Search results for " EXPERIMENTAL"

showing 10 items of 3530 documents

Neuroergonomic Assessment of Hot Beverage Preparation and Consumption: An EEG and EDA Study.

2020

Neuroergonomics is an emerging field that investigates the human brain in relation to behavioral performance in natural environments and everyday settings. This study investigated the body and brain activity correlates of a typical daily activity, hot beverage preparation, and consumption in a realistic office environment where participants performed natural daily tasks. Using wearable, battery operated and wireless Electroencephalogram (EEG) and Electrodermal activity (EDA) sensors, neural and physiological responses were measured in untethered, freely moving participants who prepared hot beverages using two different machines (a market leader and follower as determined by annual US sales)…

Brain activity and meditationWearable computerElectroencephalography050105 experimental psychologyArousallcsh:RC321-571consumer neuroscience03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineNeuroergonomicsmedicineneuroergonomics0501 psychology and cognitive sciencesValence (psychology)lcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological PsychiatryOriginal Researchmedicine.diagnostic_testbusiness.industryelectroencephalogram (EEG)05 social sciencesUsabilityHuman NeurosciencePsychiatry and Mental healthNeuropsychology and Physiological Psychologymarket researchNeurologyemotional valenceelectrodermal activity (EDA)Consumer neurosciencePsychologybusiness030217 neurology & neurosurgeryCognitive psychologyFrontiers in human neuroscience
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The relation between workload and personal well-being among university professors

2019

The aim of the present study was to explore the level of perceived workload of university professors and to evaluate related psychosocial risks, such as addiction to work, psychological distress, and work–life conflicts. A total of 252 professors, balanced for gender (Mean age = 48.1; DS: 5.1) from 10 different universities across Europe participated in the research. Participants completed the University’s Work Environment Survey, the Dutch Workaholism Scale, and The General Health Questionnaire. The results showed that workload, when linked to bureaucratic university practices, is a predisposing factor to a negative perception of work-related well-being among university professors.

Bureaucratic practiceWorkaholismAddictionmedia_common.quotation_subjecteducation05 social sciencesApplied psychologyWell-being050109 social psychologyWorkloadWorkload050105 experimental psychologyWork environmentSettore M-PSI/04 - Psicologia Dello Sviluppo E Psicologia Dell'EducazioneScale (social sciences)Well-beingSettore M-PSI/06 - Psicologia Del Lavoro E Delle Organizzazioni0501 psychology and cognitive sciencesGeneral Health QuestionnaireNegative perceptionPsychologyPsychosocialhealth care economics and organizationsGeneral Psychologymedia_common
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TRADITION AND MODERNITY OF CATALAN VAULTS: HISTORICAL AND STRUCTURAL ANALYSIS

2012

Il lavoro analizza da diversi punti di vista un particolare tipo di strutture voltate noto come volte catalane (bóveda tabicada) e la tecnica costruttiva correlata. Le volte catalane sono volte realizzate da strati alterni di mattoni e malta, caratterizzate da un esiguo spessore in relazione alle altre dimensioni. Il primo passo presentato nel lavoro è quello di investigare tutti gli aspetti storici al fine di inquadrare sia la tipologia che la tecnica costruttiva correlata all'interno della storia dell'architettura e di individuare le ragioni di un così grande successo lungo i secoli. Successivamente, è stata effettuata una campagna di indagini sperimentali, che completa una precedente, su…

Bóvedas tabicadas historical research experimental analysis constitutive model numerical analysis.Settore ICAR/08 - Scienza Delle CostruzioniVolte Catalane Ricerca storica Analisi sperimentale Modello Costituivo Analisi numerica.
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T cell-mediated cytotoxicity: discrimination between antigen recognition, lethal hit and cytolysis phase.

1974

Using a 51Cr release cytotoxicity assay, the cytotoxic effector phase of in vitro activated mouse T lymphocytes (killer cells) against 51Cr-labeled target cells has been investigated. It is shown that within 5–10 minutes of contact between killer cells and target cells, the target cells are already committed to lysis, therefore, antigen recognition and “lethal hit” must have taken place within this period of time. In contrast, target cell lysis (cytolysis phase) requires up to 3–4 h in order to be completed; it occurs independently of killer cells and it is highly temperature dependent. The killer cell-dependent phase (antigen-recognition and “lethal hit”) is dissociated into two consecutiv…

C57BL/6MaleLysisTime FactorsCell SurvivalT-LymphocytesImmunologyAntigen-Antibody ReactionsMiceAntibody SpecificityImmunology and AllergyCytotoxic T cellAnimalsCytotoxicitybiologyEffectorTemperatureNeoplasms Experimentalbiology.organism_classificationCytotoxicity Tests ImmunologicVirologyIn vitroChromium RadioisotopesCell biologyMice Inbred C57BLCytolysisKineticsMice Inbred DBAMice Inbred CBAFemaleT cell mediated cytotoxicityLymphocyte Culture Test MixedEuropean journal of immunology
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The Cytokine GM-CSF Drives the Inflammatory Signature of CCR2+ Monocytes and Licenses Autoimmunity.

2015

Granulocyte-macrophage colony-stimulating factor (GM-CSF) has emerged as a crucial cytokine produced by auto-reactive T helper (Th) cells that initiate tissue inflammation. Multiple cell types can sense GM-CSF, but the identity of the pathogenic GM-CSF-responsive cells is unclear. By using conditional gene targeting, we systematically deleted the GM-CSF receptor (Csf2rb) in specific subpopulations throughout the myeloid lineages. Experimental autoimmune encephalomyelitis (EAE) progressed normally when either classical dendritic cells (cDCs) or neutrophils lacked GM-CSF responsiveness. The development of tissue-invading monocyte-derived dendritic cells (moDCs) was also unperturbed upon Csf2r…

CCR2Myeloidmedicine.medical_treatmentInterleukin-1betaAutoimmunitymedicine.disease_causeMonocytesAutoimmunityCytokine Receptor Common beta Subunit0302 clinical medicineSTAT5 Transcription FactorImmunology and AllergyAntigens LyMyeloid CellsPhosphorylationMice Knockout0303 health sciencesReverse Transcriptase Polymerase Chain ReactionExperimental autoimmune encephalomyelitisGene targetingFlow CytometryInfectious DiseasesCytokinemedicine.anatomical_structureGranulocyte macrophage colony-stimulating factor2723 Immunology and Allergymedicine.symptommedicine.drugSignal TransductionEncephalomyelitis Autoimmune ExperimentalReceptors CCR2Immunology610 Medicine & healthInflammationMice TransgenicBiology03 medical and health sciencesmedicineAnimalsHumans030304 developmental biologyInflammation2403 ImmunologyGranulocyte-Macrophage Colony-Stimulating Factor2725 Infectious DiseasesDendritic Cellsmedicine.disease10040 Clinic for NeurologyImmunologyTranscriptome030217 neurology & neurosurgery
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Antibodies to PECAM-1 and glucocorticoids reduce leukocyte adhesion in adjuvant arthritis of the rat knee synovium in vivo.

2002

Objective and design: To demonstrate the effect of monoclonal antibodies to the adhesion-molecule PECAM-1 (CD31) and of prednisolone on leukocyte adhesion in rat adjuvant arthritis.¶Material: Adjuvant arthritis was induced in male CD®-rats (five groups of n=6) 18 days prior to measurements.¶Treatment: Mouse-monoclonal antibody to rat CD-31 at 200 μg/kg or prednisolone at 24 mg/kg were administered i.v. 15 minutes prior to measurements.¶Methods: Venules within the intact rat-knee synovium were focused by confocal laser scanning microscopy. Numbers of rolling and adherent leukocytes were assessed in vivo.¶Results: Induction of arthritis significantly increased rolling and adherent leukocytes …

CD31Malemedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentPrednisoloneImmunologyAnti-Inflammatory AgentsArthritisMonoclonal antibodyLeukocyte CountIn vivoInternal medicineSynovial FluidmedicineCell AdhesionLeukocytesAnimalsAntibodies BlockingGlucocorticoidsPharmacologyMicroscopy Confocalbusiness.industryAntibodies Monoclonalmedicine.diseaseArthritis ExperimentalRheumatologyRatsPlatelet Endothelial Cell Adhesion Molecule-1Rheumatoid arthritisImmunologyPrednisoloneJointsbusinessAdjuvantmedicine.drugInflammation research : official journal of the European Histamine Research Society ... [et al.]
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Expression of Putative Fatty Acid Transporter Genes Are Regulated by Peroxisome Proliferator-activated Receptor α and γ Activators in a Tissue- and I…

1998

Regulation of gene expression of three putative long-chain fatty acid transport proteins, fatty acid translocase (FAT), mitochondrial aspartate aminotransferase (mAspAT), and fatty acid transport protein (FATP), by drugs that activate peroxisome proliferator-activated receptor (PPAR) alpha and gamma were studied using normal and obese mice and rat hepatoma cells. FAT mRNA was induced in liver and intestine of normal mice and in hepatoma cells to various extents only by PPARalpha-activating drugs. FATP mRNA was similarly induced in liver, but to a lesser extent in intestine. The induction time course in the liver was slower for FAT and FATP mRNA than that of an mRNA encoding a peroxisomal en…

CD36 AntigensMalemedicine.medical_specialtyAdipatesOrganic Anion TransportersReceptors Cytoplasmic and NuclearPeroxisome proliferator-activated receptorWhite adipose tissueBiologyMicrobodiesBiochemistryMiceLiver Neoplasms ExperimentalDiethylhexyl PhthalateInternal medicineBrown adipose tissueTumor Cells CulturedmedicineAnimalsClofibrateRNA MessengerMolecular BiologyDNA Primerschemistry.chemical_classificationMembrane GlycoproteinsBase SequenceFatty Acid Transport ProteinsFatty acidTroglitazoneCell BiologyPeroxisomeRatsPyrimidinesEndocrinologymedicine.anatomical_structureAdipose TissueGene Expression RegulationLiverchemistryPeroxisome proliferator-activated receptor alphaTranscription Factorsmedicine.drugJournal of Biological Chemistry
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SOCS3 transactivation by PPARγ prevents IL-17-driven cancer growth.

2013

Abstract Activation of the transcription factor PPARγ by the n-3 fatty acid docosahexaenoic acid (DHA) is implicated in controlling proinflammatory cytokine secretion, but the intracellular signaling pathways engaged by PPARγ are incompletely characterized. Here, we identify the adapter-encoding gene SOCS3 as a critical transcriptional target of PPARγ. SOCS3 promoter binding and gene transactivation by PPARγ was associated with a repression in differentiation of proinflammatory T-helper (TH)17 cells. Accordingly, TH17 cells induced in vitro displayed increased SOCS3 expression and diminished capacity to produce interleukin (IL)-17 following activation of PPARγ by DHA. Furthermore, naïve CD4…

CD4-Positive T-LymphocytesCancer ResearchAngiogenesisMammary Neoplasms Experimental/genetics/pathology/prevention & controlSuppressor of Cytokine Signaling Proteinsddc:616.07BioinformaticsTransactivationMice0302 clinical medicineTumor Burden/drug effects/geneticsSOCS3Docosahexaenoic Acids/administration & dosage/pharmacologyPromoter Regions GeneticMice Knockout0303 health sciencesMice Inbred BALB CChemistryReverse Transcriptase Polymerase Chain ReactionInterleukin-17InterleukinCell DifferentiationCell biologyTumor BurdenOncology030220 oncology & carcinogenesisFemaleRNA InterferenceInterleukin 17Th17 Cells/drug effects/metabolismTranscriptional ActivationDocosahexaenoic AcidsBlotting WesternMice NudeCD4-Positive T-Lymphocytes/drug effects/metabolismProinflammatory cytokine03 medical and health sciencesSuppressor of Cytokine Signaling Proteins/genetics/metabolismCell Line TumorAnimalsTranscription factor030304 developmental biologyMammary Neoplasms ExperimentalPromoter Regions Genetic/geneticsDietMice Inbred C57BLPPAR gammaInterleukin-17/metabolismCell cultureSuppressor of Cytokine Signaling 3 ProteinCell Differentiation/drug effectsPPAR gamma/agonists/genetics/metabolismTh17 CellsCancer research
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The role of CD8+ T cells and their local interaction with CD4+ T cells in myelin oligodendrocyte glycoprotein35-55-induced experimental autoimmune en…

2013

Abstract T cells have an essential role in the induction of multiple sclerosis and its animal model experimental autoimmune encephalomyelitis (EAE). Although for CD4+ T cells it is well established that they contribute to the disease, less is known about the role of CD8+ T cells. Our aim was to determine the individual contribution of CD4+ and CD8+ T cells in myelin oligodendrocyte glycoprotein (MOG)35–55–induced EAE. We investigated MOG35–55–activated CD8+ T cells to clarify their potential to induce or attenuate EAE. We monitored the behavior of CD8+ T cells and their interaction with CD4+ T cells directly at the site of inflammation in the CNS using intravital imaging of the brainstem of…

CD4-Positive T-LymphocytesCentral Nervous SystemEncephalomyelitis Autoimmune ExperimentalT cellImmunologyMedizinCell CommunicationCD8-Positive T-LymphocytesLymphocyte ActivationInterleukin 21MiceCell MovementmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorInflammationMice KnockoutCD40biologyCD28Molecular biologyPeptide FragmentsMice Inbred C57BLmedicine.anatomical_structureImmunologybiology.proteinInterleukin 12Myelin-Oligodendrocyte GlycoproteinCD8Journal of immunology (Baltimore, Md. : 1950)
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Cross-recognition of a myelin peptide by CD8+ T cells in the CNS is not sufficient to promote neuronal damage.

2015

Multiple sclerosis (MS) is an inflammatory disease of the CNS thought to be driven by CNS-specific T lymphocytes. Although CD8+T cells are frequently found in multiple sclerosis lesions, their distinct role remains controversial because direct signs of cytotoxicity have not been confirmedin vivo. In the present work, we determined that murine ovalbumin-transgenic (OT-1) CD8+T cells recognize the myelin peptide myelin oligodendrocyte glycoprotein 40–54 (MOG40–54) bothin vitroandin vivo. The aim of this study was to investigate whether such cross-recognizing CD8+T cells are capable of inducing CNS damagein vivo. Using intravital two-photon microscopy in the mouse model of multiple sclerosis, …

CD4-Positive T-LymphocytesCentral Nervous SystemMaleEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisAutoimmunityMice TransgenicCD8-Positive T-Lymphocytesmedicine.disease_causeMyelin oligodendrocyte glycoproteinMyelinMiceIn vivomedicineCytotoxic T cellAnimalsCells CulturedCell ProliferationbiologyCell DeathGeneral NeuroscienceMultiple sclerosisArticlesmedicine.diseaseMolecular mimicrymedicine.anatomical_structureImmunologyNerve Degenerationbiology.proteinFemaleMyelin-Oligodendrocyte GlycoproteinCD8Intravital microscopyThe Journal of neuroscience : the official journal of the Society for Neuroscience
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