Search results for " Ewing"

showing 8 items of 38 documents

Ultrastructure of one Ewing's sarcoma of bone with endothelial character and a comparative review of the vessels in 27 cases of typical Ewing's sarco…

1980

An atypical variant of Ewing's sarcoma, located in the left hip of a nine-year-old girl, is discussed at optical, histochemical and electron microscopical level. The endothelial appearing cells seem to play a main role in its histogenesis. Tumoral cells of an undifferentiated blastemic nature show round nuclei and bright lucent cytoplasm, being organized in solid sheets or vascular-like profiles. Alkaline and acid phosphatases are very prominent in all tumoral cells, and some of them also show PAS positive material. Its ultrastructure demonstrates an active pinocytic capacity, cytoplasmic filaments and Weibel-Palade bodies. Simultaneously a review is performed on 27 cases of typical Ewing's…

Pathologymedicine.medical_specialtyAcid PhosphataseBone NeoplasmsSarcoma EwingBiologyHistogenesisPathology and Forensic MedicineHemangioendotheliomamedicineHumansEndotheliumChildHipEwing's sarcomaCell BiologyAnatomymedicine.diseaseCytoplasmic filamentsAlkaline PhosphataseMicroscopy ElectronCytoplasmUltrastructureBlood VesselsPinocytosisFemaleSarcomaDifferential diagnosisPathology, research and practice
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Molecular Diagnosis of Ewing Sarcoma Family of Tumors

2009

To compare the sensitivity and specificity of fluorescence in situ hybridization (FISH) with reverse transcription polymerase chain reaction (RT-PCR) in the diagnosis of Ewing sarcoma family of tumors (ESFTs) and other small round-cell tumors (SRCTs) in formalin-fixed paraffin-embedded tissue assembled in tissue microarrays (TMAs). The second objective is to confirm the value of molecular methods and immunohistochemical (IHC) assays, to perform a differential diagnosis between ESFTs and SRCTs with similar or overlapping morphology.A total of 560 cases were selected for the present study out the 806 cases collected from the PROgnosis and THerapeutic Targets in the Ewing's Family of TumorS pr…

Pathologymedicine.medical_specialtyBone NeoplasmsSarcoma EwingIn situ hybridizationBiologyTranslocation GeneticPathology and Forensic MedicinePredictive Value of TestsBiomarkers TumormedicineHumansMolecular diagnostic techniquesRNA NeoplasmParaffin embeddingMolecular BiologyIn Situ Hybridization FluorescenceParaffin Embeddingmedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionDNA NeoplasmCell Biologymedicine.diseaseReverse transcription polymerase chain reactionReal-time polymerase chain reactionMolecular Diagnostic TechniquesTissue Array AnalysisFish <Actinopterygii>SarcomaFluorescence in situ hybridizationDiagnostic Molecular Pathology
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Notch signalling is off and is uncoupled from HES1 expression in Ewing's sarcoma

2010

Notch can act as an oncogene or as a tumour suppressor and thus can either promote or inhibit tumour cell growth. To establish Notch status in Ewing's sarcoma family of tumours (ESFT), we investigated the Notch pathway by gene expression profiling meta-analysis or immunohistochemistry in samples obtained from 96 and 24 ESFT patients, respectively. We found that although Notch receptors were highly expressed, Notch did not appear to be active, as evidenced by the absence of Notch receptors in cell nuclei. In contrast, we show that Notch receptors known to be active in colon adenocarcinoma, hepatocarcinoma, and pancreatic carcinoma stain cell nuclei in these tumours. High expression of the No…

Pathologymedicine.medical_specialtyCellNotch signaling pathwayBone NeoplasmsSarcoma EwingBiologyPathology and Forensic MedicineBasic Helix-Loop-Helix Transcription FactorsTumor Cells CulturedmedicineHumansHES1HEY1Transcription factorCell ProliferationCell NucleusHomeodomain ProteinsRegulation of gene expressionReceptors NotchCell growthGene Expression ProfilingNeoplasm ProteinsGene Expression Regulation Neoplasticmedicine.anatomical_structureNeoplastic Stem CellsCancer researchTranscription Factor HES-1Cyclin-dependent kinase 8Signal TransductionThe Journal of Pathology
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Scanning electron microscopic evidence for neural differentiation in Ewing's sarcoma cell lines.

1990

A number of recent studies have suggested a relationship between Ewing's sarcoma (ES) and other small round cell tumours of childhood such as peripheral neuroepithelioma (PN). We report scanning electron microscopic studies on the character of induced neural differentiation in ES, neuroblastoma, PN, osteosarcoma and colon carcinoma. We found evidence of neural differentiation in both neural lines and in one of two Ewing's lines before treatment. After differentiation, both Ewing's and neural lines developed neuritic processes with varicosities and little arborization, except for the initially undifferentiated Ewing's line (A4573) which displayed extensive lateral sprouting from neuritic pro…

Pathologymedicine.medical_specialtyImmunocytochemistrySarcoma EwingBiologyAdenocarcinomaPathology and Forensic MedicineCell LineNeuroblastomamedicineHumansNeuroectodermal Tumors Primitive PeripheralMolecular BiologyOsteosarcomaNeural tubeEwing's sarcomaCell BiologyGeneral Medicinemedicine.diseasemedicine.anatomical_structureCell Transformation NeoplasticCell cultureColonic NeoplasmsMicroscopy Electron ScanningOsteosarcomaSarcomaFilopodiaVirchows Archiv. A, Pathological anatomy and histopathology
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Activation of TRK Genes in Ewingʼs Sarcoma Trk A Receptor Expression Linked to Neural Differentiation

1997

Trk receptors have been identified by immunohistochemical methods in primitive neuroectodermal tumor (PNET)/Ewing's sarcoma (ES). However, the presence of different members of the Trk family of receptors in PNET/ES has not been specified. We have examined whether Trk A, B, and C receptors are specifically expressed in ES both with and without features of neural differentiation. Ten ES tumors (five primary tumors of bone and five extraosseous tumors transplanted into nude mice) were investigated for expression of Trk receptors by immunohistochemistry and reverse transcription-polymerase chain reaction. One primary ES and the five grafted ES tumors exhibited signs of neural differentiation; t…

animal structuresReceptor expressionReceptors Nerve Growth FactorSarcoma EwingBiologyPathology and Forensic MedicineMiceProto-Oncogene ProteinsmedicineAnimalsNeuroectodermal Tumors PrimitiveReceptor trkCReceptor trkAReceptorReceptor Ciliary Neurotrophic FactorMolecular BiologyNeuronsMembrane ProteinsReceptor Protein-Tyrosine KinasesEwing's sarcomaCell DifferentiationCell BiologyProtein-Tyrosine Kinasesmedicine.diseaseMolecular biologyGene Expression Regulation Neoplasticenzymes and coenzymes (carbohydrates)nervous systemTrk receptorPrimitive neuroectodermal tumorembryonic structuresImmunohistochemistrySarcomaImmunostainingDiagnostic Molecular Pathology
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Translocation (10;11;22)(p14;q24;q12) Characterized by Fluorescence in Situ Hybridization in a Case of Ewing's Tumor

2001

It is well recognized that the identification by classic cytogenetics of t(11;22)(q24;q12) is a useful aid in the accurate diagnosis of Ewing's sarcoma and related tumors. This translocation induces the EWS/FLI-1 fusion transcript, which can be detected by reverse transcription-polymerase chain reaction. Recent studies have also used fluorescence in situ hybridization (FISH) to demonstrate the translocation. The authors coupled classic cytogenetics and FISH on tumor cells from the original specimen, the local recurrence, and the pulmonary metastasis as well as from the xenografted tumors in a case of extraosseous Ewing's sarcoma. FISH analysis not only confirmed the cytogenetic results but …

medicine.medical_specialtyLung NeoplasmsOncogene Proteins FusionChromosomes Human Pair 22Bone NeoplasmsChromosomal translocationSarcoma EwingBiologyTranslocation GeneticPathology and Forensic MedicineImmunoenzyme TechniquesFatal OutcomemedicineHumansChildMolecular BiologyIn Situ Hybridization FluorescenceLegmedicine.diagnostic_testChromosomes Human Pair 10Proto-Oncogene Protein c-fli-1Reverse Transcriptase Polymerase Chain ReactionChromosomes Human Pair 11CytogeneticsChromosomeEwing's tumorDNA NeoplasmSequence Analysis DNACell Biologymedicine.diseaseCombined Modality TherapyFusion transcriptKaryotypingCancer researchFemaleInterphaseSarcomaRNA-Binding Protein EWSTranscription FactorsFluorescence in situ hybridizationDiagnostic Molecular Pathology
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The utility of SATB2 immunohistochemical expression in distinguishing between osteosarcomas and their malignant bone tumor mimickers, such as Ewing s…

2016

SATB2 is commonly expressed in osteosarcomas. Although apparently being a valuable diagnostic marker for differentiating between small cell osteosarcoma (SCO) and other small round cell tumors of bone, for instance Ewing sarcoma family of tumors (ESFT), it has not been tested in a large series of ESFT and chondrosarcomas so far. We studied the immunohistochemical expression of SATB2 in 42 osteosarcomas, 31 chondrosarcomas, and 371 genetically confirmed ESFT. SATB2 positivity was detected in 90.4% of osteosarcomas, 87.5% of SCO, 91.3% of osteoblastic osteosarcomas, and in all chondroblastic and parosteal osteosarcomas. The osteoblastic and SCO subtypes expressed SATB2 more intensely than oth…

musculoskeletal diseases0301 basic medicinePathologymedicine.medical_specialtyCD99ChondrosarcomaBone NeoplasmsSarcoma EwingSensitivity and SpecificitySmall Cell OsteosarcomaPathology and Forensic MedicineDiagnosis Differential03 medical and health sciences0302 clinical medicineChondroblastic OsteosarcomaBiomarkers TumormedicineHumansRetrospective StudiesOsteosarcomabusiness.industryOsteoidMatrix Attachment Region Binding ProteinsCell Biologymusculoskeletal systemmedicine.diseaseImmunohistochemistry030104 developmental biology030220 oncology & carcinogenesisImmunohistochemistryOsteosarcomaSarcomaChondrosarcomabusinessTranscription FactorsPathology - Research and Practice
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Estudio de la heterogeneidad fenotípica del sarcoma de Ewing en xenotrasplantes

2019

Los Tumores de la Familia del Sarcoma de Ewing (TFSE) son neoplasias malignas de hueso y de partes blandas, poco frecuentes (entre el 6-8% de los tumores malignos primarios de hueso) y que afectan principalmente a pacientes en edad de crecimiento, adolescentes y adultos jóvenes (1, 2). El término engloba a tres tumores que durante años han sido considerados por separado por su morfología característica, pero que forman un espectro morfológico, de biología e histogénesis comunes (3). Dichos tumores son los Sarcomas de Ewing clásicos, los atípicos y los tumores neuroectodérmicos primitivos periféricos (PNET). Desde el punto de vista morfológico son neoplasias constituidas por células pequeñas…

sarcoma de ewingsarcomainmunohistoquímicaUNESCO::CIENCIAS MÉDICASxenotrasplantesTMA:CIENCIAS MÉDICAS [UNESCO]PDX
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