Search results for "Growth factor"

showing 10 items of 1300 documents

Growth hormone and hematopoiesis: A retrospective analysis on a large cohort of children with growth hormone deficiency

2018

Objective: Few large-scale studies regarding the impact of GH deficiency (GHD) on hematopoiesis in children have been reported. Our aim was to investigate hematopoiesis indices in a large cohort of GHD children at diagnosis and during GH treatment (GHT) and any correlation with hormonal parameters. Design: Clinical and biochemical data of children with idiopathic GHD at diagnosis and annually up to 36 months of GHT were retrospectively evaluated. Overall, 255 children reached 12 months, 140 children 24 months and 86 children 36 months of follow-up during GHT. Results: At baseline, 18.4% of GHD children and 10.1% of controls showed normocytic anemia. GHD children showed lower hemoglobin (Hb)…

MalePediatricsmedicine.medical_specialtyAnemiaEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismSettore MED/13 - EndocrinologiaGrowth hormone deficiency03 medical and health sciences0302 clinical medicineEndocrinologyHumansGrowth hormone treatmentMedicineHematopoiesiInsulin-Like Growth Factor IChildChildrenGrowth hormoneGrowth DisordersRetrospective StudiesHuman Growth Hormonebusiness.industryAnemiaNormocytic anemiamedicine.diseaseHematopoiesisGrowth hormone treatmentHaematopoiesisChild Preschool030220 oncology & carcinogenesisErythropoiesisFemaleGrowth hormone deficiencyHemoglobinbusinessHormoneGrowth Hormone & IGF Research
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Human periodontal fibroblast response to a nanostructured hydroxyapatite bone replacement graft in vitro

2007

Abstract Objective The efficacy of nanostructured hydroxyapatite (NHA) for the treatment of osseous defects has been demonstrated in recent studies, even though the underlining biological mechanism is still poorly known. This study examined the alterations in cellular adhesion and mitogenic responses in human periodontal ligament (PDL) cells treated with a novel nanostructured hydroxyapatite bone graft substitute and characterized associated changes in cellular signalling pathways. Methods Cultured PDL cells were stimulated with NHA in a surface coated form. Proliferation was determined by bromodeoxyuridine (BrdU) incorporation and cell adhesion was analysed by a colorimetric assay. In orde…

MalePeriodontal LigamentIntegrinBiocompatible MaterialsFocal adhesionstomatognathic systemCell AdhesionHumansEpidermal growth factor receptorCell adhesionProtein kinase AGeneral DentistryProtein kinase BCells CulturedCell ProliferationbiologyChemistryCell BiologyGeneral MedicineAnatomyFibroblastsNanostructuresCell biologyErbB ReceptorsDurapatiteOtorhinolaryngologyFocal Adhesion Kinase 1Mitogen-activated protein kinasebiology.proteinPhosphorylationFemaleMitogen-Activated Protein KinasesSignal TransductionArchives of Oral Biology
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Autologous Platelet-Rich Plasma in the treatment of androgenetic alopecia: results of a 54-patient prospective study

2021

The current medical treatments of androgenetic alopecia (AGA) have hardly achieved a satisfying clinical improvement. Biologic regenerative therapies, such as platelet-rich plasma (PRP) injections in the scalp, have been proposed recently. This multidisciplinary prospective study aims to explore the efficacy and safety of autologous PRP injections into the scalp of patients with AGA. Fifty-four patients with AGA (35 men and 19 women) were enrolled. Non-activated autologous PRP was injected into the androgen-related areas of the scalp. The study protocol consisted of three sessions of injections at 3-month interval. The effects were assessed by means of noninvasive methods. Safety profile, p…

MalePlatelet-Rich Plasma Autologous PRP Androgenetic Alopecia Woman AGA Man AGA Growth FactorScalpTreatment OutcomePlatelet-Rich PlasmaSettore MED/35 - Malattie Cutanee E VenereeHumansAlopeciaFemaleProspective StudiesHair
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Proneurotrophin Binding to P75 Neurotrophin Receptor (P75ntr) Is Essential for Brain Lesion Formation and Functional Impairment after Experimental Tr…

2015

Traumatic brain injury (TBI) initiates an excessive mediator release of e.g. neurotrophins, which promote neuronal survival, differentiation, and modulate synaptic plasticity. Paradoxically, mature forms of neurotrophins promote neuronal survival, whereas unprocessed forms of neurotrophins induce cell death through p75 neurotrophin receptor (p75NTR) signaling. p75NTR is widely expressed during synaptogenesis and is subsequently downregulated in adulthood. Repair mechanisms after acute cerebral insults can reactivate its expression. Therefore, the influence of p75NTR on secondary brain damage was addressed. mRNA levels of p75NTR and its ligands were quantified in brain tissue up to 7 days af…

MaleProgrammed cell deathmedicine.medical_specialtyTraumatic brain injurySynaptogenesisReceptors Nerve Growth FactorBrain damageMiceInternal medicineAnimalsMedicineLow-affinity nerve growth factor receptorRNA MessengerMice KnockoutBehavior AnimalCell Deathbiologybusiness.industrymedicine.diseaseMice Inbred C57BLDisease Models AnimalEndocrinologyBrain InjuriesSynaptic plasticitybiology.proteinFemalesense organsNeurology (clinical)medicine.symptomSignal transductionbusinessNeuroscienceProtein BindingSignal TransductionNeurotrophinJournal of Neurotrauma
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Shotgun Proteomics of Isolated Urinary Extracellular Vesicles for Investigating Respiratory Impedance in Healthy Preschoolers

2021

Urine proteomic applications in children suggested their potential in discriminating between healthy subjects from those with respiratory diseases. The aim of the current study was to combine protein fractionation, by urinary extracellular vesicle isolation, and proteomics analysis in order to establish whether different patterns of respiratory impedance in healthy preschoolers can be characterized from a protein fingerprint. Twenty-one 3–5-yr-old healthy children, representative of 66 recruited subjects, were selected: 12 late preterm (LP) and 9 full-term (T) born. Children underwent measurement of respiratory impedance through Forced Oscillation Technique (FOT) and no significant differen…

MaleProteomePharmaceutical SciencePhysiologyUrineUrineProteomicsAminopeptidasesAnalytical Chemistry0302 clinical medicineDrug DiscoveryElectric ImpedanceMedicineRespiratory systemproteomic0303 health sciencesTripeptidyl-Peptidase 1urine fractionationExtracellular vesicleTripeptidyl peptidase IRespiratory Function Testsforced oscillation techniqueChemistry (miscellaneous)Child PreschoolMolecular MedicineFemaleUrinary systemReceptors Cell SurfaceArticlelcsh:QD241-441Extracellular Vesicles03 medical and health sciencesproteomicslcsh:Organic chemistryHumansNerve Growth FactorsPhysical and Theoretical ChemistryDipeptidyl-Peptidases and Tripeptidyl-PeptidasesEye ProteinsShotgun proteomicsAngiopoietin-Like Protein 2Serpins030304 developmental biologypreschooler healthy childrenbusiness.industryOrganic ChemistryCubilinAngiopoietin-like Proteins030228 respiratory systemThy-1 Antigensextracellular vesicleSerine Proteasesbusiness
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Proteomic signature of the Dravet syndrome in the genetic Scn1a-A1783V mouse model.

2021

Abstract Background Dravet syndrome is a rare, severe pediatric epileptic encephalopathy associated with intellectual and motor disabilities. Proteomic profiling in a mouse model of Dravet syndrome can provide information about the molecular consequences of the genetic deficiency and about pathophysiological mechanisms developing during the disease course. Methods A knock-in mouse model of Dravet syndrome with Scn1a haploinsufficiency was used for whole proteome, seizure, and behavioral analysis. Hippocampal tissue was dissected from two- (prior to epilepsy manifestation) and four- (following epilepsy manifestation) week-old male mice and analyzed using LC-MS/MS with label-free quantificati…

MaleProteomics0301 basic medicineProteomeHippocampusEpilepsies MyoclonicHaploinsufficiencyScn1aHippocampusSynaptic TransmissionElevated Plus Maze TestEpilepsyMice0302 clinical medicineTandem Mass Spectrometry11-beta-Hydroxysteroid Dehydrogenase Type 1Genetic epilepsyCarbon-Nitrogen LigasesGene Knock-In TechniquesGliosisNeuronal PlasticityBehavior AnimalEpileptic encephalopathyImmunohistochemistryAstrogliosisNeurologyProteomeDisease ProgressionFemaleHaploinsufficiencySignal TransductionRC321-571Dopamine and cAMP-Regulated Phosphoprotein 32Neovascularization PhysiologicNeurosciences. Biological psychiatry. NeuropsychiatryBiologyNitric Oxide03 medical and health sciencesDravet syndromemedicineAnimalsHyperthermiaSocial Behaviorras-GRF1Proteomic Profilingmedicine.diseaseVascular Endothelial Growth Factor Receptor-2NAV1.1 Voltage-Gated Sodium ChannelDisease Models Animal030104 developmental biologyRotarod Performance TestSynaptic plasticityEpileptic Encephalopathy ; Genetic Epilepsy ; Mice ; Proteome ; Scn1aCalcium-Calmodulin-Dependent Protein Kinase Type 2Open Field TestNeuroscience030217 neurology & neurosurgeryChromatography Liquid
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Vascular effects of sildenafil in patients with pulmonary fibrosis and pulmonary hypertension: an ex vivo/in vitro study

2015

Sildenafil improves the 6-min walking distance in patients with idiopathic pulmonary fibrosis (IPF) and right-sided ventricular systolic dysfunction.We analysed the previously unexplored role of sildenafil on vasoconstriction and remodelling of pulmonary arteries from patients with IPF and pulmonary hypertension (PH) ex vivo. Pulmonary arteries from 18 donors without lung disease, nine IPF, eight PH+IPF and four PH patients were isolated to measure vasodilator and anti-contractile effects of sildenafil in isometric organ bath. Ventilation/perfusion was explored in an animal model of bleomycin lung fibrosis.Sildenafil relaxed serotonin (5-HT) pre-contracted pulmonary arteries in healthy dono…

MalePulmonary FibrosisVasodilator AgentsVasodilation030204 cardiovascular system & hematologyPulmonary Disease Chronic ObstructiveIdiopathic pulmonary fibrosischemistry.chemical_compound0302 clinical medicinePulmonary fibrosisMyofibroblastsLungrespiratory systemExtracellular Matrixmedicine.anatomical_structurecardiovascular systemCardiologymedicine.symptomSignal TransductionPulmonary and Respiratory MedicineSerotoninmedicine.medical_specialtyEndotheliumSildenafilHypertension PulmonaryMyocytes Smooth MusclePulmonary ArterySildenafil CitrateTransforming Growth Factor beta1Bleomycin03 medical and health sciencesmedicine.arteryInternal medicinemedicineAnimalsHumansRats Wistarbusiness.industryFibroblastsmedicine.diseasePulmonary hypertensionRatsrespiratory tract diseasesDisease Models Animal030228 respiratory systemchemistryVasoconstrictionPulmonary arteryEndothelium VascularbusinessVasoconstrictionEuropean Respiratory Journal
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MUC1 intracellular bioactivation mediates lung fibrosis

2019

BackgroundSerum KL6/mucin 1 (MUC1) has been identified as a potential biomarker in idiopathic pulmonary fibrosis (IPF), but the role of MUC1 intracellular bioactivation in IPF is unknown.ObjectiveTo characterise MUC1 intracellular bioactivation in IPF.Methods and resultsThe expression and phosphorylation of Thr41 and Tyr46 on the intracellular MUC1-cytoplasmic tail (CT) was increased in patients with IPF (n=22) compared with healthy subjects (n=21) and localised to fibroblasts and hyperplastic alveolar type II cells. Transforming growth factor (TGF)-β1 phosphorylated SMAD3 and thereby increased the phosphorylation of MUC1-CT Thr41 and Tyr46 in lung fibroblasts and alveolar type II cells, ac…

MalePulmonary and Respiratory MedicineCellRisk AssessmentTransforming Growth Factor beta1BleomycinMice03 medical and health sciencesIdiopathic pulmonary fibrosis0302 clinical medicinemedicineAnimalsHumansGene silencingMolecular Targeted TherapyRNA MessengerSmad3 ProteinFibroblastneoplasmsCells CulturedMUC1030304 developmental biologyMice Knockout0303 health sciencesbusiness.industryBiopsy NeedleMucin-1Fibroblastsmedicine.diseaseImmunohistochemistryIdiopathic Pulmonary Fibrosisdigestive system diseasesDisease Models Animalmedicine.anatomical_structureGene Expression Regulation030228 respiratory systemCancer researchFemalebusinessMyofibroblastIntracellularSignal TransductionTransforming growth factorThorax
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Upregulation of basic fibroblast growth factor in smokers with chronic bronchitis

2006

The aim of the study was to investigate the expression of basic fibroblast growth factor (bFGF) and its receptor, fibroblast growth factor receptor (FGFR)-1, in the central airways of smokers with chronic bronchitis. The lobar bronchi from 17 subjects undergoing thoracotomy for solitary nodules were examined. All had a history of cigarette smoking, nine had symptoms of chronic bronchitis and airflow limitation, and eight were asymptomatic with normal lung function. Using immunohistochemical methods, bFGF and FGFR-1 expression in the total airway wall and the different airway compartments, i.e. bronchial glands, submucosal vessels and smooth muscle, was quantified. Moreover, to investigate t…

MalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyChronic bronchitisAngiogenesisBasic fibroblast growth factorFibroblast growth factorchemistry.chemical_compoundGrowth factor receptorCigarette smokingHumansMedicineAgedFibrolast growth factor receptor-1business.industryChronic obstructive pulmonary diseaseFibroblast growth factor receptor 1Smokingrespiratory systemmedicine.diseaseAirway remodelling; cigarette smoking; chronic obstructive pulmonary disease; fibroblast growth factor receptor-1Airway remodellingUp-Regulationrespiratory tract diseasesBronchitis ChronicchemistryFibroblast growth factor receptorBronchitisFemaleFibroblast Growth Factor 2Airway remodelling; Chronic obstructive pulmonary disease; Cigarette smoking; Fibrolast growth factor receptor-1;business
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Hypoxia and myocardial remodeling in human cardiac allografts: a time-course study.

2009

Background: Cardiac allografts are known to develop myocardial fibrosis, which may be a cause of progressive cardiac dysfunction. Apart from the renin‐angiotensin and transforming growth factor- system, hypoxia has been proposed as an important player in the pathogenesis of fibrosis, but its significance remains unclear. This study examines the degree of myocardial fibrosis, cellular remodeling and hypoxic signaling over a time-course of 10 years after human cardiac allograft transplantation. Methods: Serial right ventricular biopsies of 57 patients were collected in 6-month intervals after cardiac transplant surgery for a total of 10 years to allow a retrospective longitudinal analysis. Ov…

MalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyTime FactorsHeart DiseasesHeart Ventriclesmedicine.medical_treatmentMuscle hypertrophychemistry.chemical_compoundFibrosisHumansTransplantation HomologousMedicineLung transplantationHypoxiaTransplantationVentricular Remodelingbusiness.industryMiddle AgedHypoxia (medical)Endomyocardial Fibrosismedicine.diseaseVascular endothelial growth factorTransplantationchemistryHypertensionCirculatory systemHeart TransplantationFemaleSurgeryMyocardial fibrosismedicine.symptomCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesThe Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation
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