Trombofīlijas patoģenēzē un folskābes metabolismā iesaistīto gēnu variantu ietekme uz embrija implantāciju un grūtniecības iznākumu

6533b7d7fe1ef96bd12692e2

RESEARCH PRODUCT

Trombofīlijas patoģenēzē un folskābes metabolismā iesaistīto gēnu variantu ietekme uz embrija implantāciju un grūtniecības iznākumu

subject

Hiperhomocisteinēmija Trombofīlija Neauglība Pārtraukusies grūtniecība Bioloģija

description

Trombofīlija ir nosliece uz pastiprinātu asins recēšanu. Tās rezultātā pacientam ir paaugstināts trombu veidošanās risks. Pastiprinātu asins recēšanu var izraisīt izmaiņas noteiktos gēnos. Visbiežāk sastopamās trombofīliju izraisošās gēnu izmaiņas ir V asinsreces faktora gēna (F5) variācijas - G1601A (rs6025), A4070G (rs1800595), un A5279G (rs118203907); viena nukleotīda nomaiņa G20210A F2 gēnā (rs1799963), SERPINE1 4G (rs1799889) homozigots variants un MTHFR gēna izmaiņas (rs1801133 un rs1801131). Minēto variantu klātbūtne palielina trombozes riskus, un šo genotipu nesējiem ir paaugstināta varbūtība nelabvēlīgai grūtniecības norisei (neveiksmīgai embrija implantācijai, grūtniecības pārtrūkšanai vai bērna augšanas aizturei grūtniecības laikā). Šajā pētījumā tika statistiski analizēta 7 visbiežāko ar trombofīliju un hiperhomocisteinēmiju saistīto gēnu variantu ietekme uz pacientu auglības stāvokli un lietotās terapijas efektivitāti. Pēc statistiskas analīzes tika spriests par pētāmo gēnu variantu ietekmi uz grūtniecības iestāšanos un norisi, kā arī uz hiperhomocisteinēmiju.

year	journal	country	edition	language
2017-01-01

https://dspace.lu.lv/dspace/handle/7/36207

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