Search results for "mediators"

showing 10 items of 245 documents

Changes of inflammatory mediators in obese patients after laparoscopic cholecystectomy.

2010

Background Obesity is associated with the impairment of immunological functions. The aim of this study was to analyze some inflammatory mediators in obese subjects who underwent laparoscopic cholecystectomy. Methods Seventeen consecutive female patients with a BMI ranging from 35 to 45 kg/m2 (obese) and 17 consecutive female patients with BMI ranging from 20 to 25 kg/m2 (nonobese) were included in the study. All patients were affected by symptomatic gallbladder stone disease and underwent laparoscopic cholecystectomy. Changes in levels of leukocytes, neutrophils, IL-6, IL-10, leptin, and adiponectin were evaluated. Results We observed a significant increase in leukocyte and neutrophil level…

Leptinmedicine.medical_specialtySurgical stressmedicine.medical_treatmentInflammationGastroenterologyBody Mass IndexLeukocyte CountLaparoscopic cholecystectomyStress PhysiologicalInternal medicinemedicineHumansLaparoscopic cholecystectomy Obesity Inflammatory mediatorsObesitybusiness.industryObesity Laparoscopic Cholecystectomy Inflammatory MediatorsInterleukin-6GallbladderCholecystolithiasisVascular surgerySweet SyndromeInflammatory mediatorsCardiac surgeryInterleukin-10Settore MED/18 - Chirurgia Generalemedicine.anatomical_structureCholecystectomy LaparoscopicCardiothoracic surgerySurgeryCholecystectomyFemaleAdiponectinmedicine.symptomInflammation MediatorsbusinessAbdominal surgery
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Voluntary distance running prevents TNF-mediated liver injury in mice through alterations of the intrahepatic immune milieu

2017

AbstractPhysical activity confers a broad spectrum of health benefits. Beyond the obvious role in metabolically driven diseases, the role of physical activity in acute liver injury is poorly explored. To study the role of physical activity in acute liver injury, a novel model of voluntary distance running in mice was developed and mice were subjected to acute liver injury induced by N-galactosamine (GalN) and lipopolysaccharide (LPS). Analyses included histological stains, immunoblotting, qRT-PCR and FACS analysis. Voluntary distance running increased to an average of 10.3 km/day after a learning curve. Running lead to a decrease in the absolute numbers of intrahepatic CD4+ T and B lymphocy…

Lifestyle modification0301 basic medicineLipopolysaccharidesMaleCancer ResearchChemokineApoptosisGalactosamineLiver Function TestsAlarminsLiver injurybiologymedicine.diagnostic_testChemotaxisNF-kappa Bmedicine.anatomical_structureLiverReceptors Pattern RecognitionModels AnimalCytokinesTumor necrosis factor alphaOriginal Articlemedicine.symptomChemokinesInflammation Mediatorsmedicine.medical_specialtyImmunologyInflammationCCL2Proinflammatory cytokine03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicinePhysical Conditioning AnimalmedicineAnimalsLiver diseasesInflammationbusiness.industryTumor Necrosis Factor-alphaMonocyteBody WeightJNK Mitogen-Activated Protein KinasesCell BiologyLiver Failure Acutemedicine.diseaseEnzyme ActivationMice Inbred C57BL030104 developmental biologyEndocrinologyImmunologybiology.proteinLiver function testsbusinessCell Death & Disease
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Modulation of protein tyrosine nitration and inflammatory mediators by isoprenylhydroquinone glucoside.

2007

The nitration of tyrosine caused by peroxynitrite and other reactive nitrogen species is clearly detrimental for some physiological processes; however, its signalling role is still open to controversy. Among the natural phenolics known for their ability to oppose free tyrosine nitration, isoprenylhydroquinone glucoside is investigated due to its unusual structure, which contains a simple hydroxybenzene alkylated by a hemiterpenoid moiety. This hydroquinone was shown to be an effective inhibitor of peroxynitrite-induced protein tyrosine nitration in 3T3 fibroblasts. When tested on bovine seroalbumin nitration, however, the potency was reduced by half and the effect was almost abolished in th…

LipopolysaccharidesCell SurvivalNeutrophilsBlotting WesternInterleukin-1betaPharmaceutical ScienceNitric Oxide Synthase Type IIHemeNitric oxidechemistry.chemical_compoundMiceGlucosideGlucosidesNitrationPeroxynitrous AcidAnimalsHumansTyrosineReactive nitrogen speciesCells CulturedNitritesNitratesbiologyCell-Free SystemReverse Transcriptase Polymerase Chain ReactionRhodaminesTumor Necrosis Factor-alphaNitrotyrosineSerum Albumin Bovine3T3 CellsHydrogen PeroxideFibroblastsStimulation ChemicalHydroquinonesNitric oxide synthasechemistryBiochemistrybiology.proteinTetradecanoylphorbol AcetateTyrosineInflammation MediatorsPeroxynitriteEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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6-Dimethylamino 1H-pyrazolo[3,4-d]pyrimidine derivatives as new inhibitors of inflammatory mediators in intact cells.

2003

The synthesis of 6-dimethylamino 1H-pyrazolo[3,4-d]pyrimidines substituted at positions 1 and 4, and their effects on murine macrophage and human neutrophil functions are described. Several compounds and especially 4b-6b are potent inhibitors of PGE2 generation in murine macrophages. This action is related to a direct effect on COX-2 activity without affecting the enzyme expression. Some of these compounds also inhibited COX-1 and COX-2 in human monocytes and 4b showed selectivity for COX-2 inhibition. © 2003 Elsevier Science Ltd. All rights reserved.

LipopolysaccharidesMagnetic Resonance SpectroscopyPyrimidineClinical BiochemistryBlotting WesternPharmaceutical ScienceBiochemistryLeukotriene B4Pyrazolopyrimidinechemistry.chemical_compoundMiceStructure-Activity RelationshipDrug DiscoverymedicineLeukocytesMacrophageAnimalsHumansCyclooxygenase InhibitorsMolecular Biologychemistry.chemical_classificationbiologyCyclooxygenase 2 InhibitorsPancreatic ElastaseMonocyteOrganic ChemistryMembrane ProteinsBiological activityIn vitroIsoenzymesEnzymemedicine.anatomical_structurePyrimidineschemistryBiochemistryEnzyme inhibitorCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesLuminescent Measurementsbiology.proteinCyclooxygenase 1Macrophages PeritonealMolecular MedicinePyrazolesInflammation MediatorsBioorganicmedicinal chemistry
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The anti-inflammatory and antinociceptive effects of NF-κB inhibitory guanidine derivative ME10092

2010

The guanidine compound ME10092 (1-(3,4-dimethoxy-2-chlorobenzylideneamino)-guanidine) is known to possess anti-radical and anti-ischemic activity but its molecular targets have not been identified. This study investigated whether ME10092 regulates the nuclear factor kappa B (NF-kappaB)-mediated signal transduction in vivo. The effect of ME10092 treatment (1-100 pmol/mouse) on nuclear translocation of NF-kappaB, activation of expression of inflammatory mediators and production of nitric oxide were measured in the lipopolysaccharide (LPS)-induced brain inflammation model in mice in vivo. The antinociceptive activity of ME10092 was tested in the formalin-induced paw licking test. ME10092 dose-…

LipopolysaccharidesMaleNecrosisTranscription GeneticLipopolysaccharidemedicine.drug_classInterleukin-1betaImmunologyAdministration OralNitric Oxide Synthase Type IIInflammationPharmacologyNitric OxideGuanidinesAnti-inflammatoryNitric oxideMicechemistry.chemical_compoundIn vivoFormaldehydemedicineAnimalsImmunology and AllergyPain MeasurementPharmacologyAnalgesicsMice Inbred ICRbiologyTumor Necrosis Factor-alphaAnti-Inflammatory Agents Non-SteroidalNF-kappa BNitric oxide synthasechemistryCyclooxygenase 2Immunologybiology.proteinEncephalitisInflammation Mediatorsmedicine.symptomLickingSignal TransductionInternational Immunopharmacology
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Beta- and gamma-melanocortins inhibit lipopolysaccharide induced nitric oxide production in mice brain.

2003

The pro-opiomelanocortin-derived peptide alpha-melanocyte stimulating hormone (alpha-MSH) mediates many diverse physiological actions, including anti-inflammatory and immunomodulatory effects. However, little is known about the physiological roles of the other melanocortins, beta- and gamma-MSH. Here, we investigated the effects of melanocortin peptides in an in vivo neuroinflammation model. Six hours following intracisternal (i.c.) administration of 10 microg lipopolysaccharide (LPS) to mice a five-fold increase in the nitric oxide (NO) level was seen in the animals' brains, when detected by electron paramagnetic resonance (EPR). All tested melanocortins, alpha-, beta-, gamma1- and gamma2-…

LipopolysaccharidesMaleendocrine systemmedicine.medical_specialtyLipopolysaccharideCentral nervous systemInflammationPharmacologyBiologyNitric OxideNitric oxidechemistry.chemical_compoundMicegamma-MSHIn vivoInternal medicinebeta-MSHmedicineAnimalsMolecular BiologyNeuroinflammationMelanocortinsFeedback PhysiologicalMice Inbred ICRintegumentary systemDose-Response Relationship DrugGeneral NeuroscienceElectron Spin Resonance SpectroscopyBrainDisease Models Animalmedicine.anatomical_structureEndocrinologychemistryalpha-MSHNeurology (clinical)Melanocortinmedicine.symptomInflammation Mediatorshormones hormone substitutes and hormone antagonistsDevelopmental BiologySignal TransductionBrain research
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Hepatic over-expression of TGF-beta1 promotes LPS-induced inflammatory cytokine secretion by liver cells and endotoxemic shock.

2005

Transforming growth factor-beta (TGF-beta) is an important suppressor of inflammation. However, TGF-beta has also been found to promote secretion of inflammatory cytokines, and transgenic mice, which constitutively express TGF-beta in liver, have been found to be more susceptible to endotoxemia. To approach this apparent paradox, we investigated the role of hepatic TGF-beta1 in endotoxemia by utilising inducible TGF-beta1-transgenic mice that express TGF-beta1 under control of the C-reactive protein promoter. In contrast to non-transgenic littermates, administration of lipopolysaccharide (LPS) induced strongly increased expression of TGF-beta and acute phase proteins in the TGF-beta1-transg…

LipopolysaccharidesMalemedicine.medical_specialtyLipopolysaccharidemedicine.medical_treatmentImmunologyInflammationMice TransgenicBiologyProinflammatory cytokineTransforming Growth Factor beta1chemistry.chemical_compoundMiceImmune systemTransforming Growth Factor betaInternal medicinemedicineImmunology and AllergyAnimalsSecretionAcute-Phase ReactionCells CulturedInterleukin-6Acute-phase proteinEndotoxemiaCytokineEndocrinologychemistryHepatocytesCytokine secretionmedicine.symptomInflammation MediatorsImmunology letters
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Inhibition of leukocyte functions by the alkaloid isaindigotone from Isatis indigotica and some new synthetic derivatives.

2001

The alkaloid isaindigotone (1a) and seven derivatives have been synthesized to study their influence on several leukocyte functions and the generation of inflammatory mediators. Isaindigotone (1a) was found to be a scavenger of superoxide generated either by the hypoxanthine/xanthine oxidase system or stimulated human neutrophils. Isaindigotone (1a) and its acetylated derivative (1b) also inhibited 5-lipoxygenase activity and leukotriene B(4) production in these cells, whereas none of the compounds affected degranulation. In RAW 264.7 macrophages stimulated with lipopolysaccharide, synthetic derivatives exerted higher inhibitory effects on prostaglandin E(2) (PGE(2)) and nitric oxide (NO) g…

LipopolysaccharidesXanthine OxidaseMagnetic Resonance SpectroscopyLeukotriene B4StereochemistryNeutrophilsmedicine.medical_treatmentPharmaceutical ScienceLeukotriene B4DinoprostoneAnalytical ChemistryNitric oxidechemistry.chemical_compoundInhibitory Concentration 50MiceStructure-Activity RelationshipAlkaloidsDrug DiscoverymedicineLeukocytesAnimalsHumansLipoxygenase InhibitorsXanthine oxidaseHypoxanthineCells CulturedPharmacologyInflammationPlants MedicinalbiologyMolecular StructureSuperoxideAlkaloidMacrophagesOrganic ChemistryFree Radical ScavengersComplementary and alternative medicineBiochemistrychemistryArachidonate 5-lipoxygenaseBrassicaceaebiology.proteinQuinazolinesMolecular MedicineChromatography Thin LayerInflammation MediatorsNitric Oxide SynthaseProstaglandin EJournal of natural products
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Inflammatory Characteristics of Monocytes from Pediatric Patients with Tuberous Sclerosis.

2015

Objective  Therapeutic options for the tuberous sclerosis complex (TSC) syndrome showed varying outcomes. Malfunctional tsc1 / tsc2 genes leave mTOR uninhibited, a positive downstream modulator of the innate proinflammatory immune system, which has not yet been described in pediatric patients with TSC. Methods  Using polymerase chain reaction (PCR) gene expression levels of monocytes after cultivation with lipopolysaccharide (LPS) or with LPS + mTOR inhibitor rapamycin, patients with TSC ( n  = 16) were compared with healthy subjects ( n  = 20). Results  Compared with monocytes from healthy controls, LPS showed a more prominent gene expression pattern in patients with TSC (CCL24, CXCL10, IL…

Lipopolysaccharidescongenital hereditary and neonatal diseases and abnormalitiesLipopolysaccharideGene ExpressionMonocytesProinflammatory cytokinechemistry.chemical_compoundTuberous sclerosisTuberous SclerosisGene expressionmedicineCXCL10HumansChildInflammationSirolimusbusiness.industryTOR Serine-Threonine KinasesInfant NewbornInfantGeneral Medicinemedicine.diseasemedicine.anatomical_structureCross-Sectional StudieschemistryChild PreschoolPediatrics Perinatology and Child HealthImmunologyCytokinesNeurology (clinical)TSC1TSC2Inflammation MediatorsbusinessCCL24Immunosuppressive AgentsNeuropediatrics
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Sex hormones modulate inflammatory mediators produced by macrophages.

1999

Lipopolysaccharidesmedicine.medical_specialtyNitric oxide biosynthesisReceptors EstradiolNitric OxideGeneral Biochemistry Genetics and Molecular BiologyCell LineHistory and Philosophy of ScienceInternal medicinemedicineAnimalsTestosteroneReceptorEstradiolbusiness.industryTumor Necrosis Factor-alphaGeneral NeuroscienceMacrophagesTumor Necrosis Factor alpha biosynthesisAndrogen MetabolismInterleukin-10EndocrinologyCell cultureReceptors AndrogenInflammation MediatorsbusinessHormoneAnnals of the New York Academy of Sciences
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