0000000000044157
AUTHOR
Profita M
CysLT1 receptor activation-induced adhesion of human eosinophils to bronchial epithelial cells: signal transduction and pharmacological modulation
Obstructive sleep apnea does not affect circulating hematopoietic precursors
AIRWAYS CELLS IN SWIMMERS: A CASE REPORT AND A REVIEW OF THE LITERATURE.
Circulating Immature Blood Cells in Obstructive Sleep Apnea Syndrome(OSAS)
Elevated expression of prostaglandin receptor and increased release of prostaglandin E maintain the survival of CD45RO T cells in the inflamed human pleural space.
Effetti dell'allenamento aerobico di 12 settimane in bambini con asma lieve intermittente
Increased markers of angiogenesis in peripheral blood in long distance runners after a race
Reticulocyte release in obstructive sleep apnea
THE M3 MUSCARINIC RECEPTOR IS INVOLVED IN THE ACTIVATION OF AIRWAY EPITHELIUM IN VITRO
Functional role of PAR-1 receptors in the epithelial cells of smokers with and without COPD: involvement of intracellular signal pathways.
Beneficial effects of aerobic training+montelukast in children with mild intermittent asthma
Pleural mesothelial cells express both BLT2 and PPARa and mount an integrated response to pleural LTB4
Role of PGE2 in the invasiveness, growth and protection of cancer cells in malignant pleuritis
Abstract The recurrence of pleural effusions is a common event in a variety of neoplastic diseases. The objective of this study was to identify the mechanisms promoting the homing and growth of cancer cells within the pleural space. A cancer cell line recovered from malignant pleural fluids (lung adenocarcinoma cell line) that constitutively expresses cyclooxygenase 2 (COX-2) and all types of prostaglandin receptors was studied. It was first demonstrated using a matrigel system, that malignant pleural fluids increase the invasiveness of adenocarcinoma cells more than congestive heart failure (CHF) pleural fluids. Moreover, exposure to exudative malignant, but not to CHF pleural fluids, incr…
Pleural LTB4 upregulates ICAM-1 molecules via STAT-1 activation on pleural mesothelial cells
The release of PGE2 by pleural macrophage prolongs the survival of T lymphocytee within the inflamed human pleural space
Erythrocyte production in obstructive sleep apnea
Increased airway inflammatory cells in endurance athletes: what do they mean?
Inflammatory cells are increased in the airways of endurance athletes, but their role in causing exercise-induced respiratory symptoms and bronchoconstriction, or their possible long-term consequences, are uncertain.To put the results of athlete studies in perspective, by analysing the pathogenesis of airway cell changes and their impact on respiratory function.Athletes of different endurance sports at rest showed increased airway neutrophils. Elite swimmers and skiers also showed large increases in airway eosinophils and lymphocytes, possibly related to chronic, exercise-related exposure to irritants or cold and dry air, respectively. Post-exercise studies reported variable responses of ai…