0000000000057341

AUTHOR

Giuseppe Cannino

showing 31 related works from this author

Effects of cadmium chloride on some mitochondria-related activity and gene expression of human MDA-MB231 breast tumor cells.

2007

It was reported that cadmium is able to exert a cytotoxic effect on tumor MDA-MB231 cells, which shows signs of "non-classical" apoptosis and is characterized by drastic changes in gene expression pattern. In this study, we have extended our knowledge of metal-breast cancer cell interactions by analyzing some mitochondria-related aspects of the stress response to CdCl(2) at either 5 or 50 microM 24- or 96-h exposure, by cytochemical, conventional PCR and Northern/Western blot techniques. We demonstrated that (i) no modification of the mitochondrial mass was detectable due to CdCl(2) exposure; (ii) the respiration activity appeared to be increased after 96-h exposures, while the production o…

Breast cancer Cadmium MitochondriaAntineoplastic AgentsApoptosisBreast NeoplasmsMitochondrionCadmium chlorideBiochemistryElectron Transport Complex IVMitochondrial Proteinscadmium mitochondria breast tumor cellsInorganic Chemistrychemistry.chemical_compoundCadmium ChlorideWestern blotCell Line TumorGene expressionmedicineHumansCytochrome c oxidaseSettore BIO/06 - Anatomia Comparata E CitologiaHeat-Shock Proteinsbiologymedicine.diagnostic_testChemistryMolecular biologyMitochondriaOxidative StressGene Expression RegulationApoptosisbiology.proteinHSP60Reactive Oxygen SpeciesIntracellular
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Mitochondrial compartment: a possible target of cadmium effects on breast epithelial cells.

2009

Cadmium–breast epithelial cell interactions were studied by analyzing some mitochondria-related aspects of stress response. We treated immortalized non-tumor breast cells with 5 or 50 μM CdCl2 for 24 or 96 h demonstrating that the exposure did not cause a significant mitochondrial proliferation, while it induced a significant increase in the respiratory activity and mitochondrial polarization. In addition, we found that hsp60 was up-regulated while hsp70 and COXII and COXIV were down-regulated. The mRNA for hsp70 remained constant and only the inducible form of the 70-kDa heat shock protein was over expressed. The mRNAs for COXII and COXIV remained constant after 24 h and increased after lo…

Clinical chemistryCadmium - Mitochondria - Stress - Breast EpithelialClinical BiochemistryCell RespirationMitochondrionBiologyCell LineElectron Transport Complex IVHeat shock proteinmedicineHumansHSP70 Heat-Shock ProteinsBreastCytotoxicityMolecular BiologyMembrane Potential MitochondrialMessenger RNAMembranesDose-Response Relationship DrugEpithelial CellsCell BiologyGeneral MedicineChaperonin 60EpitheliumCell biologyHsp70Mitochondriamedicine.anatomical_structureGene Expression RegulationHSP60FemaleCadmium
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Nuclear-mitochondrial interaction.

2007

The biogenesis of mitochondria depends on the coordinated expression of nuclear and mitochondrial genomes. Consequently, the control of mitochondrial biogenesis and function depends on extremely complex processes requiring a variety of well orchestrated regulatory mechanisms. It is clear that the interplay of transcription factors and coactivators contributes to the expression of both nuclear and mitochondrial respiratory genes. In addition, the regulation of mitochondria biogenesis depends on proteins that, interacting with messenger RNAs for mitochondrial proteins, influence their metabolism and expression. Moreover, a tight regulation of the import and final assembly of mitochondrial pro…

Cell NucleusRNA-binding proteinRNA-binding proteinsCell BiologyCell CommunicationBiologyMitochondrionCell biologyEpigenesis GeneticMitochondriamitochondrial fusionMitochondrial biogenesisNeoplasmsMolecular MedicineAnimalsHumansMitochondrial fissionMolecular BiologyTranscription factorPost-transcriptional regulationBiogenesistranscriptional factorpost-transcriptional regulationTranscription FactorsMitochondrion
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Cadmium and mitochondria

2009

The heavy metal cadmium (Cd) a pollutant associated with several modern industrial processes, is absorbed in significant quantities from cigarette smoke, water, food and air contaminations. It is known to have numerous undesirable effects on health in both experimental animals and humans, targeting kidney, liver and vascular system. The molecular mechanism accounting for most of the biological effects of Cd are not well-understood and the toxicity targets are largely unidentified. The present review focuses on important recent advances about the effects of cadmium on mitochondria of mammalian cells. Mitochondria are the proverbial powerhouses of the cell, running the fundamental biochemical…

Cellchemistry.chemical_elementMitochondrionBiologyModels BiologicalmedicineAnimalsHumansSettore BIO/06 - Anatomia Comparata E CitologiaCytotoxicityMolecular BiologyMembrane potentialMammalsPollutantCadmiumMitochondrial gene expressionApoptosiROSCell BiologyMitochondriamedicine.anatomical_structurechemistryBiochemistryApoptosisToxicityMolecular MedicineEnergy MetabolismIntracellularInner membrane ion permeabilityCadmiumMitochondrion
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Correction: DAPIT Over-Expression Modulates Glucose Metabolism and Cell Behaviour in HEK293T Cells

2015

Introduction Diabetes Associated Protein in Insulin-sensitive Tissues (DAPIT) is a subunit of mitochondrial ATP synthase and has also been found to associate with the vacuolar H+-ATPase. Its expression is particularly high in cells with elevated aerobic metabolism and in epithelial cells that actively transport nutrients and ions. Deletion of DAPIT is known to induce loss of mitochondrial ATP synthase but the effects of its over-expression are obscure. Results In order to study the consequences of high expression of DAPIT, we constructed a transgenic cell line that constitutively expressed DAPIT in human embryonal kidney cells, HEK293T. Enhanced DAPIT expression decreased mtDNA content and …

Epithelial-Mesenchymal Transitionmitochondrial metabolismBiolääketieteet - BiomedicineCellActive Transport Cell NucleusGene DosageRespiratory chainlcsh:MedicineGene ExpressionMitochondrionta3111glukoosiNeoplasmsmedicineHumansLactic Acidglucoselcsh:ScienceTranscription factorMultidisciplinaryATP synthasebiologyCell growthta1184lcsh:RHEK 293 cellsCorrectionMitochondrial Proton-Translocating ATPasesMitochondriaCell biologyHEK293 CellsDiabetes Associated Protein in Insulin-sensitive Tissuesmedicine.anatomical_structureCell culturebiology.proteinATP synthaselcsh:QResearch ArticlePLOS ONE
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Proteins participating to the post-transcriptional regulation of the mitochondrial cytochrome c oxidase subunit IV via elements located in the 3′UTR

2009

Abstract In developing rat brain cytochrome c oxidase subunit IV (COXIV) expression is also regulated at post-transcriptional level and two 3′UTR-COXIV RNA-binding factors have been identified. Here, we report the enrichment and identification of the factors from just born rat brains by affinity chromatography of biotinylated 3′UTR-COXIV RNA–protein complexes on streptavidin-conjugated paramagnetic particles. We successfully isolated two main proteins of about 86 and 42 kDa, whose sequences were highly attributable to Hsp90 and Actin. The purified proteins maintain RNA-binding ability and specificity for COXIV messenger and, interacting with the 3′UTR, then could negatively modulate mRNA tr…

Protein subunitRNA-binding proteinMitochondrionChromatography AffinityElectron Transport Complex IVMitochondrial ProteinsRats Sprague-DawleySequence Analysis ProteinSerineAnimalsCytochrome c oxidaseHSP90 Heat-Shock ProteinsPhosphorylationPost-transcriptional regulation RNA-binding proteins Mitochondria Cytochrome c oxidase COXIV 3'UTR3' Untranslated RegionsMolecular BiologyPost-transcriptional regulationMessenger RNAbiologyThree prime untranslated regionBrainRNA-Binding ProteinsTranslation (biology)Cell BiologyActinsRatsMolecular WeightAnimals NewbornGene Expression RegulationBiochemistrybiology.proteinMolecular MedicineProtein BindingMitochondrion
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Analysis of cytochrome C oxidase subunits III and IV expression in developing rat brain

2004

Abstract Cytochrome c oxidase (COX) complex is built up with both nucleus- and mitochondrion-encoded subunits. Biogenesis and assembly of the complex thus requires fine cross-talk between the two compartments. In order to shed light on the regulation of nuclear–mitochondrial interactions, we studied the expression of COXIII (mitochondrion-encoded) and COXIV (nucleus-encoded) in adult rat tissues and rat developing brain. We found that the levels of COXIV protein and mRNA are not linearly related, thus suggesting a post-transcriptional mode of regulation. In agreement with this observation, we report the presence of a protein that specifically binds to the 3′-untranslated region of COXIV mRN…

CytoplasmRNA-binding proteinProtein subunitBlotting WesternCOX IVRNA-binding proteinMitochondrionBiologyGene Expression Regulation EnzymologicElectron Transport Complex IVAnimalsCytochrome c oxidaseElectrophoresis Gel Two-DimensionalCOX III.RNA MessengerRNA Processing Post-TranscriptionalMessenger RNAGeneral NeuroscienceBrainProteinsRNABlotting NorthernMitochondriaRatsProtein TransportCytosolnucleus-mitochondrion cross-talkBiochemistryCytoplasmbiology.proteinNeuroscience
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Purification by affinity chromatography of H1 RNA-Binding Proteins from rat brain

2003

Post-transcriptional regulation of mRNA metabolism is involved in processes as different as cell fate specification in development and cell response to a large variety of environmental cues. Regulation of all steps of RNA metabolism depends on RNA-binding proteins (RBPs). By using a T1 RNase protection assay, we previously identified three H1° RNA-binding factors (p40, p70 and p110), highly expressed in the rat brain. Here we report enrichment of these factors from brain extracts, obtained by affinity chromatography of biotinylated H1° RNA-protein complexes on streptavidin-conjugated paramagnetic particles. The purified proteins maintain RNA-binding ability and preference for histone messag…

biologyCellRNA-binding proteinGeneral MedicineCell cycleCell fate determinationMolecular biologymedicine.anatomical_structureHistoneBiochemistryAffinity chromatographyBiotinylationGeneticsmedicinebiology.proteinrat brain developing brain RNA-binding factors histone variants RNA affinity chromatography streptavidin conjugated paramagnetic particlesGene
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RNA binding proteins: putative regulative role in mitochondrial proteins expression.

2006

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Mitochondrial protein expression in Ratt and in human cells

2006

In the present paper, we analyzed some aspects of the post-transcriptional regulation of two COX (cy-tochrome c oxidase) subunits, i.e. mitochondrion-encoded COXIII and nucleus-encoded COXIV. In particular, by T1 RNase protection assays, we found two proteins, present in mitochondrial extracts from adult rat brain and testis, able to bind COXIII mRNA. We also found cytoplasmic proteins present in testis, kidney and heart extracts that bind COXIV mRNA. Moreover, to study the expression of mitochondrial proteins in tumor cells, we quantitated COXIV and Hsp60 chaperonin abundance in two epithelial cell lines from human breast, one neoplastic (MDA-MB231) and the other immortalized and non-tumor…

mitochondria
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Mitochondrial proteins regulation in Rattus norvegicus and human cells

2006

mitochondrial proteins rattus norvegicus human cellsSettore BIO/06 - Anatomia Comparata E Citologia
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Possible mechanisms regulating the expression of nuclear and mitochondrial genes

2007

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Cytochemical and molecular analyses on mitochondria of immortalized and neoplastic epithelial cells of the human breast after cadmium treatments

2008

Settore BIO/06 - Anatomia Comparata E Citologiamitochondria neoplastic cells human breast cadmium
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Possible mechanisms regulating the expression of nuclear and mitochondrial genes

2008

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Mitochondrial protein expression in rattus norvegicus and human cells.

2005

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Effect of cadmium chloride on some mitochondria-related activity and gene expression of human MDA-MB231 breast tumor cells

2008

Settore BIO/06 - Anatomia Comparata E Citologiacadmium breast cancer
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Regolazione dell’espressione delle subunità III e IV della citocromo c ossidasi

2007

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Proteine di fusione, metodo di preparazione, e relativi anticorpi per la diagnosi di patologie mitocondriali

2004

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Cytochemical and molecular analyses on mitochondria of immortalized and neoplastic epithelial cells from the human breast

2007

Settore BIO/06 - Anatomia Comparata E Citologiabreast cancer mitochondria
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STUDIO DELL’ESPRESSIONE DI GENI CODIFICANTI PROTEINE MITOCONDRIALI IN RATTUS NORVEGICUS.

2004

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Mitochondria and Development

2005

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Analysis of cytochrome oxidase subunits III and IV expression in debveloping rat brain.

2005

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Effetto del cadmio sull'attività mitocondriale e sull'espressione genica in cellule tumorali e immortalizzatedi epitelio ghiandolare mammario

2007

cadmium mitochondria tumor cells
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Analysis of cytochrome c oxidase subunits III and IV in developing rat brain

2004

COXIVSettore BIO/10 - BiochimicaCOXIIIRNA-binding proteinsSettore BIO/06 - Anatomia Comparata E Citologianucleus-mitochondrion crosstalk
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Analysis of cytochrome oxidase subunits III and IV expression in developing rat brain.

2005

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Analysis of cytochrome c oxidase subunits III and IV expression in developing rat brain

2004

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Espressione del messaggero per l’Hsp56 in embrioni di Paracentrotus lividus

2007

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Mitochondrial protein expression in rattus norvegicus and human cells

2006

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Mitochondrial protein expression in rat and human cells.

2006

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Possible meccanisms regulating the expression of nuclear and mitochondrial genes

2007

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Effect of cadmium on mitochondria-related activity and gene expression in tumoral and immortalized human breast cells

2007

cadmium mitochondria cancer cells
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