Search results for "REGION"

showing 10 items of 4910 documents

TGF-beta1 in liver fibrosis: an inducible transgenic mouse model to study liver fibrogenesis.

1999

Transforming growth factor-beta1 (TGF-beta1) is a powerful stimulus for collagen formation in vitro. To determine the in vivo effects of TGF-beta1 on liver fibrogenesis, we generated transgenic mice overexpressing a fusion gene [C-reactive protein (CRP)/TGF-beta1] consisting of the cDNA coding for an activated form of TGF-beta1 under the control of the regulatory elements of the inducible human CRP gene promoter. Two transgenic lines were generated with liver-specific overexpression of mature TGF-beta1. After induction of the acute phase response (15 h) with lipopolysaccharide (100 microgram ip), plasma TGF-beta1 levels reached600 ng/ml in transgenic animals, which is100 times above normal …

Genetically modified mouseLipopolysaccharidesmedicine.medical_specialtyTranscription GeneticPhysiologyTransgeneRecombinant Fusion ProteinsMice TransgenicBiologyRegulatory Sequences Nucleic AcidLiver Cirrhosis ExperimentalMiceDownregulation and upregulationFibrosisIn vivoTransforming Growth Factor betaPhysiology (medical)Internal medicinemedicineAnimalsHumansRNA MessengerPromoter Regions GeneticRegulation of gene expressionHepatologyGastroenterologymedicine.diseaseMolecular biologyImmunohistochemistryEndocrinologymedicine.anatomical_structureC-Reactive ProteinGene Expression RegulationLiverHepatocyteHepatic stellate cellCollagenProcollagen
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A novel in vivo inducible dendritic cell ablation model in mice

2010

Abstract Dendritic cells (DCs) are involved in T cell activation via their uptake and presentation of antigens. In vivo function of DCs was analyzed using transgenic mouse models that express diphtheria toxin receptor (DTR) or the diphtheria toxin-A subunit (DTA) under the control of the CD11c/Itgax promoter. However, CD11c+ cells are heterogeneous populations that contain several DC subsets. Thus, the in vivo function of each subset of DCs remains to be elucidated. Here, we describe a new inducible DC ablation model, in which DTR expression is induced under the CD11c/Itgax promoter after Cre-mediated excision of a stop cassette (CD11c-iDTR). Crossing of CD11c-iDTR mice with CAG-Cre transge…

Genetically modified mouseT cellBiophysicsCD11cCre recombinaseMice Transgenicchemical and pharmacologic phenomenaBiologyBiochemistryMiceAntigenIn vivomedicineAnimalsPromoter Regions GeneticMolecular BiologyIntegraseshemic and immune systemsDendritic CellsCell BiologyDendritic cellMolecular biologyIn vitroCD11c Antigenmedicine.anatomical_structureModels AnimalIntercellular Signaling Peptides and ProteinsHeparin-binding EGF-like Growth FactorBiochemical and Biophysical Research Communications
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Internal limiting membrane peeling versus no peeling during primary vitrectomy for rhegmatogenous retinal detachment: A systematic review and meta-an…

2018

Background Internal limiting membrane (ILM) peeling during primary vitrectomy for rhegmatogenous retinal detachment (RRD) prevents the formation of postoperative macular epiretinal membrane (ERM). However, studies that compared vitrectomy with and without ILM peeling for RRD, have reported controversial outcomes. Objective To assess the efficacy of ILM peeling versus non-ILM peeling during vitrectomy for RRD by a systematic review and meta-analysis of published studies. Methods PubMed, Medline, Web of Science, Embase databases, and the Cochrane Library were searched up to April 2018 to identify studies that compared primary vitrectomy with and without ILM peeling for RRD with at least six m…

Genetics and Molecular Biology (all)Visual acuitygenetic structuresVisionmedicine.medical_treatmentVisual Acuitylcsh:MedicineSocial SciencesVitrectomyBiochemistrylaw.inventionDatabase and Informatics Methods0302 clinical medicineMathematical and Statistical TechniquesPostoperative ComplicationsRandomized controlled trialBiochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)lawVitrectomyMedicine and Health SciencesPsychology030212 general & internal medicineDatabase Searchinglcsh:ScienceMultidisciplinaryOphthalmic ProceduresRetinal detachmentEpiretinal MembraneResearch AssessmentMeta-analysisPhysical SciencesRetinal DisordersSensory PerceptionEpiretinal membranemedicine.symptomAnatomyStatistics (Mathematics)HumanResearch Articlemedicine.medical_specialtySystematic ReviewsSurgical and Invasive Medical ProceduresResearch and Analysis Methods03 medical and health sciencesOcular SystemOphthalmologymedicineHumansStatistical MethodsBiochemistry Genetics and Molecular Biology (all)Primary vitrectomybusiness.industryInternal limiting membranelcsh:RRetinal DetachmentBiochemistry; Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)Biology and Life Sciencesmedicine.diseaseeye diseasesbody regionsOphthalmologyAgricultural and Biological Sciences (all)030221 ophthalmology & optometryEyeslcsh:QPostoperative Complicationsense organsbusinessHeadMathematicsMeta-AnalysisNeuroscience
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Flow Cytometric Immunobead Assay for Detection of BCR-ABL1 Fusion Proteins in Chronic Myleoid Leukemia: Comparison with FISH and PCR Techniques

2015

Chronic Myeloid Leukemia (CML) is characterized by a balanced translocation juxtaposing the Abelson (ABL) and breakpoint cluster region (BCR) genes. The resulting BCR-ABL1 oncogene leads to increased proliferation and survival of leukemic cells. Successful treatment of CML has been accompanied by steady improvements in our capacity to accurately and sensitively monitor therapy response. Currently, measurement of BCR-ABL1 mRNA transcript levels by real-time quantitative PCR (RQ-PCR) defines critical response endpoints. An antibody-based technique for BCR-ABL1 protein recognition could be an attractive alternative to RQ-PCR. To date, there have been no studies evaluating whether flow-cytometr…

Genetics and Molecular Biology (all)medicine.medical_specialtyScienceFusion Proteins bcr-ablBiologyBiochemistryPolymerase Chain ReactionInternal medicinehemic and lymphatic diseasesmedicineHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)In Situ Hybridization FluorescenceImmunoassayMultidisciplinaryABLHematologymedicine.diagnostic_testMedicine (all)QRbreakpoint cluster regionMyeloid leukemiaLeukemia Myelomonocytic Chronicmedicine.diseaseFlow CytometryMolecular biologyFusion proteinLeukemiaReal-time polymerase chain reactionAgricultural and Biological Sciences (all)ImmunoassayMedicineResearch ArticlePLoS ONE
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Low frequency and rare coding variation contributes to multiple sclerosis risk

2018

AbstractMultiple sclerosis is a common, complex neurological disease, where almost 20% of risk heritability can be attributed to common genetic variants, including >230 identified by genome-wide association studies (Patsopoulos et al., 2017). Multiple strands of evidence suggest that the majority of the remaining heritability is also due to the additive effects of individual variants, rather than epistatic interactions between these variants, or mutations exclusive to individual families. Here, we show in 68,379 cases and controls that as much as 5% of this heritability is explained by low-frequency variation in gene coding sequence. We identify four novel genes driving MS risk independe…

Genetics0303 health sciencesLinkage disequilibriumMultiple sclerosisDiseaseBiologyHeritabilitymedicine.disease3. Good health03 medical and health sciences0302 clinical medicinemedicineEpistasisCoding regionGene030217 neurology & neurosurgery030304 developmental biologyGenetic association
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Ascl1 Coordinately Regulates Gene Expression and the Chromatin Landscape during Neurogenesis

2015

Summary The proneural transcription factor Ascl1 coordinates gene expression in both proliferating and differentiating progenitors along the neuronal lineage. Here, we used a cellular model of neurogenesis to investigate how Ascl1 interacts with the chromatin landscape to regulate gene expression when promoting neuronal differentiation. We find that Ascl1 binding occurs mostly at distal enhancers and is associated with activation of gene transcription. Surprisingly, the accessibility of Ascl1 to its binding sites in neural stem/progenitor cells remains largely unchanged throughout their differentiation, as Ascl1 targets regions of both readily accessible and closed chromatin in proliferatin…

Genetics0303 health sciencesNeurogenesisNeurogenesisDNABiologyGeneral Biochemistry Genetics and Molecular BiologyChromatin remodelingArticleCell biologyChromatin03 medical and health sciences0302 clinical medicinelcsh:Biology (General)[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular Biology[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyAscl1Scaffold/matrix attachment regionEnhancerlcsh:QH301-705.5Transcription factor030217 neurology & neurosurgeryChIA-PET030304 developmental biologyBivalent chromatin
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Are polymorphisms of the β3 -adrenoceptor gene associated with an altered bladder function?

2012

Aims As the presence of a Trp64Arg polymorphism of the gene encoding the β3-adrenoceptor (B3AR) has been linked to the presence of overactive bladder, we investigated whether additional polymorphisms are detectable in this gene and explore their relationships parameters related to lower urinary tract function. Methods The coding region and adjacent stretches of the B3AR gene was sequenced in 91 patients. In total, 1015 patients from a single academic hospital were genotyped for the presence of two single nucleotide polymorphisms. Symptom scores and parameters from pressure-flow studies were analyzed relative to genotype in the B3AR gene. Results No frequent novel polymorphisms were detected…

GeneticsAdrenergic receptorbusiness.industryUrology030232 urology & nephrologySingle-nucleotide polymorphismmedicine.disease03 medical and health sciences0302 clinical medicineOveractive bladder030220 oncology & carcinogenesisGenotypeMedicineCoding regionNeurology (clinical)Gene polymorphismAllelebusinessGeneNeurourology and Urodynamics
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Characterization of a Cu/Zn Superoxide dismutase-encoding gene region in Drosophila willistoni

1994

A Cu/Zn superoxide dismutase-encoding gene (Sod) from Drosophila willistoni was cloned and sequenced. The gene shows a typical structure for a fruit-fly Sod gene, with a coding region of 462 bp in two exons separated by a 417-bp intron. Comparison of the Sod sequences from D. willistoni and D. melanogaster suggests that these species are only remotely related. Downstream from the Sod gene, there is an ORF on the opposite strand that putatively encodes the last exon of an unidentified gene. The polyadenylation signals of the two genes are separated by only 61 bp in D. willistoni, conforming to the common picture of compact dipteran genomes.

GeneticsBase SequencebiologyPolyadenylationSuperoxide DismutaseMolecular Sequence DataIntronDNAGeneral Medicinebiology.organism_classificationMolecular biologyDrosophila virilisOpen Reading FramesExonGeneticsMelanogasterAnimalsCoding regionDrosophilaDrosophila willistoniAmino Acid SequenceGeneGene
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The CpG island methylator phenotype in breast cancer is associated with the lobular subtype

2014

Background: Aberrations in DNA methylation patterns are well-described in human malignancies. However, the existence of the ‘CpG island methylator phenotype’ (CIMP) in human breast cancer is still controversial. Materials & methods: Illumina's HumanMethylation 450K BeadChip was used to analyze genome-wide DNA methylation patterns. Chromosomal abnormalities were determined by array-based CGH. Results: Invasive lobular breast carcinomas exhibit the highest number of differentially methylated CpG sites and a strong inverse correlation of aberrant DNA hypermethylation and copy number alterations. Nine differentially methylated regions within seven genes discriminating the investigated subg…

GeneticsCancer ResearchCpG Island Methylator PhenotypeGene ExpressionCancerBreast NeoplasmsDNA MethylationBiologymedicine.diseaseEpigenesis GeneticPhenotypeDifferentially methylated regionsBreast cancerCpG siteTumor progressionCell Line TumorDNA methylationGeneticsCancer researchmedicineHumansCpG IslandsFemaleEpigeneticsEpigenomics
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Interphase FISH assays for the detection of translocations with breakpoints in immunoglobulin light chain loci

2002

Many B-cell malignancies bear chromosomal translocations juxtaposing immunoglobulin (IG) genes with oncogenes, resulting in deregulated expression of the latter. Translocations affecting the IG heavy chain (IGH) locus in chromosomal region 14q32 are most prevalent. However, variant translocations involving the IG kappa (IGK) locus in 2p12 or the IG lambda (IGL) locus in 22q11 occur recurrently in B-cell neoplasias. No routine methods for the detection of all breakpoints involving IG light chain loci independently of the translocation partner have been described. For this reason, we have designed 2 novel interphase fluorescence in situ hybridization (FISH) assays using differentially labeled…

GeneticsCancer Researchmedicine.medical_specialtymedicine.diagnostic_testBreakpointCytogeneticsChromosomeLocus (genetics)Chromosomal translocationBiologyImmunoglobulin light chainMolecular biologyOncologyChromosomal regionmedicineFluorescence in situ hybridizationInternational Journal of Cancer
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