Search results for "angiogenesi"

showing 10 items of 568 documents

Antitumor effect of B16 melanoma cells genetically modified with the angiogenesis inhibitor rnasin.

2001

The growth of new blood vessels is an essential condition for the development of tumors with a diameter greater than 1-2 mm and also for their metastatic dissemination. RNasin, the placental ribonuclease inhibitor, is known to have antiangiogenic activity through the inhibition of angiogenin and basic fibroblast growth factor. Nevertheless, the administration of the recombinant form of a protein poses several limitations; as a result, we have studied the antitumor effect of RNasin in a murine gene therapy model. RNasin cDNA was subcloned into the pcDNA3 expression vector, and the resulting recombinant plasmid was used to transfect the B16 murine melanoma cell line. An RNasin inverted constr…

Cancer ResearchLung NeoplasmsAngiogeninTranscription GeneticGenetic enhancementCellBasic fibroblast growth factorGenetic VectorsMelanoma ExperimentalGene ExpressionAngiogenesis InhibitorsTransfectionNeovascularizationImmunoenzyme Techniqueschemistry.chemical_compoundMiceRibonucleasesmedicineTumor Cells CulturedAnimalsHumansRNA MessengerEnzyme InhibitorsMolecular BiologyDNA PrimersNeovascularization PathologicReverse Transcriptase Polymerase Chain ReactionMelanomaGenetic Therapymedicine.diseaseAngiogenesis inhibitormedicine.anatomical_structurechemistryCell cultureCancer researchMolecular Medicinemedicine.symptomPlacental HormonesCell DivisionCancer gene therapy
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Abstract B5: A BMP7 variant inhibits angiogenesis in vitro and in vivo in part by downregulating VEGFR2 and FGFR1 expression in endothelial cells.

2013

Abstract Glioblastoma multiforme (GBM), the most aggressive glioma, requires active angiogenesis for growth and survival. Bone morphogenetic proteins (BMPs), members of the TGF-β superfamily, have numerous biological activities including control of growth, differentiation, and vascular development. Previously, we demonstrated the use of a BMP7 variant (BMP7v) to differentiate glioblastoma stem-like cells (GSLCs) and significantly reduce their tumorigenic potential (Tate and Pallini et al. 2012). Using an in vitro co-culture endothelial cord formation assay, a surrogate of angiogenesis, and its cognate in vivo model, we investigated the role of BMP7v in VEGF, basic FGF (bFGF), tumor-driven a…

Cancer ResearchMatrigelbiologyAngiogenesisSMADFibroblast growth factorReceptor tyrosine kinaseEndothelial stem cellOncologyIn vivoMothers against decapentaplegic homolog 4Immunologybiology.proteinCancer researchMolecular Cancer Therapeutics
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Evidence for characteristic vascular patterns in solid tumours: quantitative studies using corrosion casts

1999

The vascular architecture of four different tumour cell lines (CaX, CaNT, SaS, HEC-1B) transplanted subcutaneously in mice was examined by means of microvascular corrosion casting in order to determine whether there is a characteristic vascular pattern for different tumour types and whether it differs significantly from two normal tissues, muscle and gut. Three-dimensional reconstructed scanning electron microscope images were used for quantitative measurements. Vessel diameters, intervessel and interbranch distances showed large differences between tumour types, whereas the branching angles were similar. In all tumours, the variability of the vessel diameters was significantly higher than …

Cancer ResearchPathologymedicine.medical_specialtyAngiogenesisTransplantation Heterologousvascular patternNormal tissueMice NudeAdenocarcinomaBiologyCorrosion CastingVascular architectureMiceMicroscopyTumor Cells CulturedmedicineAnimalsHumansmicrovascular corrosion castingtumourCarcinomaRegular ArticleNeoplasms ExperimentalAnatomymedicine.diseaseEndometrial NeoplasmsTransplantationxenograftsOncologyVascular networkrodentsMice Inbred CBAMicroscopy Electron ScanningFemaleSarcoma ExperimentalSarcomaCorrosion CastingNeoplasm TransplantationBritish Journal of Cancer
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Angiogenesis in neuroblastoma: relationship to survival and other prognostic factors in a cohort of neuroblastoma patients.

2000

PURPOSE: To study angiogenesis in neuroblastoma, using morphometric and computerized image analysis, and correlate the results with survival and other prognostic factors. PATIENTS AND METHODS: Sixty-nine patients from the Spanish Cooperative Study for Neuroblastoma were studied. Tumoral angiogenesis was studied using an avidin-biotin immunoperoxidase technique with an anti-CD34 antibody. Vascular parameters (VPs) were analyzed by a computerized system. Statistical analysis was also performed. RESULTS: Sixty-six samples had adequate tumoral tissue, and their tumoral vessels were counted. Endothelial cells were more prominent in pure neuroblastomas than in maturing and more mature tumors. VP…

Cancer ResearchPathologymedicine.medical_specialtyImmunoperoxidaseAngiogenesisbusiness.industrymedicine.diseaseNeovascularizationOncologyNeuroblastomamedicineImmunohistochemistryHistopathologyStage (cooking)medicine.symptombusinessSurvival rate
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Vascularity, perfusion rate and local tissue oxygenation of tumors derived from ras-transformed fibroblasts.

2007

Tumors derived from ras-transformed rat fibroblasts were investigated in order to gain insight into possible interrelationships between oncogenic transformations and therapeutically relevant parameters of the metabolic micromilieu of solid tumors in vivo. Tumors grew in nude mice after injection of in vitro-passaged cells. Growth rates, early stages of angiogenesis, perfusion and tissue oxygenation were assessed. Compared with the parental cell line, both ras transformants grew very rapidly and exhibited an early onset of angiogenesis. Perfusion rates of one ras-transformed tumor line were similar to those of the parental tumors whereas reduced flow values were detected in tumors of the oth…

Cancer ResearchPathologymedicine.medical_specialtyRatónAngiogenesisPartial PressureMice NudeBiologyTransfectionCell LineMiceVascularityOxygen ConsumptionIn vivomedicineAnimalsHumansCardiac OutputFibroblastOncogeneNeovascularization PathologicOxygenationArteriesNeoplasms ExperimentalRatsPerfusionThallium Radioisotopesmedicine.anatomical_structureCell Transformation NeoplasticGenes rasOncologyOrgan SpecificityRegional Blood FlowAutoradiographymedicine.symptomPerfusionCell DivisionInternational journal of cancer
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GLUT-1 staining of squamous cell carcinomas of the uterine cervix identifies a novel element of invasion.

2010

Perturbation of the normal tissue architecture in solid malignant tumors is perceived to be the consequence of actively migrating cancer cells which invade the adjacent normal host tissue. The opposite, invasion of cancer cell clusters by a vascularized stroma, has not been considered. The latter process should, however, be expected to occur since the hypoxic cores of tumor cell aggregates, under the control of HIF-1, are known to secrete cytokines (e.g., bFGF, VEGF) which attract fibroblasts and induce blood vessel formation. In this study, the expression of glucose transporter (GLUT)-1, a major HIF-1 target gene, was examined in 51 squamous cell carcinomas of the uterine cervix by immunoh…

Cancer ResearchPathologymedicine.medical_specialtyStromal cellAngiogenesisCellUterine Cervical NeoplasmsBiologyMetastasisStromamedicineHumansNeoplasm InvasivenessProspective StudiesCell ShapeGlucose Transporter Type 1Tumor hypoxiaCell cyclemedicine.diseaseImmunohistochemistryCell Hypoxiamedicine.anatomical_structureOncologyCancer cellCarcinoma Squamous CellFemaleStromal CellsInternational journal of oncology
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Cannabinoid-associated cell death mechanisms in tumor models

2012

In recent years, cannabinoids (the active compo- nents of Cannabis sativa) and their derivatives have received considerable interest due to findings that they can affect the viability and invasiveness of a variety of different cancer cells. Moreover, in addition to their inhibitory effects on tumor growth and migration, angiogenesis and metastasis, the ability of these compounds to induce different pathways of cell death has been highlighted. Here, we review the most recent results generating interest in the field of death mechanisms induced by cannabinoids in cancer cells. In particular, we analyze the pathways triggered by cannabinoids to induce apoptosis or autophagy and investigate the …

Cancer ResearchProgrammed cell deathAngiogenesismedicine.medical_treatmentAutophagyCancerBiologyCell cyclemedicine.diseaseMetastasisCell biologyOncologyCancer cellmedicineCancer researchCannabinoidInternational Journal of Oncology
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Abstract 2935: Novel combination of repurposed drugs induces complete cell invasion arrest of glioblastoma in vitro

2019

Abstract Introduction: Glioblastoma multiforme (GBM) is an aggressive and lethal cancer with a poor prognosis even after conventional treatment (surgery, radiation, chemotherapy). Temozolomide (TMZ) is a standard cyotoxic agent used, despite resistance leading to recurrence. Therefore, additional strategies for targeting the tumor environment are needed. We demonstrate a combination of approved drugs (CAD) repurposed to target GBM leads to complete arrest of GBM cell invasion. Each drug in the combination individually targets diverse tumor pathways: 1) invasion via MMP2 (doxycycline); 2) angiogenesis, inflammation, and proliferation via p53-dependent G1 cell-cycle arrest (celecoxib); 3) aut…

Cancer ResearchProgrammed cell deathChemotherapyTumor microenvironmentMMP2TemozolomideAngiogenesisbusiness.industrymedicine.medical_treatmentCancermedicine.diseaseOncologymedicineCancer researchbusinessTamoxifenmedicine.drugCancer Research
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The protease complex consisting of dipeptidyl peptidase IV and seprase plays a role in the migration and invasion of human endothelial cells in colla…

2006

Abstract Dipeptidyl peptidase IV (DPP4/CD26) and seprase/fibroblast activation protein α are homologous type II transmembrane, homodimeric glycoproteins that exhibit unique prolyl peptidase activities. Human DPP4 is ubiquitously expressed in epithelial and endothelial cells and serves multiple functions in cleaving the penultimate positioned prolyl bonds at the NH2 terminus of a variety of physiologically important peptides in the circulation. Recent studies showed a linkage between DPP4 and down-regulation of certain chemokines and mitogenic growth factors, and degradation of denatured collagens (gelatin), suggesting a role of DPP4 in the cell invasive phenotype. Here, we found the existen…

Cancer ResearchProteasesDipeptidyl Peptidase 4medicine.medical_treatmentBiologyArticleDipeptidyl peptidaseExtracellular matrixFibroblast activation protein alphaCell MovementmedicineHumansSerine proteaseProteaseSerine EndopeptidasesAntibodies MonoclonalEndothelial CellsCell migrationdipeptidyl peptidase IV CD26 seprase fibroblast activation protein α endothelial cell migration angiogenesisExtracellular MatrixUp-RegulationEndothelial stem cellOncologyBiochemistrybiology.proteinGelatinCell Surface ExtensionsCollagenPeptide Hydrolases
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Tumor and its microenvironment: a synergistic interplay.

2013

The mutual and interdependent interaction between tumor and its microenvironment is a crucial topic in cancer research. Recently, it was reported that targeting stromal events could improve efficacies of current therapeutics and prevent metastatic spreading. Tumor microenvironment is a "complex network" of different cell types, soluble factors, signaling molecules and extracellular matrix components, which orchestrate the fate of tumor progression. As by definition, cancer stem cells (CSCs) are proposed to be the unique cell type able to maintain tumor mass and survive outside the primary tumor at metastatic sites. Being exposed to environmental stressors, including reactive oxygen species …

Cancer ResearchStromal cellEpithelial-Mesenchymal TransitionAngiogenesisCell SurvivalBiologyCancer stem cellCell MovementNeoplasmsmedicineTumor MicroenvironmentAnimalsHumansEpithelial–mesenchymal transitionNeoplasm MetastasisStem Cell NicheHypoxiaTumor microenvironmentNeovascularization Pathologicmedicine.diseaseAngiogenesis CAFs CAMs CRC CSCs ECM EMT GSH HIF Hypoxia MMPs ROS Tumor microenvironment VEGF cancer stem cells cancer-associated fibroblasts cancer-associated macrophages colorectal cancer epithelial mesenchymal transition extracellular matrix hypoxia-inducible factor matrix metalloproteinase reactive oxygen species reduced glutathione vascular endothelial growth factorPrimary tumorTumor progressionImmunologyCancer researchNeoplastic Stem CellsCancer-Associated FibroblastsOxidation-ReductionSignal Transduction
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