Search results for "Fluorescence"

showing 10 items of 2463 documents

Clinicopathological significance of cell cycle regulation markers in a large series of genetically confirmed Ewing's sarcoma family of tumors.

2010

More than 90% of all Ewing's Sarcoma Family of Tumors (ESFT) exhibit specific chromosomal rearrangements between the EWS gene on chromosome 22 and various members of the ETS gene family of transcription factors. The gene fusion type and other secondary genetic alterations, mainly involving cell cycle regulators, have been shown to be of prognostic relevance in ESFT. However, no conclusive results have been reported. We analyzed the clinicopathological significance of relevant cell cycle regulators in genetically confirmed ESFT. A total of 324 cases were analyzed for the immunohistochemical expression of p53, p21(Waf1/Cip1) , p27(Kip1) and Ki67 and the chromosomal alterations of the p53 and …

AdultMaleCancer ResearchPathologymedicine.medical_specialtyAdolescentChromosomes Human Pair 22Sarcoma EwingBiologyFusion geneCohort StudiesYoung AdultGene mappingmedicineBiomarkers TumorHumansProgression-free survivalChildIn Situ Hybridization FluorescenceAgedAged 80 and overCell CycleCancerEwing's sarcomaInfantCell cycleMiddle Agedmedicine.diseaseGenes p53ImmunohistochemistryOncologyChild PreschoolCancer researchFemaleSarcomaChromosome DeletionRNA-Binding Protein EWSChromosome 22International journal of cancer
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Association of loss of 1p and alterations of chromosome 14 in meningioma progression

2004

Meningiomas are usually benign tumors; however, they can recur after surgical resection and occasionally show histologic progression to a higher grade II and III malignancy. The second most frequently reported genetic abnormality after 22q loss is deletion of 1p, although alterations in 9q, 10q, and 14q are also implicated in meningioma progression. Fourteen tumors comprising six benign, four atypical, and four malignant meningiomas were examined by means of cytogenetic and fluorescence in situ hybridization analysis. All tumors showed losses in different regions of 1p, with 1p11, 1p13, 1p21, 1p22, 1p32, and 1q21 breakpoints; eight tumors also presented alterations of chromosome 14. Five of…

AdultMaleCancer ResearchPathologymedicine.medical_specialtyBiologyBioinformaticsMalignancyMeningiomaMonosomyGeneticsmedicine1p DeletionHumansMolecular BiologyIn Situ Hybridization FluorescenceAgedChromosomes Human Pair 14medicine.diagnostic_testBreakpointChromosomeMiddle Agedmedicine.diseaseHistologic ProgressionChromosomes Human Pair 1Tumor progressionKaryotypingFemaleChromosome DeletionMeningiomaFluorescence in situ hybridizationCancer Genetics and Cytogenetics
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Analysis of translocations that involve theNUP98 gene in patients with 11p15 chromosomal rearrangements

2004

The NUP98 gene has been reported to be fused with at least 15 partner genes in leukemias with 11p15 translocations. We report the results of screening of cases with cytogenetically documented rearrangements of 11p15 and the subsequent identification of involvement of NUP98 and its partner genes. We identified 49 samples from 46 hematology patients with 11p15 (including a few with 11p14) abnormalities, and using fluorescence in situ hybridization (FISH), we found that NUP98 was disrupted in 7 cases. With the use of gene-specific FISH probes, in 6 cases, we identified the partner genes, which were PRRX1 (PMX1; in 2 cases), HOXD13, RAP1GDS1, HOXC13, and TOP1. In the 3 cases for which RNA was a…

AdultMaleCancer Researchmedicine.medical_specialtyAdolescentMolecular Sequence DataChromosomal translocationBiologyTranslocation GeneticComplementary DNAInternal medicineGeneticsmedicineGuanine Nucleotide Exchange FactorsHumansGenetic Predisposition to DiseaseGeneIn Situ Hybridization FluorescenceHomeodomain ProteinsGeneticsNUP98 GeneLeukemiaHematologyBase Sequencemedicine.diagnostic_testChromosomes Human Pair 11BreakpointInfantMolecular biologyNuclear Pore Complex ProteinsDNA Topoisomerases Type IHOXD13Child PreschoolTranscription FactorsFluorescence in situ hybridizationGenes, Chromosomes and Cancer
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Prevalence and Clinical Outcomes for Patients With ALK-Positive Resected Stage I to III Adenocarcinoma: Results From the European Thoracic Oncology P…

2014

Purpose The prevalence of anaplastic lymphoma kinase (ALK) gene fusion (ALK positivity) in early-stage non–small-cell lung cancer (NSCLC) varies by population examined and detection method used. The Lungscape ALK project was designed to address the prevalence and prognostic impact of ALK positivity in resected lung adenocarcinoma in a primarily European population. Methods Analysis of ALK status was performed by immunohistochemistry (IHC) and fluorescent in situ hybridization (FISH) in tissue sections of 1,281 patients with adenocarcinoma in the European Thoracic Oncology Platform Lungscape iBiobank. Positive patients were matched with negative patients in a 1:2 ratio, both for IHC and for …

AdultMaleCancer Researchmedicine.medical_specialtyPathologyLung NeoplasmsPopulation610 Medicine & healthAdenocarcinoma of LungAdenocarcinomaGastroenterologyCohort Studies10049 Institute of Pathology and Molecular Pathologyhemic and lymphatic diseasesThoracic OncologyInternal medicinePrevalencemedicineHumansAnaplastic lymphoma kinase1306 Cancer ResearchAnaplastic Lymphoma KinaseLung cancereducationIn Situ Hybridization FluorescenceAgedNeoplasm Stagingeducation.field_of_studyLungbusiness.industryALK-PositiveReceptor Protein-Tyrosine KinasesMiddle Agedmedicine.diseaseImmunohistochemistryEuropeTreatment Outcomemedicine.anatomical_structureOncology10032 Clinic for Oncology and HematologyImmunohistochemistryAdenocarcinoma2730 OncologyFemalebusiness
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The 2q37-deletion syndrome: an update of the clinical spectrum including overweight, brachydactyly and behavioural features in 14 new patients

2012

International audience; The 2q37 locus is one of the most commonly deleted subtelomeric regions. Such a deletion has been identified in >100 patients by telomeric fluorescence in situ hybridization (FISH) analysis and, less frequently, by array-based comparative genomic hybridization (array-CGH). A recognizable ‘2q37-deletion syndrome’ or Albright’s hereditary osteodystrophy-like syndrome has been previously described. To better map the deletion and further refine this deletional syndrome, we formed a collaboration with the Association of French Language Cytogeneticists to collect 14 new intellectually deficient patients with a distal or interstitial 2q37 deletion characterized by FISH and …

AdultMaleCandidate geneAdolescentDNA Copy Number Variations[SDV]Life Sciences [q-bio]Chromosome DisordersLocus (genetics)BiologyFibrous Dysplasia PolyostoticBioinformaticsArticleYoung Adult03 medical and health sciences0302 clinical medicineIntellectual DisabilityGeneticsmedicineHumansChildGenetic Association StudiesGenetics (clinical)030304 developmental biologyKIF1AGeneticsBehaviorComparative Genomic Hybridization0303 health sciences[ SDV ] Life Sciences [q-bio]medicine.diagnostic_testBrachydactylyBrachydactylyChromosome MappingOverweightSubtelomeremedicine.disease[SDV] Life Sciences [q-bio]Child PreschoolChromosomes Human Pair 2AutismFemaleChromosome Deletion030217 neurology & neurosurgeryComparative genomic hybridizationFluorescence in situ hybridizationEuropean Journal of Human Genetics
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Structure, chromosomal localization, and brain expression of human Cx36 gene

1999

Rat connexin-36 (Cx36) is the first gap junction protein shown to be expressed predominantly in neuronal cells of the mammalian central nervous system. As a prerequisite for studies devoted to the investigation of the possible role of this connexin in human neurological diseases, we report the cloning and sequencing of the human Cx36 gene, its chromosomal localization, and its pattern of expression in the human brain analyzed by radioactive in situ hybridization. The determination of the human gene sequence revealed that the coding sequence of Cx36 is highly conserved (98% identity at the protein level with the mouse and rat Cx36 and 80% with the ortholog perch and skate Cx35), and that the…

AdultMaleCandidate geneAdolescentgenetic structuresMolecular Sequence DataIn situ hybridizationBiologyHippocampal formationPolymerase Chain ReactionConnexinsMiceCellular and Molecular NeurosciencemedicineAnimalsHumansCoding regionAmino Acid SequenceSkates FishCloning MolecularEye ProteinsPeptide Chain Initiation TranslationalGeneIn Situ Hybridization FluorescenceChromosomes Human Pair 15Genomic LibrarySequence Homology Amino Acidmedicine.diagnostic_testBrainChromosome MappingHuman brainMiddle AgedMolecular biologyIntronsRatsmedicine.anatomical_structureSpinal CordOrgan SpecificityPerchesCerebellar cortexFemalesense organsSequence AlignmentFluorescence in situ hybridizationJournal of Neuroscience Research
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Expanded gentamicin volume of distribution in critically ill adult patients receiving total parenteral nutrition

1995

Aminoglycoside antibiotics distribute into the extracellular fluid compartment and are eliminated by the kidney via glomerular filtration. Malnutrition and total parenteral nutrition influence the fluid and electrolyte status of the patient, and cause organ changes. The purpose of this clinical study was to characterize the kinetic behaviour of gentamicin in the parenterally fed critically ill adult patient. Eighty-six critically ill adult patients treated with gentamicin for severe Gram-negative infections were enrolled in the study (mean +/- SD): age, 60 +/- 14 years; weight, 69.4 +/- 10.2 kg; height, 163 +/- 10 cm; 22 females and 64 males. Four study groups were defined (2 x 2): total pa…

AdultMaleCritical IllnessRenal functionFluorescence PolarizationCommunicable DiseasesPharmacokineticsExtracellular fluidmedicineHumansPharmacology (medical)Infusions IntravenousAgedAntibacterial agentPharmacologyVolume of distributionbusiness.industryAminoglycosideMiddle AgedAnti-Bacterial AgentsNutrition DisordersParenteral nutritionAnesthesiaRegression AnalysisFemaleParenteral Nutrition TotalGentamicinGentamicinsbusinessmedicine.drugJournal of Clinical Pharmacy and Therapeutics
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Solid cheese consumption: Quantification of oral coating

2011

International audience; Mouth coating, defined as the residual food that sticks to the oral surface after food ingestion, plays an important role in both the delivery of food components and mouth feel and after-feel perceptions. The aim of this work was to adapt a rapid and simple instrumental method to the quantification of food remaining after the in vivo ingestion of a solid food. This method was investigated in 12 non-trained subjects who consumed four melted cheeses differing in terms of their fat and water contents. Mouth coating was determined by fluorescence measurements using curcumin, an oil-soluble dye that had been added to the cheeses during their preparation. The results obtai…

AdultMaleCurcuminSensationengineering.materialFluorescenceMouthfeelTongueCoatingCheese[SDV.IDA]Life Sciences [q-bio]/Food engineeringHumansIngestionFood componentsFood scienceSolid-foodTextureMouth mucosaGeneral DentistryAnalysis of VarianceViscosityChemistrydigestive oral and skin physiologyMouth MucosaReproducibility of ResultsCell BiologyGeneral MedicineMiddle AgedOral coatingDeglutitionOtorhinolaryngologySolid foodFatengineeringMasticationFemale
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Screening of subtelomeric rearrangements in autistic disorder: identification of a partial trisomy of 13q34 in a patient bearing a 13q;21p translocat…

2006

Within the framework of a FISH screening protocol to detect cryptic subtelomeric rearrangements in autistic disorder (AD), a patient bearing three copies of the subtelomeric portion of the q arm of chromosome 13 has been identified. Beside AD, the patient also has severe mental retardation and displays several dysmorphic features. Further FISH analyses revealed that the trisomy was caused by the translocation of a 13q subtelomeric fragment to the acrocentric tip of one chromosome 21 [46,XY.ish der(21) t(13;21) (q34;p13)(D13S1825+)]. Gene dosage experiments carried out with three multiallelic polymorphisms of the subtelomeric region of chromosome 13q showed that the putative length of the tr…

AdultMaleDerivative chromosomeAdolescentGene DosageautismChromosomal translocationTrisomyBiologyGene dosagePolymerase Chain ReactionTranslocation GeneticCellular and Molecular NeurosciencemedicineHumansAutistic DisorderChildGenetics (clinical)In Situ Hybridization FluorescenceChromosome 13GeneticsChromosomes Human Pair 13ChromosomeTelomereSubtelomeremedicine.diseasePsychiatry and Mental healthfrontal bossingFemaleTrisomyChromosome 21American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics
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Genome-wide analysis for micro-aberrations in familial exstrophy of the bladder using array-based comparative genomic hybridization

2007

OBJECTIVE: Exstrophy of the bladder (EB) is part of the bladder-exstrophy-epispadias complex (BEEC). Because familial occurrence of BEEC is rare, exogenous factors are thought to play a major role in the etiology of most BEEC cases. We aimed to investigate a possible genetic basis of BEEC in a consanguineous kindred of Moroccan origin with three members showing the same phenotypic expression of BEEC. PATIENTS AND METHODS: The three affected males (two cousins and their maternal uncle) all presenting with nonsyndromic classic EB, were born in Morocco or The Netherlands. One Moroccan patient had an open bladder surface for 22 years due to late surgical reconstruction, avoided upright posture …

AdultMaleEpispadiasAdolescentUrologyClone (cell biology)GenomeMedicineHumansAbnormalities MultipleGenetic Predisposition to DiseaseGeneIn Situ Hybridization FluorescenceOligonucleotide Array Sequence AnalysisGeneticsChromosome AberrationsGenomebusiness.industryBladder ExstrophyNucleic Acid HybridizationKaryotypeDNAmedicine.diseasePhenotypePedigreeBladder exstrophyMoroccoEtiologybusinessComparative genomic hybridizationBJU International
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