Search results for "Clero"

showing 10 items of 2052 documents

Oxidative stress in vascular disease: causes, defense mechanisms and potential therapies

2007

Endothelial cells control vascular homeostasis by generating paracrine factors that regulate vascular tone, inhibit platelet function, prevent adhesion of leukocytes, and limit proliferation of vascular smooth muscle. The dominant factor responsible for many of those effects is endothelium-derived nitric oxide (NO). Endothelial dysfunction characterized by enhanced inactivation or reduced synthesis of NO, alone or in combination, is seen in conjunction with risk factors for cardiovascular disease. Endothelial dysfunction can promote vasospasm, thrombosis, vascular inflammation, and proliferation of the intima. Vascular oxidative stress and increased production of reactive oxygen species con…

Vascular smooth muscleEndotheliumArteriosclerosisPharmacologyNitric Oxidemedicine.disease_causeAntioxidantsReceptor Angiotensin Type 1Superoxide dismutaseRisk FactorsmedicineHumansEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologybusiness.industryAnticholesteremic AgentsGeneral Medicinemedicine.diseaseOxidative Stressmedicine.anatomical_structureMitochondrial respiratory chainchemistryImmunologybiology.proteinEndothelium VascularHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessOxidative stressNature Clinical Practice Cardiovascular Medicine
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Regulation of NOS expression in vascular diseases

2020

Nitric oxide synthases (NOS) are the major sources of nitric oxide (NO), a small bioactive molecule involved in the regulation of many cellular processes. One of the most prominent functions of NO is regulation of vasodilatation and thereby control of blood pressure. Most important for vascular tone is NOS3. Endothelial NOS3-generated NO diffuses into the vascular smooth muscle cells, activates the soluble guanylate cyclase resulting in enhanced cGMP concentrations and smooth muscle cell relaxation. However, more and more evidence exist that also NOS1 and NOS2 contribute to vascular function. We summarize the current knowledge about the regulation of NOS expression in the vasculature by tra…

Vascular smooth muscleNitric Oxide Synthase Type IIINOS1CellNitric Oxide Synthase Type IIBlood PressureVasodilationInflammationNitric Oxide Synthase Type INitric OxideMuscle Smooth VascularNitric oxidechemistry.chemical_compoundmedicineAnimalsHumansProtein IsoformsVascular DiseasesRNA Processing Post-TranscriptionalInflammationRegulation of gene expressionInnate immune systemAtherosclerosisImmunity InnateCell biologyGene Expression Regulation Neoplasticmedicine.anatomical_structurechemistryNitric Oxide Synthasemedicine.symptomProtein Processing Post-TranslationalFrontiers in Bioscience-Landmark
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Factor VIII and Antithrombin III in Atherosclerosis Obliterans of the Lower Limbs

1984

In the multifactorial pathogenesis of atherosclerosis (the W.H.O. gives a list of about forty risk factors) recent acquisitions have allowed to determine in part the role carried out by the term “haemocoagulative disorders” (90) showing the possible connections between haematic components of haemocoagulation and vascular wall that up to now have been only hypothesised.

Vascular wallPathogenesismedicine.medical_specialtybusiness.industryInternal medicineAntithrombinmedicineCardiologybusinessATHEROSCLEROTIC VASCULAR DISEASEmedicine.drug
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Endothelial progenitor cells: Are they displaying a function in autoimmune disorders?

2016

Endothelial Progenitor Cells (EPCs) are bone marrow derived cells able to differentiate in mature endothelial cells (EC) contributing to the generation of new vessels, connecting to fibronectin, and forming colonies and/or colony forming units. Since circulating EPCs can be actively considered part of endothelial damage in several cardiovascular diseases and autoimmune disorders the possibility to have a measure for endothelium damage should be considered of interest to predict the patient out-come. At the same time the EPCs proliferative and regenerative role could be considered for therapeutic applications. Studies have been performed to elucidate the role of EPCs in Systemic Sclerosis an…

Vasculitis0301 basic medicineVasculitiAgingEndotheliumAutoimmune Diseases03 medical and health sciences0302 clinical medicineMedicineAnimalsHumansRheumatoid arthritisProgenitor cellRheumatoid arthritiEndothelial Progenitor Cells030203 arthritis & rheumatologyColony-forming unitScleroderma SystemicbiologyEPCs; LES; Rheumatoid arthritis; Vasculitis; Aging; Developmental Biologybusiness.industryEPCmedicine.diseaseFibronectinSettore MED/16 - Reumatologia030104 developmental biologymedicine.anatomical_structureLESEPCsImmunologyembryonic structuresbiology.proteincardiovascular systemBone marrowEndothelium VascularbusinessVasculitisFunction (biology)circulatory and respiratory physiologyDevelopmental BiologyMechanisms of ageing and development
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New Lipid Modulating Drugs: The Role of Microsomal Transport Protein Inhibitors

2011

Microsomal triglyceride transfer protein (MTP) is involved in the synthesis of very low density lipoprotein in the liver. Its deficiency results in abetalipoproteinemia. MTP inhibitors target the assembly and secretion of apolipoprotein B-containing lipoproteins. These agents may potentially play a role, alone or in combination, in the treatment of hypercholesterolemia or hypertriglyceridemia. Clinical applications of MTP inhibitors initially focused primarily on high-dose monotherapy in order to produce substantial reductions in LDL-cholesterol levels but these proved to induce significant hepatic steatosis and transaminase elevations. However, likely orphan indications for MTP inhibitors,…

Very low-density lipoproteinApolipoprotein BHypercholesterolemiaFamilial hypercholesterolemiaLipoproteins VLDLPharmacologyMicrosomal triglyceride transfer proteinHyperlipoproteinemia Type IIchemistry.chemical_compoundMicrosomesDrug DiscoveryClinical endpointHumansMedicineApolipoproteins BHypertriglyceridemiaPharmacologybiologybusiness.industryCholesterolAbetalipoproteinemiamedicine.diseaseAbetalipoproteinemiaBiochemistrychemistryMTP-inhibitors lipids lipoproteins atherosclerosis cardiovascular prevention.biology.proteinlipids (amino acids peptides and proteins)SteatosisCarrier ProteinsbusinessCurrent Pharmaceutical Design
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On the pathogenesis of atherosclerosis: enzymatic transformation of human low density lipoprotein to an atherogenic moiety.

1995

Combined treatment with trypsin, cholesterol esterase, and neuraminidase transforms LDL, but not HDL or VLDL, to particles with properties akin to those of lipid extracted from atherosclerotic lesions. Single or double enzyme modifications, or treatment with phospholipase C, or simple vortexing are ineffective. Triple enzyme treatment disrupts the ordered and uniform structure of LDL particles, and gives rise to the formation of inhomogeneous lipid droplets 10-200 nm in diameter with a pronounced net negative charge, but lacking significant amounts of oxidized lipid. Enzymatically modified LDL (E-LDL), but not oxidatively modified LDL (ox-LDL), is endowed with potent complement-activating c…

Very low-density lipoproteinArteriosclerosisImmunologyNeuraminidaseComplement Membrane Attack Complexchemistry.chemical_compoundLipid dropletmedicineExtracellularImmunology and AllergyHumansTrypsinReceptors ImmunologicComplement ActivationGlycoproteinsReceptors Lipoproteinchemistry.chemical_classificationReceptors ScavengerPhospholipase CCholesterolMacrophagesMembrane ProteinsComplement C3Complement System ProteinsArticlesScavenger Receptors Class BSterol EsteraseTrypsinLipid MetabolismLipoproteins LDLEnzymechemistryBiochemistryLow-density lipoproteinlipids (amino acids peptides and proteins)medicine.drugFoam CellsThe Journal of experimental medicine
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Periodontal treatment on patients with cardiovascular disease : systematic review and meta-analysis

2018

Podeu consultar la versió en castellà del document a: http://hdl.handle.net/2445/153580

Very low-density lipoproteinmedicine.medical_specialtyMalalties periodontalsDisease030204 cardiovascular system & hematologylaw.invention03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRandomized controlled triallawInternal medicinemedicineHumansGeneral DentistryPeriodontal DiseasesOral Medicine and PathologyMalalties cardiovascularsCholesterolbusiness.industryResearch030206 dentistryAtherosclerosis:CIENCIAS MÉDICAS [UNESCO]Confidence intervalClinical trialCardiovascular diseasesOtorhinolaryngologychemistryCardiovascular DiseasesMeta-analysisUNESCO::CIENCIAS MÉDICASEtiologySurgeryPeriodontal diseasebusinessAterosclerosi
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Vertigo, Masseter Paresis or Masseter Reflex Abnormality, and Impaired Caloric Response

1993

Symptoms and signs of acute peripheral-type vestibular abnormalities occasionally occur in patients with multiple sclerosis and brain-stem ischemia. Such patients may show reduced excitability of the lateral semicircular canal. From this observation, the question arises as to the location of the causative lesion.

Vestibular systemmedicine.medical_specialtybiologybusiness.industryMultiple sclerosisAnatomyAudiologybiology.organism_classificationmedicine.diseaseLesionLateral vestibular nucleusmedicine.anatomical_structureVestibular nucleiVertigootorhinolaryngologic diseasesmedicinesense organsmedicine.symptombusinessJaw jerk reflexParesis
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Vaskulärer Schwindel

2002

Ischemia,hemorrhages, and other vascular disorders can result in various central or peripheral vestibular syndromes with vertigo, oculomotor/balance disturbances, and nausea. The vascular vertigo syndromes listed in Table 1 can however be brought about by other causes such as demyelitizing focuses in multiple sclerosis or space-occupying lesions, so that not only localization of the damaged structure but also the various etiologies are decisive for the choice of therapy. Occasionally, combined functional disturbances of the peripheral and central vestibular system appear, such as an infarction of the inferior anterior cerebellar artery, which supplies the labyrinth and parts of the brainste…

Vestibular systemmedicine.medical_specialtybiologybusiness.industryMultiple sclerosisIschemiaInfarctionGeneral Medicinebiology.organism_classificationmedicine.diseaseLesionPsychiatry and Mental healthmedicine.anatomical_structureNeurologyVertigoInternal medicineotorhinolaryngologic diseasesmedicineCardiologysense organsNeurology (clinical)Brainstemmedicine.symptombusinessCerebellar arteryDer Nervenarzt
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Fatigue, sleep, and nocturnal complaints in patients with amyotrophic lateral sclerosis

2012

Background and purpose: Fatigue is a common symptom in amyotrophic lateral sclerosis (ALS). Although sleep disturbances are a candidate factor that may interfere with fatigue in patients with ALS, the role of sleep-related abnormalities in determining fatigue in ALS is unknown. Objective: To evaluate the frequency and determinants of fatigue in a group of 91 consecutive patients with ALS, with special attention to the relationship between fatigue and sleep problems. Methods: Measures included the Fatigue Severity Scale (FSS), Pittsburgh Sleep Quality Index (PSQI), Epworth Sleepiness Scale (ESS), ALS Functional Rating ScaleRevised (ALSFRS-R), and Beck Depression Inventory (BDI). Results: The…

Vital capacitymedicine.medical_specialtybusiness.industryEpworth Sleepiness ScaleBeck Depression Inventorymedicine.diseasePittsburgh Sleep Quality IndexPhysical medicine and rehabilitationNeurologySeverity of illnessmedicinePhysical therapyNocturiaNeurology (clinical)medicine.symptomAmyotrophic lateral sclerosisbusinessMuscle crampEuropean Journal of Neurology
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