Search results for " ACTIVATION"

showing 10 items of 1535 documents

iNOS-derived nitric oxide mediates the increase in TFF2 expression associated with gastric damage: role of HIF-1.

2009

Trefoil (TFF) peptides are involved in gastrointestinal mucosal restitution. An hypoxia inducible factor 1 (HIF-1)-dependent induction of TFF genes has been reported in gastric epithelial cells. Nitric oxide (NO) is associated with mucosal damage and modulates HIF-1 activity. The aim of the present study was to analyze the role of iNOS-derived NO in HIF-1alpha stabilization and TFF gene expression in damaged gastric mucosa. Aspirin caused gastric injury that peaked 6 h after dosing and returned to normality at 24 h. iNOS mRNA expression occurs in the corpus in parallel with damage. Blockade of iNOS activity did not modify gastric lesions induced by aspirin but delayed mucosal healing. Aspir…

MaleBenzylaminesAmidinesNitric Oxide Synthase Type IINitric OxideBiochemistryNitric oxideCell LineRats Sprague-Dawleychemistry.chemical_compoundMiceDownregulation and upregulationGene expressionGeneticsGastric mucosamedicineGene silencingAnimalsHumansRNA MessengerEnzyme InhibitorseducationMolecular BiologyDNA Primerseducation.field_of_studyWound HealingAspirinBase SequenceAnti-Inflammatory Agents Non-SteroidalTrefoil factor 2Macrophage ActivationHypoxia-Inducible Factor 1 alpha SubunitCoculture TechniquesRatsUp-RegulationMicroRNAsmedicine.anatomical_structurechemistryCell cultureGastric MucosaCancer researchTrefoil Factor-2Wound healingPeptidesBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Metronomic cyclophosphamide induces regulatory T cells depletion and PSA‐specific T cells reactivation in patients with biochemical recurrent prostat…

2019

Biochemical recurrence (BCR) occurs in up to 40% of prostate cancer patients after prostatectomy. In our study, we performed an immune monitoring study in 20 prostate cancer patients with BCR previously treated with metronomic cyclophosphamide (mCTX). We observed a decrease of regulatory T cells (Tregs) from 2 months and this was more pronounced after 6 months of mCTX treatment. This drop of Tregs was associated with increased level of activated HLADR+ CD45R0+ T cells in peripheral blood. Furthermore, a reactivation of Th1 polarized anti-PSA T-cell response was detected in BCR patients treated with mCTX. However, dendritic cell subsets counts and activation were not influenced by the treatm…

MaleBiochemical recurrenceCancer ResearchT-Lymphocytesmedicine.medical_treatmentLymphocyte ActivationT-Lymphocytes RegulatoryLymphocyte DepletionAndrogen deprivation therapy03 medical and health sciencesProstate cancer0302 clinical medicinemedicineHumansAntineoplastic Agents AlkylatingCyclophosphamideMetronomic cyclophosphamideDiminutionProstatectomybusiness.industrybreakpoint cluster regionProstatic NeoplasmsDendritic cellProstate-Specific Antigenmedicine.diseaseOncology030220 oncology & carcinogenesisAdministration MetronomicCancer researchNeoplasm Recurrence LocalbusinessInternational Journal of Cancer
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Immune activation rapidly mirrored in a secondary sexual trait.

2003

A crucial assumption underlying most models of sexual selection is that sexual advertisements honestly reflect the phenotypic and/or genetic quality of their bearers ([1][1]). Here we show that experimental activation of the immune system is rapidly mirrored in the expression of a carotenoid-based

MaleBiologyXanthophyllsSongbirdsSexual Behavior AnimalImmune systemZeaxanthinsAnimalsCryptoxanthinsGeneticsMultidisciplinaryEcologyPigmentationLuteinBeakHemagglutination Testsbeta CarotenePhenotypeCarotenoidsMate choiceSexual selectionImmune SystemAntibody FormationTraitFemaleImmunizationImmune activationScience (New York, N.Y.)
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Error Detection and Response Adjustment in Youth With Mild Spastic Cerebral Palsy

2013

This study evaluated the brain activation state during error making in youth with mild spastic cerebral palsy and a peer control group while carrying out a stimulus recognition task. The key question was whether patients were detecting their own errors and subsequently improving their performance in a future trial. Findings indicated that error responses of the group with cerebral palsy were associated with weak motor preparation, as indexed by the amplitude of the late contingent negative variation. However, patients were detecting their errors as indexed by the amplitude of the response-locked negativity and thus improved their performance in a future trial. Findings suggest that the con…

MaleBrain activationSelf-Assessmentmedicine.medical_specialtyAdolescentevent-related brain potentialsContingent Negative VariationStimulus (physiology)Cerebral palsyExecutive FunctionSpastic cerebral palsyPhysical medicine and rehabilitationerror detectionReaction TimeSpasticmedicineHumansAttentionspasticChildta515Cerebral CortexLearning DisabilitiesCerebral PalsyElectroencephalographySignal Processing Computer-AssistedNegativity effectAwarenessmedicine.diseaseContingent negative variationMemory Short-TermPattern Recognition VisualPediatrics Perinatology and Child HealthPhysical therapyFemaleNeurology (clinical)PsychologyError detection and correctionJournal of Child Neurology
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Xid-defective male (CBA/N x C57BL/6)F1 accessory cells present bovine insulin to long-term cultured F1-restricted T-cells

1982

The reactivity of H-2b-restricted murine T cells towards bovine insulin was reported to depend on the expression of Ia. W39, a private specificity of I-Ab, on antigen-presenting cells. Cells of male (CBA/N X B6)F1 mice carrying the mutation xid on the X chromosome lack IA. W39 on the cell surface. These cells are unable to present bovine insulin to primed T cells derived from female (CBA/N X B6)F1 mice. We show here that spleen cells of male (CBA/N X B6)F1 hybrids served perfectly as accessory cells for the insulin-dependent induction of a proliferative response of long-term cultured T cells with (B10 X B10.BR)F1 genotype, restricted to recognizing insulin in the context of F1-unique I-A de…

MaleC57BL/6Time FactorsT-Lymphocytesmedicine.medical_treatmentGenes MHC Class IILymphocyte CooperationImmunologyCellRats Inbred WFSpleenContext (language use)BiologyLymphocyte ActivationEpitopeMiceGeneticsmedicineAnimalsInsulinCells CulturedCrosses GeneticMHC class IIInsulinGlutamic acidbiology.organism_classificationMolecular biologyRatsMice Inbred C57BLmedicine.anatomical_structureBiochemistryMice Inbred CBAbiology.proteinCattleFemaleImmunogenetics
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The associated expression of Maspin and Bax proteins as a potential prognostic factor in intrahepatic cholangiocarcinoma

2006

Abstract Background Maspin, a member of the serpin family, is a suppressor of tumor growth, an inhibitor of angiogenesis and an inducer of apoptosis. Maspin induces apoptosis by increasing Bax, a member of the Bcl-2 family of apoptosis-regulating proteins. In this exploratory study, we investigated the associated expression of Maspin and Bax proteins as a potential prognostic factor in intrahepatic cholangiocarcinoma (IHCCA). Methods Twenty-two paraffin-embedded samples were analyzed by immunohistochemical methods using Maspin, Bax and CD34 antibodies. Maspin was scored semiquantitatively (HSCORE). Apoptosis was assessed using an antibody against cleaved caspase-3. Results The strong relati…

MaleCancer ResearchPathologymedicine.medical_specialtyAngiogenesisApoptosisCaspase 3Kaplan-Meier EstimateSerpinlcsh:RC254-282law.inventionCholangiocarcinomaBcl-2-associated X proteinlawBiomarkers TumorGeneticsmedicineHumansGenes Tumor SuppressorSerpinsIntrahepatic CholangiocarcinomaAgedbcl-2-Associated X ProteinbiologyCaspase 3business.industryMicrocirculationLiver NeoplasmsMaspinMiddle AgedPrognosislcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensNeoplasm ProteinsEnzyme ActivationOncologyApoptosisCancer researchbiology.proteinSuppressorFemalebusinessResearch ArticleBMC Cancer
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Vascular imaging of solid tumors in rats with a radioactive arsenic-labeled antibody that binds exposed phosphatidylserine.

2008

Abstract Purpose: We recently reported that anionic phospholipids, principally phosphatidylserine, become exposed on the external surface of vascular endothelial cells in tumors, probably in response to oxidative stresses present in the tumor microenvironment. In the present study, we tested the hypothesis that a chimeric monoclonal antibody that binds phosphatidylserine could be labeled with radioactive arsenic isotopes and used for molecular imaging of solid tumors in rats. Experimental Design: Bavituximab was labeled with 74As (β+, T1/2 17.8 days) or 77As (β−, T1/2 1.6 days) using a novel procedure. The radionuclides of arsenic were selected because their long half-lives are consistent w…

MaleCancer ResearchPathologymedicine.medical_specialtyBiodistributionBavituximabmedicine.drug_classPhosphatidylserinesMonoclonal antibodyArticleArsenicchemistry.chemical_compoundIn vivomedicineTumor Cells CulturedAnimalsTissue DistributionPlatelet activationRadioisotopesTumor microenvironmentbiologyNeovascularization PathologicAntibodies MonoclonalProstatic NeoplasmsPhosphatidylserineMolecular biologyXenograft Model Antitumor AssaysRatsOncologychemistryRadioimmunodetectionPositron-Emission Tomographybiology.proteinEndothelium VascularAntibodymedicine.drugClinical cancer research : an official journal of the American Association for Cancer Research
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Up-regulation of c-FLIPshort and reduction of activation-induced cell death in T-cells from patients with Type 1 diabetes

2004

AICD of T-cells is an efficient way of removing activated T-lymphocytes. In this study we investigated the molecular basis of AICD upon reactivation in peripheral T-lymphocytes from newly diagnosed T1DM patients and age-matched healthy controls. In an in vitro model system, PHA-stimulated T-cells, upon prolonged culture in IL-2, acquire a sensitive phenotype to Fas-mediated apoptosis. This phenomenon is less pronounced in T1DM T-cells. Moreover, the restimulation of activated T-cells via TCR/CD3 and/or via CD28 inhibits Fas-mediated apoptosis in T1DM in comparison to control T-cells. After Fas triggering, the generation of the active sub-units of caspase-8 is significantly reduced in T1DM T…

MaleCaspase 8Adolescenttype 1 diabetesT-LymphocytesCASP8 and FADD-Like Apoptosis Regulating ProteinIntracellular Signaling Peptides and ProteinsApoptosisLymphocyte ActivationCaspase InhibitorsSettore MED/13 - EndocrinologiaUp-RegulationDiabetes Mellitus Type 1CD28 AntigensReceptor-CD3 Complex Antigen T-CellCase-Control StudiesCaspasesHumansFemaleCarrier Proteins
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Long-term pertussis-specific immune responses to a combined diphtheria, tetanus, tricomponent acellular pertussis and hepatitis B vaccine in pre-term…

2002

Abstract Immunoglobulin G (IgG) antibody titres against pertussis antigens, Bordetella pertussis-specific proliferation and cytokine production by peripheral blood mononuclear cells (PBMCs) were evaluated at the age of 5–6 years in 13 children who had been pre-term infants with a gestational age (GA) of ≤31 weeks, 10 who had been pre-term infants with a GA of 32–37 weeks, and 15 who had been term infants with a GA of 38–42 weeks. All of the infants had been immunised with a combined diphtheria, tetanus, tricomponent acellular pertussis and hepatitis B vaccine (DTaP–HBV) at 3, 5, and 11 months of post-natal age. Our results show that the long-term immune responses induced by primary pertussi…

MaleCellular immunityBordetella pertussisTime FactorsHepatitis B vaccineWhooping CoughImmunization SecondaryIn Vitro TechniquesLymphocyte ActivationBordetella pertussisPertussismedicineHumansHepatitis B VaccinesChildDiphtheria-Tetanus-Pertussis VaccineImmunization ScheduleWhooping coughHaemophilus VaccinesImmunity CellularPre-term infantsGeneral VeterinaryGeneral Immunology and MicrobiologybiologyTetanusbusiness.industryDiphtheriaDTaP-HBV vaccine; Pertussis; Pre-term infantsAge FactorsInfant NewbornPublic Health Environmental and Occupational Healthmedicine.diseasebiology.organism_classificationAntibodies BacterialVaccinationInfectious DiseasesCase-Control StudiesChild PreschoolImmunoglobulin GImmunologybiology.proteinDTaP-HBV vaccineCytokinesMolecular MedicineFemaleAntibodybusinessInfant PrematureVaccine
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Extracellular Vesicles from Hyperammonemic Rats Induce Neuroinflammation and Motor Incoordination in Control Rats.

2020

Minimal hepatic encephalopathy is associated with changes in the peripheral immune system which are transferred to the brain, leading to neuroinflammation and thus to cognitive and motor impairment. Mechanisms by which changes in the immune system induce cerebral alterations remain unclear. Extracellular vesicles (EVs) seem to play a role in this process in certain pathologies. The aim of this work was to assess whether EVs play a role in the induction of neuroinflammation in cerebellum and motor incoordination by chronic hyperammonemia. We characterized the differences in protein cargo of EVs from plasma of hyperammonemic and control rats by proteomics and Western blot. We assessed whether…

MaleCerebellumtnfαhepatic encephalopathyArticleExtracellular VesiclesImmune systemWestern blotmedicineAnimalsHumansHyperammonemiaRats WistarReceptorlcsh:QH301-705.5NeuroinflammationInflammationMicrogliamedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaHyperammonemiaGeneral Medicinetnfα receptor tnfr1medicine.diseaseRatsMotor Skills DisordersDisease Models Animalmedicine.anatomical_structurelcsh:Biology (General)glial activationTumor necrosis factor alphaNervous System DiseasesTNF alpha receptor TNFR1businessNeuroscienceTNF alpha
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