Search results for " Immune system"

showing 10 items of 893 documents

New insights into the pathogenesis of giant cell arteritis

2017

Giant cell arteritis (GCA) is an inflammatory chronic disease occurring exclusively in elderly individuals. Until recently, the disease has been considered a unique disease resulting from the interaction in the walls of susceptible arteries, between an unknown infectious agents with local dendritic cells (DCs), activated CD4 T cells and effector macrophages. Recent evidence has shown that this view was too simplistic and has clarified many of the pathogenetic aspects of the disease. Many genetic studies recently published have identified different new genes, including cytokines, adhesion molecules and regulators of innate immunity, as crucial players in the development and progression of GC…

030203 arthritis & rheumatology0301 basic medicineImmunology and Allergy; ImmunologyInnate immune systemGiant Cell ArteritisImmunologyContext (language use)DiseaseBiologymedicine.diseasePathogenesisSettore MED/16 - Reumatologia03 medical and health sciencesGiant cell arteritis030104 developmental biology0302 clinical medicineImmune systemLymphatic systemAntigenImmunologymedicineAnimalsHumansImmunology and Allergyskin and connective tissue diseases
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Interfering with MIF-CD74 signalling on macrophages and dendritic cells with a peptide-based approach restores the immune response against metastatic…

2018

ABSTRACTMounting an effective immune response against cancer requires the activation of innate and adaptive immune cells. Metastatic melanoma is the most aggressive form of skin cancer. Immunotherapies that boost the activity of effector T cells have shown a remarkable success in melanoma treatment. Patients, however, can develop resistance to such therapies by mechanisms that include the establishment of an immune suppressive tumour microenvironment. Understanding how metastatic melanoma cells suppress the immune system is vital to develop effective immunotherapies against this disease. In this study, we find that the innate immune cells, macrophages and dendritic cells are suppressed in m…

0303 health sciencesInnate immune systemCD74Effectorbusiness.industrymedicine.medical_treatmentMelanomaanimal diseasesCancerchemical and pharmacologic phenomenaImmunotherapymedicine.diseaseHedgehog signaling pathway3. Good health03 medical and health sciences0302 clinical medicineImmune systemCancer researchmedicinebusiness030304 developmental biology030215 immunology
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Mucin induces CRISPR-Cas defence in an opportunistic pathogen

2021

AbstractParasitism by bacteriophages has led to the evolution of a variety of defense mechanisms in their host bacteria. However, it is unclear what factors lead to specific defenses being deployed upon phage infection. To explore this question, we exposed the bacterial fish pathogenFlavobacterium columnareto its virulent phage V156 in the presence of a eukaryotic host signal (mucin). All tested conditions led to some level of innate immunity, but the presence of mucin led to a dramatic increase in CRISPR spacer acquisition, especially in low nutrient conditions where over 60% of colonies had obtained at least one new spacer. Additionally, we show that the presence of a competitor bacterium…

0303 health sciencesInnate immune systembiology030306 microbiologyMucinVirulencebiology.organism_classificationMicrobiology03 medical and health sciencesImmune systemFlavobacterium columnareCRISPRPathogenBacteria030304 developmental biology
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Mast cells as initiators of immunity and host defense

2001

Until recently, mast cells have been viewed primarily as harmful because of their key role as effector cells of allergic and potentially lethal anaphylactic reactions. Their contribution to human health appeared instead to be limited to the elimination of parasites. There is, however, growing evidence for additional beneficial functions of mast cells, particularly regarding the initiation of acquired immune reactions. Thus, mast cells can phagocytize diverse particles, take up antigens, and express a number of receptors, particularly MHC class I and II antigens, ICAM-1 and -3, CD43, CD80, CD86 and CD40L which allow them to interact with T and B lymphocytes. They can also secrete numerous cy…

0303 health sciencesInnate immune systembiologyDegranulationchemical and pharmacologic phenomenaDermatologyImmunoglobulin EAcquired immune systemMast cellBiochemistry3. Good healthInterleukin 3303 medical and health sciencesClassical complement pathway0302 clinical medicineImmune systemmedicine.anatomical_structureImmunologybiology.proteinmedicineMolecular Biology030304 developmental biology030215 immunologyExperimental Dermatology
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2'-O-methylation within prokaryotic and eukaryotic tRNA inhibits innate immune activation by endosomal Toll-like receptors but does not affect recogn…

2019

Bacterial RNA has emerged as an important activator of innate immune responses by stimulating Toll-like receptors TLR7 and TLR8 in humans. Guanosine 2′-O-methylation at position 18 (Gm18) in bacterial tRNA was shown to antagonize tRNA-induced TLR7/8 activation, suggesting a potential role of Gm18 as an immune escape mechanism. This modification also occurs in eukaryotic tRNA, yet a physiological immune function remained to be tested. We therefore set out to investigate the immune modulatory role of Gm18 in both prokaryotic and eukaryotic microorganisms, Escherichia coli and Saccharomyces cerevisiae, and in human cells. Using RiboMethSeq analysis we show that mutation of trmH in E. coli, trm…

0303 health sciencesTRNA modificationInnate immune system030302 biochemistry & molecular biologyRNA[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyTLR7BiologyTLR8[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyCell biology03 medical and health sciencesImmune system[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]Transfer RNAGene expression[SDV.BBM.GTP] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]Molecular BiologyComputingMilieux_MISCELLANEOUS030304 developmental biology
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Regulatory T cell-derived adenosine induces dendritic cell migration through the Epac-Rap1 pathway.

2014

Abstract Dendritic cells (DC) are one target for immune suppression by regulatory T cells (Treg), because their interaction results in reduced T cell stimulatory capacity and secretion of inhibitory cytokines in DC. We show that DC in the presence of Treg are more mobile as compared with cocultures with conventional CD4+ T cells and form DC–Treg aggregates within 2 h of culture. The migration of DC was specifically directed toward Treg, as Treg, but not CD4+ T cells, attracted DC in Boyden chambers. Treg deficient for the ectonucleotidase CD39 were unable to attract DC. Likewise, addition of antagonists for A2A adenosine receptors abolished the formation of DC–Treg clusters, indicating a ro…

AdenosineRegulatory T cellT cellImmunologyMedizinchemical and pharmacologic phenomenaCell CommunicationBiologyT-Lymphocytes RegulatoryMiceAdenosine TriphosphateAntigens CDCell MovementmedicineImmunology and AllergyAnimalsGuanine Nucleotide Exchange FactorsDendritic cell migrationReceptors Adenosine A2Apyraserap1 GTP-Binding Proteinshemic and immune systemsDendritic CellsActin cytoskeletonAdenosineAdenosine receptorCell biologyActin Cytoskeletonmedicine.anatomical_structureRap1Signal transductionmedicine.drugSignal TransductionJournal of immunology (Baltimore, Md. : 1950)
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Adiponectin and Orexin-A as a Potential Immunity Link Between Adipose Tissue and Central Nervous System

2018

Adipose tissue (AT) is strongly associated with development and progression of immune disorders through adipokines secretion, such as adiponectin. This protein has beneficial energetic properties and is involved in inflammation and immunity processes. Three oligomers of circulating Adiponectin with different molecular weight are described: High (HMW), Medium (MMW) and Low (LMW). The HMW is the most biologically active oligomers. On binding to its receptors AdipoR1, AdipoR2, and T-cadherin, adiponectin acts on both innate and acquired immunity. The suppression of NF-kB activation and pro-inflammatory cytokine expression in macrophages is mediated by AdipoR1. AdipoR2 mediates polarization of …

Adiponectin; Adipose tissue; Central nervous system; Immunity; Orexin-A0301 basic medicineNervous systemPhysiologyorexin-AAdipokineAdipose tissueInflammationReviewBiologylcsh:Physiology03 medical and health sciencesImmune systemImmunityPhysiology (medical)medicineAdiponectinadiponectinlcsh:QP1-981biochemical phenomena metabolism and nutritionAcquired immune systemcentral nervous systemimmunityadipose tissue030104 developmental biologymedicine.anatomical_structureImmunologymedicine.symptomFrontiers in Physiology
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Neuroimmune Activation and Myelin Changes in Adolescent Rats Exposed to High-Dose Alcohol and Associated Cognitive Dysfunction: A Review with Referen…

2014

Aims: The aim of the study was to assess whether intermittent ethanol administration to adolescent rats activates innate immune response and TLRs signalling causing myelin disruption and long-term cognitive and behavioural deficits. Methods: We used a rat model of intermittent binge-like ethanol exposure during adolescence. Results: Binge-like ethanol administration to adolescent rats increased the gene expression of TLR4 and TLR2 in the prefrontal cortex (PFC), as well as inflammatory cytokines TNF alpha and IL-1 beta. Up-regulation of TLRs and inflammatory mediators were linked with alterations in the levels of several myelin proteins in the PFC of adolescent rats. These events were assoc…

AdolescentAlcohol DrinkingGene ExpressionPrefrontal CortexBinge drinkingImpulsivityProinflammatory cytokineMyelinmedicineAnimalsHumansPrefrontal cortexMyelin SheathNeuroinflammationInnate immune systemEthanolGeneral MedicineImmunity InnateToll-Like Receptor 2RatsToll-Like Receptor 4medicine.anatomical_structureImmunologyTLR4Inflammation Mediatorsmedicine.symptomCognition DisordersPsychologySignal TransductionAlcohol and Alcoholism
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Endogenous CD83 Expression in CD4+ Conventional T Cells Controls Inflammatory Immune Responses

2020

Abstract The glycoprotein CD83 is known to be expressed by different immune cells including activated CD4+Foxp3+ regulatory T cells (Tregs) and CD4+Foxp3− conventional T cells. However, the physiological function of endogenous CD83 in CD4+ T cell subsets is still unclear. In this study, we have generated a new CD83flox mouse line on BALB/c background, allowing for specific ablation of CD83 in T cells upon breeding with CD4-cre mice. Tregs from CD83flox/flox/CD4-cretg/wt mice had similar suppressive activity as Tregs from CD83flox/flox/CD4-crewt/wt wild-type littermates, suggesting that endogenous CD83 expression is dispensable for the inhibitory capacity of Tregs. However, CD83-deficient CD…

Adoptive cell transferCD40biologyChemistryT cellImmunologyMedizinCD11cFOXP3chemical and pharmacologic phenomenahemic and immune systemsEndogenyMolecular biologyIn vitro03 medical and health sciences0302 clinical medicineImmune systemmedicine.anatomical_structurebiology.proteinmedicineImmunology and Allergy030215 immunologyThe Journal of Immunology
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Increasing Functional Avidity of T Cell Receptor (TCR)-Redirected T Cells by Removing Defined N-Glycoslyation Sites in the Constant Domain of Introdu…

2007

Abstract Adoptive transfer of T lymphocytes transduced with a TCR to impart tumor reactivity has been reported as potential strategy to redirect immune responses to target cancer cells. However, the affinities of most TCRs specific for shared tumor antigens that can be isolated are usually low, in part reflecting the nature of the targeted tumor antigens which are self-proteins. Thus strategies that can increase the affinity or functional avidity of TCRs to be used in therapy to transduce T cells might be therapeutically beneficial. However, current strategies for increasing TCR affinity require extensive and usually random mutagenesis followed by screening the many derived mutations, and m…

Adoptive cell transferChemistryImmunologyT-cell receptorhemic and immune systemschemical and pharmacologic phenomenaCell BiologyHematologyBiochemistryCell biologyImmune systemAntigenCancer cellCytotoxic T cellAvidityCD8Blood
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