0000000000343355

AUTHOR

Sergio Portal-núñez

0000-0003-0659-1545

showing 3 related works from this author

Oxidative stress, autophagy, epigenetic changes and regulation by miRNAs as potential therapeutic targets in osteoarthritis

2015

Aging is a natural process characterized by the declining ability of the different organs and tissues to respond to stress, increasing homeostatic imbalance and risk of disease. Osteoarthritis (OA) is a multifactorial disease in which cartilage degradation is a central feature. Aging is the main risk factor for OA. In OA cartilage, a decrease in the number of chondrocytes and in their ability to regenerate the extracellular matrix and adequately respond to stress has been described. OA chondrocytes show a senescence secretory phenotype (SSP) consisting on the overproduction of cytokines (interleukins 1 and 6), growth factors (e.g., epidermal growth factor) and matrix metalloproteinases (MMP…

0301 basic medicineSenescenceMAPK/ERK pathwayAgingProgrammed cell deathDNA damageBiologymedicine.disease_causeBiochemistryChondrocyteEpigenesis Genetic03 medical and health sciencesChondrocytesOsteoarthritisAutophagymedicineAnimalsHumansMolecular Targeted TherapyEpigeneticsCellular SenescencePharmacologyAutophagyDNA MethylationCell biologyMicroRNAsOxidative Stress030104 developmental biologymedicine.anatomical_structureImmunologyReactive Oxygen SpeciesOxidative stressDNA DamageBiochemical Pharmacology
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Adverse Effects of Diabetes Mellitus on the Skeleton of Aging Mice

2016

In the present study, the possibility that a diabetic (DM) status might worsen age-related bone deterioration was explored in mice. Male CD-1 mice aged 2 (young control group) or 16 months, nondiabetic or made diabetic by streptozotocin injections, were used. DM induced a decrease in bone volume, trabecular number, and eroded surface, and in mineral apposition and bone formation rates, but an increased trabecular separation, in L1-L3 vertebrae of aged mice. Three-point bending and reference point indentation tests showed slight changes pointing to increased frailty and brittleness in the mouse tibia of diabetic old mice. DM was related to a decreased expression of both vascular endothelial …

Male0301 basic medicineSenescenceAgingmedicine.medical_specialtyBone densityOsteoporosisDiabetes Mellitus ExperimentalBone remodelingMice03 medical and health scienceschemistry.chemical_compoundSDG 3 - Good Health and Well-beingBone DensityOsteogenesisDiabetes mellitusInternal medicineAnimalsMedicineLumbar Vertebraebusiness.industryStreptozotocinmedicine.diseaseVascular endothelial growth factor030104 developmental biologyEndocrinologymedicine.anatomical_structurechemistryOsteoporosisBone RemodelingBone marrowGeriatrics and GerontologyTomography X-Ray Computedbusinessmedicine.drugThe Journals of Gerontology Series A: Biological Sciences and Medical Sciences
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Oxidative stress in osteoarticular diseases

2016

chemistry.chemical_classificationReactive oxygen speciesbusiness.industryOsteoporosisSynovial hyperplasiaOsteoarthritismedicine.diseasemedicine.disease_causechemistry.chemical_compoundImmune systemchemistryRheumatoid arthritisImmunologyMedicinebusinessReactive nitrogen speciesOxidative stress
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