0000000001063260

AUTHOR

Francesco Prattichizzo

0000-0002-2959-2658

showing 2 related works from this author

Circulating miR-21, miR-146a and Fas ligand respond to postmenopausal estrogen-based hormone replacement therapy--a study with monozygotic twin pairs.

2014

Biological aging is associated with physiological deteriorations and its’ remodeling, which are partly due to changes in the hormonal profile. MicroRNAs are known to post-transcriptionally regulate various cellular processes associated with cell senescence; differentiation, replication and apoptosis. Measured from the serum, microRNAs have the potential to serve as noninvasive markers for diagnostics/prognostics and therapeutic targets. We analysed the association of estrogen-based hormone replacement therapy (HRT) with selected microRNAs and inflammation markers from the serum, leukocytes and muscle tissue biopsy samples obtained from 54-62 year-old postmenopausal monozygotic twins (n=11 p…

SenescenceAdultmedicine.medical_specialtyAgingFas Ligand Proteinmedicine.drug_classmedicine.medical_treatmentMonozygotic twinInflammationApoptosisBiologyta3111Fas ligand“Inflamm-aging”Internal medicinemicroRNAmedicineestrogenHumansmicrornasMuscle SkeletalHormone therapyCellular SenescenceInflammationmicroRNAEstrogen Replacement TherapyapoptosisHormone replacement therapy (menopause)ta3141Cell DifferentiationEstrogenstwinsTwins MonozygoticMiddle AgedPostmenopauseAgeinghormone replacement therapyMicroRNAsEndocrinologyEstrogenFemalemedicine.symptomBiomarkersDevelopmental BiologyHormoneMechanisms of ageing and development
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Cellular Senescence and Inflammaging in Age-Related Diseases

2018

0301 basic medicineAgingArticle SubjectImmunologyCellular senescenceCell BiologyBiologymedicine.disease_causeBioinformatics03 medical and health sciencesOxidative Stress030104 developmental biologyEditorialAge relatedmedicinelcsh:PathologyAnimalsHumansSettore MED/05 - Patologia ClinicaCellular Senescence Inflammaging Age-Related DiseasesOxidative stressCellular Senescencelcsh:RB1-214Mediators of Inflammation
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