Search results for " EXPERIMENTAL"

showing 10 items of 3530 documents

Effects of streptozotocin-induced diabetes, physical training and their combination on collagen biosynthesis in rat skeletal muscle.

1995

The effects of streptozotocin-induced diabetes, physical training and their combination on the activities of prolyl 4-hydroxylase (PH) and galactosylhydroxylysyl glucosyl-transferase (GGT), both marker enzymes of collagen biosynthesis, and on the concentration of hydroxyproline (Hyp) were studied in vastus lateralis, rectus femoris and gastrocnemius muscles in rats. The experimental period was 12-16 weeks. Diabetes had an overall decreasing effect on specific PH activity in all muscles studied, whereas specific GGT activity remained at control level. Total PH and GGT activities decreased in all three muscles in the diabetic animals (P < 0.001). Training caused an increase in PH and GGT acti…

Blood GlucoseMalemedicine.medical_specialtyPhysiologyProcollagen-Proline DioxygenasePhysical exerciseProcollagen glucosyltransferaseDiabetes Mellitus ExperimentalRats Sprague-DawleyHydroxyprolinechemistry.chemical_compoundInternal medicineDiabetes mellitusPhysical Conditioning AnimalGlycosyltransferasemedicineAnimalsInsulinMuscle Skeletalchemistry.chemical_classificationbiologyBody WeightSkeletal musclemedicine.diseaseStreptozotocinRatsHydroxyprolineEnzymemedicine.anatomical_structureEndocrinologychemistryGlucosyltransferasesbiology.proteinCollagenmedicine.drugActa physiologica Scandinavica
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Effects of acute exercise, exercise training, and diabetes on the expression of lymphangiogenic growth factors and lymphatic vessels in skeletal musc…

2007

Blood and lymphatic vessels together form the circulatory system, allowing the passage of fluids and molecules within the body. Recently we showed that lymphatic capillaries are also found in the capillary bed of skeletal muscle. Exercise is known to induce angiogenesis in skeletal muscle, but it is not known whether exercise has effects on lymphangiogenesis or lymphangiogenic growth factors. We studied lymphatic vessel density and expression of the main lymphangiogenic growth factors VEGF-C and VEGF-D and their receptor VEGFR-3 in response to acute running exercise and endurance exercise training in the skeletal muscle of healthy and diabetic mice. VEGF-C mRNA expression increased after t…

Blood GlucoseMalemedicine.medical_specialtyTime FactorsPhysiologyPhysical ExertionVascular Endothelial Growth Factor CVascular Endothelial Growth Factor DPhysical exercisePolymerase Chain ReactionDiabetes Mellitus ExperimentalMiceEndurance trainingPhysiology (medical)Internal medicinemedicineLymphatic vesselAnimalsRNA MessengerLymphangiogenesisMuscle SkeletalLymphatic Vesselsbusiness.industryBody WeightSkeletal muscleVascular Endothelial Growth Factor Receptor-3ImmunohistochemistryLymphangiogenesisLymphatic systemEndocrinologymedicine.anatomical_structureVascular endothelial growth factor CGene Expression RegulationCirculatory systemCardiology and Cardiovascular MedicinebusinessAmerican journal of physiology. Heart and circulatory physiology
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Time course of changes in serum glucose, insulin, lipids and tissue lipase activities in macrosomic offspring of rats with streptozotocin-induced dia…

1999

The aim of this investigation was to determine the time course of changes in serum glucose, insulin and lipid levels, as well as lipid and protein content and lipolytic activities in insulin target organs (liver, adipose tissue and muscle), in macrosomic offspring of streptozotocin-induced mildly hyperglycaemic rats. Food intake and nutritional efficiency were also evaluated. Mild hyperglycaemia in pregnant rats was induced by intraperitoneal injection of streptozotocin (40 mg/kg body weight) on day 5 of gestation. Control pregnant rats were injected with citrate buffer. At birth, macrosomic pups (birth weight &gt; 1.7 S.D. greater than the mean value for the control pups) had higher serum …

Blood GlucoseMalemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentLipolysisAdipose tissueBiologyDiabetes Mellitus ExperimentalFetal Macrosomiachemistry.chemical_compoundEatingInsulin resistanceSex FactorsDiabetes mellitusAdipocyteInternal medicinemedicineFetal macrosomiaAnimalsInsulinObesityRats WistarMuscle SkeletalInsulinLipid metabolismGeneral MedicineLipasemedicine.diseaseStreptozotocinLipidsRatsEndocrinologychemistryAdipose TissueLiverFemalemedicine.drugClinical science (London, England : 1979)
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Vascular Dysfunction in Experimental Diabetes Is Improved by Pentaerithrityl Tetranitrate but Not Isosorbide-5-Mononitrate Therapy

2011

OBJECTIVE Diabetes is associated with vascular oxidative stress, activation of NADPH oxidase, and uncoupling of nitric oxide (NO) synthase (endothelial NO synthase [eNOS]). Pentaerithrityl tetranitrate (PETN) is an organic nitrate with potent antioxidant properties via induction of heme oxygenase-1 (HO-1). We tested whether treatment with PETN improves vascular dysfunction in the setting of experimental diabetes. RESEARCH DESIGN AND METHODS After induction of hyperglycemia by streptozotocin (STZ) injection (60 mg/kg i.v.), PETN (15 mg/kg/day p.o.) or isosorbide-5-mononitrate (ISMN; 75 mg/kg/day p.o.) was fed to Wistar rats for 7 weeks. Oxidative stress was assessed by optical methods and o…

Blood GlucoseMalemedicine.medical_specialtyXanthine OxidaseEndocrinology Diabetes and MetabolismVasodilator AgentsOxidative phosphorylationIsosorbide Dinitratemedicine.disease_causeWeight GainNitric oxideDiabetes Mellitus Experimentalchemistry.chemical_compoundEnosInternal medicineInternal MedicinemedicineAnimalsPentaerythritol TetranitrateGene SilencingEndothelial dysfunctionRats WistarXanthine oxidaseGTP CyclohydrolaseNADPH oxidasebiologyNADPH Oxidasesmedicine.diseasebiology.organism_classificationStreptozotocinPharmacology and TherapeuticsRatsOxidative StressEndocrinologychemistryVasoconstrictionbiology.proteinEndothelium VascularReactive Oxygen SpeciesOxidative stressHeme Oxygenase-1medicine.drugDiabetes
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Autoimmune Diabetes Induced by the β-cell Toxin STZ: Immunity to the 60-kDa Heat Shock Protein and to Insulin

1994

Administered at a suitably low dose, the toxin streptozotocin (STZ) can trigger an autoimmune process leading to destruction of the beta-cells of the pancreatic islets. In this study, we examined specific immunological reactions in mice before and during the development of STZ-induced autoimmune diabetes. We now report that the development of spontaneous autoantibodies to insulin can serve as a marker of susceptibility to a low dose of STZ. Susceptible male mice of the C57BL/KsJ strain manifested such anti-insulin antibodies, and resistant female mice did not. Administration of a low dose of STZ (five daily doses each of 30 mg/kg) induced transient hyperglycemia approximately 20-30 days lat…

Blood GlucoseMalemedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentBiologyActive immunizationmedicine.disease_causeStreptozocinAutoimmune DiseasesDiabetes Mellitus ExperimentalAutoimmunityMiceInternal medicineDiabetes mellitusInternal MedicinemedicineAnimalsInsulinHeat-Shock ProteinsAutoantibodiesAutoimmune diseaseMice Inbred BALB CPancreatic isletsInsulinnutritional and metabolic diseasesmedicine.diseaseStreptozotocinMice Inbred C57BLEndocrinologymedicine.anatomical_structureFemaleImmunizationBeta cellmedicine.drugDiabetes
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Comparison of the Effects of Glibenclamide on Metabolic Parameters, GLUT1 Expression, and Liver Injury in Rats With Severe and Mild Streptozotocin-In…

2012

Background and Objective. Glucose transport via GLUT1 protein could be one of additional mechanisms of the antidiabetic action of sulfonylureas. Here, the GLUT1 gene and the protein expression was studied in rats in the course of severe and mild streptozotocin-induced diabetes mellitus and under glibenclamide treatment. Material and Methods. Severe and mild diabetes mellitus was induced using different streptozotocin doses and standard or high fat chow. Rats were treated with glibenclamide (2 mg/kg daily, per os for 6 weeks). The therapeutic effect of glibenclamide was monitored by measuring several metabolic parameters. The GLUT1 mRNA and the protein expression in the kidneys, heart, and l…

Blood GlucoseMalemedicine.medical_specialtymedicine.medical_treatmentGene ExpressionDiabetes Mellitus ExperimentalGlibenclamideInternal medicineDiabetes mellitusGlyburideInsulin SecretionmedicineAnimalsHypoglycemic AgentsInsulinRats WistarLiver injuryGlucose Transporter Type 1Kidneybiologybusiness.industryInsulinGlucose transporternutritional and metabolic diseasesGeneral Medicinemedicine.diseaseStreptozotocinRatsSulfonylurea Compoundsmedicine.anatomical_structureEndocrinologyLiverProtein Biosynthesisglibenclamide; GLUT1; kidney; streptozotocin; expressionbiology.proteinGLUT1businessmedicine.drugMedicina; Volume 48; Issue 10; Pages: 78
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The sodium-glucose co-transporter 2 inhibitor empagliflozin improves diabetes-induced vascular dysfunction in the streptozotocin diabetes rat model b…

2014

Objective In diabetes, vascular dysfunction is characterized by impaired endothelial function due to increased oxidative stress. Empagliflozin, as a selective sodium-glucose co-transporter 2 inhibitor (SGLT2i), offers a novel approach for the treatment of type 2 diabetes by enhancing urinary glucose excretion. The aim of the present study was to test whether treatment with empagliflozin improves endothelial dysfunction in type I diabetic rats via reduction of glucotoxicity and associated vascular oxidative stress. Methods Type I diabetes in Wistar rats was induced by an intravenous injection of streptozotocin (60 mg/kg). One week after injection empagliflozin (10 and 30 mg/kg/d) was adminis…

Blood GlucoseMalemedicine.medical_treatmentReceptor for Advanced Glycation End Productslcsh:MedicineGene ExpressionType 2 diabetesmedicine.disease_causeVascular MedicineGlucosidesMedicine and Health SciencesMedicineInsulinEndothelial dysfunctionReceptors Immunologiclcsh:ScienceMultidisciplinaryType 1 DiabetesCytokinesInflammation Mediatorsmedicine.drugSignal TransductionResearch Articlemedicine.medical_specialtyCardiologyBlood sugarStreptozocinCardiovascular PharmacologyDiabetes Mellitus ExperimentalDiabetes ComplicationsInternal medicineDiabetes mellitusEmpagliflozinDiabetes MellitusAnimalsRNA MessengerVascular DiseasesBenzhydryl CompoundsSodium-Glucose Transporter 2 InhibitorsPharmacologybusiness.industryInsulinlcsh:RHemodynamicsStreptozotocinmedicine.diseaseRatsOxidative StressEndocrinologyGlucoseMetabolic Disorderslcsh:QbusinessOxidative stressDiabetic AngiopathiesPloS one
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Intraportal transplantation of allogenic pancreatic islets encapsulated in barium alginate beads in diabetic rats.

2003

The survival of microencapsulated islets transplanted into the unmodified peritoneal cavity is limited, even if capsular overgrowth is restricted to a minimum, due to an insufficient oxygen supply to the islets. Therefore, research efforts should focus on finding or creating a transplantation site, which permits a closer contact between the encapsulated islets and the blood. For this reason, the liver could be an interesting candidate. The aim of the present study was to test the hypothesis that the intraportal transplantation of allogenic islets encapsulated in small-sized barium alginate beads is safe and succeeds to induce normoglycemia in diabetic rats. The intraportal transplantation o…

Blood Glucoseendocrine systemmedicine.medical_specialtyAlginatesDrug CompoundingBiomedical EngineeringIslets of Langerhans TransplantationMedicine (miscellaneous)Blood sugarBioengineeringDiabetes Mellitus ExperimentalBiomaterialsRats Sprague-Dawleychemistry.chemical_compoundPeritoneal cavityIslets of LangerhansGlucuronic AcidInternal medicineDiabetes mellitusmedicineAnimalsgeographyDrug Carriersgeography.geographical_feature_categoryCell DeathPancreatic isletsHexuronic AcidsGeneral MedicineGlucuronic acidIsletmedicine.diseaseSurgeryRatsTransplantationPortal Systemmedicine.anatomical_structureEndocrinologychemistryLiverRats Inbred LewDrug carrierArtificial organs
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Peroxisome Proliferator-Activated Receptor Deficiency Increases the Risk of Maternal Abortion and Neonatal Mortality in Murine Pregnancy with or with…

2006

We assessed the implication of peroxisome proliferator-activated receptor (PPAR) alpha deficiency in pregnancy outcome and neonatal survival and in the modulation of T cell differentiation in murine diabetic pregnancy and their offspring. Pregnant wild-type (WT) and PPAR alpha-null mice of C57BL/6J genetic background were rendered diabetic by five low doses of streptozotocin. We observed that, in the absence of diabetes, PPAR alpha deficiency resulted in an increase in abortion rate, i.e. 0% in WT mice vs. 20% in PPAR alpha-null mice [odds ratio (OR) = 14.33; P = 0.013]. Under diabetic conditions, the abortion rate was enhanced, i.e. 8.3% in WT mice vs. 50% in PPAR alpha-null mice (OR = 4.2…

Blood Glucosemedicine.medical_specialtyOffspringRatónT-LymphocytesPeroxisome proliferator-activated receptorBiologyPeroxisomeDiabetes Mellitus ExperimentalInterferon-gammaMiceEndocrinologyTh2 CellsDownregulation and upregulationPregnancyDiabetes mellitusInternal medicinemedicineAnimalsInsulinPPAR alphaLymphocyte CountRNA MessengerReceptorFetal Deathchemistry.chemical_classificationMice KnockoutPregnancy[SCCO.NEUR]Cognitive science/NeuroscienceCell DifferentiationTh1 CellsStreptozotocinmedicine.diseaseLipidsInterleukin-10Abortion SpontaneousMice Inbred C57BLPregnancy ComplicationsEndocrinologychemistry[ SCCO.NEUR ] Cognitive science/NeuroscienceCytokinesInterleukin-2FemaleInterleukin-4Spleenmedicine.drug
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Modulation of lipid metabolism by n-3 polyunsaturated fatty acids in gestational diabetic rats and their macrosomic offspring.

2005

The time course of changes in lipid metabolism by dietary n−3 PUFAs (polyunsaturated fatty acids) in streptozotocin-induced diabetic rats during pregnancy (days 12 and 21) and their macrosomic offspring at birth (day 0) and through adulthood (days 60 and 90) was studied with respect to adipose tissue, liver and serum lipid concentrations, and fatty acid composition. Glucose and insulin levels were also assessed in order to characterize the diabetic state of macrosomic offspring. Pregnant diabetic and control rats were fed either an Isio-4 or EPAX diet (enriched with n−3 PUFA). The same diets were also consumed by pups at weaning. Compared with control rats, during pregnancy diabetic rats ha…

Blood Glucosemedicine.medical_specialtyOffspringmedicine.medical_treatmentBiologyDiabetes Mellitus ExperimentalFetal Macrosomiachemistry.chemical_compoundDietary Fats UnsaturatedPregnancyDiabetes mellitusInternal medicinemedicineAnimalsInsulinRats Wistarchemistry.chemical_classificationTriglycerideCholesterolInsulinBody WeightLipid metabolismGeneral MedicineOrgan Sizemedicine.diseaseLipid MetabolismRatsDiabetes GestationalEndocrinologychemistryAdipose TissueAnimals NewbornLiverDocosahexaenoic acidlipids (amino acids peptides and proteins)FemalePolyunsaturated fatty acidClinical science (London, England : 1979)
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