Search results for " CYTOKINES"

showing 10 items of 104 documents

IL-25 dampens the growth of human germinal center-derived B-cell non Hodgkin Lymphoma by curtailing neoangiogenesis

2018

Interleukin (IL)-25, a member of the IL-17 cytokine superfamily, is produced by immune and non-immune cells and exerts type 2 pro-inflammatory effects in vitro and in vivo. The IL-25 receptor(R) is composed of the IL-17RA/IL-17RB subunits. Previous work showed that germinal centre (GC)-derived B-cell non Hodgkin lymphomas (B-NHL) expressed IL-17AR, formed by IL-17RA and IL-17RC subunits, and IL-17A/IL-17AR axis promoted B-NHL growth by stimulating neoangiogenesis. Here, we have investigated expression and function of IL-25/IL-25R axis in lymph nodes from human GC-derived B-NHL, i.e. Follicular Lymphoma (FL,10 cases), Diffuse Large B Cell Lymphoma (6 cases) and Burkitt Lymphoma (3 cases). Tu…

0301 basic medicinelcsh:Immunologic diseases. AllergyPathologymedicine.medical_specialtyAngiogenesismedicine.medical_treatmentImmunologyFollicular lymphomalcsh:RC254-282Angiogenesis; B lymphocytes; B-NHL; Cytokines; IL-25; Tumor immunology; Immunology and Allergy; Immunology; Oncology03 medical and health sciencesangiogenesisIL-25immune system diseaseshemic and lymphatic diseasesmedicineImmunology and AllergyCytokineOriginal ResearchB lymphocyteChemistryGerminal centerInterleukinmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenscytokinesLymphomaAngiogenesi030104 developmental biologyCytokineOncologyCancer researchB-Cell Non-Hodgkin LymphomaTumor immunologyB-NHLb lymphocyteslcsh:RC581-607Diffuse large B-cell lymphoma
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Structure–Activity Relationship of Plesiomonas shigelloides Lipid A to the Production of TNF-α, IL-1β, and IL-6 by Human and Murine Macrophages

2017

Plesiomonas shigelloides is a Gram-negative bacterium that is associated with diarrheal disease in humans. Lipopolysaccharide (LPS) is the main surface antigen and virulence factor of this bacterium. The lipid A (LA) moiety of LPS is the main region recognized by target cells of immune system. Here, we evaluated the biological activities of P. shigelloides LA for their abilities to induce the productions of proinflammatory cytokines (TNF-α, IL-1β, and IL-6) by human and murine macrophages [THP-1 macrophages and immortalized murine bone marrow-derived macrophages (iBMDM)]. Four native P. shigelloides LA preparations differing in their phosphoethanolamine (PEtn) substitution, length, number, …

0301 basic medicinelcsh:Immunologic diseases. AllergyReceptor complexLipopolysaccharideImmunologymedicine.disease_causeVirulence factorProinflammatory cytokineLipid A03 medical and health scienceschemistry.chemical_compoundmedicineImmunology and AllergyEscherichia colilipid AOriginal ResearchbiologyChemistrylipopolysaccharidebiology.organism_classificationBMDMMolecular biologyIn vitro030104 developmental biologyPlesiomonas shigelloidesproinflammatory cytokinesPlesiomonasTHP-1lcsh:RC581-607Frontiers in Immunology
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Systemic Oxidative Stress and Visceral Adipose Tissue Mediators of NLRP3 Inflammasome and Autophagy Are Reduced in Obese Type 2 Diabetic Patients Tre…

2020

Obesity is a low-grade inflammatory condition affecting a range of individuals, from metabolically healthy obese (MHO) subjects to type 2 diabetes (T2D) patients. Metformin has been shown to display anti-inflammatory properties, though the underlying molecular mechanisms are unclear. To study whether the effects of metformin are mediated by changes in the inflammasome complex and autophagy in visceral adipose tissue (VAT) of obese patients, a biopsy of VAT was obtained from a total of 68 obese patients undergoing gastric bypass surgery. The patients were clustered into two groups: MHO patients and T2D patients treated with metformin. Patients treated with metformin showed decreased levels o…

0301 basic medicinemedicine.medical_specialtyvisceral adipose tissue (VAT)obesityautophagyendocrine system diseasesPhysiologyinflammatory cytokinesClinical BiochemistryATG5Adipose tissue030209 endocrinology & metabolismLeukocyte homeostasisType 2 diabetesBiochemistryArticleProinflammatory cytokine03 medical and health sciences0302 clinical medicineInternal medicinemedicineoxidative stressMolecular Biologytype 2 diabetes (T2D)business.industrylcsh:RM1-950nutritional and metabolic diseasesInflammasomeCell Biologymedicine.diseaseMetforminlcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologybusinessmetforminInflammasome complexmedicine.drugAntioxidants (Basel, Switzerland)
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Repeated, Intermittent Social Defeat across the Entire Juvenile Period Resulted in Behavioral, Physiological, Hormonal, Immunological, and Neurochemi…

2016

The developing brain is vulnerable to social defeat during the juvenile period. As complements of human studies, animal models of social defeat provide a straightforward approach to investigating the functional and neurobiological consequences of social defeats. Taking advantage of agonist behavior and social defeat in male golden hamster, a set of 6 experiments was conducted to investigate the consequences at multiple levels in young adulthood resulting from repeated, intermittent social defeats or “social threats” across the entire juvenile period. Male hamsters at postnatal day 28 (P28) were randomly assigned to either the social defeat, “social threat”, or arena control group, and they …

0301 basic medicinesocial threatCognitive NeuroscienceHippocampusContext (language use)cortisolpro-inflammatory cytokinesrepeated intermittent social defeatDevelopmental psychologySocial defeat03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineNeurochemicalmonoamine neurotransmittersJuvenileYoung adultOriginal Researchmale golden hamstersSocial relationadolescent bullying030104 developmental biologyjuvenileNeuropsychology and Physiological PsychologyPsychology030217 neurology & neurosurgeryGolden hamsterNeuroscienceFrontiers in Behavioral Neuroscience
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CD4 T lymphocyte autophagy is upregulated in the salivary glands of primary Sjögren’s syndrome patients and correlates with focus score and disease a…

2017

Background Primary Sjögren’s syndrome (pSS) is a common chronic autoimmune disease characterized by lymphocytic infiltration of exocrine glands and peripheral lymphocyte perturbation. In the current study, we aimed to investigate the possible pathogenic implication of autophagy in T lymphocytes in patients with pSS. Methods Thirty consecutive pSS patients were recruited together with 20 patients affected by sicca syndrome and/or chronic sialoadenitis and 30 healthy controls. Disease activity and damage were evaluated according to SS disease activity index, EULAR SS disease activity index, and SS disease damage index. T lymphocytes were analyzed for the expression of autophagy-specific marke…

AdultCD4-Positive T-LymphocytesMale0301 basic medicinePathologymedicine.medical_specialtylcsh:Diseases of the musculoskeletal systemAutophagy; Cytokines; Lymphocytes; Sjögren syndrome; Immunology and Allergy; Rheumatology; ImmunologyLymphocyteImmunologySjögren syndromeSalivary GlandsPathogenesis03 medical and health sciencesImmune systemRheumatologystomatognathic systemSicca syndromeAutophagymedicineImmunology and AllergyHumansLymphocytesCytokineAgedSjögren syndrome; Autophagy; Lymphocytes; CytokinesAutoimmune diseaseSalivary glandbusiness.industryAutophagyT lymphocyteMiddle Agedmedicine.diseaseSjögren syndromeUp-RegulationSettore MED/16 - Reumatologiastomatognathic diseasesSjogren's Syndrome030104 developmental biologymedicine.anatomical_structureImmunologyCytokinesLymphocyteFemalelcsh:RC925-935businessResearch ArticleArthritis Research & Therapy
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The transcription factor IFN regulatory factor–4 controls experimental colitis in mice via T cell–derived IL-6

2008

The proinflammatory cytokine IL-6 seems to have an important role in the intestinal inflammation that characterizes inflammatory bowel diseases (IBDs) such as Crohn disease and ulcerative colitis. However, little is known about the molecular mechanisms regulating IL-6 production in IBD. Here, we assessed the role of the transcriptional regulator IFN regulatory factor-4 (IRF4) in this process. Patients with either Crohn disease or ulcerative colitis exhibited increased IRF4 expression in lamina propria CD3+ T cells as compared with control patients. Consistent with IRF4 having a regulatory function in T cells, in a mouse model of IBD whereby colitis is induced in RAG-deficient mice by transp…

AdultCD4-Positive T-LymphocytesMaleAdoptive cell transferRecombinant Fusion ProteinsT-LymphocytesCD3T cellAdoptive Transfer; Adult; Animals; Apoptosis; CD4-Positive T-Lymphocytes; Colitis; Cytokines; DNA-Binding Proteins; Female; Gene Expression Regulation; Humans; Inflammatory Bowel Diseases; Interferon Regulatory Factors; Interleukin-6; Intestinal Mucosa; Male; Mice; Mice Inbred C57BL; Mice Knockout; Middle Aged; Oxazolone; Receptors Interleukin-6; Recombinant Fusion Proteins; T-Lymphocytes; Trinitrobenzenesulfonic AcidApoptosisProinflammatory cytokineMiceIntestinal mucosamedicineAnimalsHumansIntestinal MucosaColitisInterleukin 6Mice KnockoutbiologyInterleukin-6OxazoloneGeneral MedicineMiddle AgedColitisInflammatory Bowel Diseasesmedicine.diseaseAdoptive TransferReceptors Interleukin-6Ulcerative colitisDNA-Binding ProteinsMice Inbred C57BLmedicine.anatomical_structureGene Expression RegulationTrinitrobenzenesulfonic AcidInterferon Regulatory FactorsImmunologybiology.proteinCytokinesFemaleResearch ArticleJournal of Clinical Investigation
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CD40 activation in human pancreatic islets and ductal cells.

2008

Aims/hypothesis: CD40 expression on non-haematopoietic cells is linked to inflammation. We previously reported that CD40 is expressed on isolated human and non-human primate islets and its activation results in secretion of IL-8, macrophage inflammatory protein 1-beta (MIP-1β) and monocyte chemoattractant protein-1 (MCP-1) through nuclear factor-κB and extracellularly regulated kinases 1/2 pathways. The objective of this study was to identify the pattern of gene expression, and to study viability and functionality affected by CD40-CD40 ligand (CD40L) interaction in human islets. Furthermore, we have studied the CD40-mediated cytokine/chemokine profile in pancreatic ductal cells, as they are…

AdultChemokinemedicine.medical_specialtyDuctal cellsCell SurvivalEndocrinology Diabetes and Metabolismmedicine.medical_treatmentChemokine CXCL1CD40 Chemokines Cytokines Ductal cells Inflammation Insulin Islets of Langerhans Microarray Quantitative RT-PCRCD40 LigandEnzyme-Linked Immunosorbent AssayIslets of LangerhansYoung AdultInternal medicineInternal MedicinemedicineHumansCD40 AntigensMacrophage inflammatory proteinOligonucleotide Array Sequence AnalysisCD40biologySettore BIO/16 - Anatomia UmanaReverse Transcriptase Polymerase Chain ReactionPancreatic isletsPancreatic DuctsMiddle AgedFlow CytometryMolecular biologyCXCL1CXCL2Endocrinologymedicine.anatomical_structureCytokinebiology.proteinCytokinesChemokinesDiabetologia
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Genome-based in silico identification of new Mycobacterium tuberculosis antigens activating polyfunctional CD8+ T cells in human tuberculosis.

2011

Although CD8(+) T cells help control Mycobacterium tuberculosis infection, their M. tuberculosis Ag repertoire, in vivo frequency, and functionality in human tuberculosis (TB) remains largely undefined. We have performed genome-based bioinformatics searches to identify new M. tuberculosis epitopes presented by major HLA class I supertypes A2, A3, and B7 (covering 80% of the human population). A total of 432 M. tuberculosis peptides predicted to bind to HLA-A*0201, HLA-A*0301, and HLA-B*0702 (representing the above supertypes) were synthesized and HLA-binding affinities determined. Peptide-specific CD8(+) T cell proliferation assays (CFSE dilution) in 41 M. tuberculosis-responsive donors ide…

AdultIntracellular FluidMaleTuberculosisT cellImmunologyEpitopes T-LymphocyteHuman leukocyte antigenCD8-Positive T-LymphocytesLymphocyte ActivationEpitopeTuberculosis CD8 T cells cytokinesMycobacterium tuberculosis03 medical and health sciencesAntigenifn-gamma protective efficacy binding-affinity dormancy regulon subunit vaccine transgenic mice hla-b epitopes infection responsesPredictive Value of TestsmedicineImmunology and AllergyCytotoxic T cellHumansTuberculosis030304 developmental biologyAged0303 health sciencesAntigens Bacterialbiology030306 microbiologyGenome HumanComputational BiologyMycobacterium tuberculosisMiddle Agedbiology.organism_classificationmedicine.diseaseVirology3. Good healthmedicine.anatomical_structureFemaleCD8Genome BacterialJournal of immunology (Baltimore, Md. : 1950)
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LPS-mediated production of pro/anti-inflammatory cytokines and eicosanoids in whole blood samples: Biological effects of +896A/G TLR4 polymorphism in…

2011

Toll-like receptors (TLRs) are the principal mediators of rapid microbial recognition: the lipopolysaccharide (LPS) receptor TLR4 seems to have a paradigmatic role. Single nucleotide polymorphisms (SNPs) in the TLR4 gene, such as +896A/G, known to attenuate receptor signaling, have been described. The +896A/G SNP is significantly less frequent in patients with myocardial infarction, Alzheimer's disease or prostate cancer, whereas it is overrepresented in centenarians. To clarify and confirm the biological effects of +896A/G SNP and its role in the pathophysiology of age-related diseases and longevity, we assessed the levels of IL-6, TNF-α, IL-10 and eicosanoids (LTB4 and PGE2) in LPS-stimul…

AdultLipopolysaccharidesMaleAgingAgeing Cytokines Eicosanoids Genetics Inflammation Longevity TLR4PopulationInflammationSingle-nucleotide polymorphismBiologyLeukotriene B4Polymorphism Single NucleotideDinoprostonemedicineHumansSNPeducationReceptorSettore MED/04 - Patologia Generaleeducation.field_of_studyMiddle AgedToll-Like Receptor 4ItalyEicosanoidImmunologyTLR4CytokinesFemalelipids (amino acids peptides and proteins)medicine.symptomDevelopmental BiologyEicosanoid ProductionMechanisms of Ageing and Development
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Downregulation of miRNA17-92 cluster marks Vγ9Vδ2 T cells from patients with rheumatoid arthritis

2018

Abstract Background We aimed to evaluate the phenotype, function, and microRNA (miRNA)17–92 cluster expression in Vγ9Vδ2 T-cell subsets and the correlation with immune response in rheumatoid arthritis (RA) patients. Methods Peripheral blood from 10 early RA untreated patients and 10 healthy donors (HD) was obtained. Polyclonal Vγ9Vδ2 T-cell lines were generated and analysed by flow cytometry. Analysis of miRNA17–92 cluster expression was performed by real-time polymerase chain reaction (RT-PCR), and expression of mRNA target genes was also studied. Results A remarkable change in the distribution of Vγ9Vδ2 T-cell functional subsets was observed in the peripheral blood of RA patients compared…

AdultMale0301 basic medicinemiRNA17–92lcsh:Diseases of the musculoskeletal systemInflammatory cytokineImmunologyDown-RegulationBiologyγδ T cellsProinflammatory cytokineFlow cytometryArthritis RheumatoidPathogenesis03 medical and health sciences0302 clinical medicineImmune systemRheumatologyT-Lymphocyte SubsetsInflammatory cytokines; miRNA17-92; Rheumatoid arthritis; γδ T cells; Rheumatology; Immunology and Allergy; ImmunologymicroRNAmedicineHumansImmunology and AllergyRheumatoid arthritisRheumatoid arthritiγδ T cellmedicine.diagnostic_testEffectorInterleukinMiddle AgedInflammatory cytokinesPhenotypemiRNA17-92MicroRNAsSettore MED/16 - Reumatologia030104 developmental biology030220 oncology & carcinogenesisImmunologyFemalelcsh:RC925-935Research Article
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