Search results for " ALPHA"

showing 10 items of 1610 documents

A questionnaire to assess parental perception of barriers towards active commuting to school (PABACS): Reliability and validity

2019

Abstract Introduction Parental barriers are a key factor to determine physical activity behaviors, such as active commuting to school in young people. The purpose of this study was to test the reliability and validity of a questionnaire which addresses parental barriers towards active commuting to school. Methods This paper describes the development, following the Delphi method, and the validation of a Likert-scale questionnaire of 23 items (Parental Perception of Barriers towards Active Commuting to School (PABACS)) designed to determine the main parental barriers. These barriers are categorized into 3 scales (general, walking, and cycling barriers) and the overall questionnaire. A total o…

Predictive validityHealth PolicyPublic Health Environmental and Occupational HealthDelphi methodHuman factors and ergonomicsPoison control030209 endocrinology & metabolismTransportationPollutionTest (assessment)03 medical and health sciences0302 clinical medicineCronbach's alphaWalkability030212 general & internal medicineSafety Risk Reliability and QualityPsychologySafety ResearchReliability (statistics)Clinical psychologyJournal of Transport & Health
researchProduct

Refinement and validation of a comprehensive scale for measuring HR practices aimed at performance-enhancement and employee-support

2019

Abstract The purpose of this paper is to refine and validate a Human Resource practices (HRP) scale to measure employees' perceptions and test a two-tier model structured in eight practices and two bundles. In a sample of 554 employees, an EFA (Exploratory Factor Analysis) offered six factors that explained about 70% of the variance. Then, with 1647 employees (from 41 Spanish organizations), first- and second-order models were tested with Confirmatory Factor Analysis (CFA). The former encompasses eight practices. The latter grouped the practices in two bundles, one on enhancing performance and the other on supporting employees. The Cronbach's alpha, Rho coefficient (Composite Reliability Co…

Predictive validityIMPACTSATISFACTIONWORK SYSTEMSStrategy and ManagementApplied psychologyBEHAVIORSHRM assessmentBUNDLESCronbach's alpha0502 economics and businessATTITUDESHUMAN-RESOURCE MANAGEMENTOUTCOMES05 social sciencesScale validationVariance (accounting)ReliabilityConfirmatory factor analysisExploratory factor analysisHuman resource managementScale (social sciences)050211 marketingHuman Resource PracticesPsychologyWork systems050203 business & managementEuropean Management Journal
researchProduct

Some Decision Results on Nonrepetitive Words

1985

The paper addresses some generalizations of the Thue Problem such as: given a word u, does there exist an infinite nonrepetitive overlap free (or square free) word having u as a prefix? A solution to this as well as to related problems is given for the case of overlap free words on a binary alphabet.

PrefixCombinatoricsTheoryofComputation_MATHEMATICALLOGICANDFORMALLANGUAGESComputer Science::Discrete MathematicsUnique factorization domainComputer Science::Computation and Language (Computational Linguistics and Natural Language and Speech Processing)Square-free integerComputer Science::Formal Languages and Automata TheoryBinary alphabetWord (computer architecture)Mathematics
researchProduct

Regulation of tumour cell sensitivity to TNF-induced oxidative stress and cytotoxicity: Role of glutathione

1998

Glutathione (GSH) and the rate of cellular proliferation determine tumour cell sensitivity to tumour necrosis factor (TNF). Buthionine sulphoximine (BSO), a selective inhibitor of GSH synthesis, inhibits tumour growth and increases recombinant human TNF (rhTNF)-alpha cytoxicity in vitro. Administration of sublethal doses of rhTNF-alpha to Ehrlich ascites-tumour (EAT)-bearing mice induces oxidative stress (as measured by increases in intracellular peroxide levels, O2.- generation and mitochondrial GSSG). ATP-induced selective GSH depletion, when combined with rhTNF-alpha administration, affords a 61% inhibition of tumour growth and results in a significant extent of host survival. Administra…

Programmed cell deathCell SurvivalClinical BiochemistryMitochondrionPharmacologyBiologymedicine.disease_causeBiochemistryMicechemistry.chemical_compoundSuperoxidesmedicineAnimalsHumansCarcinoma Ehrlich TumorGlutathione DisulfideTumor Necrosis Factor-alphaGeneral MedicineGlutathioneGlutathioneRecombinant ProteinsOxidative StresschemistryBiochemistryCancer cellMolecular MedicineGlutathione disulfideTumor necrosis factor alphaOxidative stressIntracellularBioFactors
researchProduct

Cell death and hepatocarcinogenesis: Dysregulation of apoptosis signaling pathways

2011

Hepatocellular carcinoma (HCC) remains a disease with a poor prognosis despite recent advances in the pathophysiology and treatment. Although the disease is biologically heterogeneous, dysregulation of cellular proliferation and apoptosis both occur frequently and contribute to the malignant phenotype. Chronic liver disease is associated with intrahepatic inflammation which promotes dysregulation of cellular signaling pathways; this triggers proliferation and thus lays the ground for expansion of premalignant cells. Cancer emerges when immunological control fails and transformed cells develop resistance against cell death signaling pathways. The same mechanisms underlie the poor responsiven…

Programmed cell deathCell signalingHepatologybiologybusiness.industryGastroenterologyFas receptorCell biologyApoptosisbiology.proteinMedicineTumor necrosis factor alphaFADDSignal transductionbusinessTranscription factorJournal of Gastroenterology and Hepatology
researchProduct

Lysosomal degradation of the carboxydextran shell of coated superparamagnetic iron oxide nanoparticles and the fate of professional phagocytes

2010

Contrast agents based on dextran-coated superparamagnetic iron oxide nanoparticles (SPIO) are internalized by professional phagocytes such as hepatic Kupffer cells, yet their role in phagocyte biology remains largely unknown. Here we investigated the effects of the SPIO ferucarbotran on murine Kupffer cells and human macrophages. Intravenous injection of ferucarbotran into mice led to rapid accumulation of the particles in phagocytes and to long-lasting increased iron deposition in liver and kidneys. Macrophages incorporate ferucarbotran in lysosomal vesicles containing α-glucosidase, which is capable of degrading the carboxydextran shell of the ferucarbotran particles. Intravenous injectio…

Programmed cell deathMaterials sciencePhagocyteKupffer Cellsmedicine.medical_treatmentIntracellular SpaceBiophysicsApoptosisBioengineeringProinflammatory cytokineBiomaterialsMiceEdaravonemedicineAnimalsHumansMacrophageMagnetite Nanoparticleschemistry.chemical_classificationPhagocytesReactive oxygen speciesTumor Necrosis Factor-alphaDextransFree Radical ScavengersMagnetic Resonance ImagingCell biologyKineticsmedicine.anatomical_structureCytokineLiverchemistryBiochemistryMechanics of MaterialsApoptosisCeramics and CompositesNanoparticlesTumor necrosis factor alphaLysosomesReactive Oxygen SpeciesAntipyrineBiomaterials
researchProduct

Hypoxia inducible factor-1alpha inactivation unveils a link between tumor cell metabolism and hypoxia-induced cell death.

2008

Hypoxia and the acquisition of a glycolytic phenotype are intrinsic features of the tumor microenvironment. The hypoxia inducible factor-1alpha (HIF-1alpha) pathway is activated under hypoxic conditions and orchestrates a complex transcriptional program that enhances cell survival. Although the consequences of HIF-1alpha inactivation in cancer cells have been widely investigated, only a few studies have addressed the role of HIF-1alpha in the survival of cancer cells endowed with different glycolytic capacities. In this study, we investigated this aspect in ovarian cancer cells. Hypoxia-induced toxicity was increased in highly glycolytic cells compared with poorly glycolytic cells; it was a…

Programmed cell deathMice SCIDBiologyPathology and Forensic MedicineMiceCell Line TumormedicineAnimalsHumansGene SilencingRNA Small InterferingCell ProliferationOvarian NeoplasmsTumor microenvironmentCell DeathCell growthLentivirusHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitCell HypoxiaCell biologyPhenotypeHypoxia-inducible factorsApoptosisCell cultureCancer cellFemalemedicine.symptomRegular ArticlesThe American journal of pathology
researchProduct

Apoptosis of T cells and the control of inflammatory bowel disease: therapeutic implications.

2007

Inflammatory bowel diseases (IBDs) such as Crohn’s disease and ulcerative colitis are the result of an imbalanced mucosal T cell response. Despite the identification of a genetic susceptibility region in the NOD2/CARD15 (nucleotide-binding oligomerisation domain 2/caspase recruitment domain 15) gene, the aetiology is still unclear. Thus, the hunt for disease-initiating factors such as defects in the mucosal barrier or pathogenic microorganisms is ongoing. By contrast, the immunopathogenesis in IBDs is better understood. The identification of cytokines that are involved in T cell and monocyte signalling led to specific therapeutic concepts. Recent data have clearly shown that the most powerf…

Programmed cell deathNecrosisCell Survivalmedicine.medical_treatmentT cellT-LymphocytesApoptosisImmune systemCrohn DiseaseNOD2AzathioprinemedicineHumansIntestinal MucosaMesalamineImmunity Mucosalbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaAnti-Inflammatory Agents Non-SteroidalGastroenterologyRecent Advances in Basic ScienceInflammatory Bowel DiseasesInterleukin-12Immunosuppressive drugmedicine.anatomical_structureApoptosisImmunologyTumor necrosis factor alphamedicine.symptombusinessImmunosuppressive AgentsSignal TransductionGut
researchProduct

Caspase-8 regulates TNF-alpha induced epithelial necroptosis and terminal ileitis

2011

Two groups identify the regulation of death-receptor-induced necroptosis as an epithelial intrinsic mechanism that is important for the maintenance of immune homeostasis and the prevention of intestinal inflammation in mice. Welz et al. describe an unexpected physiological function for FADD (Fas-associated protein with death domain), an adaptor protein required for death-receptor-induced apoptosis. Mice with intestinal epithelial specific knockout of FADD develop severe colon inflammation due to increased death of FADD-deficient colonic epithelial cells. Gunther et al. report a novel and unexpected function of caspase-8 in maintaining immune homeostasis in the gut. Caspase-8 expression by g…

Programmed cell deathPaneth CellsNecroptosisInflammationApoptosisBiologyIn Vitro Techniquesdigestive systemArticle03 medical and health sciencesMiceNecrosis0302 clinical medicineCrohn DiseasemedicineAnimalsHumansFADD030304 developmental biology0303 health sciencesCaspase 8MultidisciplinaryInnate immune systemTumor Necrosis Factor-alphaColitisIntestinal epithelium3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesPaneth cellImmunologybiology.proteinCancer researchTumor necrosis factor alphaGoblet Cellsmedicine.symptomGene DeletionNature
researchProduct

Selective targeting of activated T cells in chronic intestinal inflammation

2009

Programmed cell death (apoptosis) has been implicated in normal biological processes as well as in the pathology of human diseases.1 The characterisation of genes involved in apoptosis has been pursued intensively and led to the identification of two major classes of genes: the bcl-2 family and the caspase family. Caspases are proteases that cleave their target substrates at specific peptide sequences and during apoptosis the activation of caspases takes place in a cascade fashion, leading to nuclear engulfment and cell death. Thus, caspases represent key functional components of the apoptosis pathway in human cells. Resistance against apoptosis is a key phenomenon in various chronic inflam…

Programmed cell deathRecombinant Fusion ProteinsT-LymphocytesT cellApoptosisLymphocyte ActivationProinflammatory cytokineImmune systemmedicineAnimalsHumansIntestinal MucosaCaspasebiologyCaspase 3Intrinsic apoptosisGastroenterologyColitisCell biologymedicine.anatomical_structureApoptosisChronic DiseaseModels Animalbiology.proteinInterleukin-2Tumor necrosis factor alphaGut
researchProduct