Search results for " Autoimmune"

showing 10 items of 252 documents

IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice

2009

The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE. To study the impact of IL-17A on CNS inflammation, we generated transgenic mice in which high levels of expression of IL-17A could be initiated after Cre-mediated recombination. Although ubiquitous overexpression of IL-17A led to skin inflammation and granulocytosis, T cell–specific IL-17A overexpression did not have a perceptible impact on the development and health of the mice. In the context of EAE, neither the T cell–driven overexpression of IL-17A nor its complete loss had a major impact on the development of clinical disease. Sin…

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellEncephalomyelitisPopulation610 Medicine & healthMice TransgenicInflammation2700 General Medicine10263 Institute of Experimental ImmunologyMyelin oligodendrocyte glycoproteinMicemedicineAnimalseducationCells CulturedGlycoproteinseducation.field_of_studybiologybusiness.industryInterleukin-17General MedicineTh1 Cellsmedicine.diseasePeptide FragmentsMice Inbred C57BLCytokinemedicine.anatomical_structureImmunologybiology.protein570 Life sciences; biologyExperimental pathologyFemaleMyelin-Oligodendrocyte GlycoproteinInterleukin 17medicine.symptombusinessGranulocytesResearch Article
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Regulatory T cells--the renaissance of the suppressor T cells.

2007

Immune reactions are stringently regulated and balanced by complex interactions of stimulating and suppressing mechanisms. Dysfunctions of this sophisticated immune regulatory network can lead to a variety of diseases such as autoimmunity, allergy, cancer, and pregnancy disorders. The rediscovery of suppressor T cells a decade ago--now designated as T regulatory cells--set off a huge avalanche of research activities leading to a multitude of preclinical and clinical studies. Herein, we give a comprehensive review about this research on T regulatory cells and the relevance of this suppressive T cell population for the development of innovative immune therapeutic strategies.

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellPopulationAutoimmunitymedicine.disease_causeInfectionsT-Lymphocytes RegulatoryAutoimmunitylaw.inventionMiceImmune systemlawPregnancyT-Lymphocyte SubsetsTransplantation ImmunologyNeoplasmsmedicineSuppressor Factors ImmunologicAnimalsHumanseducationeducation.field_of_studybusiness.industryModels ImmunologicalGeneral MedicineT lymphocyteImmunotherapyInflammatory Bowel DiseasesTransplantationDisease Models Animalmedicine.anatomical_structureDiabetes Mellitus Type 1ImmunologySuppressorFemaleImmunotherapybusinessAnnals of medicine
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IL-6 controls Th17 immunity in vivo by inhibiting the conversion of conventional T cells into Foxp3+regulatory T cells

2008

The conditions leading to the induction of adaptive Foxp3+regulatory T cells (T-regs) from peripheral T cells in vivo are incompletely understood. Here, we show that unresponsiveness of T cells to IL-6 by T cell-selective deletion of gp130 or immunization of wild-type mice with antigen in incomplete Freund's adjuvant (IFA), which fails to induce IL-6, promotes the conversion of peripheral CD4+T cells into adaptive Foxp3+T-regs. Thus, both T cell-conditional gp130 knockout (KO) mice immunized with MOG35-55 in complete Freund's adjuvant (CFA) and wild-type mice immunized with MOG35-55 in IFA develop overwhelming antigen-specific T-reg responses and are protected from experimental autoimmune e…

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentchemical and pharmacologic phenomenaInflammationBiologyT-Lymphocytes RegulatoryMiceInterleukin 21Antigenimmune system diseasesmedicineAnimalsCytotoxic T cellIL-2 receptorMice KnockoutMultidisciplinaryInterleukin-6Experimental autoimmune encephalomyelitisFOXP3Forkhead Transcription Factorshemic and immune systemsT-Lymphocytes Helper-InducerBiological Sciencesmedicine.diseaseCytokineImmunologymedicine.symptomProceedings of the National Academy of Sciences
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Endoscopic ultrasonography findings in autoimmune pancreatitis

2011

Endoscopic ultrasonography is an established diagnostic tool for pancreatic masses and chronic pancreatitis. In recent years there has been a growing interest in the worldwide medical community in autoimmune pancreatitis (AIP), a form of chronic pancreatitis caused by an autoimmune process. This paper reviews the current available literature about the endoscopic ultrasonographic findings of AIP and the role of this imaging technique in the management of this protean disease.

Endoscopic ultrasoundmedicine.medical_specialtyAUTOIMMUNEEndoscopic ultrasonographyDiseaseGastroenterologyAutoimmune DiseasesEndosonographyPANCREATITIS; AUTOIMMUNE; ENDOSCOPIC ULTRASOUND; IgG4 CHOLANGITISAutoimmune ProcessInternal medicinemedicineENDOSCOPIC ULTRASOUNDTopic HighlightPancreasIgG4 CHOLANGITISAutoimmune pancreatitismedicine.diagnostic_testbusiness.industryGastroenterologyGeneral Medicinemedicine.diseasemedicine.anatomical_structurePancreatitisPancreatitisImaging techniqueBile DuctsPancreasbusiness
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Crucial role of aspartic acid at position 265 in the CH2 domain for murine IgG2a and IgG2b Fc-associated effector functions.

2008

Abstract Replacement of aspartic acid by alanine at position 265 (D265A) in mouse IgG1 results in a complete loss of interaction between this isotype and low-affinity IgG Fc receptors (FcγRIIB and FcγRIII). However, it has not yet been defined whether the D265A substitution could exhibit similar effects on the interaction with two other FcγR (FcγRI and FcγRIV) and on the activation of complement. To address this question, 34-3C anti-RBC IgG2a and IgG2b switch variants bearing the D265A mutation were generated, and their effector functions and in vivo pathogenicity were compared with those of the respective wild-type Abs. The introduction of the D265A mutation almost completely abolished the…

ErythrocytesAspartic Acid/genetics/physiologyAntibodies Monoclonal/toxicityImmunologyMutantReceptors Fcddc:616.07Complement Activation/genetics/immunologyAlanine/geneticsMiceStructure-Activity RelationshipProtein structureImmunoglobulin G/chemistry/metabolismProtein Isoforms/chemistry/deficiency/genetics/physiologyAspartic acidImmunology and AllergyAnimalsProtein IsoformsErythrocytes/immunologyReceptorComplement ActivationAutoantibodiesAlanineMice KnockoutAspartic AcidMice Inbred BALB CAlaninebiologyAnemia Hemolytic Autoimmune/genetics/immunologyAntibodies MonoclonalReceptors Fc/chemistry/deficiency/genetics/physiologyFragment crystallizable regionIsotypeAmino Acid Substitution/genetics/physiologySialic Acids/geneticsProtein Structure TertiaryMice Inbred C57BLBiochemistryAmino Acid SubstitutionImmunoglobulin Gbiology.proteinSialic AcidsAutoantibodies/toxicityAnemia Hemolytic AutoimmuneAntibodyProtein Structure Tertiary/genetics/physiologyJournal of immunology (Baltimore, Md. : 1950)
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Anti-Inflammatory Action of Heterogeneous Nuclear Ribonucleoprotein A2/B1 in Patients with Autoimmune Endocrine Disorders

2019

Our previous studies documented that human fibroblast-limbal stem cells (f-LSCs) possess immunosuppressive capabilities, playing a role in regulating T-cell activity. This study highlights the molecular activities by which human f-LSCs can attenuate the inflammatory responses of self-reactive peripheral blood mononuclear cells (PBMCs) collected from patients with autoimmune endocrine diseases (AEDs). Anti-CD3 activated PBMCs from twenty healthy donors and fifty-two patients with AEDs were cocultured on f-LSC monolayer. 2D-DIGE proteomic experiments, mass spectrometry sequencing and functional in vitro assays were assessed in cocultured PBMCs. We identified the downmodulation of several huma…

Gene isoformInflammationfibroblast-limbal stem cells Autoimmune Endocrine DiseaseNF-ĸB interaction.Peripheral blood mononuclear cellArticleSettore MED/13 - EndocrinologiaAutoimmune Endocrine Diseases03 medical and health sciencesNF-ĸB interaction0302 clinical medicinemedicineGene silencingIL-2 receptorSettore BIO/06 - Anatomia Comparata E CitologiaHeterogeneous Nuclear Ribonucleoprotein A2/B1hnRNP A2/B1030304 developmental biology0303 health sciencesSettore MED/30 - Malattie Apparato Visivobusiness.industryautoimmunityGeneral Medicinefibroblast-limbal stem cellsSettore BIO/18 - Genetica030220 oncology & carcinogenesisCancer researchfibroblast-limbal stem cells Autoimmune Endocrine Diseasesmedicine.symptomStem cellbusinessCD8immunotoleranceJournal of Clinical Medicine
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Adherence and Reactogenicity to Vaccines against SARS-COV-2 in 285 Patients with Neuropathy: A Multicentric Study

2022

Background: The safety of the new vaccines against SARS-CoV-2 have already been shown, although data on patients with polyneuropathy are still lacking. The aim of this study is to evaluate the adherence to SARS-CoV-2 vaccination, as well as the reactogenicity to those vaccines in patients affected by neuropathy. Methods: A multicentric and web-based cross-sectional survey was conducted among patients affected by neuropathy from part of South Italy. Results: Out of 285 responders, n = 268 were included in the final analysis and n = 258 of them (96.3%) were fully vaccinated. Adherence to vaccination was higher in patients with hereditary neuropathies compared to others, while it was lower in …

General NeuroscienceSARS-CoV-2 infectionCIDP COVID-19 vaccines SARS-CoV-2 infection autoimmune neuropathy hereditary neuropathy neuropathy reactogenicity vaccine hesitancy vaccine safetyreactogenicityvaccine hesitancySARS-CoV-2 infection; COVID-19 vaccines; reactogenicity; vaccine safety; vaccine hesitancy; neuropathy; autoimmune neuropathy; hereditary neuropathy; CIDPneuropathyvaccine safetyCIDPhereditary neuropathyCOVID-19 vaccineautoimmune neuropathy
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Impairment of TGF-β signaling in T cells increases susceptibility to experimental autoimmune hepatitis in mice

2002

In autoimmune hepatitis, strong TGF-beta1 expression is found in the inflamed liver. TGF-beta overexpression may be part of a regulatory immune response attempting to suppress autoreactive T cells. To test this hypothesis, we determined whether impairment of TGF-beta signaling in T cells leads to increased susceptibility to experimental autoimmune hepatitis (EAH). Transgenic mice of strain FVB/N were generated expressing a dominant-negative TGF-beta type II receptor in T cells under the control of the human CD2 promoter/locus control region. On induction of EAH, transgenic mice showed markedly increased portal and periportal leukocytic infiltrations with hepatocellular necroses compared wit…

Genetically modified mouseCellular immunityPhysiologyT-LymphocytesEnzyme-Linked Immunosorbent AssayMice TransgenicAutoimmune hepatitisBiologyMiceImmune systemTransforming Growth Factor betaPhysiology (medical)medicineAnimalsHomeostasisCells CulturedAutoimmune diseaseHepatitisB-LymphocytesHepatologyGastroenterologyT lymphocyteBlotting Northernmedicine.diseaseImmunohistochemistryHepatitis AutoimmunePhenotypeNeutrophil InfiltrationImmunologySignal transductionSignal TransductionAmerican Journal of Physiology-Gastrointestinal and Liver Physiology
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Clinical update on risks and efficacy of anti-SARS-CoV-2 vaccines in patients with autoimmune hepatitis and summary of reports on post-vaccination li…

2022

Patients with liver diseases, especially those with cirrhosis, have an increased mortality risk when infected by SARS-CoV-2 and therefore anti-SARS-CoV-2 vaccine has been recommended by leading Scientific Associations for all patients with chronic liver diseases. However, previous reports have shown a reduced antibody response following the full course of vaccination in immunosuppressed patients, including liver transplant recipients and several rheumatic diseases. This document, drafted by an expert panel of hepatologists appointed by the Italian Association for the Study of the Liver (AISF), aims to present the updated scientific data on the safety and efficacy of anti-SARS-CoV-2 mRNA vac…

Hepatitis AutoimmuneSettore MED/12 - GastroenterologiaCOVID-19 VaccinesHepatologySARS-CoV-2Liver DiseasesVaccinationGastroenterologyCOVID-19HumansSARS-CoV-2 mRNA vaccines autoimmune hepatitis immunosoppressionDigestive and Liver Disease
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Humoral Mechanisms in T cell Vaccination: Induction and Functional Characterization of Anti-lymphocytic Autoantibodies

1997

T cell vaccination, the application of syngeneic attenuated T cells, has been shown to prevent effectively and treat experimental autoimmune diseases, but its mechanisms of action are poorly understood. Here we present data on the induction of a humoral anti-T cell response by T cell vaccination, capable of strongly inhibiting T cell proliferation and of ameliorating experimental autoimmune disease. T cell vaccination in the Lewis rat induced autoantibodies reactive with several syngeneic T cell proteins. These autoantibodies were not detectable in normal Lewis sera as assessed by immunoblotting and flow cytometry with intact syngeneic T cells. The autoantibody reactivity was not restricted…

IdiotypeEncephalomyelitis Autoimmune ExperimentalT-LymphocytesT cellImmunologyT-cell vaccinationBiologyLymphocyte ActivationImmunoglobulin IdiotypesmedicineAnimalsImmunology and AllergyCytotoxic T cellAntilymphocyte SerumAutoantibodiesImmune SeraVaccinationAutoantibodyMembrane ProteinsT lymphocyteClone CellsRatsmedicine.anatomical_structureRats Inbred LewImmunologyHumoral immunitybiology.proteinAntibodyJournal of Autoimmunity
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