Search results for "GMP"

showing 10 items of 121 documents

Biphasic 24-Hour Variations in Cyclic GMP Accumulation in the Rat Pineal Gland Are Due to Corresponding Changes in the Activity of Cytosolic and Part…

2006

Various parameters of the rat pineal gland display a 24-h rhythm. However, nothing is known about possible 24-h variations in cyclic GMP (cGMP) metabolism. In the present study, 24-h variations in pineal gland cGMP accumulation were investigated by determining the increase in cGMP level with and without inhibitors of phosphodiesterase at different time points over a light/dark cycle (12/12 h). Furthermore, the activity of guanylate cyclase (GC) was determined under substrate-saturated conditions regarding the cytosolic and particulate forms of the enzyme. It has been found that cGMP accumulation and GC activity display biphasic 24-h variations with two peaks--one approximately 7 h after lig…

Malemedicine.medical_specialtyTime FactorsBiologyArginineNitric OxidePineal GlandBiochemistryNitric oxideRats Sprague-DawleyCellular and Molecular NeurosciencePineal glandchemistry.chemical_compoundCytosolInternal medicinemedicineAnimalsCircadian rhythmCyclic GMPchemistry.chemical_classificationomega-N-MethylargininePhosphodiesteraseMetabolismCircadian RhythmRatsEnzyme ActivationCytosolEnzymeEndocrinologymedicine.anatomical_structurechemistryBiochemistryGuanylate CyclaseEndocrine glandJournal of Neurochemistry
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Microvascular effects of the inhibition of dipeptidylpeptidase IV by linagliptin in nondiabetic hypertensive patients

2015

Recent studies suggest vascular benefits of dipeptidylpeptidase IV (DPP-IV) inhibition in patients with diabetes mellitus. Only little is known about potential vascular effects of DPP-IV inhibitors in nondiabetic individuals. The aim of this study was to investigate the effect of DPP-IV inhibition in a nondiabetic hypertensive population.This was a double-blinded, randomized, placebo-controlled, mechanistic study, comparing microvascular effects of the DPP-IV inhibitor linagliptin with placebo in nondiabetic individuals with a history of arterial hypertension. Twenty-one patients received 5 mg linagliptin (5 women; age 67.6 ± 6.0 years; mean ± SD), whereas 22 patients were randomized to pla…

Malemedicine.medical_specialtyanimal structuresPhysiologyMEDLINELinagliptin030209 endocrinology & metabolism030204 cardiovascular system & hematologyArginineLinagliptinlaw.inventionTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineDouble-Blind MethodRandomized controlled triallawDiabetes mellitusInternal medicineInternal MedicinemedicineHumansArterial PressureIn patientCystatin CCyclic GMPDipeptidylpeptidase ivAgedGlycated HemoglobinDipeptidyl-Peptidase IV Inhibitorsbusiness.industryRetinal VesselsMiddle Agedmedicine.diseaseC-Reactive ProteinEndocrinologyRegional Blood FlowHypertensionMicrovesselsFemaleCardiology and Cardiovascular Medicinebusinessmedicine.drugJournal of Hypertension
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Nebivolol inhibits superoxide formation by NADPH oxidase and endothelial dysfunction in angiotensin II-treated rats.

2006

Nebivolol is a β 1 -receptor antagonist with vasodilator and antioxidant properties. Because the vascular NADPH oxidase is an important superoxide source, we studied the effect of nebivolol on endothelial function and NADPH oxidase activity and expression in the well-characterized model of angiotensin II–induced hypertension. Angiotensin II infusion (1 mg/kg per day for 7 days) caused endothelial dysfunction in male Wistar rats and increased vascular superoxide as detected by lucigenin-derived chemiluminescence, as well as dihydroethidine staining. Vascular NADPH oxidase activity, as well as expression at the mRNA and protein level, were markedly upregulated, as well as NOS III uncoupled, …

Malerac1 GTP-Binding Proteinmedicine.medical_specialtyLuminescenceEndotheliumNitric Oxide Synthase Type IIIAdrenergic beta-AntagonistsNitric OxideFluorescenceCell LineNebivololchemistry.chemical_compoundHemoglobinsSuperoxidesInternal medicineInternal MedicinemedicineAnimalsHumansBenzopyransRats WistarCyclic GMPNitritesOxidase testNADPH oxidaseLuminescent AgentsbiologyChemistrySuperoxideAngiotensin IIMyocardiumNADPH OxidasesDicarbethoxydihydrocollidinePhosphoproteinsAngiotensin IINebivololRatsNitric oxide synthasemedicine.anatomical_structureEndocrinologyEthanolaminesNOX1biology.proteinAcridinesBlood VesselsLuminolEndothelium Vascularmedicine.drugSignal TransductionHypertension (Dallas, Tex. : 1979)
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Evidence for a relationship between mitochondrial Complex I activity and mitochondrial aldehyde dehydrogenase during nitroglycerin tolerance: effects…

2012

The medical use of nitroglycerin (GTN) is limited by patient tolerance. The present study evaluated the role of mitochondrial Complex I in GIN biotransformation and the therapeutic effect of mitochondrial antioxidants. The development of GIN tolerance (in rat and human vessels) produced a decrease in mitochondrial 02 consumption. Co-incubation with the mitochondria-targeted antioxidant mitoquinone (MQ 10(-6) mol/L) or with glutathione ester (GEE, 10(-4) mol/L) blocked GTN tolerance and the effects of GTN on mitochondrial respiration and aldehyde dehydrogenase 2 (ALDH-2) activity. Biotransformation of GTN depended on the mitochondria being functionally active, particularly mitochondrial Comp…

Mitochondrial ROSMaleAntioxidantmedicine.medical_treatmentAldehyde dehydrogenaseMitochondrionmedicine.disease_causeBiochemistryAntioxidantsRats Sprague-Dawleychemistry.chemical_compoundMiceNitroglycerinCyclic GMPAortaBiotransformationbiologyDrug ToleranceGlutathioneMitochondriaVasodilationBiochemistrycardiovascular systemAntioxidantcirculatory and respiratory physiologyBiophysicsIn Vitro TechniquesALDH-2Nitric oxideCell LineOxygen ConsumptionRotenoneRespirationmedicineHuman Umbilical Vein Endothelial CellsAnimalsHumansElectron Transport Complex IDose-Response Relationship DrugNitric oxideGlutathioneCell BiologyAldehyde DehydrogenaseRatschemistryOxidative stressMutationbiology.proteinReactive Oxygen SpeciesOxidative stressBiochimica et biophysica acta
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Reciprocal regulation of endothelial nitric-oxide synthase and NADPH oxidase by betulinic acid in human endothelial cells.

2007

Nitric oxide (NO) produced by endothelial NO synthase (eNOS) is a protective principle in the vasculature. Many cardiovascular diseases are associated with reduced NO bioactivity and eNOS uncoupling due to oxidative stress. Compounds that reverse eNOS uncoupling and increase eNOS expression are of therapeutic interest. Zizyphi Spinosi semen (ZSS) is one of the most widely used traditional Chinese herbs with protective effects on the cardiovascular system. In human umbilical vein endothelial cells (HUVEC) and HUVEC-derived EA.hy 926 cells, an extract of ZSS increased eNOS promoter activity, eNOS mRNA and protein expression, and NO production in a concentration- and time-dependent manner. Maj…

Nitric Oxide Synthase Type IIIBlotting Westernmedicine.disease_causeNitric OxideGene Expression Regulation EnzymologicNitric oxidechemistry.chemical_compoundEnosBetulinic acidmedicineHumansNitric Oxide DonorsEnzyme InhibitorsBetulinic AcidCyclic GMPCells CulturedPharmacologyNADPH oxidaseBetulinbiologyDose-Response Relationship DrugReverse Transcriptase Polymerase Chain ReactionNOX4AcetophenonesEndothelial CellsNADPH OxidasesZiziphusSaponinsbiology.organism_classificationTriterpenesNG-Nitroarginine Methyl EsterchemistryBiochemistryNADPH Oxidase 4biology.proteinMolecular MedicineSpermineP22phoxPentacyclic TriterpenesReactive Oxygen SpeciesOxidative stressDrugs Chinese HerbalThe Journal of pharmacology and experimental therapeutics
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Effects of fenspiride on human bronchial cyclic nucleotide phosphodiesterase isoenzymes: functional and biochemical study.

1998

We have investigated the role of human bronchial cyclic nucleotide phosphodiesterases in the effects of fenspiride, a drug endowed with bronchodilator and anti-inflammatory properties. Functional studies on human isolated bronchi showed that fenspiride (10(-6)-3 x 10(-3) M, 30 min) induced a shift to the left of the concentration-response curves for isoprenaline and sodium nitroprusside with -logEC50 values of 4.1+/-0.1 (n = 7) and 3.5+/-0.2 (n = 8), respectively. Biochemical studies were carried out on three human bronchi in which separation of cyclic nucleotide phosphodiesterase isoenzymes was performed by ion exchange chromatography followed by determination of phosphodiesterase activity…

NitroprussideMuscle RelaxationVasodilator AgentsPhosphodiesterase 3FenspirideBronchimedicineHumansSpiro CompoundsPharmacologyCyclic nucleotide phosphodiesterasebiologyDose-Response Relationship DrugChemistryIsoproterenolPhosphodiesteraseBronchodilator AgentsIsoenzymesBiochemistryEnzyme inhibitor3'5'-Cyclic-AMP PhosphodiesterasescGMP-specific phosphodiesterase type 5biology.proteinPhosphodiesterase 2Sodium nitroprussidemedicine.drugMuscle ContractionEuropean journal of pharmacology
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Postsynaptic NO/cGMP Increases NMDA Receptor Currents via Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels in the Hippocampus

2013

The nitric oxide (NO)/cyclic guanosine monophosphate (cGMP) signaling cascade participates in the modulation of synaptic transmission. The effects of NO are mediated by the NO-sensitive cGMP-forming guanylyl cyclases (NO-GCs), which exist in 2 isoforms with indistinguishable regulatory properties. The lack of long-term potentiation (LTP) in knock-out (KO) mice deficient in either one of the NO-GC isoforms indicates the contribution of both NO-GCs to LTP. Recently, we showed that the NO-GC1 isoform is located presynaptically in glutamatergic neurons and increases the glutamate release via hyperpolarization-activated cyclic nucleotide (HCN)-gated channels in the hippocampus. Electrophysiologi…

Patch-Clamp TechniquesCognitive NeuroscienceLong-Term PotentiationIn Vitro TechniquesNeurotransmissionNitric OxideReceptors N-Methyl-D-AspartateMiceCellular and Molecular Neurosciencechemistry.chemical_compoundCyclic nucleotidePostsynaptic potentialHyperpolarization-Activated Cyclic Nucleotide-Gated ChannelsHCN channelAnimalsAnesthetics LocalCA1 Region HippocampalCyclic GMPCyclic guanosine monophosphateMice KnockoutNeuronsbiologyLidocaineTetraethylammoniumLong-term potentiationHyperpolarization (biology)Electric StimulationPyrimidinesAnimals Newbornnervous systemchemistryGuanylate CyclaseBiophysicsbiology.proteinNMDA receptorExcitatory Amino Acid AntagonistsNeuroscienceCerebral Cortex
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Nitric oxide and excitatory postsynaptic currents in immature rat sympathetic preganglionic neurons in vitro.

1997

Neuronal nitric oxide synthase immunoreactivity was localized to sympathetic preganglionic neurons of the intermediolateral cell column and cyclic GMP immunoreactivity to nerve fibers projecting into the intermediolateral cell column of 20-25-day-old rats. Whole-cell patch-clamp recordings were made from sympathetic preganglionic neurons in spinal cord slices of immature rats and the role of nitric oxide and cyclic GMP on excitatory postsynaptic currents was studied. Superfusing the slices with the nitric oxide precursor L-arginine (300 microM) increased the amplitude of evoked excitatory postsynaptic currents as well as the frequency of spontaneous miniature excitatory postsynaptic current…

Patch-Clamp TechniquesPostsynaptic CurrentArginineNitroarginineSynaptic TransmissionNitric oxideRats Sprague-Dawleychemistry.chemical_compoundHemoglobinsNerve Fibers1-Methyl-3-isobutylxanthinemedicineAnimalsPhosphodiesterase inhibitorCyclic GMPEvoked PotentialsNeuronsGanglia SympatheticbiologyGeneral NeuroscienceGlutamate receptorImmunohistochemistryElectric StimulationRatsNitric oxide synthasechemistryBiochemistrySpinal Cordbiology.proteinExcitatory postsynaptic potentialBiophysicsNMDA receptorSodium nitroprussideNitric Oxide Synthasemedicine.drugNeuroscience
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Inflammation in the Human Periodontium Induces Downregulation of the α1- and β1-Subunits of the sGC in Cementoclasts

2021

Nitric oxide (NO) binds to soluble guanylyl cyclase (sGC), activates it in a reduced oxidized heme iron state, and generates cyclic Guanosine Monophosphate (cGMP), which results in vasodilatation and inhibition of osteoclast activity. In inflammation, sGC is oxidized and becomes insensitive to NO. NO- and heme-independent activation of sGC requires protein expression of the &alpha

Periodontium0301 basic medicinealveolar bonecementoclastslcsh:Chemistrychemistry.chemical_compound0302 clinical medicineCathepsin Kheterocyclic compoundsperiodontitisCyclic GMPlcsh:QH301-705.5SpectroscopyGeneral MedicineComputer Science ApplicationsResorptionCell biologymedicine.anatomical_structurecardiovascular systemOxidation-Reductioncementuminorganic chemicalsPeriodontal LigamentIronAntigens Differentiation MyelomonocyticHemeArticleCatalysisNitric oxideInorganic Chemistry03 medical and health sciencesstomatognathic systemAntigens CDnitric oxideOsteoclastmedicineAnimalsHumansddc:610CementumPhysical and Theoretical ChemistryMolecular BiologyCyclic guanosine monophosphateInflammationOrganic Chemistrysoluble guanylyl cyclase030206 dentistryPeriodontiumcGMPosteoclasts030104 developmental biologyGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999chemistrySoluble guanylyl cyclaseInternational Journal of Molecular Sciences
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Effects of N2, O2'-dibutyril cyclic GMP on the nucleoside phosphotransferase activity of the retina of the chick embryos.

1977

In the retina of the chick embryo, 2 different forms of nucleoside phosphotransferase take part in the phosphorylation of thymidine. One is an unstable form with higher molecular weight. The other with lower m. wt is a stable form. This paper shows that N2, O2′-dibutyril cyclic GMP causes a marked decrement of the activity of the unstable nucleoside phosphotransferase.

PharmacologyRetinaNucleoside phosphotransferase activityPhosphotransferasesEmbryoCell BiologyChick EmbryoBiologyRetinaCellular and Molecular Neurosciencechemistry.chemical_compoundCyclic gmpmedicine.anatomical_structureBiochemistrychemistryNucleoside phosphotransferasemedicineMolecular MedicinePhosphorylationAnimalsThymidineMolecular BiologyNucleosideCyclic GMPThymidineExperientia
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