Search results for "Herpes"

showing 10 items of 307 documents

Impact of infectious burden on extent and long-term prognosis of atherosclerosis.

2002

Background — Recent findings suggest a causative role of infections in the pathogenesis of atherosclerosis. In hypothesizing an association between infectious agents and the development of atherosclerosis, we would expect a correlation to the extent of atherosclerosis. Moreover, this effect could be multiplied by the number of pathogens to which an individual had been exposed. Methods and Results — In 572 patients, IgG or IgA antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-Barr virus, Hemophilus influenzae , Chlamydia pneumoniae , Mycoplasma pneumoniae , and Helicobacter pylori were measured. The extent of atherosclerosis was determined by coronary angiography, carotid…

Human cytomegalovirusMalemedicine.medical_specialtyHerpesvirus 4 HumanTime FactorsArteriosclerosisCarotid arteriesCytomegalovirusCoronary diseasemedicine.disease_causeAntibodies ViralHerpesviridaePathogenesisRisk FactorsPhysiology (medical)medicineHumansSimplexvirusIntensive care medicineAgedHelicobacter pyloribusiness.industryBacterial InfectionsChlamydophila pneumoniaeMiddle Agedmedicine.diseasePrognosisAntibodies BacterialHaemophilus influenzaeSurvival AnalysisDNA Virus InfectionsImmunoglobulin AMycoplasma pneumoniaeSurvival RateC-Reactive ProteinLogistic ModelsHerpesvirus hominisImmunoglobulin GImmunologyMultivariate AnalysisVirusesFemaleCardiology and Cardiovascular MedicinebusinessCirculation
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Frequent coinfection of cells explains functional in vivo complementation between cytomegalovirus variants in the multiply infected host.

2005

In contrast to many other virus infections, primary cytomegalovirus (CMV) infection does not fully protect against reinfection. Accordingly, clinical data have revealed a coexistence of multiple human CMV variants/ strains in individual patients. Notably, the phenomenon of multiple infection was found to correlate with increased virus load and severity of CMV disease. Although of obvious medical relevance, the mechanism underlying this correlation is unknown. A weak immune response in an individual could be responsible for a more severe disease and for multiple infections. Alternatively, synergistic contributions of variants that differ in their biological properties can lead to qualitative…

Human cytomegalovirusMuromegalovirusImmunologyPopulationGreen Fluorescent ProteinsBiologymedicine.disease_causeMicrobiologyHerpesviridaeVirusMiceViral ProteinsBetaherpesvirinaeVirologymedicineAnimalseducationLungeducation.field_of_studyMice Inbred BALB CIntegrasesVirulenceGenetic VariationInborn immunodeficiencyCytomegalovirusmedicine.diseasebiology.organism_classificationVirologyGenetic Diversity and EvolutionInsect ScienceImmunologyCytomegalovirus InfectionsCoinfectionNIH 3T3 CellsFemaleSpleenJournal of virology
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Experimental Preemptive Immunotherapy of Murine Cytomegalovirus Disease with CD8 T-Cell Lines Specific for ppM83 and pM84, the Two Homologs of Human …

2001

ABSTRACTCD8 T cells are the principal antiviral effectors controlling cytomegalovirus (CMV) infection. For human CMV, the virion tegument protein ppUL83 (pp65) has been identified as a source of immunodominant peptides and is regarded as a candidate for cytoimmunotherapy and vaccination. Two sequence homologs of ppUL83 are known for murine CMV, namely the virion protein ppM83 (pp105) expressed late in the viral replication cycle and the nonstructural protein pM84 (p65) expressed in the early phase. Here we show that ppM83, unlike ppUL83, is not delivered into the antigen presentation pathway after virus penetration before or in absence of viral gene expression, while other virion proteins o…

Human cytomegalovirusMuromegalovirusmedicine.medical_treatmentImmunologyImmunodominanceCD8-Positive T-LymphocytesBiologyMicrobiologyCell LineViral Matrix ProteinsInterferon-gammaMiceImmune systemAntigenVirologyVaccines and Antiviral AgentsmedicineAnimalsCytotoxic T cellMice Inbred BALB CHerpesviridae InfectionsImmunotherapyPhosphoproteinsmedicine.diseaseAdoptive TransferVirologyPeptide FragmentsDisease Models AnimalViral replicationInsect ScienceImmunologyFemaleCytokine secretionImmunologic MemoryJournal of Virology
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Human cytomegalovirus glycoprotein B genotypes in immunocompetent, immunocompromised, and congenitally infected Italian populations

2003

Human cytomegalovirus (HCMV) strains, obtained from immunocompetent and immunocompromised Italian hosts, were typed with glycoprotein B (gB) gene restriction analysis. A predominant circulation of HCMV strains with gB type 2 and 3 was detected in both the immunocompetent host with a primary HCMV infection and the immunocompromised host with or without HCMV disease. No association between gB types and subjects with different risks of developing HCMV disease was found. All four gB genotypes were capable of causing congenital infection in Italian babies, with gB type 1 accounting for 50% of the strains examined in symptomatic infants and a remarkable incidence of gB type 4 viruses.

Human cytomegalovirusSettore MED/07 - Microbiologia E Microbiologia Clinicamedicine.medical_specialtyGenotypevirusesRestriction MappingCongenital cytomegalovirus infectionCytomegalovirusHIV Infectionsmedicine.disease_causePolymerase Chain ReactionHerpesviridaeVirusImmunocompromised HostMedical microbiologyViral Envelope ProteinsBetaherpesvirinaeVirologyGenotypemedicineHumansBone Marrow TransplantationbiologyInfant Newbornvirus diseasesGeneral Medicinebiology.organism_classificationmedicine.diseaseKidney TransplantationVirologyHuman cytomegalovirus immunocompromised gB genotypes ItalyCytomegalovirus InfectionsViral diseaseImmunocompetenceArchives of Virology
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Inhibition of CD1 antigen presentation by human cytomegalovirus.

2008

ABSTRACTThe betaherpesvirus human cytomegalovirus (HCMV) encodes several molecules that block antigen presentation by the major histocompatibility complex (MHC) proteins. Humans also possess one other family of antigen-presenting molecules, the CD1 family; however, the effect of HCMV on CD1 expression is unknown. The majority of CD1 molecules are classified on the basis of homology as group 1 CD1 and are present almost exclusively on professional antigen-presenting cells such as dendritic cells, which are a major target for HCMV infection and latency. We have determined that HCMV encodes multiple blocking strategies targeting group 1 CD1 molecules. CD1 transcription is strongly inhibited by…

Human cytomegalovirusTranscription GeneticvirusesImmunologyAntigen presentationCD1Cytomegaloviruschemical and pharmacologic phenomenaMajor histocompatibility complexmedicine.disease_causeMicrobiologycomplex mixturesCell LineAntigens CD1Immune systemAntigenVirologyMHC class ImedicineHumansCells CulturedAntigen PresentationbiologyImmunityhemic and immune systemsmedicine.diseaseVirologyProtein TransportHerpes simplex virusGene Expression RegulationInsect Sciencebiology.proteinPathogenesis and Immunitylipids (amino acids peptides and proteins)Journal of virology
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The viral chemokine MCK-2 of murine cytomegalovirus promotes infection as part of a gH/gL/MCK-2 complex.

2013

Human cytomegalovirus (HCMV) forms two gH/gL glycoprotein complexes, gH/gL/gO and gH/gL/pUL(128,130,131A), which determine the tropism, the entry pathways and the mode of spread of the virus. For murine cytomegalovirus (MCMV), which serves as a model for HCMV, a gH/gL/gO complex functionally homologous to the HCMV gH/gL/gO complex has been described. Knock-out of MCMV gO does impair, but not abolish, virus spread indicating that also MCMV might form an alternative gH/gL complex. Here, we show that the MCMV CC chemokine MCK-2 forms a complex with the glycoprotein gH, a complex which is incorporated into the virion. We could additionally show that mutants lacking both, gO and MCK-2 are not ab…

Human cytomegalovirusViral DiseasesMuromegalovirusChemokinevirusesMurine Cytomegalovirus ; viral chemokine MCK-2 ; gH/gL/MCK-2 complexMiceViral Envelope ProteinsBiology (General)Cells Culturedchemistry.chemical_classificationMice Inbred BALB Cvirus diseasesHerpesviridae InfectionsRecombinant ProteinsSpecific Pathogen-Free OrganismsInfectious DiseasesLiverChemokines CCMedicineFemaleResearch ArticleQH301-705.5ImmunologyBiologyMicrobiologyVirusCell LineViral ProteinsMuromegalovirusGlycoprotein complexVirologyGeneticsmedicineAnimalsBiologyMolecular BiologyTropismMacrophagesVirionVirus InternalizationRC581-607medicine.diseasebiology.organism_classificationVirologyImmunity InnatechemistryCell cultureMutationMacrophages Peritonealbiology.proteinParasitologyProtein MultimerizationImmunologic diseases. AllergyGlycoprotein
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Longitudinal analysis of human cytomegalovirus glycoprotein B (gB)-specific and neutralizing antibodies in AIDS patients either with or without cytom…

2001

Serum neutralizing and glycoprotein B (gB)-specific antibody levels were monitored prospectively in AIDS patients who either did or did not develop human cytomegalovirus (HCMV) end-organ disease, to delineate further the role of antibodies in protecting against HCMV disease. Antibody levels declined substantially (at least 4-fold) only in patients who developed HCMV disease; this decline in turn occurred concurrently with antigenemia. Nevertheless, AIDS patients who remained free of HCMV disease and did not become antigenemic during the follow-up period maintained stable levels of serum antibodies, with only minor fluctuations. The impact of HAART on the levels of functional anti-HCMV antib…

Human cytomegalovirusbiologybusiness.industryvirusesAntibody titerCongenital cytomegalovirus infectionvirus diseasesmedicine.disease_causemedicine.diseasebiology.organism_classificationVirologyHerpesviridaeInfectious DiseasesBetaherpesvirinaeVirologyImmunopathologyImmunologymedicinebiology.proteinViral diseaseAntibodybusinessJournal of Medical Virology
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Identification of a Conserved HLA-A2-Restricted Decapeptide from the IE1 Protein (pUL123) of Human Cytomegalovirus

2002

Abstract Control of human cytomegalovirus (HCMV) infection is predominantly mediated by cytolytic CD8 + T lymphocytes (CTL). Among the roughly 200 HCMV-encoded polypeptides, the tegument protein pp65 (ppUL83) and the nonstructural IE1 protein are considered to be dominant CTL targets. Yet the importance of CTL against IE1 for protective immunity against HCMV reactivation and disease has remained elusive. Analyses have been difficult, as all MHC class I presented peptides of IE1 defined so far are located in parts of the protein that are variable between viral strains. In this study a conserved decameric peptide from IE1 (P6, IE1 354–363 ) that bound to HLA-A2 was identified. Using peptide-p…

Human cytomegalovirusherpesvirusesViral proteinvirusesMolecular Sequence DataIE1CytomegalovirusEpitopes T-Lymphocytecytotoxic T lymphocytesmedicine.disease_causeImmediate early proteinCell LineImmediate-Early ProteinsViral Proteinsconserved CTL epitopesVirologyHLA-A2 AntigenMHC class ImedicineHumansCytotoxic T cellAmino Acid SequenceConserved SequencebiologyELISPOTvirus diseasesHLA-A2biochemical phenomena metabolism and nutritionCytotoxicity Tests Immunologicmedicine.diseaseVirologyPeptide FragmentsVirus LatencyCTL*human cytomegalovirusCytomegalovirus InfectionsImmunologybiology.proteinPeptidesCD8T-Lymphocytes CytotoxicVirology
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Non-redundant and redundant roles of cytomegalovirus gH/gL complexes in host organ entry and intra-tissue spread

2015

Herpesviruses form different gH/gL virion envelope glycoprotein complexes that serve as entry complexes for mediating viral cell-type tropism in vitro; their roles in vivo, however, remained speculative and can be addressed experimentally only in animal models. For murine cytomegalovirus two alternative gH/gL complexes, gH/gL/gO and gH/gL/MCK-2, have been identified. A limitation of studies on viral tropism in vivo has been the difficulty in distinguishing between infection initiation by viral entry into first-hit target cells and subsequent cell-to-cell spread within tissues. As a new strategy to dissect these two events, we used a gO-transcomplemented ΔgO mutant for providing the gH/gL/gO…

Human cytomegalovirusherpesvirusesvirusesgH/FL complexesCytomegalovirusMiceViral Envelope ProteinsMedizinische FakultätBiology (General)In Situ Hybridization0303 health sciencesMice Inbred BALB CMembrane GlycoproteinsImmunohistochemistrycytomegalovirus ; gH/FL complexes ; gO ; MCK-2 ; herpesvirusesCytomegalovirus InfectionsFemaleMCK-2BIOMEDICINA I ZDRAVSTVO. Temeljne medicinske znanosti.Research ArticleQH301-705.5Immunology-BiologyMicrobiologyVirus03 medical and health sciencesgOViral entryIn vivoVirologyGeneticsmedicineAnimalsddc:610Molecular BiologyTropism030304 developmental biology030306 microbiologyBIOMEDICINE AND HEALTHCARE. Basic Medical Sciences.RC581-607medicine.diseaseVirologyHerpesvirus glycoprotein BDisease Models AnimalViral TropismCell cultureTissue tropismParasitologyImmunologic diseases. Allergy
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Clinically-based determination of safe DNAemia cutoff levels for preemptive therapy or human cytomegalovirus infections in solid organ and hematopoie…

2004

Transplantation Centers using human cytomegalovirus (HCMV) antigenemia-based preemptive therapy will need to replace in the near future the antigenemia assay with a more standardized and automatable assay, such as a molecular assay quantifying HCMV DNA in blood (DNAemia). Thus, in view of replacing antigenemia with clinically safe cutoff values, DNAemia levels corresponding to antigenemia cutoffs guiding HCMV preemptive therapy were determined retrospectively in solid organ and hematopoietic stem cell transplant recipients (HSCTR) using an "in-house" quantitative PCR (QPCR) method. Since preemptive therapy had prevented appearance of HCMV disease in all patients tested, DNA cutoffs determin…

Human cytomegalovirusmedicine.medical_specialtymedicine.medical_treatmentCytomegalovirusHematopoietic stem cell transplantationAntiviral AgentsPolymerase Chain ReactionOrgan transplantationPostoperative ComplicationsPredictive Value of TestsBetaherpesvirinaeVirologyPositive predicative valuemedicineHumansCutoffViremiaAntigens ViralRetrospective StudiesAntiviral Agentbiologybusiness.industryHematopoietic Stem Cell Transplantationvirus diseasesCytomegaloviruOrgan Transplantationbiology.organism_classificationmedicine.diseaseVirologyTransplantationInfectious DiseasesPredictive value of testsCytomegalovirus InfectionsDNA Viralbusiness
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