Wood-derived estrogens: studies in vitro with breast cancer cell lines and in vivo in trout.

1996

The wood-derived compound, beta-sitosterol (purity > 90%), was shown to be estrogenic in fish. It induced the expression of the vitellogenin gene in the liver of juvenile and methyltestosterone-treated rainbow trout. Structural similarities to beta-sitosterol notwithstanding, cholesterol, citrostadienol, beta-sitostanol, and 5-androstene-3 beta,17 beta-diol, an estrogenic member of the androstenic steroid group, were inactive. An abietic acid mixture (37% abietic acid, 6% dehydroabietic acid, and a remainder of unknown compounds) showed slight hormonal activity in feed, but it was completely inactive when given intraperitoneally in implants. The estrogenic component of the abietic acid prep…

medicine.medical_specialtymedicine.drug_classIsorhapontigeninPinosylvinEstrogen receptorGene ExpressionBreast NeoplasmsToxicologyVitellogeninchemistry.chemical_compoundVitellogeninsInternal medicinemedicineTumor Cells CulturedBioassayAnimalsHumansAbietic acidPharmacologyBetulinbiologyPhytosterolsEstrogensBlotting NorthernWoodEndocrinologychemistryEstrogenOncorhynchus mykissbiology.proteinRNACell DivisionWater Pollutants ChemicalToxicology and applied pharmacology
researchProduct

Inducibility of the avidin gene by progesterone is suppressed during estrogen-induced cytodifferentiation.

1992

Abstract We have studied epithelial differentiation of the chick oviduct as induced by diethylstilbestrol (DES) and 17β-estradiol (E 2 ). The proportion of goblet cells in the oviduct was slightly higher after E 2 than after DES treatment. Also avidin induction by progesterone was stronger following DES than E 2 priming. In the estrogen pretreated oviduct epithelium, avidin expression was induced by progesterone in the surface epithelial cells, protodifferentiated gland cells and tubular gland cells, but not in goblet cells. During prolonged estrogen treatment, however, the inducibility of avidin by progesterone ceased in tubular gland cells but not in surface epithelial cells. The estrogen…

medicine.medical_specialtymedicine.drug_classOvalbuminEndocrinology Diabetes and MetabolismClinical BiochemistryDiethylstilbestrolEstrogen receptorOviductsBiologyBiochemistryEpitheliumImmunoenzyme TechniquesEndocrinologystomatognathic systemInternal medicineProgesterone receptormedicineAnimalsTubular glandMolecular BiologyDiethylstilbestrolIn Situ HybridizationProgesteroneEstradiolCell DifferentiationEpithelial CellsCell BiologyAvidinEpitheliummedicine.anatomical_structureEndocrinologyGene Expression RegulationEstrogenbiology.proteinMolecular MedicineOviductChickenshormones hormone substitutes and hormone antagonistsmedicine.drugAvidinThe Journal of steroid biochemistry and molecular biology
researchProduct

Estrogen receptor agonists and immune system in ovariectomized mice.

2006

Several data implicate the immune system in bone lost after estrogen deficiency, however, some of the effects on the immune system of estrogen deficiency or of estrogen receptor (ER) modulation are not well established. In this study, the effect of ER agonists on the immune system in ovariectomized mice is analyzed. Mice were ovariectomized and were administered 17β-estradiol (E2), raloxifene (RAL) or genistein (GEN). The effect of a 4-week treatment on bone turnover and on several parameters that reflect the status of the immune system was studied. Results show that ovariectomy provoked both uterine atrophy and thymic hypertrophy. Although RAL corrected thymic hypertrophy, only E2 correct…

medicine.medical_specialtymedicine.drug_classOvariectomyImmunologyEstrogen receptorGenistein03 medical and health scienceschemistry.chemical_compoundEstrogen-related receptor alphaMice0302 clinical medicineImmune systemInternal medicinemedicineImmunology and AllergyAnimalsRaloxifeneEstrogen receptor betaCell ProliferationDNA PrimersPharmacologyBase SequenceEstradiolbusiness.industryReverse Transcriptase Polymerase Chain ReactionGenisteinMice Inbred C57BLEndocrinologychemistryReceptors EstrogenEstrogen030220 oncology & carcinogenesisImmune SystemRaloxifene HydrochlorideOvariectomized ratFemalebusiness030215 immunologymedicine.drugInternational journal of immunopathology and pharmacology
researchProduct

Comparative effects of estradiol, raloxifene, and genistein on the uterus of ovariectomized mice.

2005

To explore the uterine effects of administration of compounds that exert their bone-sparing functions through estrogen receptors, we administered 17beta-E2, raloxifene, or genistein to ovariectomized mice and analyzed the uterus weight and histology 4 weeks after beginning the treatments. Results indicated that raloxifene and genistein have partial agonistic properties on the uterus in estrogen-depleted mice, and that genistein induced apoptosis and several atypias in the glandular epithelium of endometrium, as demonstrated in hematoxylin-eosin-stained histological sections.

medicine.medical_specialtymedicine.drug_classOvariectomyUterusGenisteinEstrogen receptorBiologyEndometriumchemistry.chemical_compoundMiceInternal medicinemedicineAnimalsRaloxifeneintegumentary systemEstradiolBody WeightUterusObstetrics and GynecologyAntiestrogenGenisteinMice Inbred C57BLmedicine.anatomical_structureEndocrinologyReproductive MedicinechemistryEstrogenRaloxifene HydrochlorideOvariectomized ratFemalemedicine.drugFertility and sterility
researchProduct

The role of fibrate treatment in dyslipidemia: an overview.

2012

Dyslipidemia, and especially atherogenic dyslipidemia, a combination of small low-density lipoproteins cholesterol (LDL-C), decreased high-density lipoprotein cholesterol (HDL-C) and increased triglyceride (TG) concentrations, represents a major cardiovascular (CV) risk factor. Nuclear receptor peroxisome proliferator-activated receptors (PPARs) are involved in the regulation of lipid metabolism; PPAR ligands are used to treat dyslipidemias. Fibrates have a major impact on TG metabolism as well as on modulating LDL size and subclasses. Fibrates target atherogenic dyslipidemia by increasing plasma HDL-C concentrations and decreasing small dense LDL (sdLDL) particles and TGs, thus contributin…

medicine.medical_specialtymedicine.drug_classPeroxisome proliferator-activated receptorAdipokineFibratechemistry.chemical_compoundInternal medicineDrug DiscoverymedicineAnimalsHumansTriglyceridesDyslipidemiasHypolipidemic AgentsPharmacologychemistry.chemical_classificationTriglycerideCholesterolbusiness.industryFibric Acidsnutritional and metabolic diseasesLipid metabolismCholesterol LDLmedicine.diseaseFibrates dyslipidemia cardiovascular risk diabetes.EndocrinologychemistryCardiovascular Diseaseslipids (amino acids peptides and proteins)Metabolic syndromebusinessDyslipidemiaCurrent pharmaceutical design
researchProduct

CHF2819: Pharmacological profile of a novel acetylcholinesterase inhibitor

2002

CHF2819 is a novel orally active acetylcholinesterase inhibitor (AChEI) developed for the treatment of Alzheimer's disease (AD). CHF2819 is a selective inhibitor of AChE, it is 115 times more potent against this enzyme than against butyrylcholinesterase (BuChE). Moreover, CHF2819 is more selective for inhibition of central (brain) AChE than peripheral (heart) AChE. In vivo CHF2819, 0.5, 1.5, and 4.5 mg/kg p.o., significantly and in dose-dependent manner increased acetylcholine (ACh) levels in hippocampus of young adult rats. Moreover, aging animals, with lower basal ACh levels than young adult rats, also exhibit a marked increase in hippocampal levels of this neurotransmitter after administ…

medicine.medical_specialtymedicine.drug_classPhenylcarbamatesPharmacologyHippocampusArticleCyclic N-Oxideschemistry.chemical_compoundNeurochemicalAlzheimer DiseaseDopamineInternal medicinemedicineAnimalsBiogenic MonoaminesAmino AcidsNeurotransmitterButyrylcholinesteraseCholinesterasePharmacologybiologybusiness.industryGlutamate receptoracetylcholinesterase inhibitors; alzheimer's disease; amino acids; chf2819; ganstigmine; neurotransmitters; rat hippocampusAcetylcholineRatsNeuropsychology and Physiological PsychologyEndocrinologyAcetylcholinesterase inhibitorchemistrybiology.proteinCarbamatesCholinesterase InhibitorsbusinessAcetylcholinemedicine.drug
researchProduct

Constitutive androstane receptor activation stimulates faecal bile acid excretion and reverse cholesterol transport in mice.

2010

The constitutive androstane receptor (CAR) is a nuclear receptor expressed in the liver and involved in xenobiotic metabolism. The aim of this study was to assess whether pharmacological CAR activation could affect neutral sterol and bile acid elimination under conditions of cholesterol overload.Wild type, Car-/-, ApoE-/-, and low-density lipoprotein receptor (Ldlr)-/- mice fed a western-type diet were treated with the CAR agonist TCPOBOP.CAR activation was associated with a decrease in faecal cholesterol output related to the repression of the Abcg5/g8 cholesterol transporters. In contrast, TCPOBOP treatment induced a marked increase (up to three fold, p0.01) in the elimination of faecal b…

medicine.medical_specialtymedicine.drug_classPyridinesLipoproteinsBiological Transport ActiveGene ExpressionReceptors Cytoplasmic and NuclearHyperlipidemiasBiologyCholesterol 7 alpha-hydroxylaseBile Acids and Saltschemistry.chemical_compoundFecesMiceApolipoproteins EInternal medicineConstitutive androstane receptormedicineAnimalsHomeostasisATP Binding Cassette Transporter Subfamily G Member 5Liver X receptorConstitutive Androstane ReceptorMice KnockoutHepatologyBile acidCholesterolReverse cholesterol transportATP Binding Cassette Transporter Subfamily G Member 8Cholesterol HDLAtherosclerosisSterolMice Inbred C57BLEndocrinologyCholesterolchemistryLiverReceptors LDLLDL receptorlipids (amino acids peptides and proteins)ATP-Binding Cassette TransportersJournal of hepatology
researchProduct

A stinging wasp in the stomach: accidental endoscopic diagnosis of a systemic anaphylactic reaction.

2015

of a proton pump inhibitor and prokinetic treatment. Because of her medical history, an upper gastrointestinal (GI) endoscopy was performed. We discovered, in the proximalpartof theantrumonthegreater curvature, an intact dead wasp with its sting visible and an underlying slightly swollen red welt in the area of the sting

medicine.medical_specialtymedicine.drug_classWaspsProton-pump inhibitorwasp endoscopy anaphylaxisGastroscopymedicineUpper gastrointestinalAnimalsHumansMedical historyAnaphylaxismedicine.diagnostic_testbusiness.industryStomachStomachGastroenterologyAnaphylactic reactionInsect Bites and StingsMiddle Agedeye diseasesSurgeryEndoscopyDeglutitionSettore MED/18 - Chirurgia GeneraleStingmedicine.anatomical_structureAccidentalFemalebusinessEndoscopy
researchProduct

The search for novel avenues for the therapy and prevention of Alzheimer's disease.

2006

The prevention and therapy of neurodegenerative disorders in the elderly is one of the greatest challenges facing molecular medicine today. Alzheimer's is an excellent example of a disease being studied by many groups worldwide. Indeed, while many molecular details of this disorder have been elucidated in the last two decades, there are still no strictly causal therapies available. While certain symptomatic pharmacological treatments are frequently employed, current molecular medicine research is focused on central Alzheimer-associated biochemical changes to find the key switch that turns the detrimental Alzheimer process on. Although amyloid beta proteins and tau proteins are the focus of …

medicine.medical_specialtymedicine.drug_classbusiness.industryFemale sexDiseaseBioinformaticsMolecular medicineEndocrinologyAmyloid beta proteinsEstrogenGSK-3Alzheimer DiseaseInternal medicinemedicineAnimalsHumansNeuroprotective factorsbusinessHormoneDrug newsperspectives
researchProduct