Search results for "cancer cell"

showing 10 items of 756 documents

Metal drugs and the anticancer immune response

2018

The immune system deploys a multitude of innate and adaptive mechanisms not only to ward off pathogens but also to prevent malignant transformation ("immune surveillance"). Hence, a clinically apparent tumor already reflects selection for those malignant cell clones capable of evading immune recognition ("immune evasion"). Metal drugs, besides their well-investigated cytotoxic anticancer effects, massively interact with the cancer-immune interface and can reverse important aspects of immune evasion. This topic has recently gained intense attention based on combination approaches with anticancer immunotherapy (e.g., immune checkpoint inhibitors), a strategy recently delivering first exciting…

Metal Drugs Immune Response Anticancer cisplatinanimal diseasesmedicine.medical_treatmentEvasion (network security)chemical and pharmacologic phenomenaAntineoplastic Agents010402 general chemistry01 natural sciencesMalignant transformationImmune systemImmunityCoordination ComplexesNeoplasmsmedicineHumansLymphocytesTumor microenvironment010405 organic chemistryChemistryGeneral ChemistryImmunotherapybiochemical phenomena metabolism and nutritionAcquired immune systemImmunity Innate0104 chemical sciencesGastrointestinal MicrobiomeMetalsSettore CHIM/03 - Chimica Generale E InorganicaCancer cellbacteriaNanoparticlesImmunotherapyNeuroscience
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O6-methylguanine DNA methyltransferase and p53 status predict temozolomide sensitivity in human malignant glioma cells

2006

Temozolomide (TMZ) is a methylating agent which prolongs survival when administered during and after radiotherapy in the first-line treatment of glioblastoma and which also has significant activity in recurrent disease. O6-methylguanine DNA methyltransferase (MGMT) is a DNA repair enzyme attributed a role in cancer cell resistance to O6-alkylating agent-based chemotherapy. Using a panel of 12 human glioma cell lines, we here defined the sensitivity to TMZ in acute cytotoxicity and clonogenic survival assays in relation to MGMT, mismatch repair and p53 status and its modulation by dexamethasone, irradiation and BCL-X(L). We found that the levels of MGMT expression were a major predictor of T…

MethyltransferaseCell Survivalbcl-X ProteinBcl-xLTransfectionBiochemistryDNA methyltransferaseO(6)-Methylguanine-DNA MethyltransferaseCellular and Molecular NeuroscienceCell Line TumorGliomaTemozolomidemedicineHumansCytotoxicityAntineoplastic Agents AlkylatingneoplasmsTumor Stem Cell AssayTemozolomideCell DeathbiologyGliomamedicine.diseaseCarmustinedigestive system diseasesDacarbazineEnzyme ActivationGene Expression Regulation NeoplasticCancer cellbiology.proteinCancer researchDNA mismatch repairTumor Suppressor Protein p53medicine.drugJournal of Neurochemistry
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Itraconazole inhibits nuclear delivery of extracellular vesicle cargo by disrupting the entry of late endosomes into the nucleoplasmic reticulum

2021

ABSTRACT Extracellular vesicles (EVs) are mediators of intercellular communication under both healthy and pathological conditions, including the induction of pro‐metastatic traits, but it is not yet known how and where functional cargoes of EVs are delivered to their targets in host cell compartments. We have described that after endocytosis, EVs reach Rab7+ late endosomes and a fraction of these enter the nucleoplasmic reticulum and transport EV biomaterials to the host cell nucleoplasm. Their entry therein and docking to outer nuclear membrane occur through a tripartite complex formed by the proteins VAP‐A, ORP3 and Rab7 (VOR complex). Here, we report that the antifungal compound itracona…

Models MolecularHistologyAntifungal AgentsEndosomeNuclear EnvelopeNucleoplasmic reticulumActive Transport Cell NucleusVesicular Transport ProteinsHost cell nucleoplasmEndosomesEndocytosisFatty Acid-Binding ProteinsExosomeCell LineExtracellular VesiclesCell MovementSettore BIO/13 - Biologia ApplicataHumanscancerexosomemetastasisendosomeResearch ArticlesCholestenonesmicro‐vesicleQH573-671Chemistryrab7 GTP-Binding ProteinsCell BiologyExtracellular vesicleSaponinsEndocytosisCell biologyKetoconazoleCancer cellintercellular communicationnucleoplasmic reticulumcancer endosome exosome intercellular communication metastasis micro-vesicle nucleoplasmicreticulumItraconazoleCytologyIntracellularResearch ArticleJournal of Extracellular Vesicles
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Double-Network-Structured Graphene Oxide-Containing Nanogels as Photothermal Agents for the Treatment of Colorectal Cancer.

2017

Here, we reported the production of hyaluronic acid/polyaspartamide-based double-network nanogels for the potential treatment of colorectal carcinoma. Graphene oxide, thanks to the huge aromatic surface area, allows to easily load high amount of irinotecan (33.0% w/w) and confers to the system hyperthermic properties when irradiated with a near-infrared (NIR) laser beam. We demonstrate that the release of antitumor drug is influenced both by the pH of the external medium and the NIR irradiation process. In vitro biological studies, conducted on human colon cancer cells (HCT 116), revealed that nanogels are uptaken by the cancer cells and, in the presence of the antitumor drug, can produce a…

Models MolecularPolymers and PlasticsColorectal cancerInfrared RaysProtein ConformationNanogelsBioengineeringNanotechnologyAntineoplastic Agents02 engineering and technology010402 general chemistry01 natural scienceslaw.inventionPolyethylene GlycolsBiomaterialschemistry.chemical_compoundgraphene oxide hydrogel nenogel cancer therapylawHyaluronic acidMaterials ChemistrymedicineCytotoxic T cellHumansPolyethyleneimineHyaluronic AcidGrapheneChemistryOxidesPhotothermal therapy021001 nanoscience & nanotechnologymedicine.diseaseHCT116 CellsIn vitro0104 chemical sciencesIrinotecanPhotochemotherapyCancer cellCancer researchGraphite0210 nano-technologyColorectal Neoplasmsmedicine.drugBiomacromolecules
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Palladium(II)-Stabilized Pyridine-2-Diazotates: Synthesis, Structural Characterization, and Cytotoxicity Studies

2018

Well-defined diazotates are scarce. Here we report the synthesis of unprecedented homoleptic palladium(II) diazotate complexes. The palladium(II)-mediated nitrosylation of 2-aminopyridines with NaNO2 results in the formation of metal-stabilized diazotates, which were found to be cytotoxic to human ovarian cancer cells.

Models Molecularinorganic chemicalsCell SurvivalPyridinesPyrazine Diazohydroxidechemistry.chemical_elementAntineoplastic AgentsorganometalliyhdisteetCrystallography X-Ray010402 general chemistry01 natural sciencesInorganic ChemistryStructure-Activity Relationshipchemistry.chemical_compoundCoordination ComplexesCell Line Tumororganometallic compoundsPyridineHumansCytotoxic T cellsytotoksisuusPhysical and Theoretical ChemistryHomolepticCytotoxicityta116Cell ProliferationDose-Response Relationship DrugMolecular Structure010405 organic chemistryNitrosylationCombinatorial chemistry0104 chemical sciencesHEK293 Cellssyöpäsolutchemistrycancer cellsOvarian cancer cellscytotoxicityDrug Screening Assays AntitumorAzo CompoundsPalladiumPalladiumInorganic Chemistry
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The triad hsp60-mirnas-extracellular vesicles in brain tumors: Assessing its components for understanding tumorigenesis and monitoring patients

2021

Brain tumors have a poor prognosis and progress must be made for developing efficacious treatments, but for this to occur their biology and interaction with the host must be elucidated beyond current knowledge. What has been learned from other tumors may be applied to study brain tumors, for example, the role of Hsp60, miRNAs, and extracellular vesicles (EVs) in the mechanisms of cell proliferation and dissemination, and resistance to immune attack and anticancer drugs. It has been established that Hsp60 increases in cancer cells, in which it occurs not only in the mitochondria but also in the cytosol and plasma-cell membrane and it is released in EVs into the extracellular space and in cir…

Molecular chaperonesCellBrain tumorBiologymedicine.disease_causelcsh:Technologylcsh:Chemistry03 medical and health sciences0302 clinical medicineImmune systemHigh-grade gliomaExtracellularmedicineGeneral Materials Sciencelcsh:QH301-705.5Instrumentation030304 developmental biologyFluid Flow and Transfer Processes0303 health sciencesLiquid biopsylcsh:TProcess Chemistry and TechnologyGeneral EngineeringCancerTumor biomarkersChaperonopathiesExtracellular vesiclesmedicine.diseaseHsp60lcsh:QC1-999Computer Science ApplicationsCrosstalk (biology)medicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999lcsh:TA1-2040030220 oncology & carcinogenesisCancer cellCancer researchChaperone systemMiRNAslcsh:Engineering (General). Civil engineering (General)CarcinogenesisGlioblastomaMeningiomalcsh:Physics
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Impact of exogenous lactate on survival and radioresponse of carcinoma cells in vitro

2009

Tumour lactate levels have been shown to correlate with high radioresistance in tumour models in vivo. The study aimed to evaluate the impact of pathophysiological extracellular lactate concentrations and acidosis on the in vitro survival and radioresponse of various cancer cell lines.HCT-116, HT29 (colorectal) and FaDu (HNSCC) carcinoma cells were studied. Lactate release rates were determined, and expression of the monocarboxylate transporter MCT1 and its cofactor CD147 were monitored by immunofluorescence and flow cytometry. Colony formation was compared for cells exposed to 20 mM exogenous lactate, acidosis (pH 6.4) and lactate plus acidosis relative to control and dose response curves …

Monocarboxylic Acid TransportersCell SurvivalIn Vitro TechniquesRadiation ToleranceFlow cytometryAndrologyCell Line TumorRadioresistanceExtracellularmedicineHumansRadiology Nuclear Medicine and imagingLactic AcidClonogenic assayTumor Stem Cell AssayAcidosisMonocarboxylate transporterSymportersRadiological and Ultrasound Technologybiologymedicine.diagnostic_testCarcinomaHydrogen-Ion ConcentrationHCT116 CellsDose–response relationshipGlucoseBiochemistryCancer cellBasiginbiology.proteinmedicine.symptomAcidosisHT29 CellsInternational Journal of Radiation Biology
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Zinc oxide nanorod based immunosensing platform for the determination of human leukemic cells.

2019

Zinc oxide (ZnO) based nanostructures owing unique physical properties – high photoluminescence, bio- compatibility and other characteristics, therefore, they attract attention as building blocks suitable for biosensor development. In this research as a target we have used human leukemic cell line IM9 (IM9). IM9 was derived from the patient with a multiple myeloma and expressed cluster of differentiation proteins СD19 on the surface of 85–95% here investigated cancer cells. As a control sample healthy human's peripheral blood mononuclear cells (PBMC) were used and the expression of CD19 protein was found only in 5–9% of these cells. Two types of antibodies labeled by f…

Monoclonal antibodymedicine.drug_class02 engineering and technologyCell SeparationMonoclonal antibody01 natural sciencesCD19Analytical ChemistryFlow cytometrychemistry.chemical_compoundHuman lymphocytesmedicineHumansFlow cytometryZnO-nanorodsFluorescein isothiocyanatePhotoluminescenceCells CulturedImmunoassayB-LymphocytesLeukemiaNanotubesCluster of differentiationmedicine.diagnostic_testbiology010401 analytical chemistryhemic and immune systemsВ-lymphoblast cells021001 nanoscience & nanotechnologyFlow CytometryMolecular biologyhumanities0104 chemical scienceschemistryCell cultureCancer cell:NATURAL SCIENCES [Research Subject Categories]biology.proteinAntibodyZinc Oxide0210 nano-technologyhuman activitiesBiomarkersTalanta
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Evolutionary dynamics of imatinib-treated leukemic cells by stochastic approach

2008

The evolutionary dynamics of a system of cancerous cells in a model of chronic myeloid leukemia (CML) is investigated by a statistical approach. Cancer progression is explored by applying a Monte Carlo method to simulate the stochastic behavior of cell reproduction and death in a population of blood cells which can experience genetic mutations. In CML front line therapy is represented by the tyrosine kinase inhibitor imatinib which strongly affects the reproduction of leukemic cells only. In this work, we analyze the effects of a targeted therapy on the evolutionary dynamics of normal, first-mutant and cancerous cell populations. Several scenarios of the evolutionary dynamics of imatinib-tr…

Monte Carlo simulation stochastic approach Evolutionary dynamicsMutation rate87.23.kgmedicine.drug_classQC1-999medicine.medical_treatmentPopulationGeneral Physics and AstronomyBiologyTyrosine-kinase inhibitorTargeted therapyhemic and lymphatic diseasesmedicine87.10.mncomplex systemsQuantitative Biology - Populations and EvolutioneducationEvolutionary dynamicseducation.field_of_studycancer evolutionPhysicsstochastic dynamics87.19.xjPopulations and Evolution (q-bio.PE)Myeloid leukemiaImatinibSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)FOS: Biological sciencesCancer cellCancer research87.10.rtmedicine.drugOpen Physics
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Bleomycin Exerts Ambivalent Antitumor Immune Effect by Triggering Both Immunogenic Cell Death and Proliferation of Regulatory T Cells

2013

International audience; Bleomycin (BLM) is an anticancer drug currently used for the treatment of testis cancer and Hodgkin lymphoma. This drug triggers cancer cell death via its capacity to generate radical oxygen species (ROS). However, the putative contribution of anticancer immune responses to the efficacy of BLM has not been evaluated. We make here the observation that BLM induces immunogenic cell death. In particular, BLM is able to induce ROS-mediated reticulum stress and autophagy, which result in the surface exposure of chaperones, including calreticulin and ERp57, and liberation of HMBG1 and ATP. BLM induces anti-tumor immunity which relies on calreticulin, CD8(+) T cells and inte…

MouseCancer TreatmentCD8-Positive T-LymphocytesT-Lymphocytes RegulatoryHematologic Cancers and Related DisordersMice0302 clinical medicineTransforming Growth Factor beta[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyCytotoxic T cellImmune Response0303 health sciencesMultidisciplinaryCell DeathbiologyQRFOXP3Animal ModelsHematology3. Good healthCell biologyOncology030220 oncology & carcinogenesisMedicine[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunogenic cell deathFemaleLymphomasOncology AgentsResearch ArticleTumor Immunologycongenital hereditary and neonatal diseases and abnormalitiesProgrammed cell death[SDV.IMM] Life Sciences [q-bio]/ImmunologyScienceImmunologyAntineoplastic Agentschemical and pharmacologic phenomenaBleomycin03 medical and health sciencesModel OrganismsImmune systemCell Line TumorAnimalsHumansBiologyCell Proliferation030304 developmental biologyHodgkin Lymphomaurogenital systemCell growthImmunitynutritional and metabolic diseasesImmunologic SubspecialtiesChemotherapy and Drug TreatmentImmunity InnateCancer cellbiology.proteinClinical ImmunologyCalreticulinPLoS ONE
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