Search results for "oxidative stress."

showing 10 items of 1575 documents

Different strategies to achieve Pb-tolerance by the two Trebouxia algae coexisting in the lichen Ramalina farinacea.

2012

Lichen thalli are permeable to airborne substances, including heavy metals, which are harmful to cell metabolism. Ramalina farinacea shows a moderate tolerance to Pb. This lichen comprises two Trebouxia phycobionts, provisionally referred to as TR1 and TR9, with distinct physiological responses to acute oxidative stress. Thus, there is a more severe decay in photosynthesis and photosynthetic pigments in TR1 than in TR9. Similarly, under oxidative stress, antioxidant enzymes and HSP70 protein decrease in TR1 but increase in TR9. Since Pb toxicity is associated with increased ROS formation, we hypothesized greater Pb tolerance in this phycobiont. Accordingly, the aim of the present study was …

TrebouxiaChlorophyllAntioxidantLichensPhysiologymedicine.medical_treatmentBOTANICAGlutathione reductasePlant SciencePhotosynthesisAntioxidantsFluorescenceLichen microalgaeRamalina farinaceaSuperoxide dismutaseElectron TransportAscorbate PeroxidasesSpecies SpecificityChlorophytaStress PhysiologicalBotanymedicineHSP70 Heat-Shock ProteinsPhotosynthesisSymbiosisChlorophyll fluorescencePlant ProteinsBIOLOGIA VEGETALbiologySuperoxide DismutaseStress responsebiology.organism_classificationAPXCatalaseOxidative StressGlutathione ReductaseBiochemistryLeadTrebouxia algaebiology.proteinReactive Oxygen SpeciesHeavy metal toleranceAgronomy and Crop ScienceJournal of plant physiology
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Unraveling the complex trait of low temperature adaptation in the wine yeast Saccharomyces cerevisiae

2016

1. Introducción Se cree que las uvas fueron domesticadas entre el Mar Negro e Irán durante el periodo del 7000-4000 aC. Las primeras evidencias de elaboración de vino provienen de la presencia de ácido tartárico en un tarro antiguo que data de 5400 - 5000 aC en el yacimiento neolítico de Tepe en Mesopotamia y de los restos de la extracción del zumo de uva en el yacimiento neolítico de Dikili Tash en Grecia (5000 aC). La colonización de los romanos extendió la elaboración del vino por todo el Mediterráneo; en el 500 aC el vino ya se producía en Italia, Francia, España, Portugal y el norte de África. Posteriormente también fue extendido a los Balcanes, Alemania y otras partes del norte de Eur…

UNESCO::CIENCIAS DE LA VIDAmolecular biologyoxidative stress responseyeastlow temperaturewinequantitative trait analysis:CIENCIAS DE LA VIDA [UNESCO]phenotype-genotype interaction
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Oxidative stress, a new hallmark in the pathophysiology of Lafora progressive myoclonus epilepsy

2015

12 páginas, 4 figuras, 1 tabla

Ubiquitin-Protein LigasesFree radicalsBiologymedicine.disease_causeBiochemistryAntioxidantsLafora diseasechemistry.chemical_compoundLaforinPhysiology (medical)medicineHumansLafora diseaseProteostasis DeficienciesGlycogenAutophagyProtein Tyrosine Phosphatases Non-ReceptorMalinmedicine.diseaseOxidative StressProteostasisLafora DiseaseBiochemistrychemistryProteasomeOxidative stressMutationProteostasisUnfolded protein responseCarrier ProteinsLaforinGlycogenOxidative stressFree Radical Biology and Medicine
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An inter-laboratory validation of methods of lipid peroxidation measurement in UVA-treated human plasma samples

2010

Lipid peroxidation products like malondialdehyde, 4-hydroxynonenal and F2-isoprostanes are widely used as markers of oxidative stress in vitro and in vivo. This study reports the results of a multi-laboratory validation study by COST Action B35 to assess inter-laboratory and intra-laboratory variation in the measurement of lipid peroxidation. Human plasma samples were exposed to UVA irradiation at different doses (0, 15 J, 20 J), encoded and shipped to 15 laboratories, where analyses of malondialdehyde, 4-hydroxynonenal and isoprostanes were conducted. The results demonstrate a low within-day-variation and a good correlation of results observed on two different days. However, high coefficie…

Ultraviolet RaysClinical Chemistry TestsEnzyme-Linked Immunosorbent AssayIsoprostanesmedicine.disease_causeF2-isoprostanesSensitivity and SpecificityBiochemistryHigh-performance liquid chromatographyMass Spectrometry4-HydroxynonenalLipid peroxidationPlasmachemistry.chemical_compoundIn vivoMalondialdehydemedicineHumansChromatography High Pressure LiquidAldehydesChromatographyChemistryReproducibility of Resultsoxidative stress; F2-Isoprostanes; 4.-hydroxynonenal; malondialdehydeGeneral MedicineOxidative stress; F2-isoprostanes; 4-hydroxynonenal; malondialdehydeMalondialdehydeIsoprostanes4-hydroxynonenalF2-IsoprostanesBiochemistryOxidative stressLipid PeroxidationOxidative stressChromatography Liquid
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Antioxidant betalains from cactus pear (Opuntia ficus-indica) inhibit endothelial ICAM-1 expression.

2004

It has been suggested that some pigments would have antioxidant properties and that their presence in dietary constituents would contribute to reduce the risk of oxidative stress–correlated diseases. Among others, inflammatory response depends on redox status and may implicate oxidative stress. Vascular endothelial cells are a direct target of oxidative stress in inflammation. We have tested the impact of the free radical scavenger and antioxidant properties of betalains from the prickle pear in an in vitro model of endothelial cells. Here we show the capacity of betalains to protect endothelium from cytokine- induced redox state alteration, through ICAM-1 inhibition. KEYWORDS: endothelial …

Umbilical VeinsAntioxidantEndotheliumICAM-1Pyridinesmedicine.medical_treatmentAnti-Inflammatory AgentsBetalainsInflammationOxidative phosphorylationBiologymedicine.disease_causeModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyAntioxidantschemistry.chemical_compoundHistory and Philosophy of ScienceSettore BIO/10 - BiochimicamedicineHumansCells CulturedInflammationICAM-1Dose-Response Relationship DrugPlant ExtractsGeneral NeurosciencebetalainOpuntiaFree radical scavengerFlow CytometryIntercellular Adhesion Molecule-1BetaxanthinsQuaternary Ammonium CompoundsOxidative Stressmedicine.anatomical_structurechemistryBiochemistryendothelial cellendothelial cells; ICAM-1; betalains; antiinflammatory drugsCytokinesEndothelium Vascularantiinflammatory drugsmedicine.symptomIndicaxanthinOxidation-ReductionOxidative stressAnnals of the New York Academy of Sciences
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Glyceraldehyde-3-phosphate dehydrogenase regulates endothelin-1 expression by a novel, redox-sensitive mechanism involving mRNA stability

2008

17 pages.-- PMID: 18809573 [PubMed].-- Printed version published on Dec 2008.

Untranslated regionUmbilical VeinsRNA StabilityRNA StabilityGlyceraldehyde-3'-phosphate dehydrogenase (GAPDH)Plasma protein bindingstomatognathic systemHumansmRNA stabilityS-Glutathionylation3' Untranslated RegionsMolecular BiologyGlyceraldehyde 3-phosphate dehydrogenaseRegulation of gene expressionMessenger RNAEndothelin-1biologyThree prime untranslated regionGlyceraldehyde-3-Phosphate DehydrogenasesArticlesCell BiologyGlutathioneOxidative StressGene Expression RegulationBiochemistryEndothelin-1 (ET-1)biology.proteinOxidation-ReductionProtein Binding
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Urinary metals and metal mixtures and oxidative stress biomarkers in an adult population from Spain: The Hortega Study

2019

Introduction: Few studies have investigated the role of exposure to metals and metal mixtures on oxidative stress in the general population. Objectives: We evaluated the cross-sectional association of urinary metal and metal mixtures with urinary oxidative stress biomarkers, including oxidized to reduced glutathione ratio (GSSG/GSH), malondialdehyde (MDA), and 8-oxo-7,8-dihydroguanine(8-oxo-dG), in a representative sample of a general population from Spain (Hortega Study). Methods: Urine antimony (Sb), barium (Ba), cadmium (Cd), chromium (Cr), cobalt (Co), copper (Cu), molybdenum (Mo), vanadium (V) and zinc (Zn) were measured by ICPMS in 1440 Hortega Study participants. Results: The geometr…

Urine metalsMetal mixturesOxidative stressPopulation-based
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Oxidative stress in vascular disease: causes, defense mechanisms and potential therapies

2007

Endothelial cells control vascular homeostasis by generating paracrine factors that regulate vascular tone, inhibit platelet function, prevent adhesion of leukocytes, and limit proliferation of vascular smooth muscle. The dominant factor responsible for many of those effects is endothelium-derived nitric oxide (NO). Endothelial dysfunction characterized by enhanced inactivation or reduced synthesis of NO, alone or in combination, is seen in conjunction with risk factors for cardiovascular disease. Endothelial dysfunction can promote vasospasm, thrombosis, vascular inflammation, and proliferation of the intima. Vascular oxidative stress and increased production of reactive oxygen species con…

Vascular smooth muscleEndotheliumArteriosclerosisPharmacologyNitric Oxidemedicine.disease_causeAntioxidantsReceptor Angiotensin Type 1Superoxide dismutaseRisk FactorsmedicineHumansEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologybusiness.industryAnticholesteremic AgentsGeneral Medicinemedicine.diseaseOxidative Stressmedicine.anatomical_structureMitochondrial respiratory chainchemistryImmunologybiology.proteinEndothelium VascularHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessOxidative stressNature Clinical Practice Cardiovascular Medicine
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Organic Nitrate Therapy, Nitrate Tolerance, and Nitrate-Induced Endothelial Dysfunction: Emphasis on Redox Biology and Oxidative Stress

2015

Abstract Organic nitrates, such as nitroglycerin (GTN), isosorbide-5-mononitrate and isosorbide dinitrate, and pentaerithrityl tetranitrate (PETN), when given acutely, have potent vasodilator effects improving symptoms in patients with acute and chronic congestive heart failure, stable coronary artery disease, acute coronary syndromes, or arterial hypertension. The mechanisms underlying vasodilation include the release of •NO or a related compound in response to intracellular bioactivation (for GTN, the mitochondrial aldehyde dehydrogenase [ALDH-2]) and activation of the enzyme, soluble guanylyl cyclase. Increasing cyclic guanosine-3′,-5′-monophosphate (cGMP) levels lead to an activation of…

Vascular smooth musclePhysiologyVasodilator AgentsClinical BiochemistryVasodilationPharmacologymedicine.disease_causeBiochemistryCalcium in biologyComprehensive Invited ReviewmedicineAnimalsHumansEndothelial dysfunctionMolecular BiologyGeneral Environmental ScienceNitratesChemistryCell Biologymedicine.diseaseOxidative StressCardiovascular DiseasesGeneral Earth and Planetary SciencesEndothelium VascularIsosorbide dinitrateReactive Oxygen SpeciesSoluble guanylyl cyclaseOxidation-ReductionOxidative stressIntracellularmedicine.drugAntioxidants & Redox Signaling
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Pro-inflammatory effects of interleukin-17A on vascular smooth muscle cells involve NAD(P)H- oxidase derived reactive oxygen species.

2010

T cells are known for their contribution to the inflammatory element of atherosclerosis. Recently, it has been demonstrated that the Th17 derived cytokine IL-17 is involved in the pro-inflammatory response of vascular smooth muscle cells (VSMC). The aim of the present study was to examine whether reactive oxygen species (ROS) might be involved in this context. The effect of IL-17A on ROS generation was examined using the fluorescent dye 2′7′-dichlorodihydrofluorescein (H<sub>2</sub>DCF) in primary murine VSMC. IL-17A induced an increase in H<sub>2</sub>DCF fluorescence in VSMC, and this effect was blocked by the NAD(P)H-oxidase inhibitor apocynin and siRNA targeting …

Vascular smooth musclePhysiologymedicine.medical_treatmentAorta Thoracicmedicine.disease_causep38 Mitogen-Activated Protein KinasesMuscle Smooth Vascularchemistry.chemical_compoundMiceCell MovementmedicineAnimalsEnzyme InhibitorsRNA Small InterferingCells Culturedchemistry.chemical_classificationReactive oxygen speciesNADPH oxidaseMembrane GlycoproteinsbiologyInterleukin-17AcetophenonesNADPH OxidasesCell DifferentiationMolecular biologyMice Inbred C57BLOxidative StressCytokinechemistryBiochemistryNAD(P)H oxidaseNADPH Oxidase 4ApocyninNADPH Oxidase 2cardiovascular systembiology.proteinCytokinesNAD+ kinaseCardiology and Cardiovascular MedicineReactive Oxygen SpeciesOxidative stressJournal of vascular research
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