Search results for "energy homeostasis"

showing 10 items of 44 documents

Fasting enhances the response of arcuate neuropeptide Y-glucose-inhibited neurons to decreased extracellular glucose

2009

0363-6143 (Print) Comparative Study In Vitro Journal Article Research Support, N.I.H., Extramural; Fasting increases neuropeptide Y (NPY) expression, peptide levels, and the excitability of NPY-expressing neurons in the hypothalamic arcuate (ARC) nucleus. A subpopulation of ARC-NPY neurons ( approximately 40%) are glucose-inhibited (GI)-type glucose-sensing neurons. Hence, they depolarize in response to decreased glucose. Because fasting enhances NPY neurotransmission, we propose that during fasting, GI neurons depolarize in response to smaller decreases in glucose. This increased excitation in response to glucose decreases would increase NPY-GI neuronal excitability and enhance NPY neurotr…

LeptinMalemedicine.medical_specialtyArcuate Nucleus/cytology/*metabolismPhysiologyGlucose/*deficiencyAMP-Activated Protein Kinases/metabolismAMP-Activated Protein KinasesIn Vitro TechniquesNeurotransmissionBiologySynaptic TransmissionEnergy homeostasisMembrane PotentialsRats Sprague-Dawley03 medical and health sciences0302 clinical medicineNeuropeptide Y/*metabolismArcuate nucleusInternal medicinemental disordersmedicineAnimalsHomeostasisNeuropeptide YNervous System Cell BiologyFasting/*metabolismNeurons/enzymology/*metabolism030304 developmental biologyNeuronsMembrane potential0303 health sciencesLeptinArcuate Nucleus of HypothalamusLeptin/metabolismNeural InhibitionFastingCell BiologyNeuropeptide Y receptorhumanitiesRatsGlucosemedicine.anatomical_structureEndocrinologyNeuronSprague-DawleyEnergy Metabolism030217 neurology & neurosurgeryHomeostasis
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Leptin and the OB-receptor as anti-obesity target: recent in silico advances in the comprehension of the protein-protein interaction and rational dru…

2014

The OB-receptor or leptin receptor (LR) is crucial for energy homeostasis and regulation of food uptake. Leptin is a 16 kDa hormone that is mainly secreted by fat cells into the bloodstream. Under normal circumstances, circulating leptin levels are proportionate to the fat body mass. Sensing of elevated leptin levels by the hypothalamic neuro-circuitry activates a negative feedback loop resulting in reduced food intake and increased energy expenditure. Decreased leptin concentrations lead to opposite effects. Therefore, rational design of leptin agonists/antagonists could be an appealing challenge in the battle against obesity. The Leptin/LR interactions have been studied in several works b…

LeptinModels Molecularmedicine.medical_specialtyIn silicoAdipose tissueDrug designBiologyEnergy homeostasisRisk FactorsInternal medicineDrug DiscoverymedicineHumansObesityReceptorleptin receptorPharmacologyleptin receptor agonists/antagonistLeptin receptorLeptindigestive oral and skin physiologyBody WeightRational designInfant Newbornprotein/protein dockingSettore CHIM/08 - Chimica Farmaceuticamolecular modellingEndocrinologyDrug DesignReceptors LeptinHomology modellingAnti-Obesity Agentshormones hormone substitutes and hormone antagonistsProtein BindingSignal TransductionCurrent pharmaceutical design
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The role of the leptin in reproduction.

2006

Since its discovery in 1994, leptin has appeared to be a pleiotrophic hormone, governing energy homeostasis and affecting many tissues in the body. Numerous pieces of evidence have accumulated showing that leptin potentially plays an important role in the control of the reproductive function.This review presents the major concepts for the role of leptin in the modulation of reproductive function. As a marker of the nutritional status, leptin affects the hypothalamo-pituitary-gonadal axis, regulating gonadotrophin-releasing hormone and luteinising hormone secretion, and appears to be a permissive factor in the onset of the puberty. This protein and its receptor have been found in the reprodu…

Leptinmedicine.medical_specialtyLeptin receptorLeptinmedia_common.quotation_subjectReproductiondigestive oral and skin physiologyOvaryObstetrics and GynecologyBiologyEnergy homeostasisEndometriumEndocrinologyPregnancyInternal medicinemedicineHumansFemaleEmbryo ImplantationReproductionhormones hormone substitutes and hormone antagonistsHormonemedia_commonCurrent opinion in obstetricsgynecology
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Molecular dynamics, dynamic site mapping, and highthroughput virtual screening on leptin and the Ob receptor as anti-obesity target.

2014

Body weight control is a mechanism finely regulated by several hormonal, metabolic, and nervous pathways. The leptin receptor (Ob-R) is crucial for energy homeostasis and regulation of food uptake. Leptin is a 16 kDa hormone that is mainly secreted by fat cells into the bloodstream, and under normal circumstances, circulating levels are proportionate to the fat body mass. Sensing of elevated leptin levels by the hypothalamic neurocircutry activates a negative feedback loop resulting in reduced food intake and increased energy expenditure. Decreased concentrations lead to opposite effects. Therefore rational design of leptin agonists constitute an appealing challenge in the battle against ob…

Leptinmedicine.medical_specialtyProtein ConformationAdipose tissueDrug designBiologyMolecular Dynamics SimulationDynamic SiteMapping HTVS Leptin Molecular Dynamics Obesity Protein/protein docking Multivariate analysis Ob ReceptorCatalysisEnergy homeostasisInorganic ChemistryStructure-Activity RelationshipInternal medicinemedicineMolecular Targeted TherapyPhysical and Theoretical ChemistryReceptorVirtual screeningLeptin receptorBinding SitesMolecular StructureLeptindigestive oral and skin physiologyOrganic ChemistryHydrogen BondingSettore CHIM/08 - Chimica FarmaceuticaComputer Science ApplicationsHigh-Throughput Screening AssaysMolecular Docking SimulationEndocrinologyComputational Theory and MathematicsDocking (molecular)Drug DesignMultivariate AnalysisComputer-Aided DesignReceptors LeptinAnti-Obesity AgentsHydrophobic and Hydrophilic InteractionsProtein BindingJournal of molecular modeling
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Inactivation of Socs3 in the Hypothalamus Enhances the Hindbrain Response to Endogenous Satiety Signals via Oxytocin Signaling.

2012

Leptin is an adipocyte-derived hormone that controls energy balance by acting primarily in the CNS, but its action is lost in common forms of obesity due to central leptin resistance. One potential mechanism for such leptin resistance is an increased hypothalamic expression of Suppressor of cytokine signaling 3 (Socs3), a feedback inhibitor of the Jak-Stat pathway that prevents Stat3 activation. Ample studies have confirmed the important role of Socs3 in leptin resistance and obesity. However, the degree to which Socs3 participates in the regulation of energy homeostasis in nonobese conditions remains largely undetermined. In this study, using adult mice maintained under standard diet, we d…

Leptinmedicine.medical_specialty[SDV.OT]Life Sciences [q-bio]/Other [q-bio.OT][SDV]Life Sciences [q-bio][ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionHypothalamusHindbrainSuppressor of Cytokine Signaling ProteinsBiologyOxytocinDevazepideSatiety ResponseEnergy homeostasis03 medical and health sciencesEatingMice0302 clinical medicineHormone AntagonistsInternal medicinemedicineAnimals[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]SOCS3[ SDV.OT ] Life Sciences [q-bio]/Other [q-bio.OT]ComputingMilieux_MISCELLANEOUS030304 developmental biology2. Zero hunger0303 health sciences[SDV.OT] Life Sciences [q-bio]/Other [q-bio.OT]General NeuroscienceLeptindigestive oral and skin physiologyArticlesRhombencephalonEndocrinologyOxytocinHypothalamusSuppressor of Cytokine Signaling 3 Protein[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Cholecystokinin[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryHomeostasisHormonemedicine.drugSignal Transduction
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Functional Evaluation of THIQ, a Melanocortin 4 Receptor Agonist, in Models of Food Intake and Inflammation

2007

The central melanocortinergic system plays an important role in regulating different aspects of energy homeostasis and the immunomodulatory response. In the present study, we evaluated the in vivo activities of food intake suppression and anti-inflammatory activity of THIQ, which has been proposed to possess high and selective melanocortin-4 receptor agonistic activity in vitro. The results showed that THIQ (0.1, 0.3 and 1 nmol/rat, intracerebroventricularly) is less effective in reducing food intake and body weights of rats than the non-selective melanocortin receptor agonist melanotan II. Electron paramagnetic resonance measurements in mice brain tissue showed that THIQ at doses of 0.001 …

LipopolysaccharidesMaleAgonistmedicine.medical_specialtymedicine.drug_classAnti-Inflammatory AgentsBiologyNitric OxideToxicologyPeptides CyclicEnergy homeostasisEatingMiceMelanocortin receptorIn vivoTetrahydroisoquinolinesInternal medicinemedicineAnimalsRats WistarReceptorInjections IntraventricularInflammationPharmacologyMice Inbred ICRDose-Response Relationship DrugBody Weightdigestive oral and skin physiologyElectron Spin Resonance SpectroscopyBrainMelanotan IIGeneral MedicineTriazolesRatsMelanocortin 4 receptorDisease Models AnimalEndocrinologyalpha-MSHTHIQReceptor Melanocortin Type 4medicine.drugBasic & Clinical Pharmacology & Toxicology
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The neural feedback loop between the brain and adipose tissues

2009

Communication également publiée dans le livre "Adipose tissue development: from animal models to clinical conditions" (ISBN 978-3-8055-9450-9) de C. Levy-Marchal et L. Pénicaud (eds); There are more and more data supporting the importance of nervous regulation of both white and brown adipose tissue mass. This short paper will review the different physiological parameters which are regulated such as metabolism (lipolysis and thermogeneis), secretory activity (leptin and other adipokines) but also to plasticity of adipose tissues (proliferation differentiation and apoptosis). The sensory innervation of white adipose issue and its putative role will be also described. Altogether these results …

MESH: Feedback Physiological[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionPhysiologicalAdipokineAdipose tissueWhite adipose tissueBiologyAutonomic Nervous SystemMESH : Adipose TissueEnergy homeostasisMESH : Autonomic Nervous SystemFeedbackMESH: Autonomic Nervous System[ SDV.NEU.SC ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesMESH: BrainBrown adipose tissuemedicineLipolysisAnimalsHumansMESH: AnimalsComputingMilieux_MISCELLANEOUSFeedback PhysiologicalMESH: HumansLeptinMESH : HumansMESH: Energy MetabolismBrain[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesMESH : Feedback PhysiologicalNeurosecretory SystemsCell biologyMESH : Energy MetabolismAutonomic nervous systemmedicine.anatomical_structureMESH : BrainAdipose TissueMESH: Neurosecretory SystemsMESH : AnimalsEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Neurosecretory Systems[SDV.NEU.SC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesMESH: Adipose Tissue
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Transient Receptor Potential Canonical 3 (TRPC3) Channels Are Required for Hypothalamic Glucose Detection and Energy Homeostasis

2017

Fil: Chrétien, Chloé. University of Bourgogne Franche-Comté. Institut National de la Recherche Agronomique. Centre des Sciences du Goût et de l’Alimentation; France Fil: Fenech, Claire. University of Bourgogne Franche-Comté. Institut National de la Recherche Agronomique. Centre des Sciences du Goût et de l’Alimentation; France Fil: Liénard, Fabienne. University of Bourgogne Franche-Comté. Institut National de la Recherche Agronomique. Centre des Sciences du Goût et de l’Alimentation; France Fil: Grall, Sylvie. University of Bourgogne Franche-Comté. Institut National de la Recherche Agronomique. Centre des Sciences du Goût et de l’Alimentation; France Fil: Chevalier, Charlène. University of …

Male0301 basic medicine[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and Metabolismmedicine.medical_treatmentsourisTRPC3proopiomelanocortin neuronsEnergy homeostasisRats Sprague-DawleyEatingMiceTransient receptor potential channelneuropeptide-yTRPC3synaptic-transmissionneuroneInsulin SecretionHomeostasisInsulinarcuate nucleusNeurons2. Zero hungerneuropeptide ydiabetesion channelsnoyau arquémuscle squelettiqueFastingfood-intakeprise alimentaire16. Peace & justiceNeuropeptide Y receptorcation channelsproopiomelanocortine3. Good healthMedicina BásicaAlimentation et NutritionEndocrinologie et métabolismemedicine.medical_specialtyCIENCIAS MÉDICAS Y DE LA SALUDBlotting Westernarcuate nucleus;food-intake;synaptic-transmission;endothelial-cell;skeletal-muscle;cation channel;neuropeptide-y;ion channel;mouse;proopiomelanocortin neuronHypothalamusInmunologíaMédecine humaine et pathologieBiologyNeurotransmissionReal-Time Polymerase Chain ReactionHOMEOSTASIS ENERGETICA03 medical and health sciencesCalcium imagingInternal medicineInternal MedicinemedicineFood and NutritionAnimalsskeletal-musclecanal ioniquemouseTRPC Cation ChannelsEndocrinology and metabolismInsulinBody Weighttransmission synaptiqueGlucose Tolerance TestRatsMice Inbred C57BLGlucose030104 developmental biologyEndocrinologyendothelial-cellsGLUCOSA HIPOTALAMICAHypothalamic glucose sensingAnorecticHuman health and pathologyCANALES IONICOSEnergy Metabolismcellule endotheliale[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Importance of mitochondrial dynamin-related protein 1 in hypothalamic glucose sensitivity in rats.

2012

International audience; AIMS: Hypothalamic mitochondrial reactive oxygen species (mROS)-mediated signaling has been recently shown to be involved in the regulation of energy homeostasis. However, the upstream signals that control this mechanism have not yet been determined. Here, we hypothesize that glucose-induced mitochondrial fission plays a significant role in mROS-dependent hypothalamic glucose sensing. RESULTS: Glucose-triggered translocation of the fission protein dynamin-related protein 1 (DRP1) to mitochondria was first investigated in vivo in hypothalamus. Thus, we show that intracarotid glucose injection induces the recruitment of DRP1 to VMH mitochondria in vivo. Then, expressio…

MaleEnergy-Generating Resourcesnervous-systemPhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionClinical BiochemistryneuronsMitochondrionBiochemistryinvolvementEnergy homeostasisDNM1L0302 clinical medicineInsulin-Secreting CellsInsulin SecretionInsulinGeneral Environmental Science2. Zero hungerchemistry.chemical_classification0303 health sciencesTransport proteinMitochondriaProtein TransportHypothalamusGene Knockdown TechniquesMitochondrial MembranesMitochondrial fissionRNA InterferenceDynaminsmedicine.medical_specialtyendocrine systembrainmechanismCarbohydrate metabolismBiology03 medical and health sciencesOxygen ConsumptionInternal medicineexpressionmedicineAnimalsRats WistarMolecular Biologyenergy homeostasis030304 developmental biologyReactive oxygen speciesAppetite RegulationArcuate Nucleus of HypothalamusCell Biologyislet blood-flowRatsEndocrinologyGlucosechemistryVentromedial Hypothalamic NucleusGeneral Earth and Planetary SciencesactivationReactive Oxygen Species[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryinsulin-secretion
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Hypothalamic eIF2 alpha signaling regulates food intake

2014

International audience; The reversible phosphorylation of the a subunit of eukaryotic initiation factor 2 (eIF2 alpha) is a highly conserved signal implicated in the cellular adaptation to numerous stresses such as the one caused by amino acid limitation. In response to dietary amino acid deficiency, the brain-specific activation of the eIF2 alpha kinase GCN2 leads to food intake inhibition. We report here that GCN2 is rapidly activated in the mediobasal hypothalamus (MBH) after consumption of a leucine-deficient diet. Furthermore, knockdown of GCN2 in this particular area shows that MBH GCN2 activity controls the onset of the aversive response. Importantly, pharmacological experiments demo…

Male[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEukaryotic Initiation Factor-2neuronsEatingMicepiriform cortex0302 clinical medicineGene Knockdown Techniquesarcuate nucleusamino-acid deficiency;arcuate nucleus;translational control;energy homeostasis;piriform cortex;cancer cachexia;protein-intake;transfer-rna;mechanism;neuronsPhosphorylationlcsh:QH301-705.52. Zero hungerchemistry.chemical_classification0303 health sciencesGene knockdownalimentationtranslational controlamino-acid deficiencyEukaryotic Initiation Factor-2Amino acidtransfer-rnaGene Knockdown TechniquesAlimentation et NutritionPhosphorylation[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Signal transductionmedicine.symptomSignal Transductioncancer cachexiamedicine.medical_specialtyCellular adaptationHypothalamusmechanismAnorexiaBiologyProtein Serine-Threonine KinasesGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciencesLeucineInternal medicinemedicineFood and NutritionAnimalsenergy homeostasis030304 developmental biologyNeurosciencesArcuate Nucleus of Hypothalamusprotein-intakeMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologychemistrylcsh:Biology (General)Neurons and Cognition[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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