Search results for "Prom"

showing 10 items of 2286 documents

Selective Activation of Trophoblast-specific PLAC1 in Breast Cancer by CCAAT/Enhancer-binding Protein β (C/EBPβ) Isoform 2

2009

The trophoblast-specific gene PLAC1 (placenta-specific 1) is ectopically expressed in a wide range of human malignancies, most frequently in breast cancer, and is essentially involved in cancer cell proliferation, migration, and invasion. Here we show that basal activity of the PLAC1 promoter is selectively controlled by ubiquitous transcription factor SP1 and isoform 2 of CCAAT/enhancer-binding protein beta that we found to be selectively expressed in placental tissue and cancer cells. Binding of both factors to their respective elements within the PLAC1 promoter was essential to attain full promoter activity. Estrogen receptor alpha (ERalpha) signaling further augmented transcription and …

Gene isoformSp1 Transcription FactorMolecular Sequence DataEstrogen receptorBreast NeoplasmsPregnancy ProteinsBiologyBiochemistryTransactivationMolecular Basis of Cell and Developmental BiologyTranscription (biology)Cell Line TumorGene expressionHumansProtein IsoformsPromoter Regions GeneticMolecular BiologyCell ProliferationSp1 transcription factorBase SequenceCcaat-enhancer-binding proteinsCCAAT-Enhancer-Binding Protein-betaEstrogen Receptor alphaEstrogensCell BiologyMolecular biologyTrophoblastsGene Expression Regulation NeoplasticChromatin immunoprecipitationJournal of Biological Chemistry
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Autoregulation of NFATc1/A Expression Facilitates Effector T Cells to Escape from Rapid Apoptosis

2002

AbstractThreshold levels of individual NFAT factors appear to be critical for apoptosis induction in effector T cells. In these cells, the short isoform A of NFATc1 is induced to high levels due to the autoregulation of the NFATc1 promoter P1 by NFATs. P1 is located within a CpG island in front of exon 1, represents a DNase I hypersensitive chromatin site, and harbors several sites for binding of inducible transcription factors, including a tandemly arranged NFAT site. A second promoter, P2, before exon 2, is not controlled by NFATs and directs synthesis of the longer NFATc1/B+C isoforms. Contrary to other NFATs, NFATc1/A is unable to promote apoptosis, suggesting that NFATc1/A enhances eff…

Gene isoformTranscription GeneticMolecular Sequence DataImmunologyApoptosisBiologyT-Lymphocytes RegulatoryJurkat CellsMiceExonAnimalsDeoxyribonuclease IHomeostasisHumansImmunology and AllergyPromoter Regions GeneticTranscription factorMice Inbred BALB CBase SequenceNFATC Transcription Factorsintegumentary systemEffectorNuclear ProteinsNFATDNA MethylationMolecular biologyChromatinDNA-Binding ProteinsAlternative SplicingInfectious DiseasesCpG siteApoptosisElectrophoresis Polyacrylamide GelPoly ATranscription FactorsImmunity
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Regulation of the expression of inducible nitric oxide synthase

2004

The role of nitric oxide (NO) generated by the inducible isoform of nitric oxide synthase (iNOS) is very complex. Induction of iNOS expression and hence NO production has been described to have beneficial antiviral, antiparasital, microbicidal, immunomodulatory, and antitumoral effects. However, induced at the wrong place or at the wrong time, iNOS has detrimental consequences and seems to be involved in the pathophysiology of different human diseases. The pathways regulating iNOS expression seem to vary in different cells or different species. In general, activation of the transcription factors nuclear factor (NF)-kappaB and signal transducer and activator of transcription (STAT)-1alpha an…

Gene isoformTranscription GeneticNitric Oxide Synthase Type IIBiologyGene Expression Regulation EnzymologicstatNitric oxidechemistry.chemical_compoundAnimalsHumansRNA MessengerPromoter Regions GeneticTranscription factorPharmacologyRegulation of gene expressionMolecular biologyCell biologyNitric oxide synthasechemistryProtein BiosynthesisSTAT proteinbiology.proteinNitric Oxide SynthaseSignal transductionSignal TransductionTranscription FactorsEuropean Journal of Pharmacology
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PTHrP in differentiating human mesenchymal stem cells: Transcript isoform expression, promoter methylation, and protein accumulation

2013

Human PTHrP gene displays a complex organization with nine exons producing diverse mRNA variants due to alternative splicing at 5' and 3' ends and the existence of three different transcriptional promoters (P1, P2 and P3), two of which (P2 and P3) contain CpG islands. It is known that the expression of PTHrP isoforms may be differentially regulated in a developmental stage- and tissue-specific manner. To search for novel molecular markers of stemness/differentiation, here we have examined isoform expression in fat-derived mesenchymal stem cells both maintained in stem conditions and induced toward adipo- and osteogenesis. In addition, the expression of the splicing isoforms derived from P2 …

Gene isoformTranscription GeneticPTHrPCellular differentiationpromoter methylationBiologyOsteocytesBiochemistryGene expressionAdipocytesHumansProtein IsoformsadipogenesiSettore BIO/06 - Anatomia Comparata E CitologiaPromoter Regions Geneticmesenchymal stem cellCells CulturedMessenger RNAMesenchymal stem cellAlternative splicingParathyroid Hormone-Related ProteinCell DifferentiationMesenchymal Stem CellsExonsGeneral MedicineMethylationDNA MethylationosteogenesiMolecular biologyIntronsPTHrP; mesenchymal stem cells; osteogenesis; adipogenesis; gene expression; promoter methylationAlternative SplicingSettore BIO/18 - GeneticaGene Expression Regulationgene expressionCpG IslandsStem cellBiochimie
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Claudin-18 gene structure, regulation, and expression is evolutionary conserved in mammals

2011

Claudin-18 isoform 2 (CLDN18.2) is one of the few members of the human claudin family of tight junction molecules with strict restriction to one cell lineage. The objective of the current study was to compare molecular structure and tissue distribution of this gastrocyte specific molecule in mammals. We show here that the CLDN18.2 protein sequence is highly conserved, in particular with regard to functionally relevant domains in mouse, rat, rabbit, dog, monkey and human and also in lizards. Moreover, promoter regions of orthologs are highly homologous, including the binding site of the transcription factor cyclic AMP-responsive element binding protein (CREB), which is known to regulate acti…

Gene isoformmiceMolecular Sequence DataGene Expressionmolecular structureMammals/geneticsBiologyphylogenyRATSConserved sequenceEvolution MolecularDogsProtein Isoforms/geneticsSequence Homology Nucleic AcidGene expressionGeneticsProtein IsoformsAnimalsTissue DistributionAmino Acid SequenceMembrane Proteins/geneticsBinding sitePromoter Regions GeneticClaudinGeneTranscription factorConserved SequenceGastric Mucosa/metabolismMammalsRegulation of gene expressionGeneticsBinding SitesBase SequenceStomachStomach/cytologyMembrane ProteinsCREB-Binding Protein/metabolismHaplorhiniGeneral MedicineCREB-Binding ProteinGene Expression RegulationGastric MucosaOrgan SpecificityMultigene FamilyClaudinsRabbitsGene
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Hsp70 is required for optimal cell proliferation in mouse A6 mesoangioblast stem cells.

2009

Mouse Hsp70 (70 kDa heat shock protein) is preferentially induced by heat or stress stimuli. We previously found that Hsp70 is constitutively expressed in A6 mouse mesoangioblast stem cells, but its possible role in these cells and the control of its basal transcription remained unexplored. Here we report that in the absence of stress, Ku factor is able to bind the HSE (heat shock element) consensus sequence in vitro, and in vivo it is bound to the proximal hsp70 promoter. In addition, we show that constitutive hsp70 transcription depends on the co-operative interaction of different factors such as Sp1 (specificity protein 1) and GAGA-binding protein with Ku factor, which binds the HSE cons…

Gene knockdownMesoangioblastBinding SitesGeneral transcription factorCell growthStem CellsCell BiologyBiologyFlow CytometryBiochemistryMolecular biologyHsp70MiceTranscription (biology)Heat shock proteinAnimalsBlood VesselsHSP70 Heat-Shock ProteinsRNA InterferenceStem cellmesoangioblast RNAi doubling timePromoter Regions GeneticMolecular BiologyCell ProliferationTranscription FactorsThe Biochemical journal
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LEVEL OF ACTIVATION AND GOAL-DIRECTED BEHAVIOUR: The effect of chlorpromazine on aggressive, achievement oriented, and affiliation oriented verbal re…

1963

The purpose was to investigate the effect of chlorpromazine on verbal response contents in sentence completion tests. 30 male subjects participated in two chlorpromazine and one placebo session. It was found that the amount of aggressive, achievement and affiliation oriented responses was significantly reduced as an effect of drug intake. The results are discussed on the basis of the theories of conflict motivation and of arousal.

General MedicineVerbal responsePlaceboSentence completion testsArousalDevelopmental psychologyArts and Humanities (miscellaneous)Developmental and Educational PsychologymedicineSession (computer science)Drug intoxicationChlorpromazinePsychologyGeneral Psychologymedicine.drugClinical psychologyScandinavian Journal of Psychology
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Functional Significance of Microsatellite Markers

2014

The review summarizes literature data on the positive results of association studies between the length of microsatellite repeats and predisposition to pathologies. Actually, the data can be classified according to the localization of the microsatellite: in the gene promoter, in the part of exon 1 coding the signal sequence, in gene introns, in the coding areas of genes, and in 3'-untranslated regions. The functional significance of microsatellite length changes can be evaluated in many cases. The authors came up to the conclusion that further studies on microsatellite associations with diseases remain prospective as they reflect changes in the gene functional activity.

Genetic MarkersGeneticsUntranslated regionbusiness.industryIntronPromoterExonsGeneral MedicineIntronsExonHumansFunctional significanceMicrosatelliteMedicineGenetic Predisposition to DiseasePromoter Regions Geneticbusiness3' Untranslated RegionsGeneMicrosatellite RepeatsGenetic associationMedicina
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The human fascin gene promoter is highly active in mature dendritic cells due to a stage-specific enhancer.

2003

Abstract Dendritic cells (DC), regarded as the most efficient APCs of the immune system, are capable of activating naive T cells. Thus, DC are primary targets in immunotherapy. However, little is known about gene regulation in DC, and for efficient transcriptional targeting of human DC, a suitable promoter is still missing. Recently, we successfully used the promoter of the murine actin-bundling protein fascin to transcriptionally target DC by DNA vaccination in mice. In this study, we report on isolation of the human fascin promoter and characterization of its regulatory elements. The actively expressed gene was distinguished from a conserved inactive genomic locus and a continuous region …

Genetic MarkersRetroelementsTATA boxImmunologyMolecular Sequence DataCAAT boxRegulatory Sequences Nucleic AcidCell LineTumor Cells CulturedImmunology and AllergyHumansAmino Acid SequenceGene SilencingEnhancerPromoter Regions GeneticGene3' Untranslated RegionsCells CulturedConserved SequenceFascinRegulation of gene expressionbiologyBase SequenceGenome HumanMicrofilament ProteinsPromoterCell DifferentiationDendritic CellsExonsMolecular biologyIntronsEnhancer Elements GeneticGene Expression RegulationRegulatory sequencebiology.proteinCarrier ProteinsPseudogenesJournal of immunology (Baltimore, Md. : 1950)
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Overexpression of bone morphogenetic protein-6 (BMP-6) in murine epidermis suppresses skin tumor formation by induction of apoptosis and downregulati…

2001

Bone morphogenetic protein-6 (BMP-6) is a member of the transforming growth factor-beta superfamily. In murine skin, BMP-6 is highly expressed in postmitotic keratinocytes from day 15.5 p.c. till day 6 p.p. Expression in adult skin remains at very low levels, but pathological conditions such as wounding induce the expression of BMP-6. We demonstrate that tumor promotion by TPA (12-O-tetradecanoylphorbol-13-acetate) also induces expression of BMP-6 in suprabasal keratinocytes. This induction is due to post-transcriptional regulation since the level of BMP-6 mRNA remained unchanged. We performed two-stage skin carcinogenesis experiments with transgenic mice epidermally overexpressing BMP-6. T…

Genetically modified mouseKeratinocytesCancer ResearchSkin NeoplasmsBone Morphogenetic Protein 6Transgene910-Dimethyl-12-benzanthraceneDown-RegulationApoptosisMice TransgenicBiologymedicine.disease_causeMiceDownregulation and upregulationGenes junGeneticsmedicineIn Situ Nick-End LabelingTumor Cells CulturedAnimalsRNA MessengerMolecular BiologyIn Situ Hybridizationintegumentary systemActivator (genetics)Reverse Transcriptase Polymerase Chain ReactionGenes fosImmunohistochemistryCell biologyBone morphogenetic protein 6ApoptosisImmunologyBone Morphogenetic ProteinsMutationTetradecanoylphorbol AcetateTumor promotionEpidermisCarcinogenesisCell DivisionOncogene
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