6533b7d0fe1ef96bd125b8f2

RESEARCH PRODUCT

Reversible inhibition of C1Q release from guinea pig macrophages by 2,2'-dipyridyl: Evidence for a posttranslational hydroxylation step in the biosynthesis of C1Q, a subcomponent of the first component of complement (C1).

Hartmut Hanauske-abelWolfgang MüllerMichael Loos

subject

PyridinesMacrophagesGuinea PigsBiophysicsCell BiologyBiologyHydroxylationBiochemistryGuinea pigHydroxylationchemistry.chemical_compound22'-DipyridylBiochemistryBiosynthesischemistryStructural BiologyComplement C1GeneticsAnimalsReversible inhibitionMolecular BiologyCells Cultured10.1016/0014-5793(78)80372-xhttps://pubmed.ncbi.nlm.nih.gov/566678