Search results for "Dipeptidase"

showing 10 items of 52 documents

Polymorphism insertion/deletion of the ACE gene and ambulatory blood pressure circadian variability in essential hypertension

2001

OBJECTIVE The objective of the present study was to analyze the influence of the insertion/deletion (I/D) polymorphism of the angiotensin-converting enzyme on ambulatory blood pressure values and circadian variability in untreated patients with hypertension. MATERIAL AND METHODS Ninety-nine essential hypertensive patients, less than 50 years old (mean age 39.5+/-7.0 years), previously untreated with antihypertensive drugs were included. Twenty-four hour ambulatory blood pressure monitoring (ABPM) was performed with a Spacelabs (90202 and 90207) monitor, during a regular working day in unrestricted ambulatory conditions. The I/D polymorphism of the ACE was determined by PCR. RESULTS The dist…

AdultMalemedicine.medical_specialtyAmbulatory blood pressureGenotypeBlood PressurePeptidyl-Dipeptidase AAssessment and DiagnosisEssential hypertensionPolymorphism (computer science)Internal medicineGenotypeInternal MedicinemedicineHumansCircadian rhythmAlleleSequence DeletionAdvanced and Specialized NursingPolymorphism Geneticbusiness.industryGeneral MedicineBlood Pressure Monitoring AmbulatoryMiddle Agedmedicine.diseaseCircadian RhythmMutagenesis InsertionalEndocrinologyBlood pressureHypertensionAmbulatoryFemaleCardiology and Cardiovascular MedicinebusinessBlood Pressure Monitoring
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Polymorphisms of the renin-angiotensin system influence height in normotensive women in a Spanish population.

2004

The objective of this study was to analyze the influence of the polymorphisms G-6A of the angiotensinogen gene, insertion/deletion (I/D) of the angiotensin-converting enzyme, and C573T of the angiotensin II AT1 receptor gene on a healthy, middle-age population. A total of 370 (194 women) healthy normotensive Caucasian subjects, aged 25-50 yr old, were selected from the general population. A significant association was found between height and the C573T polymorphism in women (P0.001). After adjustment for age, this association remained significant (P0.002). Thus, the lowest height values were from subjects carrying TT genotype (CC, 1.627 +/- 0.008 m; CT, 1.595 +/- 0.006 m; TT, 1.586 +/- 0.01…

AdultMalemedicine.medical_specialtyAngiotensinsEndocrinology Diabetes and MetabolismClinical BiochemistryPopulationPeptidyl-Dipeptidase ABiochemistryReceptor Angiotensin Type 1EndocrinologyPolymorphism (computer science)Reference ValuesInternal medicineRenin–angiotensin systemGenotypeMedicineHumanseducationeducation.field_of_studySex CharacteristicsAngiotensin II receptor type 1Polymorphism Geneticbusiness.industryBiochemistry (medical)Case-control studyMiddle AgedAngiotensin IIBlood pressureEndocrinologySpainCase-Control StudiesFemalebusinessThe Journal of clinical endocrinology and metabolism
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Angiotensin converting enzyme has an inhibitory role in CGRP metabolism in human skin

2005

The neutral endopeptidase (NEP) is important for calcitonin gene related peptide (CGRP) degradation, while the role of angiotensin converting enzyme (ACE) remains unclear. By using dermal microdialysis we explored the effect of phosphoramidon (NEP blocker), captopril (ACE blocker) and a mixture of both drugs on the intensity of electrically-induced CGRP-mediated neurogenic flare. The results reveal that phosphoramidon elevated flare intensity, but that this was not further increased by adding captopril. In contrast, neurogenic flare was decreased when the drug mixture was applied in compared to NEP only. Electrically released CGRP levels could be measured directly in perfusates containing p…

AdultMalemedicine.medical_specialtyCaptoprilPhysiologyCalcitonin Gene-Related PeptideNeuropeptideAngiotensin-Converting Enzyme InhibitorsPeptidyl-Dipeptidase ACalcitonin gene-related peptideBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundEndocrinologyInternal medicineRetrograde DegenerationLaser-Doppler FlowmetrymedicineHumansNeprilysinSkinintegumentary systembiologyChemistryCatabolismfungiPhosphoramidonGlycopeptidesAngiotensin-converting enzymeCaptoprilMetabolismrespiratory systemVasodilationEndocrinologybiology.proteinFemalemedicine.drugPeptides
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Angiotensin-converting enzyme gene deletion polymorphism determines an increase in frequency of migraine attacks in patients suffering from migraine …

2000

Many authors have reported an association between the angiotensin-converting enzyme (ACE)-D allele and coronary heart disease and other cardiovascular diseases. The mechanism underlying the positive associations between the ACE-D alleles and diseases are not yet clear. Previous reports showed an association between migraine without aura and ACE-D allele polymorphism. The study is aimed to evaluate if the DD genotype could also be associated with the frequency and duration of migraine without aura. In 302 patients suffering from migraine without aura (at least for 1 year), with no history of cardiovascular diseases and major risk factors for ischemic events, the genotypes of the ACE gene, pl…

AdultMalemedicine.medical_specialtyGenotypeAuraMigraine DisordersPeptidyl-Dipeptidase AGastroenterologyCentral nervous system diseasePolymorphism (computer science)Internal medicineGenotypeMedicineHumansAllelesPolymorphism Geneticbiologybusiness.industryVascular diseaseAngiotensin-converting enzymeMiddle Agedmedicine.diseaseEndocrinologyNeurologyMigrainebiology.proteinFemaleNeurology (clinical)Gene polymorphismChromosome DeletionbusinessEuropean neurology
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Genetic predisposition to thrombophilia in inflammatory bowel disease

2011

BACKGROUND Inflammatory bowel disease (IBD) is linked to a definite risk of thromboembolic events (TE), but data on the role of prothrombotic genetic mutations are conflicting. STUDY Fourteen genetic factors involved in TE pathogenesis were investigated in a homogeneous cohort of Sicilian patients with IBD with and without history of TE and in healthy controls. Forty IBD patients (21 CD, 19 UC) and 20 healthy individuals were enrolled. Genetic testing was based on the reverse hybridization principle by a commercial assay that analyzes 14 polymorphisms involved in thrombophilia and cholesterol metabolism. The rate of genetic polymorphisms and mutations was compared between IBD patients and h…

AdultMalemedicine.medical_specialtyIBDAngiotensinogenPeptidyl-Dipeptidase AThrombophiliaInflammatory bowel diseaseGastroenterologyPolymorphism (computer science)Risk FactorsInternal medicineDiabetes mellitusGenotypeGenetic predispositionmedicineHumansThrombophiliaGenetic Predisposition to DiseaseAllele frequencySicilyGenetic testingAgedPolymorphism Geneticmedicine.diagnostic_testbusiness.industryGastroenterologyMiddle Agedmedicine.diseaseInflammatory Bowel Diseasesdigestive system diseasesMutationFemalebusiness
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Inhibition of neutral endopeptidase (NEP) facilitates neurogenic inflammation

2005

Neutral endopeptidase (NEP) and angiotensin-converting enzyme (ACE) are involved in neuropeptide degradation and may modulate neurogenic inflammation. We therefore explored the effect of specific blockers of NEP and ACE on the intensity of neurogenic inflammation. We investigated eight subjects on three occasions. Two pairs of microdialysis fibers equipped with intraluminal wires were inserted intracutaneously into the volar forearms and electrical stimuli were delivered via the intraluminal electrodes. The microdialysis fibers were perfused either with normal saline, phosphoramidon (NEP inhibitor), or captopril (ACE inhibitor). CGRP release was assessed in the microdialysis eluate via a sp…

AdultMalemedicine.medical_specialtyMicrodialysisCaptoprilTime FactorsCalcitonin Gene-Related PeptideMicrodialysisPeptidyl-Dipeptidase AImmunoenzyme Techniqueschemistry.chemical_compoundDevelopmental NeuroscienceInternal medicineLaser-Doppler FlowmetrymedicineHumansDrug InteractionsEnzyme InhibitorsSkinNerve Fibers UnmyelinatedNeurogenic inflammationbiologyPhosphoramidonGlycopeptidesCaptoprilAngiotensin-converting enzymeElectric StimulationVasodilationAllodyniaEndocrinologyNeurologychemistryHyperalgesiaACE inhibitorHyperalgesiabiology.proteinFemaleNeprilysinNeurogenic Inflammationmedicine.symptommedicine.drugExperimental Neurology
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Relationship between circulating E-selectin, DD genotype of angiotensin-converting-enzyme, and cardiovascular damage in central obese subjects

2003

Fifty-six young central obese patients were investigated to evaluate relationships between soluble E-selectin (sE-S), angiotensin-converting enzyme (ACE) gene polymorphism, left ventricular function and structure, and carotid morphology by determination of sE-S and ACE genotypes. Our results indicated that central obese subjects with concomitant higher levels of sE-S and ACE DD genotype may be characterized by early cardiovascular alterations and then considered a particular subset of subjects at higher risk of cardiovascular disease.

AdultMalemedicine.medical_specialtySettore MED/09 - Medicina Internaangiotensin-converting-enzyme cardiovascular damageGenotypeArteriosclerosisEndocrinology Diabetes and MetabolismBlood PressureDiseasecentral obese subjectsPeptidyl-Dipeptidase ABody Mass Indexcirculating E-selectin genotype; angiotensin-converting-enzyme cardiovascular damage; central obese subjectsEndocrinologyRisk FactorsInternal medicineGenotypeE-selectinmedicineHumansInsulinObesityAllelesbiologyVentricular functionHemodynamicsHeartAngiotensin-converting enzymeGlucose Tolerance TestSettore MED/45 - Scienze Infermieristiche Generali Cliniche E PediatricheIsoenzymesCarotid ArteriesEndocrinologyCardiovascular DiseasesEchocardiographyConcomitantbiology.proteinRegression AnalysisFemaleObese subjectsGene polymorphismE-Selectincirculating E-selectin genotypeMetabolism
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Relationship between ACE-DD polymorphism and diastolic performance in healthy subjects.

2004

Background—The ACE-D allele has been associated with cardiovascular disease. The study evaluates the relationship between the ACE-ID genotypes and diastolic function in healthy subjects after 6 years of follow-up. Methods—Two hundred and seventy-five healthy volunteers aged 25–55 years had normal physical examination, 12-lead ECG, acceptable echocardiographic windows and echocardiogram at entry. Venous blood was drawn for DNA analysis. Results—Two hundred and forty-two subjects completed 6 years of follow-up. Three genetically distinct groups were obtained: ACE-DD group (n=71, 26F/45M, mean age 48±7 years); ACE-ID (n=115, 39F/76M, mean age 40±7 years); and ACE-II (n=56, 20F/36M, mean age 47…

AdultMalemedicine.medical_specialtyTime FactorsGenotypeDiastolePhysical examinationPeptidyl-Dipeptidase ARisk AssessmentWhite PeopleCohort StudiesSex FactorsPolymorphism (computer science)DiastolePredictive Value of TestsReference ValuesInternal medicinemedicineHumansProspective StudiesProspective cohort studyAllelesProbabilityAnalysis of VariancePolymorphism Geneticmedicine.diagnostic_testbusiness.industryAge FactorsHemodynamicsVenous bloodMiddle AgedEchocardiography DopplerSurgeryPredictive value of testsMultivariate AnalysisCardiologyFemaleAnalysis of varianceCardiology and Cardiovascular MedicinebusinessCohort studyFollow-Up StudiesScandinavian cardiovascular journal : SCJ
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The serum protease network—one key to understand complex regional pain syndrome pathophysiology

2019

Complex regional pain syndrome (CRPS) develops after fracture. The acute CRPS phenotype resembles exaggerated inflammation, which is explained by local and systemic activation of a proinflammatory network including peptides and cytokines. Epidemiologic data suggest that inactivation of the peptidase angiotensin-converting enzyme in patients treated for hypertension increases the odds to develop CRPS. This hint leads us to investigate the serum protease network activity in patients with CRPS vs respective controls. For this purpose, we developed a dabsyl-bradykinin (DBK)-based assay and used it to investigate patients with CRPS, as well as healthy and pain (painful diabetic neuropathy [dPNP]…

AdultMalemedicine.medical_treatmentPainInflammationPeptidyl-Dipeptidase ABradykininProinflammatory cytokine03 medical and health sciences0302 clinical medicineDiabetic Neuropathies030202 anesthesiologyHealthy controlHumansMedicinePain MeasurementInflammationProteasebusiness.industryMiddle Agedmedicine.diseasePathophysiology3. Good healthReflex Sympathetic DystrophyAnesthesiology and Pain MedicineComplex regional pain syndromeNeurologyPainful diabetic neuropathyImmunologyCytokinesFemaleNeurology (clinical)medicine.symptomEpidemiologic databusinessComplex Regional Pain Syndromes030217 neurology & neurosurgeryPeptide HydrolasesPain
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Focus on clinical practice: angiotensin-converting enzyme 2 and corona virus disease 2019: pathophysiology and clinical implications.

2020

: ACE2 receptor has a broad expression pattern in the cellular membrane and provides a protective action against the development of cardiovascular diseases. Recently, this enzyme has become of extreme interest during the pandemic infection of COVID-19 (coronavirus disease 2019). This virus invades alveolar epithelium and cardiomyocytes using ACE2 as a transmembrane receptor. ACE2 is a counter-regulatory peptide that degrades Ang II into Ang 1-7, thereby attenuating the biological effects of the AT1 receptor. The binding between the spike protein of COVID-19 and the enzyme is crucial for the virus to enter the target cells, but whether an increase in ACE2 activity could facilitate the infect…

Angiotensin receptorARDSMyocarditisPneumonia ViralAngiotensin-Converting Enzyme Inhibitors030204 cardiovascular system & hematologyPharmacologyPeptidyl-Dipeptidase AVirusSettore MED/11Renin-Angiotensin System03 medical and health sciencesAngiotensin Receptor AntagonistsBetacoronavirus0302 clinical medicinemedicineHumans030212 general & internal medicineReceptorPandemicsDisseminated intravascular coagulationAngiotensin II receptor type 1business.industrySARS-CoV-2COVID-19General MedicineVirus Internalizationmedicine.diseaseCardiovascular DiseasesAngiotensin-converting enzyme 2Spike Glycoprotein CoronavirusAngiotensin-Converting Enzyme 2Cardiology and Cardiovascular MedicinebusinessCoronavirus Infectionshormones hormone substitutes and hormone antagonistsJournal of cardiovascular medicine (Hagerstown, Md.)
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