Search results for "Calcium Channel"

showing 10 items of 204 documents

Calcium dependence of the contraction produced by endothelin (ET-1) in isolated guinea-pig trachea.

1990

Endothelin (ET-1, 1 pM to 0.1 microM) produced a concentration-dependent contraction of isolated guinea-pig trachea. BAY K 8644 (1 microM) did not significantly alter the concentration-response curve for ET-1. Incubation with nicardipine (10 microM) partly inhibited responses to low concentrations (10 pM to 1 nM) of ET-1 while verapamil (10 microM) and diltiazem (10 microM) were ineffective. La3+ (10 microM) and Cd2+ (10 microM) preferentially depressed the responses evoked by high concentrations (30 nM-0.1 microM) of ET-1 without affecting the responses evoked by low concentrations of the peptide. Incubation in Ca2(+)-free (with EDTA, 1 mM) medium resulted in suppression of the responses e…

Malemedicine.medical_specialtyContraction (grammar)NicardipineGuinea PigsIndomethacinchemistry.chemical_elementCalciumBiologyIn Vitro TechniquesPotassium ChlorideGuinea pigInternal medicinemedicineExtracellularAnimalsDiltiazemPharmacologyEndothelinsMuscle Smooth3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterCalcium Channel BlockersTracheaEndocrinologychemistryVerapamilCalciumEndothelin receptormedicine.drugMuscle ContractionEuropean journal of pharmacology
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U-46619-induced potentiation of noradrenergic constriction in the human saphenous vein: antagonism by thromboxane receptor blockade.

2001

Objective: We investigated the potentiating effect of U-46619, a synthetic analogue of thromboxane A2 (TXA2), on the adrenergic responses in human saphenous vein. Methods: Saphenous vein rings were obtained from 35 patients undergoing coronary artery bypass surgery. The rings were suspended in organ bath chambers for isometric recording of tension. Results: U-46619 (10−10–3×10−7 mol/l) produced concentration-dependent and endothelium-independent contractile responses. U-46619 (10−10 mol/l) potentiated the contractions elicited by electrical stimulation and potassium chloride, and produced leftward shifts of the concentration–response curve for noradrenaline. The TXA2 receptor antagonist SQ-…

Malemedicine.medical_specialtyDihydropyridinesNifedipinePhysiologymedicine.drug_classThromboxaneReceptors ThromboxaneAdrenergicIn Vitro TechniquesPotassium ChlorideThromboxane receptorThromboxane A2chemistry.chemical_compoundNorepinephrineThromboxane A2Physiology (medical)Internal medicinemedicineHumansVasoconstrictor AgentsSaphenous VeinAgedVoltage-dependent calcium channelDose-Response Relationship DrugChemistryCalcium channelDihydropyridineDrug SynergismMiddle AgedReceptor antagonistCalcium Channel BlockersElectric StimulationStimulation ChemicalEndocrinology15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidFemaleEndothelium VascularCardiology and Cardiovascular Medicinemedicine.drugCardiovascular research
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Effects of calcium channel blockers on gastric emptying and acid secretion of the rat in vivo.

1986

Abstract Experiments were designed to evaluate the effects of three calcium channel blockers (verapamil, diltiazem and cinnarizine) on gastric emptying and secretion in the rat. Pretreatment with the calcium blockers delayed gastric emptying of phenol red in a dose-dependent manner. Verapamil was the most effective of the agents tested. Verapamil and diltiazem inhibited gastric acid secretion in the pylorus-ligated rat without affecting pepsin output. Cinnarizine was ineffective in this model. When the perfused lumen of the anaesthetized rat was used, verapamil was found to inhibit responses to carbachol or histamine more than those to pentagastrin. Further, we found a greater sensitivity t…

Malemedicine.medical_specialtyGastric motilitychemistry.chemical_elementBlood PressureCalciumBiologyGastric AcidInternal medicinemedicineAnimalsAnesthesiaDiltiazemPylorusPharmacologyGastric emptyingDose-Response Relationship DrugCalcium channeldigestive oral and skin physiologyRats Inbred StrainsCalcium Channel BlockersRatsPentagastrinPerfusionEndocrinologychemistryGastric Emptyingcardiovascular systemGastric acidVerapamilFemalemedicine.drugResearch ArticleBritish journal of pharmacology
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Chronic pharmacological treatment in takotsubo cardiomyopathy.

2008

Abstract Background Takotsubo cardiomyopathy is a disorder that has been appreciated only recently. In most of reported cases, this syndrome mimes an acute myocardial infarction. Till this moment no data are available from literature about the treatment in the acute phase of this disease. Aim of the study In our multicentric experience we have retrospectively looked at the benefits of a treatment with ACE-inhibitors, beta-blockers, Aspirin and calcium channels blockers, started until the early phases of the disease and continued for 30 days, in 36 patients affected by Takotsubo cardiomyopathy. We chose as endpoint of the study the efficacy of the used drug in improving left ventricular myoc…

Malemedicine.medical_specialtyHeart diseaseAdrenergic beta-AntagonistsCardiomyopathyAngiotensin-Converting Enzyme InhibitorsTakotsubo CardiomyopathyInternal medicineMedicineHumansMyocardial infarctionAgedRetrospective StudiesAspirinEjection fractionAspirinbusiness.industryRetrospective cohort studymedicine.diseaseCalcium Channel BlockersSurgeryTreatment OutcomeACE inhibitorCardiologyFemaleDifferential diagnosisCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Evidence for the neuronal origin of immunoreactive interleukin-1 beta released by rat hypothalamic explants.

1996

In this study, we have investigated the release of immunoreactive interleukin-1 beta (irIL-1 beta) from the rat hypothalamus in vitro. It was found that (1) tissue explants release sizable amounts of irIL-1 beta (ranging from 0.43 to 0.52 pg/mg of wet tissue) in 20 min incubations; (2) basal release in significantly increased by depolarization induced with 56 mM KCl; (3) K(+)-induced irIL-1 beta release is inhibited by the specific blocker of N-type calcium channels, omega-conotoxin, and by verapamil, but not by nifedipine; (4) K(+)-induced release is also inhibited by the Na+ channel blockers tetrodotoxin and lidocaine; (5) irIL-1 beta release is significantly increased by noradrenalin; su…

Malemedicine.medical_specialtyHypothalamusRadioimmunoassaychemistry.chemical_elementPropranololCalciumIn Vitro TechniquesPotassium ChlorideNorepinephrinePhentolamineNifedipineInternal medicinemedicineAnimalsChannel blockerRats WistarNeuronsVoltage-dependent calcium channelGeneral NeuroscienceDepolarizationCalcium Channel BlockersRatsElectrophysiologyEndocrinologychemistryVerapamilmedicine.drugInterleukin-1Sodium Channel BlockersNeuroscience letters
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Increased calcium influx is responsible for the sustained mechanical tone in colon from dystrophic (mdx) mice

2001

Abstract Background & Aims: Proximal colon from dystrophic mice develops spontaneous tone increment, but the mechanisms involved in its development have not been investigated. This study examined whether alterations in the properties of cell membrane calcium channels and/or sarcoplasmic reticular (SR) Ca 2+ -adenosine triphosphatase (ATPase) contribute to tone development. Methods: Effects of calcium-free solution, nifedipine, pinaverium (calcium channel blockers), and cyclopiazonic acid (CPA; SR Ca 2+ -ATPase inhibitor) on the contractile activity of colon from mdx and control mice were determined. Results Calcium-free solution abolished spontaneous contractions in both preparations, but d…

Malemedicine.medical_specialtyIndolesNifedipineColonSarcoplasmchemistry.chemical_elementCalciumMuscular DystrophiesCalcium in biologyMicechemistry.chemical_compoundNifedipineInternal medicinemedicineAnimalsHepatologyVoltage-dependent calcium channelCalcium channelGastroenterologyPinaveriumMice Inbred C57BLEndocrinologychemistryMice Inbred mdxCalciumCyclopiazonic acidMuscle Contractionmedicine.drugGastroenterology
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Characterization of two different Ca2+ entry pathways dependent on depletion of internal Ca2+ pools in rat aorta

1998

Ryanodine (10 microM), thapsigargin (1 microM) and cyclopiazonic acid (10 microM) produced a slow, sustained contractile response in rat aorta that only can be observed in Ca2+-containing solution. In Ca2+-free medium, no response to the drugs was obtained, which suggests that the contraction elicited in presence of Ca2+ is mainly due to the contribution of extracellular influx. This Ca2+ entry does not depend on the opening of dihydropyridine-dependent Ca2+-channels for nimodipine does not affect this. Noradrenaline (1 microM) induced a biphasic response in Ca2+-free medium that was mediated by two different Ca2+ compartments, one of which is common to caffeine (10 mM), and is also deplete…

Malemedicine.medical_specialtyIndolesThapsigarginContraction (grammar)Phosphodiesterase InhibitorsVasodilator AgentsAorta ThoracicIn Vitro TechniquesMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundCaffeineInternal medicinemedicine.arterymedicineExtracellularAnimalsVasoconstrictor AgentsRats WistarCa2 entryNimodipinePharmacologyAortaRyanodineRyanodine receptorGeneral MedicineRatsEndocrinologychemistryBiophysicsThapsigarginCalciumCalcium ChannelsCyclopiazonic acidMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Effects of antidepressants in adrenergic neurotransmission of human vas deferens

2000

Objectives. To evaluate the effects of sertraline, fluoxetine, and amitriptyline on the contractile responses of the human vas deferens muscle elicited by norepinephrine, electrical field stimulation, and KCl, because the therapeutic action of antidepressants may be accompanied by sexual dysfunction related to the contractility of the vas deferens smooth muscle. Methods. Ring segments of the epididymal part of the vas deferens were taken from 32 elective vasectomies and mounted in organ baths for isometric recording of tension. We then studied the effects of sertraline, fluoxetine, and amitriptyline on the neurogenic and agonist-induced contractile responses. Results. Amitriptyline caused c…

Malemedicine.medical_specialtyNifedipineAdrenergic receptorAmitriptylineUrologyAdrenergicSynaptic TransmissionNorepinephrine (medication)Vas DeferensNifedipineCulture TechniquesFluoxetineSertralineInternal medicinemedicineHumansAmitriptylineSertralineFluoxetineDose-Response Relationship Drugbusiness.industryVas deferensCalcium Channel BlockersAntidepressive AgentsReceptors AdrenergicEndocrinologymedicine.anatomical_structureCalciumbusinessMuscle Contractionmedicine.drugUrology
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Effect of Divalent Cations on the Contractile Response of Rat Aorta to Depolarization before and after Nifedipine Treatment

1996

The influence of the divalent cations, Ca2+, Mg2+ and Ba2+, on the contractile response of the rat aorta to KCl and on the recovery of this response after nifedipine treatment was analyzed. KCl (80 mmol/l) promoted a two-phase (phasic and tonic) contractile response in Krebs solution but, as expected, no contractile response in Ca(2+)-free medium. In Mg(2+)-free medium, the phasic response to KCl was unaffected but the tonic one decreased slowly, suggesting that a long incubation time in the absence of Mg2+ (65 min) promotes a loss of or a change in the intracellular distribution of this ion that modifies Ca2+ entry through L channels or Ca2+ handling. Ba2+ (1.8 mmol/l) contracted the rat a…

Malemedicine.medical_specialtyNifedipineCations DivalentAorta ThoracicMuscle Smooth VascularPotassium ChlorideDivalentTonic (physiology)NifedipineInternal medicinemedicine.arterymedicineAnimalsRats WistarPharmacologychemistry.chemical_classificationAnalysis of VarianceAortaChemistryContractile responseDepolarizationGeneral MedicineCalcium Channel BlockersRatsEndocrinologyKrebs solutionIntracellularMuscle Contractionmedicine.drugPharmacology
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Endothelin-1-induced potentiation of adrenergic responses in the rabbit pulmonary artery: role of thromboxane A(2).

2001

Abstract To examine whether low concentrations of endothelin-1 potentiate the vasocontrictor response to adrenergic stimulation, we recorded the isometric response of rings of rabbit pulmonary artery to electrical stimulation and noradrenaline. Endothelin-1 (10 −10 M) potentiated the contractions induced by electrical stimulation and noradrenaline. The endothelin ET B receptor antagonist (2,6-dimethylpiperidinecarbonyl-γ-methyl-Leu- N in -[Methoxycarbonyl]- d -Trp- d -Nle) (BQ-788, 10 −6 M), but not the endothelin ET A receptor antagonist cyclo( d -Asp-Pro- d -Val-Leu- d -TRP) (BQ-123, 10 −6 M), inhibited the potentiating effects of endothelin-1. Pretreatment with the cyclooxygenase inhibit…

Malemedicine.medical_specialtyNifedipineThromboxanemedicine.drug_classAdrenergicPulmonary ArteryThromboxane A2chemistry.chemical_compoundNorepinephrineThromboxane A2PiperidinesInternal medicinemedicineAnimalsVasoconstrictor AgentsAntihypertensive AgentsPharmacologybiologyDose-Response Relationship DrugEndothelin-1Receptors EndothelinReceptor antagonistCalcium Channel BlockersEndothelin 1Receptor Endothelin BElectric StimulationEndocrinologychemistryVasoconstrictionbiology.proteinCyclooxygenaseThromboxane-A synthaseRabbitsEndothelin receptorOligopeptidesEuropean journal of pharmacology
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