Search results for " Gene Expression"

showing 5 items of 695 documents

Limited antibody specificity compromises epitranscriptomic analyses

2019

International audience; A controversial discussion on the occurrence of the RNA modification m1A in mRNA takes a new turn, as an antibody with a central role in modification mapping was shown to also bind mRNA cap structures.

0301 basic medicineScienceGeneral Physics and Astronomy02 engineering and technologyPlasma protein bindingAntibodiesGeneral Biochemistry Genetics and Molecular BiologyEpigenesis GeneticTranscriptome03 medical and health sciencesAntibody Specificity[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]AnimalsHumansRNA Messengerlcsh:ScienceEpigenesisRegulation of gene expressionMessenger RNAMultidisciplinarybiologyCommentQRNA[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyGeneral ChemistryDNA MethylationRNA modification021001 nanoscience & nanotechnologyCell biology030104 developmental biologyGene Expression RegulationDNA methylationbiology.proteinRNAlcsh:QAntibodyTranscriptome0210 nano-technologyProtein Binding
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MBOAT7 rs641738 variant and hepatocellular carcinoma in non-cirrhotic individuals

2017

AbstractNonalcoholic fatty liver disease (NAFLD) represents an emerging cause of hepatocellular carcinoma (HCC), especially in non-cirrhotic individuals. The rs641738 C > T MBOAT7/TMC4 variant predisposes to progressive NAFLD, but the impact on hepatic carcinogenesis is unknown. In Italian NAFLD patients, the rs641738 T allele was associated with NAFLD-HCC (OR 1.65, 1.08–2.55; n = 765), particularly in those without advanced fibrosis (p < 0.001). The risk T allele was linked to 3’-UTR variation in MBOAT7 and to reduced MBOAT7 expression in patients without severe fibrosis. The number of PNPLA3, TM6SF2, and MBOAT7 risk variants was associated with NAFLD-HCC independently of clinical fa…

0301 basic medicineLiver CirrhosisMaleAlcoholic liver diseasePathologyCirrhosisliver diseasesGastroenterology0302 clinical medicineSettore BIO/13 - Biologia ApplicataNon-alcoholic Fatty Liver DiseaseRisk FactorsNonalcoholic fatty liver diseasefatty liver-disease; cirrhosis; liver cancer; hepatitis C; hepatocellular carcinoma; liver diseases; fibrosis; carcinogenesis; fatty liver; allelesHCCProspective cohort studySettore MED/12 - GastroenterologiaMultidisciplinaryLiver NeoplasmsQRhepatocellular carcinomaSingle NucleotideMiddle Aged3. Good healthItalyfatty liver-diseaseHepatocellular carcinomaCohortMedicine030211 gastroenterology & hepatologyFemalecarcinogenesisAcyltransferases; Adult; Aged; Carcinoma Hepatocellular; Female; Gene Expression Regulation; Genotype; Humans; Italy; Liver Cirrhosis; Liver Neoplasms; Male; Membrane Proteins; Middle Aged; Non-alcoholic Fatty Liver Disease; Polymorphism Single Nucleotide; Risk Factors; Alleles; Genetic Association Studies; Genetic Predisposition to Disease; Genetic VariationAdultmedicine.medical_specialtyCarcinoma HepatocellularGenotypeSciencePolymorphism Single NucleotideArticleliver cancer03 medical and health sciencesInternal medicinemedicineHumansGenetic Predisposition to DiseaseAllelePolymorphismAllelesGenetic Association Studiesfatty liverAgedSettore MED/06 - ONCOLOGIA MEDICAbusiness.industrycirrhosisfibrosisCarcinomaGenetic VariationMembrane ProteinsHepatocellularmedicine.diseasedigestive system diseases030104 developmental biologyGene Expression Regulationhepatitis CbusinessAcyltransferasesTM6SF2Scientific Reports
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The mRNA degradation factor Xrn1 regulates transcription elongation in parallel to Ccr4

2019

Abstract Co-transcriptional imprinting of mRNA by Rpb4 and Rpb7 subunits of RNA polymerase II (RNAPII) and by the Ccr4–Not complex conditions its post-transcriptional fate. In turn, mRNA degradation factors like Xrn1 are able to influence RNAPII-dependent transcription, making a feedback loop that contributes to mRNA homeostasis. In this work, we have used repressible yeast GAL genes to perform accurate measurements of transcription and mRNA degradation in a set of mutants. This genetic analysis uncovered a link from mRNA decay to transcription elongation. We combined this experimental approach with computational multi-agent modelling and tested different possibilities of Xrn1 and Ccr4 acti…

Ribosomal ProteinsSaccharomyces cerevisiae ProteinsRNA StabilitymRNAMutantRNA polymerase IISaccharomyces cerevisiaeBiology03 medical and health sciencesGenomic Imprinting0302 clinical medicineRibonucleasesRibosomal proteinTranscription (biology)Gene Expression Regulation FungalGeneticsGenomesGene030304 developmental biologyRegulation of gene expression0303 health sciencesMessenger RNAGene regulation Chromatin and EpigeneticsFungal geneticsCell biologyExoribonucleasesbiology.proteinRNARNA Polymerase IIGenome FungalTranscriptional Elongation Factors030217 neurology & neurosurgery
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The PVT-1 oncogene is a Myc protein target that is overexpressed in transformed cells

2007

The human PVT-1 gene is located on chromosome 8 telomeric to the c-Myc gene and it is frequently involved in the translocations occurring in variant Burkitt's lymphomas and murine plasmacytomas. It has been proposed that PVT-1 regulates c-Myc gene transcription over a long distance. To get new insights into the functional relationships between the two genes, we have investigated PVT-1 and c-Myc expression in normal human tissues and in transformed cells. Our findings indicate that PVT-1 expression is restricted to a relative low number of normal tissues compared to the wide distribution of c-Myc mRNA, whereas the gene is highly expressed in many transformed cell types including neuroblastom…

PhysiologyClinical BiochemistryBiologyCell LineProto-Oncogene Proteins c-mycGenes ReporterNeoplasmsC-MYCAnimalsHumansTissue DistributionPromoter Regions GeneticGeneGENE-EXPRESSIONRegulation of gene expressionReporter geneOncogeneProteinsCell BiologyTransfectionMolecular biologyPVT1Cell Transformation NeoplasticGene Expression RegulationPVT-1Cell cultureRNA Long NoncodingChromatin immunoprecipitationJournal of Cellular Physiology
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Intra-individual Gene Expression Profiling of Peripheral Blood CD34+ Hematopoietic Stem Cells Mobilized by Two Different Protocols

2014

Hematopoietic Stem Cells Gene Expression Profiling Mobilization.
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