Search results for " Breast Cancer"

showing 10 items of 427 documents

Surveillance of spontaneous breast cancer metastasis by TRAIL-expressing CD34⁺ cells in a xenograft model

2012

Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL), delivered as a membrane-bound molecule expressed on the surface of adenovirus-transduced CD34+ cells (CD34-TRAIL+), was analyzed for its apoptotic activity in vitro on 12 breast cancer cell lines representing estrogen receptor-positive, HER2+ and triple-negative (TN) subtypes and for its effect on tumor growth, vascularization, necrosis, and lung metastasis incidence in NOD/SCID mice xenografted with the TN breast cancer line MDA-MB-231. Mesenchymal TN cell lines, which are the richest in putative tumor stem cells among the different breast cancer cell subtypes, were the most susceptible to apoptosis induced by CD34-TRAIL+ cel…

PathologyCancer ResearchCD34Antigens CD34ApoptosisMice SCIDMetastasisMetastasisTNF-Related Apoptosis-Inducing LigandMicePreclinical StudyMice Inbred NODNeoplasm Metastasisskin and connective tissue diseasesHeterologousTumorbiologyCell DeathCancer stem cellsTumor BurdenOncologyNeoplastic Stem CellsTumor necrosis factor alphaFemalemedicine.medical_specialtyTRAIL BREAST CANCERTransplantation HeterologousBreast NeoplasmsSCIDCell LineTriple-negative breast cancerCancer stem cellCell Line TumormedicineAnimalsHumansAntigensTransplantationCD44Mesenchymal stem cellmedicine.diseaseApoptosisCell culturebiology.proteinCancer researchInbred NODCD34Settore MED/36 - Diagnostica Per Immagini E RadioterapiaAnimals; Antigens CD34; Apoptosis; Breast Neoplasms; Cell Death; Cell Line Tumor; Female; Humans; Mice; Mice Inbred NOD; Mice SCID; Neoplasm Metastasis; Neoplastic Stem Cells; TNF-Related Apoptosis-Inducing Ligand; Transplantation Heterologous; Tumor BurdenmTRAIL
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Proteomic patterns of cultured breast cancer cells and epithelial mammary cells.

2002

: Breast cancer is one of the leading causes of death from cancer among women in western countries. The different types of breast cancer are grouped into invasive and noninvasive forms. Among the invasive types, ductal infiltrating carcinoma (DIC) is the most common and aggressive form. Using an in vitro model consisting of a DIC-derived cell line (8701-BC) and a nontumoral mammary epithelial cell line (HB2), we used the proteomics approach to search for homology and differences in protein expression patterns between tumoral and nontumoral phenotypes. Within an analysis window comprising 1,750 discernible spots we have currently catalogued 140 protein spots of potential interest. Fifty-eigh…

Pathologymedicine.medical_specialtyProteomeBreast NeoplasmsBiologyProteomicsGeneral Biochemistry Genetics and Molecular Biologyductal infiltrating carcinomaBreast cancerbreast cancerHistory and Philosophy of ScienceGene expressionTumor Cells Culturedmedicineproteomics; breast cancer; ductal infiltrating carcinomaHumansBreastSettore BIO/06 - Anatomia Comparata E CitologiaproteomicGeneral NeuroscienceEpithelial Cellsmedicine.diseasePhenotypeCell cultureProteomeCancer cellCancer researchNeoplastic cell
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Discordance rate of HER2 status in primary breast carcinomas versus synchronous axillary lymph node metastases: a multicenter retrospective investiga…

2014

Antonio Ieni,1 Valeria Barresi,1 Rosario Caltabiano,2 Anna Maria Cascone,3 Rachele Del Sordo,4 Daniela Cabibi,5 Pio Zeppa,3 Salvatore Lanzafame,2 Angelo Sidoni,4 Vito Franco,5 Giovanni Tuccari1 1Department of Human Pathology Gaetano Barresi, Section of Anatomic Pathology, University of Messina, Messina, 2Department GF Ingrassia, Section of Anatomic Pathology, University of Catania, Catania, 3Department of Medicine and Surgery, University of Salerno, Salerno, 4Institute of Pathologic Anatomy and Histology, Division of Cancer Research, University of Perugia, Perugia, 5Department of Human Pathology, Section of Anatomic Pathology, University of Palermo, Palermo, Italy Background: Human epiderma…

Pathologymedicine.medical_specialtysynchronous lymph nodeslcsh:RC254-282OncoTargets and TherapyBreast cancer Human epidermal growth factor receptor 2 Metastases Prognosis Synchronous lymph nodesText miningBreast cancerbreast cancermedicinebreast cancer; human epidermal growth factor receptor 2; metastases; prognosis; synchronous lymph nodesPharmacology (medical)metastasesskin and connective tissue diseasesneoplasmsLymph nodeHuman Epidermal Growth Factor Receptor 2Original Researchbusiness.industryhuman epidermal growth factor receptor 2; breast cancer; synchronous lymph nodes; metastases; prognosishuman epidermal growth factor receptor 2lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseasemedicine.anatomical_structureOncologysynchronous lymph nodemetastaseprognosisbusinessBreast carcinoma
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Insight on pyrimido[5,4-g]indolizine and pyrimido[4,5-c]pyrrolo[1,2-a]azepine systems as promising photosensitizers on malignant cells

2022

Searching for new small molecules as photosensitizing agents, we have developed a class of twenty-five pyrimido[5,4-g]indolizine and pyrimido[4,5-c]pyrrolo[1,2-a]azepines with a good substitution pattern defining a versatile synthetic pathway to approach the title ring system. All compounds were evaluated for their photocytotoxicity on a triple negative human breast cancer cell line (MDA-MB-231) in the dark and under UVA light (2.0 J/cm2). The most effective compounds exhibited a photoantiproliferative activity with IC50 values up to nanomolar ranges. Interestingly, these new developed compounds showed high selectivity towards cancerous cells with respect to non-cancerous ones. Moreover, fo…

PharmacologyMDA-MB-231Triple negative human breast cancerOrganic ChemistryPhototoxic activityIndolizinesAntineoplastic AgentsApoptosisTriple Negative Breast Neoplasms4-g]indolizinespyrimido[4General MedicineAzepinespyrimido[54-g]indolizinespyrimido[45-c]pyrrolo[12-a]azepinesTriple negative human breast cancerMDA-MB-231Photosensitizing agentsPhototoxic activitypyrimido[5Photosensitizing agents5-c]pyrrolo[1pyrimido[45-c]pyrrolo[12-a]azepinesCell Line Tumor2-a]azepinesTriple negative human breast cancerMDA-MB-231Photosensitizing agentsPhototoxic activityDrug DiscoveryHumanspyrimido[54-g]indolizines
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Physiologically-Based Pharmacokinetic/Pharmacodynamic Model of MBQ-167 to Predict Tumor Growth Inhibition in Mice

2020

MBQ-167 is a dual inhibitor of the Rho GTPases Rac and Cdc42 that has shown promising results as an anti-cancer therapeutic at the preclinical stage. This drug has been tested in vitro and in vivo in metastatic breast cancer mouse models. The aim of this study is to develop a physiologically based pharmacokinetic/pharmacodynamic (PBPK-PD) model of MBQ-167 to predict tumor growth inhibition following intraperitoneal (IP) administration in mice bearing Triple Negative and HER2+ mammary tumors. PBPK and Simeoni tumor growth inhibition (TGI) models were developed using the Simcyp V19 Animal Simulator. Our developed PBPK framework adequately describes the time course of MBQ-167 in each of the mo…

Physiologically based pharmacokinetic modellinglcsh:RS1-441Pharmaceutical ScienceSpleenPharmacologyArticlelcsh:Pharmacy and materia medica03 medical and health sciences0302 clinical medicinebreast cancerPharmacokineticsIn vivomedicinePotency030304 developmental biology0303 health sciencesMammary tumorbusiness.industryMBQ-167medicine.diseaseMetastatic breast cancermedicine.anatomical_structure030220 oncology & carcinogenesisPharmacodynamicsRac inhibitorphysiologically based pharmacokinetic modelingbusinessPharmaceutics
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Type V collagen regulates the expression of apoptotic and stress response genes by breast cancer cells.

2004

Type V collagen is a "minor" component of normal human breast stroma, which is subjected to over-deposition in cases of ductal infiltrating carcinoma (DIC). We reported that, if used as a culture substrate for the DIC cell line 8701-BC, it exhibited poorly-adhesive properties and restrained the proliferative and motile behavior of the cell subpopulation able to attach onto it. Moreover, this collagen species was able to trigger DNA fragmentation and impair survival of 8701-BC cells. In this study, we have extended our investigation with the aim to obtain further evidence that the death induced by type V collagen was of the apoptotic type by (i) microscopic detection and quantitation of Apop…

PhysiologyClinical BiochemistryCellApoptosisBreast NeoplasmsEnzyme activatorCell Line TumormedicineHumansSettore BIO/06 - Anatomia Comparata E CitologiaCaspaseHeat-Shock ProteinsbiologyCarcinoma Ductal BreastCell BiologyMolecular biologyIn vitroEnzyme ActivationGene Expression Regulation Neoplasticmedicine.anatomical_structurecollagen breast cancer gene expressionApoptosisCell cultureCaspasesbiology.proteinDNA fragmentationHSP60FemaleCollagen Type VJournal of cellular physiology
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Control of dataset bias in combined Affymetrix cohorts of triple negative breast cancer

2014

AbstractHeterogenous subtypes of breast cancer need to be analyzed separately. Pooling of datasets can provide reasonable sample sizes but dataset bias is an important concern. We assembled a combined dataset of 579 Affymetrix microarrays from triple negative breast cancer (TNBC) in Gene Expression Omnibus (GEO) series GSE31519. We developed a method for selecting comparable datasets and to control for the amount of dataset bias of individual probesets.

Poolinglcsh:QH426-470MicroarrayPoolingComputational biologyMicroarrayBiologycomputer.software_genreBiochemistryBreast cancerBreast cancerData in BriefGeneticsmedicineddc:610Affymetrix microarraysTriple-negative breast cancerGene expression omnibusmedicine.diseaselcsh:GeneticsSample size determinationDataset biasMolecular MedicineGene expressionData miningcomputerBiotechnologyGenomics Data
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Effect of transfection with PLP2 antisense oligonucleotides on gene expression of cadmium-treated MDA-MB231 breast cancer cells

2012

Emerging evidence indicates that cadmium (Cd) is able to regulate gene expression, drastically affecting the pattern of transcriptional activity in human normal and pathological cells. We have already shown that exposure of MDA-MB231 breast cancer cells to 5 μM CdCl(2) for 96 h, apart from significantly affecting mitochondrial metabolism, induces modifications of the expression level of genes coding for members of stress response-, mitochondrial respiration-, MAP kinase-, NF-κB-, and apoptosis-related pathways. In the present study, we have expanded the knowledge on the biological effects of Cd-breast cancer cell interactions, indicating PLP2 (proteolipid protein-2) as a novel member of the…

ProteolipidsApoptosisBreast NeoplasmsTransfectionBiochemistryAnalytical ChemistryCadmium ChlorideStress PhysiologicalCell Line TumorGene expressionHumansRNA MessengerSettore BIO/06 - Anatomia Comparata E Citologiaskin and connective tissue diseasesGeneCaspaseHeat-Shock ProteinsMARVEL Domain-Containing Proteinsbiologycadmium PLP2 breast cancer differential display-PCR caspase gene expressionTransfectionSuicide geneOligonucleotides AntisenseMolecular biologyGene Expression Regulation NeoplasticApoptosisMitogen-activated protein kinaseCaspasesCancer cellbiology.proteinNucleic Acid ConformationFemale
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LARGE-SCALE COMPARATIVE PROTEOMICS OF BREAST CANCER SURGICAL TISSUES

2009

Proteomics BREAST CANCERSettore BIO/06 - Anatomia Comparata E Citologia
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Proteomic modulation induced in fibroblasts by breast cancer cells (8701‐BC)paracrine factors.

2009

Proteomics fibroblast breast cancerSettore BIO/06 - Anatomia Comparata E Citologia
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