Search results for "homeostasi"

showing 10 items of 636 documents

The ubiquitin-specific protease USP8 is critical for the development and homeostasis of T cells

2015

The modification of proteins by ubiquitin has a major role in cells of the immune system and is counteracted by various deubiquitinating enzymes (DUBs) with poorly defined functions. Here we identified the ubiquitin-specific protease USP8 as a regulatory component of the T cell antigen receptor (TCR) signalosome that interacted with the adaptor Gads and the regulatory molecule 14-3-3β. Caspase-dependent processing of USP8 occurred after stimulation of the TCR. T cell-specific deletion of USP8 in mice revealed that USP8 was essential for thymocyte maturation and upregulation of the gene encoding the cytokine receptor IL-7Rα mediated by the transcription factor Foxo1. Mice with T cell-specifi…

Regulatory T cellT-LymphocytesImmunologyReceptors Antigen T-Cell610 Medicine & healthBiologyCD8-Positive T-LymphocytesJurkat cellsJurkat CellsMiceddc:570EndopeptidasesmedicineImmunology and AllergyAnimalsHomeostasisHumans10239 Institute of Laboratory Animal ScienceIL-2 receptorAdaptor Proteins Signal Transducing2403 ImmunologyReceptors Interleukin-7ThymocytesEndosomal Sorting Complexes Required for TransportForkhead Box Protein O1ZAP70T-cell receptorCD28Cell DifferentiationForkhead Transcription FactorsColitisCell biologyThymocytemedicine.anatomical_structure2723 Immunology and Allergy570 Life sciences; biology590 Animals (Zoology)Ubiquitin ThiolesteraseCD8
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Mboat7 down-regulation by hyper-insulinemia induces fat accumulation in hepatocytes.

2020

Background: Naturally occurring variation in Membrane-bound O-acyltransferase domain-containing 7 (MBOAT7), encoding for an enzyme involved in phosphatidylinositol acyl-chain remodelling, has been associated with fatty liver and hepatic disorders. Here, we examined the relationship between hepatic Mboat7 down-regulation and fat accumulation. Methods: Hepatic MBOAT7 expression was surveyed in 119 obese individuals and in experimental models. MBOAT7 was acutely silenced by antisense oligonucleotides in C57Bl/6 mice, and by CRISPR/Cas9 in HepG2 hepatocytes. Findings: In obese individuals, hepatic MBOAT7 mRNA decreased from normal liver to steatohepatitis, independently of diabetes, inflammatio…

Research paperTGFβ Transforming Growth Factor BetaIntracellular SpaceCRISPR Clustered Regularly Interspaced Short Palindromic RepeatshHEPS Human HepatocytesMice0302 clinical medicineLPIAT1DAG Diacylglyceroli.p. Intraperitonealmedia_commonFatty AcidsGeneral Medicine3. Good health030220 oncology & carcinogenesisHOMA-IR homeostasis Model Assessment of Insulin ResistanceMPO morpholinolcsh:Medicine (General)medicine.medical_specialtyPE Phosphatidyl-EthanolamineNashGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesTNFα tumor Necrosis Factor AlphaLDL Low Density LipoproteinsHyperinsulinismNAFLDSD Standard Dietmedia_common.cataloged_instanceHumansCPT1 Carnitine Palmitoyltransferase IPhosphatidylinositolGene SilencingEuropean unionVLDL Very Low Density Lipoproteinlcsh:RhHSC Human Hepatic Stellate Cellsmedicine.diseaseLipid MetabolismOA Oleic AcidCI Confidence IntervalMboat7 Membrane bound O-acyltransferase domain containing 7MCD methionine choline deficient diet030104 developmental biologyEndocrinologychemistryCDP Cytidine-DiphosphateFOXO1 Forkhead Box protein O1NAFLD nonalcoholic fatty liver diseaseSteatohepatitisBMI Body Mass IndexCL CardiolipinAcyltransferases0301 basic medicineAlcoholic liver diseaseCXCL10 C-X-C Motif Chemokine 10lcsh:Medicinechemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseIFG Impaired Fasting GlucoseAPOB Apolipoprotein BNonalcoholic fatty liver diseasePIP Phosphatidyl-Inositol-PhosphateSteatohepatitisqRT-PCR quantitative Real Time Polymerase Chain ReactionMice Knockoutlcsh:R5-920ORO Oil Red O StainingPI PhosphatidylinositolFatty liverTM6SF2 Transmembrane 6 Superfamily Member 2PhospholipidTAG TriglyceridesNASH Nonalcoholic SteatohepatitisLipogenesisLPA Lyso-Phosphatidic AcidPhosphatidylinositolSignal TransductionPS Phosphatidyl-SerinePA Palmitic AcidALD alcoholic liver diseasePC Phosphatidylcholinei.v. IntravenousFATP1 Fatty Acid Transport Protein 1Models BiologicalInternal medicinemedicineAnimalsNonalcoholic fatty liver diseasePPARα Peroxisome Proliferator-Activated Receptor alphaObesityG3P Glyceraldehyde-3-PhosphateSREBP1c Sterol Regulatory Element-Binding Protein 1HDL High Density Lipoproteinsbusiness.industryPI3K Phosphatidylinositol 3 KinaseMembrane ProteinsNHEJ Non-Homologues End JoiningPNPLA3 Patatin-like Phospholipase Domain-containing-3MTTP Microsomal Triglyceride Transfer ProteinLPIAT1 Lysophosphatidylinositol Acyltransferase 1TMC4 Transmembrane Channel-Like 4Disease Models AnimalGene Expression RegulationHepatocytesFOXA2 Forkhead Box A2mTOR mammalian target of RapamycinSteatosisInsulin ResistancebusinessPG Phosphatidyl-GlycerolFABP1 Fatty Acid-Binding Protein 1 FAS Fatty Acid SynthaseT2DM Type 2 Diabetes MellitusEBioMedicine
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Genetic regulation of iron homeostasis in sideropenic patients with mild COVID-19 disease under a new oral iron formulation: Lessons from a different…

2022

Background: Severe Acute Respiratory Syndrome Coronavirus-2 (SARS-CoV-2) needs iron to replicate itself. Coronaviruses are able to upregulate Chop/Gadd153 and Arg1 genes, consequently leading to CD8 lymphocytes decrease, degradation of asparagine and decreased nitric oxide (NO), thus impairing immune response and antithrombotic functions. Little is known about regulation of genes involved in iron metabolism in pauci symptomatic patients with COVID-19 disease or in patients with iron deficiency treated with sucrosomial iron. Methods: Whole blood was taken from the COVID-19 patients and from patients with sideropenic anemia, treated or not (control group) with iron supplementations. Enrolled …

SARS-CoV-2IronMacrophagesImmunologyCovid-19 Hepcidin Arginase-1 Iron metabolism Chop/gadd153 Sucrosomial ironImmunology and AllergyHumansCOVID-19HomeostasisHematologyIron DeficienciesFerric CompoundsImmunobiology
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SERCA activity is reduced in DJ-1 mutant flies and human cells due to oxidative modification

2020

ABSTRACTDJ-1 is a causative gene for familial Parkinson’s disease (PD) with different functions, standing out its role against oxidative stress (OS). Accordingly, PD model flies harboring a mutation in the DJ-1β gene (the Drosophila ortholog of human DJ-1) show high levels of OS markers like protein carbonylation, a common post-translational modification that may alter protein function. To increase our understanding of PD pathogenesis as well as to discover potential therapeutic targets for pharmacological intervention, we performed a redox proteomic assay in DJ-1β mutant flies. Among the proteins that showed increased carbonylation levels in PD model flies, we found SERCA, an endoplasmic r…

SERCAChemistryActivator (genetics)Endoplasmic reticulumProtein CarbonylationMutantmedicineOxidative phosphorylationmedicine.disease_causeOxidative stressHomeostasisCell biology
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Potassium uptake system Trk2 is crucial for yeast cell viability during anhydrobiosis

2013

Yeasts grow at very different potassium concentrations, adapting their intracellular cation levels to changes in the external environment. Potassium homeostasis is maintained with the help of several transporters mediating the uptake and efflux of potassium with various affinities and mechanisms. In the model yeast Saccharomyces cerevisiae, two uptake systems, Trk1 and Trk2, are responsible for the accumulation of a relatively high intracellular potassium content (200-300 mM) and the efflux of surplus potassium is mediated by the Tok1 channel and active exporters Ena ATPase and Nha1 cation/proton antiporter. Using a series of deletion mutants, we studied the role of individual potassium tra…

Saccharomyces cerevisiae ProteinsATPaseAntiporterPotassiumSaccharomyces cerevisiaechemistry.chemical_elementSaccharomyces cerevisiaeMicrobiologyGeneticsHomeostasisViability assayDesiccationCation Transport ProteinsMolecular BiologySequence DeletionMicrobial ViabilitybiologyBiological Transportbiology.organism_classificationYeastBiochemistrychemistryPotassiumbiology.proteinEffluxIntracellularFEMS Microbiology Letters
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Homeostatic control of polyamine levels under long-term salt stress in Arabidopsis

2011

Salt stress has been frequently studied in its first osmotic phase. Very often, data regarding the second ionic phase is missing. It has also been suggested that Putrescine or/and Spermine could be responsible for salt resistance. In order to test this hypothesis under long-term salt stress, we obtained Arabidopsis thaliana transgenic plants harboring pRD29A::oatADC or pRD29A::GUS construction. Although Putrescine was the only polyamine significantly increased after salt acclimation in pRD29A::oatADC transgenic lines, this rendered in no advantage to this kind of stress. The higher Spermine levels found in WT and transgenic lines when compared to control conditions along with no increment o…

SalinityCarboxy-lyasesAvenaCarboxy-LyasesAcclimatizationArabidopsisSperminePlant ScienceAcclimatizationchemistry.chemical_compoundStress PhysiologicalArabidopsisPolyaminesHomeostasisArabidopsis thalianaPromoter Regions GeneticAbscisic acidbiologyArabidopsis ProteinsPlants Genetically Modifiedbiology.organism_classificationchemistryBiochemistryPutrescinePolyamineResearch PaperAbscisic AcidSignal TransductionPlant Signaling & Behavior
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Salivary apoptotic cells in oral (pre-) cancer as a potential diagnostic means

2015

Background: Apoptosis is a genetically programmed form of cell death which is indispensable for development and homeostasis of multi-cellular organism. Objectives: The aim of this study was to find out the salivary apoptotic cells in oral precancerous and cancerous patients and furthermore to observe the potential diagnostic value of salivary apoptotic cells in detection of oral pre-cancer and cancer. Materials and Methods: Unsimulated saliva was collected from a group of 103 subjects diagnosed with oral (pre-) cancer and a control group of 30 healthy age- and gender-matched individuals. The test group diagnosed with (pre-) cancer was further subdivided in 4 lesion groups oral squamous cell…

SalivaProgrammed cell deathPathologymedicine.medical_specialtyOral Medicine and Pathologybusiness.industryResearchCancerOdontología:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseCiencias de la saludLesionstomatognathic diseasesApoptosisFibrosisUNESCO::CIENCIAS MÉDICASmedicineOral lichen planusmedicine.symptombusinessGeneral DentistryHomeostasisJournal of Clinical and Experimental Dentistry
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Magnesium homeostasis and aging.

2010

Aging is very often associated with magnesium (Mg) deficit. Total plasma magnesium concentrations are remarkably constant in healthy subjects throughout life, while total body Mg and Mg in the intracellular compartment tend to decrease with age. Dietary Mg deficiencies are common in the elderly population. Other frequent causes of Mg deficits in the elderly include reduced Mg intestinal absorption, reduced Mg bone stores, and excess urinary loss. Secondary Mg deficit in aging may result from different conditions and diseases often observed in the elderly (i.e. insulin resistance and/or type 2 diabetes mellitus) and drugs (i.e. use of hypermagnesuric diuretics). Chronic Mg deficits have been…

SenescenceAdultmedicine.medical_specialtyAgingClinical BiochemistryType 2 diabetesmedicine.disease_causeBiochemistryIntestinal absorptionYoung AdultInsulin resistanceRisk FactorsDiabetes mellitusInternal medicinemedicineHomeostasisHumansMagnesiumMolecular BiologyAgedAged 80 and overInflammationbusiness.industryType 2 Diabetes MellitusMiddle Agedmedicine.diseaseDietOxidative StressEndocrinologySarcopeniabusinessMagnesium DeficiencyOxidative stressDNA DamageMagnesium research
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Are control of extracellular acid-base balance and regulation of skeleton genes linked to resistance to ocean acidification in adult sea urchins?

2020

SCOPUS: ar.j

Settore BIO/07 - EcologiaCO2 ventsEnvironmental EngineeringClimate ChangeOceans and SeasMechanical propertiesAcid–base homeostasisEnvironnement et pollutionDICGene expressionExtracellularEnvironmental ChemistryAnimalsSeawaterWaste Management and DisposalGeneSkeletonCO2 ventAcid-Base EquilibriumResistance (ecology)ChemistryOcean acidificationOcean acidificationCarbon DioxideHydrogen-Ion ConcentrationbiomineralizationPollutionSkeleton (computer programming)adult sea urchinsCell biologyTechnologie de l'environnement contrôle de la pollutionqPCRSea UrchinsTraitement des déchetsGene expressionEchinodermsThe Science of the total environment
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Salivary gland proteins alterations in the diabetic milieu

2021

AbstractSalivary glands are considered the chief exocrine glands of the mouth and physiologically contribute to the maintenance of the homeostasis of the oral cavity. They consist of the parotid, submandibular and sublingual glands, which come in pairs and are collectively called the major glands, and the minor glands, which are much smaller and are dispersed throughout the buccal cavity. Salivary glands are distinguished by their size, amount of saliva secretion and their location in the oral cavity. Salivary glands pathophysiology has been a subject of interest in various worldwide metabolic disorders, including diabetes mellitus. Diabetes mellitus (DM), a global health concern, with a pa…

Settore BIO/17 - Istologia0301 basic medicineExocrine glandHistologyPhysiologySaliva secretionMuscarinic receptorsPhysiologyContext (language use)Salivary glandsModels Biological03 medical and health sciences0302 clinical medicineDiabetes mellitusstomatognathic systemDiabetes mellitusMedicineAnimalsHumansSalivary Proteins and PeptidesPeriodontitisReview PaperSalivary glandbusiness.industry030206 dentistryCell BiologyGeneral Medicinemedicine.diseasePathophysiologyOxidative Stress030104 developmental biologymedicine.anatomical_structureScreeningbusinessHomeostasisBiomarkersJournal of Molecular Histology
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