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RESEARCH PRODUCT

Integrating psychological and neurobiological considerations regarding the development and maintenance of specific Internet-use disorders: An Interaction of Person-Affect-Cognition-Execution (I-PACE) model

Matthias BrandMatthias BrandChristian LaierKimberly S. YoungMarc N. PotenzaKlaus Wölfling

subject

Internet addictionProcess (engineering)Cognitive Neurosciencemedia_common.quotation_subjectCue-reactivityAffect (psychology)Executive functions03 medical and health sciencesBehavioral NeuroscienceCognition0302 clinical medicineddc:150Internet-use disordersHumansPornographySituational ethicsInhibitory controlInternet-gaming disordermedia_commonInternetbusiness.industryAddictionCognitionKognition [Fakultät für Ingenieurwissenschaften » Informatik und Angewandte Kognitionswissenschaft » Angewandte Kognitions- und Medienwissenschaft » Allgemeine Psychologie]Cognitive biasForschungszentren » Erwin L. Hahn Institute for Resonance Imaging030227 psychiatryBehavior AddictiveNeuropsychology and Physiological PsychologyVideo Gamesddc:15PsychologieGamblingThe InternetbusinessPsychologySocial psychology030217 neurology & neurosurgeryCognitive psychology

description

Within the last two decades, many studies have addressed the clinical phenomenon of Internet-use disorders, with a particular focus on Internet-gaming disorder. Based on previous theoretical considerations and empirical findings, we suggest an Interaction of Person-Affect-Cognition-Execution (I-PACE) model of specific Internet-use disorders. The I-PACE model is a theoretical framework for the processes underlying the development and maintenance of an addictive use of certain Internet applications or sites promoting gaming, gambling, pornography viewing, shopping, or communication. The model is composed as a process model. Specific Internet-use disorders are considered to be the consequence of interactions between predisposing factors, such as neurobiological and psychological constitutions, moderators, such as coping styles and Internet-related cognitive biases, and mediators, such as affective and cognitive responses to situational triggers in combination with reduced executive functioning. Conditioning processes may strengthen these associations within an addiction process. Although the hypotheses regarding the mechanisms underlying the development and maintenance of specific Internet-use disorders, summarized in the I-PACE model, must be further tested empirically, implications for treatment interventions are suggested. CA Brand

https://doi.org/10.1016/j.neubiorev.2016.08.033