Search results for " Adhesion"

showing 10 items of 980 documents

Visualizing Leukocyte Rolling and Adhesion in Angiotensin II-Infused Mice: Techniques and Pitfalls

2018

Epifluorescence intravital video microscopy (IVM) of blood vessels is an established method to evaluate the activation of immune cells and their ability to role and adhere to the endothelial layer. Visualization of circulating cells by injection of fluorescent dyes or fluorophore-coupled antibodies is commonly used. Alternatively, fluorescent reporter mice can be used. Interactions of leukocytes, in particular lysozyme M+ (LysM+) monocytes, with the vessel wall play pivotal roles in promoting vascular dysfunction and arterial hypertension. We here present the technique to visualize and quantify leukocyte rolling and adhesion in carotid arteries in angiotensin II (AngII)-induced hypertension…

0301 basic medicineMaleEndotheliumendotheliumGeneral Chemical EngineeringImmunologyLeukocyte RollingMice TransgenicMonocytesGeneral Biochemistry Genetics and Molecular BiologyGreen fluorescent protein03 medical and health scienceschemistry.chemical_compoundMiceintravital microscopymedicineacridine orangeCell AdhesionLeukocytesAnimalsLeukocyte RollingCell adhesionGeneral Immunology and Microbiologycarotid arteryAngiotensin IIGeneral NeuroscienceAcridine orangeAngiotensin IICell biologyIssue 131030104 developmental biologymedicine.anatomical_structureCarotid ArterieschemistryHypertensioncardiovascular systemdouble-fluorescent Cre reporter mouseCell activationIntravital microscopyJournal of Visualized Experiments
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Gut Microbiota Restricts NETosis in Acute Mesenteric Ischemia-Reperfusion Injury.

2020

Objective: Recruitment of neutrophils and formation of neutrophil extracellular traps (NETs) contribute to lethality in acute mesenteric infarction. To study the impact of the gut microbiota in acute mesenteric infarction, we used gnotobiotic mouse models to investigate whether gut commensals prime the reactivity of neutrophils towards formation of neutrophil extracellular traps (NETosis). Approach and Results: We applied a mesenteric ischemia-reperfusion (I/R) injury model to germ-free (GF) and colonized C57BL/6J mice. By intravital imaging, we quantified leukocyte adherence and NET formation in I/R-injured mesenteric venules. Colonization with gut microbiota or monocolonization with Esch…

0301 basic medicineMaleExtracellular TrapsMesenteric infarctionLipopolysaccharideNeutrophilsGut floraExtracellular Traps03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAcute mesenteric ischemiaVenulesmedicineCell AdhesionEscherichia coliLeukocytesAnimalsGerm-Free LifeLeukocyte RollingMesenteryCells CulturedMice Knockoutbiologybusiness.industryNeutrophil extracellular trapsbiology.organism_classificationmedicine.diseaseGastrointestinal MicrobiomeMice Inbred C57BLToll-Like Receptor 4Disease Models Animal030104 developmental biologychemistryNeutrophil Infiltration030220 oncology & carcinogenesisMesenteric IschemiaReperfusion InjuryImmunologyHost-Pathogen InteractionsFemaleCardiology and Cardiovascular MedicinebusinessReperfusion injuryBacillus subtilisSignal TransductionArteriosclerosis, thrombosis, and vascular biology
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The Commensal Microbiota Enhances ADP-Triggered Integrin αIIbβ3 Activation and von Willebrand Factor-Mediated Platelet Deposition to Type I Collagen

2020

The commensal microbiota is a recognized enhancer of arterial thrombus growth. While several studies have demonstrated the prothrombotic role of the gut microbiota, the molecular mechanisms promoting arterial thrombus growth are still under debate. Here, we demonstrate that germ-free (GF) mice, which from birth lack colonization with a gut microbiota, show diminished static deposition of washed platelets to type I collagen compared with their conventionally raised (CONV-R) counterparts. Flow cytometry experiments revealed that platelets from GF mice show diminished activation of the integrin αIIbβ3 (glycoprotein IIbIIIa) when activated by the platelet agonist adenosine diphosphate (ADP). Fu…

0301 basic medicineMaleGene Expression030204 cardiovascular system & hematologyvon Willebrand factorlcsh:Chemistrychemistry.chemical_compoundMice0302 clinical medicinePlateletToll-like receptor-2lcsh:QH301-705.5SpectroscopyMice KnockoutbiologyChemistryBrief ReportαIIbβ3General MedicineArteriesComputer Science ApplicationsCell biologyAdenosine DiphosphatePlatelet Glycoprotein GPIb-IX Complexgerm-freeplateletsFemaleType I collagenBlood PlateletsIntegrinPrimary Cell CulturePlatelet Glycoprotein GPIIb-IIIa ComplexCatalysisCollagen Type IInorganic Chemistry03 medical and health sciencesVon Willebrand factormedicineCell AdhesionmicrobiotaAnimalsGerm-Free LifeHumansPhysical and Theoretical ChemistryThrombusSymbiosisMolecular Biologyα<sub>IIb</sub>β<sub>3</sub>Innate immune systemOrganic ChemistryThrombosismedicine.diseaseImmunity InnateToll-Like Receptor 2Gastrointestinal MicrobiomeMice Inbred C57BLAdenosine diphosphateTLR2030104 developmental biologylcsh:Biology (General)lcsh:QD1-999biology.proteinInternational Journal of Molecular Sciences
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Functional role of endothelial CXCL16/CXCR6-platelet-leucocyte axis in angiotensin II-associated metabolic disorders.

2018

Aims Angiotensin-II (Ang-II) is the main effector peptide of the renin-angiotensin system (RAS) and promotes leucocyte adhesion to the stimulated endothelium. Because RAS activation and Ang-II signalling are implicated in metabolic syndrome (MS) and abdominal aortic aneurysm (AAA), we investigated the effect of Ang-II on CXCL16 arterial expression, the underlying mechanisms, and the functional role of the CXCL16/CXCR6 axis in these cardiometabolic disorders. Methods and results Results from in vitro chamber assays revealed that CXCL16 neutralization significantly inhibited mononuclear leucocyte adhesion to arterial but not to venous endothelial cells. Flow cytometry and immunofluorescence s…

0301 basic medicineMaleRHOAPhysiologyMice Knockout ApoE030204 cardiovascular system & hematology0302 clinical medicineLeukocytesReceptorCells CulturedMetabolic SyndromebiologyChemistryAngiotensin IIMiddle AgedAortic AneurysmVascular endothelial growth factor ALosartanmedicine.anatomical_structurecardiovascular systemFemaleCardiology and Cardiovascular Medicinemedicine.drugSignal TransductionAdultBlood Plateletsmedicine.medical_specialtyEndothelium03 medical and health sciencesPhysiology (medical)Internal medicinemedicineCell AdhesionAnimalsHumansPlatelet activationReceptors CXCR6Angiotensin II receptor type 1Endothelial CellsChemokine CXCL16Platelet ActivationAngiotensin IICoculture TechniquesMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologyCase-Control Studiesbiology.proteinAngiotensin II Type 1 Receptor BlockersCardiovascular research
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Tomato sauce enriched with olive oil exerts greater effects on cardiovascular disease risk factors than raw tomato and tomato sauce: a randomized tri…

2016

Epidemiological studies have observed a negative association between tomato intake and the incidence of cardiovascular disease. As tomato sauces are usually cooked with the addition of oil, some studies have pointed out that both processes may increase the bioavailability of the bioactive compounds. However, the effect of consumption of raw tomatoes and tomato sauces on inflammation biomarkers and adhesion molecules related to atherosclerosis remains unknown. The aim of this study was to test the postprandial effects of a single dose of raw tomatoes (RT), tomato sauce (TS) and tomato sauce with refined olive oil (TSOO) on cardiovascular disease risk factors. We performed an open, prospectiv…

0301 basic medicineMaleTime FactorsLymphocyteCD36postprandial030204 cardiovascular system & hematologytomatochemistry.chemical_compound0302 clinical medicineSolanum lycopersicumRisk FactorsTomàquetsFood scienceCookingProspective StudiesProspective cohort studyNutrition and DieteticsCross-Over Studiesbiologycardiovascularcookedfood and beveragesPostprandial PeriodLipidsInflamaciótomato; postprandial; cardiovascular; cooked; food matrix; bioavailabilityOli d'olivaPostprandialmedicine.anatomical_structureCardiovascular diseasesEstudi de casosCardiovascular DiseasesFemaleInflammation Mediatorslcsh:Nutrition. Foods and food supplyNutritive ValueAdultAdolescentlcsh:TX341-641Article03 medical and health sciencesYoung AdultTomatoesmedicineHumansSugarOlive OilInflammation030109 nutrition & dieteticsCholesterolbusiness.industryMalalties cardiovascularsProtective FactorsCrossover studyBioavailabilityDietchemistrySpainFruitbiology.proteinCase studiesbusinessEnergy IntakebioavailabilityCell Adhesion MoleculesBiomarkersfood matrixOlive oilFood Science
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Isoform-specific function of calpains in cell adhesion disruption: studies in postlactational mammary gland and breast cancer.

2016

Cleavage of adhesion proteins is the first step for physiological clearance of undesired cells during postlactational regression of the mammary gland, but also for cell migration in pathological states such as breast cancer. The intracellular Ca2+-dependent proteases, calpains (CAPNs), are known to cleave adhesion proteins. The isoform-specific function of CAPN1 and CAPN2 was explored and compared in two models of cell adhesion disruption: mice mammary gland during weaning-induced involution and breast cancer cell lines according to tumor subtype classification. In both models, E-cadherin, β-catenin, p-120, and talin-1 were cleaved as assessed by western blot analysis. Both CAPNs were able …

0301 basic medicineMammary glandBreast NeoplasmsProximity ligation assayBiochemistry03 medical and health sciencesMiceWestern blotmedicineCell AdhesionAnimalsHumansLactationInvolution (medicine)BreastCell adhesionMolecular Biologybiologymedicine.diagnostic_testCalpainCalpainCell migrationCell BiologyCytosol030104 developmental biologymedicine.anatomical_structurebiology.proteinCancer researchFemaleThe Biochemical journal
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A poly-L-lactic acid/ collagen/glycosaminoglycan matrix for tissue engineering applications

2017

Adhesion of tissue cells to biomaterials is a prerequisite of paramount importance for the effectiveness of a tissue engineering construct (cell and scaffolds). Functionalization of polymeric scaffolds with organic polymers, such as collagen or proteoglycans, is a promising approach in order to improve the cytocompatibility. As a matter of fact, organic polymers, isolated directly from the extracellular matrix, contain a multitude of surface ligand (fibronectin, laminin, vitronectin) and arginine–glycine–aspartic acid-containing peptides that promote cell adhesion. In tissue engineering, the combination of organic and synthetic polymers gives rise to scaffolds characterized simultaneously …

0301 basic medicineMaterials Chemistry2506 Metals and AlloysMaterials sciencePolymers and PlasticsBiocompatibilitypoly-L-lactic acid02 engineering and technologyMatrix (biology)Extracellular matrix03 medical and health sciencesTissue engineeringMaterials ChemistryglycosaminoglycanCell adhesionSettore ING-IND/24 - Principi Di Ingegneria ChimicaPolymers and PlasticPolymer sciencebiologyChemistry (all)General ChemistryAdhesion021001 nanoscience & nanotechnologyBiomaterialFibronectin030104 developmental biologySettore ING-IND/22 - Scienza E Tecnologia Dei Materialitissue engineeringBiophysicsbiology.proteinSurface modificationphase separation0210 nano-technology
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The Gut Microbiota in Cardiovascular Disease and Arterial Thrombosis

2019

The gut microbiota has emerged as a contributing factor in the development of atherosclerosis and arterial thrombosis. Metabolites from the gut microbiota, such as trimethylamine N-oxide and short chain fatty acids, were identified as messengers that induce cell type-specific signaling mechanisms and immune reactions in the host vasculature, impacting the development of cardiovascular diseases. In addition, microbial-associated molecular patterns drive atherogenesis and the microbiota was recently demonstrated to promote arterial thrombosis through Toll-like receptor signaling. Furthermore, by the use of germ-free mouse models, the presence of a gut microbiota was shown to influence the syn…

0301 basic medicineMicrobiology (medical)CellDiseaseReview030204 cardiovascular system & hematologyGut floraarterial thrombosisMicrobiologydigestive systemlaw.invention03 medical and health sciencesProbiotic0302 clinical medicinelawcardiovascular diseaseVirologyMedicinevascular inflammationbiologygut microbiotabusiness.industryCell adhesion moleculeblood pressure regulationReceptor signalingbiology.organism_classificationmedicine.diseaseThrombosis030104 developmental biologymedicine.anatomical_structureImmunologyImmune reactionbusinessMicroorganisms
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Evaluation of cytocompatibility of calcium silicate-based endodontic sealers and their effects on the biological responses of mesenchymal dental stem…

2015

Aim To investigate in vitro the cytocompatibility of the calcium silicate-containing endodontic sealers MTA Fillapex and TotalFill BC Sealer on human periodontal ligament stem cells (hPDLSCs) by assaying their biological responses and compare them with that observed when using an epoxy resin-based sealer (AH Plus). Methodology Specimens from the three different endodontic sealers were eluated with culture medium for 24 h. The cytotoxicity of these eluates was evaluated using the MTT assay. In addition, an in vitro scratch wound healing model was used to determine their effects on cell migration. Cell adhesion to collagen type I after treatment with the different sealer eluates was also meas…

0301 basic medicineMineral trioxide aggregateMaterials sciencePeriodontal ligament stem cellsCell SurvivalPeriodontal LigamentCell morphologyAndrologyRoot Canal Filling Materials03 medical and health sciences0302 clinical medicineCell MovementMaterials TestingCell AdhesionHumansMTT assayCytotoxicityGeneral DentistryCells CulturedCell ProliferationCell growthSilicatesStem CellsMesenchymal stem cell030206 dentistryAdhesionCalcium Compounds030104 developmental biologyBiomedical engineeringInternational endodontic journal
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The mitochondrial antioxidant SS-31 increases SIRT1 levels and ameliorates inflammation, oxidative stress and leukocyte-endothelium interactions in t…

2018

AbstractThere is growing focus on mitochondrial impairment and cardiovascular diseases (CVD) in type 2 diabetes (T2D), and the development of novel therapeutic strategies in this context. It is unknown whether mitochondrial-targeting antioxidants such as SS-31 protect sufficiently against oxidative damage in diabetes. We aimed to evaluate if SS-31 modulates SIRT1 levels and ameliorates leukocyte-endothelium interactions, oxidative stress and inflammation in T2D patients. Anthropometric and metabolic parameters were studied in 51 T2D patients and 57 controls. Production of mitochondrial reactive oxygen species (ROS), mitochondrial membrane potential, glutathione content, leukocyte-endotheliu…

0301 basic medicineMitochondrial ROSMaleAntioxidantendocrine system diseasesmedicine.medical_treatmentMitochondrionPharmacologymedicine.disease_causeAntioxidantsLeukocyte-endothelial Interactionschemistry.chemical_compoundSirtuin 1Leukocyteschemistry.chemical_classificationMembrane Potential MitochondrialMultidisciplinaryQRMiddle AgedMitochondriaUp-RegulationMedicineFemalemedicine.symptomOligopeptidesRolling FluxScienceInflammationContext (language use)SIRT1 LevelsArticle03 medical and health sciencesmedicineCell AdhesionHuman Umbilical Vein Endothelial CellsHumansAgedInflammationReactive oxygen speciesTranscription Factor RelAGlutathioneSirtuins (SIRT1)Oxidative Stress030104 developmental biologychemistryDiabetes Mellitus Type 2Case-Control StudiesReactive Oxygen SpeciesLeukocyte Rolling VelocityOxidative stressScientific Reports
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