Search results for "phenotype"

showing 10 items of 1875 documents

De novo 15q21.1q21.2 deletion identified through FBN1 MLPA and refined by 244K array-CGH in a female teenager with incomplete Marfan syndrome

2010

International audience; Interstitial deletions involving the 15q21.1 band are very rare. Only 4 of these cases have been studied using molecular cytogenetic techniques in order to confirm the deletion of the whole FBN1 gene. The presence of clinical features of the Marfan syndrome (MFS) spectrum associated with mental retardation has been described in only 2/4 patients. Here we report on a 16-year-old female referred for suspicion of MFS (positive thumb and wrist sign, scoliosis, joint hyperlaxity, high-arched palate with dental crowding, dysmorphism, mitral insufficiency with dystrophic valve, striae). She had therefore 3 minor criteria according to the Ghent nosology. She also had speech …

AdultMalemusculoskeletal diseasesProbandMarfan syndromecongenital hereditary and neonatal diseases and abnormalitiesAdolescent[SDV]Life Sciences [q-bio]Fibrillin-1BiologyFibrillinsBioinformaticsPolymerase Chain ReactionMarfan SyndromeLoss of heterozygosity03 medical and health sciencesTransforming Growth Factor betaIntellectual DisabilityGeneticsmedicineHumansMultiplex ligation-dependent probe amplificationAlleleChildGeneIn Situ Hybridization FluorescenceGenetics (clinical)Oligonucleotide Array Sequence AnalysisSequence Deletion030304 developmental biologyGeneticsChromosomes Human Pair 15Comparative Genomic Hybridization0303 health sciencesMicrofilament Proteins030305 genetics & heredityGeneral Medicinemedicine.diseasePedigree3. Good healthPhenotypeMutationMicrosatelliteFemaleDNA ProbesHaploinsufficiencyMicrosatellite RepeatsEuropean Journal of Medical Genetics
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Can palpation-induced muscle pain pattern contribute to the differential diagnosis among temporomandibular disorders, primary headaches phenotypes an…

2015

Background The evaluation of possible differences in the distribution or characteristics of palpation-induced pain in the masticatory muscles could be valuable in terms of diagnostic assessment. The aim of this study was to evaluate the impact of different combinations of anterior temporalis (AT) and masseter palpation-induced pain in the diagnostic of temporomandibular disorder (TMD), primary headaches and bruxism. Material and Methods A total of 1200 dental records of orofacial pain adult patients were analyzed. The outcomes were dichotomously classified (presence/absence) as following: a) AT and/or masseter palpation-induced pain; b) myogenous TMD; c) temporomandibular joint (TMJ) arthra…

AdultMalemyalgiamedicine.medical_specialtyOdontologíaPalpationDiagnosis Differential03 medical and health sciences0302 clinical medicinePrimary headacheFacial PainmedicineHumansGeneral DentistryRetrospective StudiesOrthodonticsPalpationOral Medicine and Pathologymedicine.diagnostic_testResearchTemporomandibular disorderHeadacheRetrospective cohort studyMyalgia030206 dentistryTemporomandibular Joint Disorders:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludMasticatory forcestomatognathic diseasesCross-Sectional StudiesPhenotypeOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASPhysical therapyDiagnostic assessmentBruxismFemaleSurgeryDifferential diagnosismedicine.symptomPsychology030217 neurology & neurosurgeryMedicina Oral Patología Oral y Cirugia Bucal
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Monocyte Chemoattractant Protein-1 Serum Levels in Patients with Breast Cancer

2003

The chemokine monocyte chemoattractant protein (MCP)-1 is thought to be involved in breast carcinogenesis. We evaluated MCP-1 serum levels in patients with breast cancer (n = 135), ductal carcinoma in situ (DCIS) I-III (n = 30), benign breast lesions (n = 143) and in healthy women (n = 27). We determined the value of MCP-1 serum levels as a differentiation marker between malignant, preinvasive and benign breast diseases and as a predictive marker for the biological phenotype of breast carcinoma. Median (range) MCP-1 serum levels in patients with breast cancer, DCIS I-III, benign breast lesions and healthy women were 200 (57-692) pg/ml, 194 (58-525) pg/ml, 174 (39-529) pg/ml and 175 (67-425)…

AdultOncologymedicine.medical_specialtyCA 15-3Breast NeoplasmsBreast DiseasesBreast cancerReference ValuesInternal medicineBiomarkers TumormedicineCarcinomaHumansNeoplasm Invasivenessskin and connective tissue diseasesLymph nodeChemokine CCL2AgedRetrospective StudiesPredictive markerbusiness.industryCase-control studyGeneral MedicineMiddle AgedDuctal carcinomamedicine.diseaseCarcinoma Intraductal NoninfiltratingCell Transformation NeoplasticPhenotypemedicine.anatomical_structureCase-Control StudiesLymphatic MetastasisFemaleBreast carcinomabusinessTumor Biology
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Mild androgen phenotypes.

2006

Mild androgen phenotypes are found in 30-40% of patients referred to an endocrine clinic because of suspected hyperandrogenic syndrome. These disorders are characterized by clinical or biological signs of hyperandrogenism in women with normal ovulatory menstrual cycles. Three main mild androgen disorders may be distinguished: ovulatory polycystic ovarian syndrome (PCOS), idiopathic hyperandrogenism, and idiopathic hirsutism. Ovulatory PCOS includes ovulatory hyperandrogenic patients presenting with polycystic ovaries. Using ESHRE/ASRM criteria for diagnosis of PCOS, this disorder is now part of PCOS spectrum. While in vivo and in vitro studies have confirmed the similarities between the two…

AdultOvulationRiskmedicine.medical_specialtyHirsutismendocrine system diseasesmedicine.drug_classEndocrinology Diabetes and Metabolismmedia_common.quotation_subjectEndocrinologyInsulin resistanceInternal medicinemedicineEndocrine systemHumansOvulationhirsutismmedia_commonUltrasonographybusiness.industryHyperandrogenismOvarymedicine.diseaseAndrogenPhenotypePolycystic ovaryEndocrinologyPhenotypeCardiovascular DiseasesFemalebusinessHyperandrogenismPolycystic Ovary SyndromeBest practiceresearch. Clinical endocrinologymetabolism
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Clinical and endocrine characteristics of the main polycystic ovary syndrome phenotypes

2009

Abstract OBJECTIVE: To evaluate the clinical and endocrine differences between main polycystic ovary syndrome (PCOS) phenotypes. DESIGN: To evaluate clinical and hormone parameters in a large group of consecutive women with PCOS diagnosed according Rotterdam criteria and divided according their phenotype. SETTING: University department of medicine. PATIENT(S): Three hundred eighty-two consecutive women with PCOS and 85 ovulatory controls. INTERVENTION(S): Evaluation of clinical and hormone parameters. MAIN OUTCOME MEASURE(S): Blood levels of gonadotropins, testosterone, sex-hormone-binding globulin, dehydroepiandrosterone sulfate, 17α-hydroxyprogesterone, progesterone, glucose, and insulin,…

AdultPCOS Lipids Insulin resistance Fertility Androgens Obesitymedicine.medical_specialtySettore MED/09 - Medicina Internaendocrine system diseasesEndocrine SystemSettore MED/13 - EndocrinologiaAnovulationYoung Adultchemistry.chemical_compoundDehydroepiandrosterone sulfateSex Hormone-Binding GlobulinInternal medicinePrevalencemedicineHumansTestosteroneRetrospective StudiesDehydroepiandrosterone SulfateFree androgen indexbusiness.industryHyperandrogenismObstetrics and GynecologyLuteinizing Hormonemedicine.diseaseSettore MED/40 - Ginecologia E OstetriciaPolycystic ovaryfemale genital diseases and pregnancy complicationsPhenotypeEndocrinologyReproductive MedicinechemistryCase-Control StudiesAndrogensFemaleFollicle Stimulating HormoneHyperandrogenismLuteinizing hormonebusinessPolycystic Ovary SyndromeHormoneFertility and Sterility
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Genetic Imbalances in Precursor Lesions of Endometrial Cancer Detected by Comparative Genomic Hybridization

2000

Endometrial hyperplasia is regarded as a precursor lesion of endometrioid adenocarcinomas of the endometrium. The genetic events involved in the multistep process from normal endometrial glandular tissue to invasive endometrial carcinomas are primarily unknown. We chose endometrial hyperplasia as a model for identifying chromosomal aberrations occurring during carcinogenesis. Comparative genomic hybridization (CGH) was performed on 47 formalin-fixed, paraffin-embedded specimens of endometrial hyperplasia using the microdissection technique to increase the number of tumor cells in the samples and reduce contamination from normal cells. CGH analysis revealed that 24 out of 47 (51%) samples ha…

AdultPathologymedicine.medical_specialtyGenotypeShort CommunicationsBiologyAdenocarcinomaPathology and Forensic MedicinemedicineAtypiaHumansMicrodissectionAgedNeoplasm StagingAged 80 and overEndometrial cancerNucleic Acid HybridizationHyperplasiaMiddle Agedmedicine.diseaseEndometrial hyperplasiaEndometrial NeoplasmsPhenotypeDysplasiaAdenocarcinomaFemalePrecancerous ConditionsComparative genomic hybridization
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Desmin-related myopathies

1997

Desmin-related myopathies are marked by accumulation of desmin, which is often familial and associated with cardiomyopathy. When multifocal this excess is characterized by inclusions such as cytoplasmic or spheroid bodies, when disseminated the excess is called granulofilamentous material. Excess of desmin might represent an abnormal type of protein metabolism.

AdultPathologymedicine.medical_specialtyGranulofilamentous materialCardiomyopathyChromosome DisordersGenes Recessivemacromolecular substancesBiologyDesminMuscular DiseasesmedicineHumansChildMuscle SkeletalGenotype-Phenotype CorrelationsGenes DominantChromosome AberrationsInclusion BodiesDESMIN-RELATED MYOPATHYMyocardiumMolecular pathogenesismusculoskeletal systemmedicine.diseaseActin CytoskeletonNeurologyCytoplasmDesminNeurology (clinical)CardiomyopathiesCurrent Opinion in Neurology
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Congenital neutropenia with retinopathy, a new phenotype without intellectual deficiency or obesity secondary toVPS13Bmutations

2013

Over one hundred VPS13B mutations are reported in Cohen syndrome (CS). Most cases exhibit a homogeneous phenotype that includes intellectual deficiency (ID), microcephaly, facial dysmorphism, slender extremities, truncal obesity, progressive chorioretinal dystrophy, and neutropenia. We report on a patient carrying two VPS13B splicing mutations with an atypical phenotype that included microcephaly, retinopathy, and congenital neutropenia, but neither obesity nor ID. RNA analysis of the IVS34+2T_+3AinsT mutation did not reveal any abnormal splice fragments but mRNA quantification showed a significant decrease in VPS13B expression. RNA sequencing analysis up- and downstream from the IVS57+2T>C…

AdultPathologymedicine.medical_specialtyMicrocephalyNeutropeniaDNA Mutational AnalysisVesicular Transport ProteinsNeutropeniamedicine.disease_causeRetinal DiseasesIntellectual DisabilityGene OrderGeneticsmedicineCongenital Bone Marrow Failure SyndromesHumansObesityCongenital NeutropeniaGenetics (clinical)GeneticsMutationCohen syndromebusiness.industryFaciesSyndromemedicine.diseasePhenotypePedigreeVPS13BPhenotypeMutationFemalebusinessRetinopathyAmerican Journal of Medical Genetics Part A
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Current state of clinical and morphological features in human NCL.

2004

The neuronal ceroid lipofuscinoses (NCL) are large group of autosomal recessive lysosomal storage disorders with both enzymatic deficiency and structural protein dysfunction. Previously, diagnosis of (NCL) was based on age at onset clinicopathological (C‐P) findings described 4 forms, classified as infantile (INCL) (2), late‐infantile (LINCL) (5), juvenile (JNCL) (6), and adult (ANCL) most patients with NCL have progressive ocular and cerebral dysfunvtion, including cognitive/motor dysfunction and uncontrolled seizures. After reviewing 520 patients with NCL, we found that about 104 (20%) did not fit this classification of NCL With further research, 4 additional forms have been recognized: F…

AdultPathologymedicine.medical_specialtymedicine.disease_causeArticlePathology and Forensic MedicineEpilepsyNeuronal Ceroid-LipofuscinosesGenotypemedicineHumansPalmitoyl protein thioesteraseAge of OnsetChildInclusion BodiesMutationbiologyTripeptidyl-Peptidase 1General NeurosciencePPT1Infantmedicine.diseasePhenotypeCLN8Child PreschoolMutationbiology.proteinNeurology (clinical)Age of onsetBrain pathology (Zurich, Switzerland)
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Distribution and phenotype ofGJB2mutations in 102 Sicilian patients with congenital non syndromic sensorineural hearing loss

2014

Objective: To evaluate the frequency of GJB2 mutations and their correlation with phenotype in Sicilian non-syndromic sensorineural hearing loss (NSHL) patients. Design: Sequencing of the coding region, basal promoter, exon 1, and donor splice site of the GJB2 gene; screening for the presence of the two common GJB6 deletions. Study sample: A cohort of 102 Sicilian NSHL patients. Results: Fifteen different mutations in GJB2 and seventeen different genotypes were detected. No GJB6 mutations were found. The hearing impairment was profound in the 64.72% of probands (mean PTA 0.25 – 4 kHz of 88.82 26.52 dB HL). A total of 81.37% of patients harboured at least one c.35delG allele; c.167delT and c…

AdultProbandLinguistics and Languagemedicine.medical_specialtyAdolescentGenotypeHearing Loss SensorineuralAudiologyConnexinsLanguage and LinguisticsYoung AdultSpeech and HearingExonBasal (phylogenetics)Genotypeotorhinolaryngologic diseasesHumansMedicineAlleleChildSicilyAgedRetrospective Studiesbiologybusiness.industrySensorineural hearing loss; GJB2; Genotype-Phenotype; SicilyMiddle Agedmedicine.diseaseGJB2Settore MED/32 - AudiologiaConnexin 26Settore MED/31 - OtorinolaringoiatriaPhenotypeSettore MED/03 - Genetica MedicaSensorineural hearing loGenotype-PhenotypeMutationCohortbiology.proteinSensorineural hearing lossbusinessGJB6International Journal of Audiology
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