Search results for " Cellular"

showing 10 items of 384 documents

A Language Shift Simulation Based on Cellular Automata

2011

Language extinction is a widespread social phenomenon affecting several million people throughout the world today. By the end of this century, more than 5100 of the approximately 6000 languages currently spoken around the world will have disappeared. This is mainly because of language shifts, i.e., because a community of speakers stops using their traditional language and speaks a new one in all communication settings. In this study, the authors present the properties of a cellular automaton that incorporates some assumptions from the Gaelic-Arvanitika model of language shifts and the findings on the dynamics of social impacts in the field of social psychology. To assess the cellular automa…

Theoretical computer scienceStochastic cellular automatonLanguage shiftComputer scienceSimulation basedCellular automatonMobile automaton
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Diagrammatic approach to cellular automata and the emergence of form with inner structure

2018

We present a diagrammatic method to build up sophisticated cellular automata (CAs) as models of complex physical systems. The diagrams complement the mathematical approach to CA modeling, whose details are also presented here, and allow CAs in rule space to be classified according to their hierarchy of layers. Since the method is valid for any discrete operator and only depends on the alphabet size, the resulting conclusions, of general validity, apply to CAs in any dimension or order in time, arbitrary neighborhood ranges and topology. We provide several examples of the method, illustrating how it can be applied to the mathematical modeling of the emergence of order out of disorder. Specif…

Theoretical computer scienceStructure (category theory)Physical systemFOS: Physical sciencesPattern Formation and Solitons (nlin.PS)01 natural sciences010305 fluids & plasmasOperator (computer programming)0103 physical sciences010306 general physicsTopology (chemistry)Mathematical PhysicsMathematicsComplement (set theory)Numerical AnalysisHierarchy (mathematics)Applied MathematicsCellular Automata and Lattice Gases (nlin.CG)Mathematical Physics (math-ph)Nonlinear Sciences - Pattern Formation and SolitonsCellular automatonNonlinear Sciences - Adaptation and Self-Organizing SystemsDiagrammatic reasoningModeling and SimulationAlgorithmAdaptation and Self-Organizing Systems (nlin.AO)Nonlinear Sciences - Cellular Automata and Lattice Gases
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Quantum versus Probabilistic One-Way Finite Automata with Counter

2001

The paper adds the one-counter one-way finite automaton [6] to the list of classical computing devices having quantum counterparts more powerful in some cases. Specifically, two languages are considered, the first is not recognizable by deterministic one-counter one-way finite automata, the second is not recognizable with bounded error by probabilistic one-counter one-way finite automata, but each recognizable with bounded error by a quantum one-counter one-way finite automaton. This result contrasts the case of one-way finite automata without counter, where it is known [5] that the quantum device is actually less powerful than its classical counterpart.

TheoryofComputation_COMPUTATIONBYABSTRACTDEVICESNested wordComputer scienceTimed automatonBüchi automatonω-automatonNondeterministic finite automaton with ε-movesTuring machinesymbols.namesakeDFA minimizationDeterministic automatonContinuous spatial automatonQuantum finite automataDeterministic system (philosophy)Two-way deterministic finite automatonNondeterministic finite automatonDiscrete mathematicsFinite-state machineQuantum dot cellular automatonNonlinear Sciences::Cellular Automata and Lattice GasesMobile automatonTheoryofComputation_MATHEMATICALLOGICANDFORMALLANGUAGESDeterministic finite automatonProbabilistic automatonsymbolsAutomata theoryComputer Science::Formal Languages and Automata TheoryQuantum cellular automaton
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Minimal forbidden words and factor automata

1998

International audience; Let L(M) be the (factorial) language avoiding a given antifactorial language M. We design an automaton accepting L(M) and built from the language M. The construction is eff ective if M is finite. If M is the set of minimal forbidden words of a single word v, the automaton turns out to be the factor automaton of v (the minimal automaton accepting the set of factors of v). We also give an algorithm that builds the trie of M from the factor automaton of a single word. It yields a non-trivial upper bound on the number of minimal forbidden words of a word.

TheoryofComputation_COMPUTATIONBYABSTRACTDEVICESfailure functionfactor code[INFO.INFO-DS]Computer Science [cs]/Data Structures and Algorithms [cs.DS]Büchi automatonComputerApplications_COMPUTERSINOTHERSYSTEMS[INFO.INFO-DS] Computer Science [cs]/Data Structures and Algorithms [cs.DS]0102 computer and information sciencesavoiding a wordω-automaton01 natural sciencesfactorial languageReversible cellular automatonCombinatoricsDeterministic automatonanti-factorial languageNondeterministic finite automaton0101 mathematicsMathematicsfactor automatonPowerset constructionLevenshtein automaton010102 general mathematicsforbidden wordComputer Science::Computation and Language (Computational Linguistics and Natural Language and Speech Processing)16. Peace & justiceNonlinear Sciences::Cellular Automata and Lattice GasesTheoryofComputation_MATHEMATICALLOGICANDFORMALLANGUAGES010201 computation theory & mathematicsProbabilistic automatonPhysics::Accelerator PhysicsComputer Science::Programming LanguagesHigh Energy Physics::ExperimentComputer Science::Formal Languages and Automata Theory
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Digital calculus: Cellular automata dynamics in closed form

2015

A simple mathematical expression for the universal map for cellular automata is found in closed form with the help of a digit function, whose most basic properties are established. This result is found after proving a theorem on the composition of functions on finite sets. The expression (and the technique used to obtain it) opens the possibility of gaining mathematical insight in any cellular automaton rule since it constitutes at the same time a simple and fast algorithm to implement any such rule.

TheoryofComputation_MATHEMATICALLOGICANDFORMALLANGUAGESCellular Automata and Lattice Gases (nlin.CG)FOS: Physical sciencesNonlinear Sciences::Cellular Automata and Lattice GasesNonlinear Sciences - Cellular Automata and Lattice Gases
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Serine- and Threonine/Valine-Dependent Activation of PDK and Tor Orthologs Converge on Sch9 to Promote Aging

2014

Dietary restriction extends longevity in organisms ranging from bacteria to mice and protects primates from a variety of diseases, but the contribution of each dietary component to aging is poorly understood. Here we demonstrate that glucose and specific amino acids promote stress sensitization and aging through the differential activation of the Ras/cAMP/PKA, PKH1/2 and Tor/S6K pathways. Whereas glucose sensitized cells through a Ras-dependent mechanism, threonine and valine promoted cellular sensitization and aging primarily by activating the Tor/S6K pathway and serine promoted sensitization via PDK1 orthologs Pkh1/2. Serine, threonine and valine activated a signaling network in which Sch…

ThreonineCancer ResearchAgingSerineMice0302 clinical medicineSettore BIO/13 - Biologia ApplicataGene Expression Regulation FungalMolecular Cell BiologySerineSignaling in Cellular ProcessesThreonineGenetics (clinical)Cellular Stress Responses0303 health sciencesageing longevity Sch9 Tor Pkhs nutrients amino acidssurvival stress resistanceMechanisms of Signal TransductionValineCell biologyBiochemistryPhosphorylationSignal transductionResearch ArticleSignal TransductionSaccharomyces cerevisiae Proteinslcsh:QH426-470Adenylyl Cyclase Signaling PathwayLongevityP70-S6 Kinase 1Ras SignalingSaccharomyces cerevisiaeBiologyMicrobiologySignaling Pathways3-Phosphoinositide-Dependent Protein Kinases03 medical and health sciencesModel OrganismsStress PhysiologicalGeneticsAnimalsGene NetworksProtein kinase AMolecular BiologyTranscription factorBiologyEcology Evolution Behavior and Systematics030304 developmental biologySerine/threonine-specific protein kinase[SDV.GEN]Life Sciences [q-bio]/GeneticsCyclic AMP-Dependent Protein Kinaseslcsh:GeneticsGlucoseFoodTor SignalingProtein Kinases030217 neurology & neurosurgeryTranscription Factors
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Caspase-3 contributes to ZO-1 and Cl-5 tight-junction disruption in rapid anoxic neurovascular unit damage.

2011

BACKGROUND: Tight-junction (TJ) protein degradation is a decisive step in hypoxic blood-brain barrier (BBB) breakdown in stroke. In this study we elucidated the impact of acute cerebral ischemia on TJ protein arrangement and the role of the apoptotic effector protease caspase-3 in this context. METHODOLOGY/PRINCIPAL FINDINGS: We used an in vitro model of the neurovascular unit and the guinea pig whole brain preparation to analyze with immunohistochemical methods the BBB properties and neurovascular integrity. In both methodological approaches we observed rapid TJ protein disruptions after 30 min of oxygen and glucose deprivation or middle cerebral artery occlusion, which were accompanied by…

Time FactorsAnatomy and Physiologylcsh:MedicineMiceMolecular Cell BiologyPathologySignaling in Cellular ProcessesHypoxia Brainlcsh:ScienceCells CulturedNeuropathologyApoptotic SignalingMultidisciplinaryTight junctionCaspase 3ChemistryAnimal ModelsCell biologyTransport proteinProtein Transportmedicine.anatomical_structureNeurologyBlood-Brain BarrierMedicineResearch ArticleSignal TransductionClinical Research DesignCerebrovascular DiseasesGuinea PigsIschemiaContext (language use)Caspase 3Protein degradationBlood–brain barrierNeurological SystemTight JunctionsCapillary PermeabilityModel OrganismsDiagnostic MedicinemedicineAnimalsTransient Ischemic AttacksAnimal Models of DiseaseClaudinBiologyIschemic Strokelcsh:REndothelial CellsMembrane ProteinsPhosphoproteinsmedicine.diseaseAnatomical PathologyClaudinsImmunologyZonula Occludens-1 ProteinNervous System Componentslcsh:QPLoS ONE
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Multipotent Neural Stem Cells Reside into the Rostral Extension and Olfactory Bulb of Adult Rodents

2002

The lateral walls of the forebrain lateral ventricles are the richest source of stem cells in the adult mammalian brain. These stem cells give rise to new olfactory neurons that are renewed throughout life. The neurons originate in the subventricular zone (SVZ), migrate within the rostral extension (RE) of the SVZ along the rostral migratory stream (RMS) within tube-like structures formed of glial cells, to eventually reach the olfactory bulb (OB). We demonstrate that, contrary to the current view, multipotential (neuronal-astroglial-oligodendroglial) precursors with stem cell features can be isolated not only from the SVZ but also from the entire RE, including the distal portion within the…

Time FactorsRostral migratory streamanimal diseasesCell Culture TechniquesSubventricular zoneCell SeparationBiologyCell LineMiceCell MovementLateral VentriclesSpheroids CellularNeurospheremedicineAnimalsARTICLEGrowth SubstancesCells CulturedNeuronsNeurotransmitter AgentsStem CellsGeneral NeuroscienceNeurogenesisCell DifferentiationOlfactory BulbNeural stem cellClone CellsNeuroepithelial cellOligodendrogliaPhenotypemedicine.anatomical_structurenervous systemAstrocytesStem cellNeuroscienceCell DivisionAdult stem cellThe Journal of Neuroscience
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Role of tir and intimin in the virulence of rabbit enteropathogenic Escherichia coli serotype O103:H2.

2000

ABSTRACT Attaching and effacing (A/E) rabbit enteropathogenic Escherichia coli (REPEC) strains belonging to serogroup O103 are an important cause of diarrhea in weaned rabbits. Like human EPEC strains, they possess the locus of enterocyte effacement clustering the genes involved in the formation of the A/E lesions. In addition, pathogenic REPEC O103 strains produce an Esp-dependent but Eae (intimin)-independent alteration of the host cell cytoskeleton characterized by the formation of focal adhesion complexes and the reorganization of the actin cytoskeleton into bundles of stress fibers. To investigate the role of intimin and its translocated coreceptor (Tir) in the pathogenicity of REPEC, …

Time Factors[SDV]Life Sciences [q-bio]MutantAdministration OralPATHOGENICITEmedicine.disease_causeBacterial AdhesionMICROSCOPIE ELECTRONIQUE A TRANSMISSIONFecesCytoskeleton0303 health sciencesVirulenceEscherichia coli ProteinsEnterobacteriaceae3. Good health[SDV] Life Sciences [q-bio]IntestinesInfectious DiseasesMolecular and Cellular PathogenesisRabbitsLocus of enterocyte effacementBacterial Outer Membrane ProteinsImmunologyMolecular Sequence DataVirulenceReceptors Cell SurfaceBiologyMicrobiologydigestive systemMicrobiologyCell Line03 medical and health sciencesBacterial ProteinsIleummedicineEscherichia coliAnimalsHumansEnteropathogenic Escherichia coliAdhesins BacterialEscherichia coli030304 developmental biologyIntiminModels Genetic030306 microbiologyGenetic Complementation TestEpithelial Cellsbiochemical phenomena metabolism and nutritionbiology.organism_classificationActin cytoskeleton[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyActinsKineticsMicroscopy ElectronMicroscopy FluorescenceMutagenesisParasitologyCarrier ProteinsHeLa CellsInfection and immunity
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T-bet and mucosal Th1 responses in the gastrointestinal tract

2002

T cells play an essential role in regulating mucosal immune responses in the gastrointestinal tract. Recent observations on T helper cell differentiation and activation by regulatory transcription factors-especially T-bet-in chronic inflammatory diseases have provided new perspectives for understanding mucosal immunity. Here we summarise recent advances in the field of transcription factors and discuss the implications of these findings for future therapeutic approaches in inflammatory bowel diseases. In particular, we have focused on the role of T-bet in controlling mucosal Th1 responses in the gastrointestinal tract.

Transcription GeneticCellular differentiationGene Expressionchemical and pharmacologic phenomenaInflammationLeading ArticleBiologyInterferon-gammaMiceImmune systemImmunopathologymedicineAnimalsHumansT-helper cell differentiationImmunity MucosalTranscription factorImmunity CellularGastrointestinal tractT-cell receptorGastroenterologyCell DifferentiationTh1 CellsInflammatory Bowel DiseasesGastric MucosaImmunologyCytokinesmedicine.symptomT-Box Domain ProteinsDigestive SystemInterleukin-1Transcription FactorsGut
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