Search results for " Models"

showing 10 items of 4240 documents

High expression of QSOX1 reduces tumorogenesis, and is associated with a better outcome for breast cancer patients.

2012

International audience; ABSTRACT: INTRODUCTION: The gene quiescin/sulfhydryl oxidase 1, QSOX1, encodes an enzyme directed to the secretory pathway and excreted into the extracellular space. QSOX1 participates in the folding and stability of proteins and thus could regulate the biological activity of its substrates in the secretory pathway and/or outside the cell. The involvement of QSOX1 in oncogenesis has been studied primarily in terms of its differential expression in systemic studies. QSOX1 is overexpressed in prostate cancers and in pancreatic adenocarcinoma. In contrast, QSOX1 gene expression is repressed in endothelial tumors. In the present study, we investigated the role of QSOX1 i…

CellGene ExpressionBreast Neoplasms[SDV.CAN]Life Sciences [q-bio]/CancerBiologymedicine.disease_causeMetastasis[ SDV.CAN ] Life Sciences [q-bio]/CancerMice03 medical and health sciences0302 clinical medicineBreast cancer[SDV.CAN] Life Sciences [q-bio]/CancerCell MovementCell Line TumormedicineExtracellularAnimalsHumansOxidoreductases Acting on Sulfur Group DonorsRNA MessengerNeoplasm MetastasisCell ProliferationRetrospective Studies030304 developmental biologyMedicine(all)0303 health sciencesCell growthCancermedicine.diseaseExtracellular MatrixTumor Burden3. Good healthPatient Outcome AssessmentDisease Models AnimalProtein TransportCell Transformation Neoplasticmedicine.anatomical_structure030220 oncology & carcinogenesisCancer researchHeterograftsAdenocarcinomaFemaleNeoplasm GradingCarcinogenesisResearch Article
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Dihydrocucurbitacin B Inhibits Delayed Type Hypersensitivity Reactions by Suppressing Lymphocyte Proliferation

2007

We have studied the effects of dihydrocucurbitacin B, a triterpene isolated from Cayaponia tayuya roots, on different models of delayed type hypersensitivity (DTH) in mice, as well as on T-lymphocyte proliferation and the mediators involved. In experiments with mice, dihydrocucurbitacin B inhibited the inflammatory reactions induced by oxazolone, dinitrofluorobenzene, and sheep red blood cells, reducing both the edema and cell infiltration. Moreover, the analysis of inflamed tissues showed that dihydrocucurbitacin B reduced the presence of the most relevant cytokines implicated in these processes, including interleukin-1 beta, interleukin-4, and tumor necrosis factor-alpha. Dihydrocucurbita…

CellLymphocyte proliferationLymphocyte ActivationResting Phase Cell CycleOxazoloneMicechemistry.chemical_compoundCyclinsmedicineAnimalsHypersensitivity DelayedCyclinInflammationPharmacologyNFATC Transcription FactorsbiologyNFATCell cyclebiology.organism_classificationMolecular biologyTriterpenesCayaponia tayuyaDisease Models Animalmedicine.anatomical_structurechemistryImmunologyCytokinesMolecular MedicineTumor necrosis factor alphaJournal of Pharmacology and Experimental Therapeutics
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Pyrrolo[2',3':3,4]cyclohepta[1,2-d][1,2]oxazoles, a New Class of Antimitotic Agents Active against Multiple Malignant Cell Types

2020

A new class of pyrrolo[2',3':3,4]cyclohepta[1,2-d][1,2]oxazoles was synthesized for the treatment of hyperproliferative pathologies, including neoplasms. The new compounds were screened in the 60 human cancer cell lines of the NCI drug screen and showed potent activity with GI50 values reaching the nanomolar level, with mean graph midpoints of 0.08-0.41 μM. All compounds were further tested on six lymphoma cell lines, and eight showed potent growth inhibitory effects with IC50 values lower than 500 nM. Mechanism of action studies showed the ability of the new [1,2]oxazoles to arrest cells in the G2/M phase in a concentration dependent manner and to induce apoptosis through the mitochondrial…

CellsMitosisAntineoplastic AgentsApoptosisAntimitotic AgentsDrug Screening Assays[12]oxazoles antimitotic agents lymphoma tubulin polymerization inhibitorsDose-Response RelationshipStructure-Activity Relationshipchemistry.chemical_compoundModelsDrug DiscoverymedicineHumansStructure–activity relationshipColchicineOxazolesAntimitotic Agents; Antineoplastic Agents; Apoptosis; Cell Proliferation; Cells Cultured; Dose-Response Relationship Drug; Drug Screening Assays Antitumor; G2 Phase Cell Cycle Checkpoints; HeLa Cells; Humans; Mitosis; Models Molecular; Molecular Structure; Oxazoles; Structure-Activity RelationshipCell Proliferationchemistry.chemical_classificationReactive oxygen speciesCulturedMolecular StructureChemistryMolecularDepolarizationAntitumorMolecular biologyG2 Phase Cell Cycle CheckpointsMechanism of actionApoptosisCell cultureMolecular MedicineAntimitotic AgentDrugmedicine.symptomHeLa Cells
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Evidence for a common progenitor of epithelial and mesenchymal components of the liver

2013

Tissues of the adult organism maintain the homeostasis and respond to injury by means of progenitor/stem cell compartments capable to give rise to appropriate progeny. In organs composed by histotypes of different embryological origins (e.g. The liver), the tissue turnover may in theory involve different stem/precursor cells able to respond coordinately to physiological or pathological stimuli. In the liver, a progenitor cell compartment, giving rise to hepatocytes and cholangiocytes, can be activated by chronic injury inhibiting hepatocyte proliferation. The precursor compartment guaranteeing turnover of hepatic stellate cells (HSCs) (perisinusoidal cells implicated with the origin of the …

Cellular differentiationLiver Stem CellDesminMice0302 clinical medicineMESH: AnimalsMESH: Nerve Tissue ProteinsHepatic stellate cellCells Cultured0303 health sciencesMesenchymal Stromal CellStem CellsCell DifferentiationCell biologyEndothelial stem cellMESH: DesminMESH: Models AnimalLiverMESH: Epithelial CellsDifferentiationModels Animal030211 gastroenterology & hepatologyStem cellMESH: Stem Cell Transplantationhepatic stellate cell; cell transplantation; liver stem cell; differentiationMESH: Cells CulturedMESH: Cell DifferentiationCell transplantation; Differentiation; Hepatic stellate cell; Liver stem cell; Animals; Cell Differentiation; Cell Line; Cell Lineage; Cell Proliferation; Cells Cultured; Desmin; Epithelial Cells; Glial Fibrillary Acidic Protein; In Vitro Techniques; Liver; Mesenchymal Stromal Cells; Mice; Mice Nude; Models Animal; Nerve Tissue Proteins; Stem Cell Transplantation; Stem Cells; Cell Biology; Molecular BiologyClinical uses of mesenchymal stem cellsMice NudeNerve Tissue ProteinsMESH: Stem Cells[SDV.BC]Life Sciences [q-bio]/Cellular BiologyBiologyIn Vitro TechniquesCell Line03 medical and health sciencesStem CellMESH: Cell ProliferationGlial Fibrillary Acidic ProteinMESH: Mice NudeAnimalsCell LineageProgenitor cellMESH: MiceMolecular Biology030304 developmental biologyCell ProliferationOriginal PaperEpithelial CellAnimalIn Vitro TechniqueMesenchymal stem cellEpithelial CellsMesenchymal Stem CellsCell BiologyMESH: Cell LineageMESH: Cell LineLiver stem cellNerve Tissue ProteinHepatic stellate cellMESH: Mesenchymal Stromal CellsCell transplantationMESH: LiverStem Cell Transplantation
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On the Interaction between 1D Materials and Living Cells

2020

One-dimensional (1D) materials allow for cutting-edge applications in biology, such as single-cell bioelectronics investigations, stimulation of the cellular membrane or the cytosol, cellular capture, tissue regeneration, antibacterial action, traction force investigation, and cellular lysis among others. The extraordinary development of this research field in the last ten years has been promoted by the possibility to engineer new classes of biointerfaces that integrate 1D materials as tools to trigger reconfigurable stimuli/probes at the sub-cellular resolution, mimicking the in vivo protein fibres organization of the extracellular matrix. After a brief overview of the theoretical models r…

Cellular membraneMaterials sciencelcsh:BiotechnologyBiomedical EngineeringTheoretical modelsregenerative medicineNanotechnologyBiointerface02 engineering and technologyReviewbioelectronics010402 general chemistry01 natural sciencesRegenerative medicineBiomaterials1D materialsbiointerfacelcsh:TP248.13-248.651D materials CNTs bioelectronics biointerface photocatalysis polymers regenerative medicineMicroscale chemistrypolymerslcsh:R5-920BioelectronicsCNTs021001 nanoscience & nanotechnology0104 chemical sciencesProtein fibreslcsh:Medicine (General)0210 nano-technologyphotocatalysisMaterial chemistryJournal of Functional Biomaterials
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Intrinsic TNFR2 signaling in T regulatory cells provides protection in CNS autoimmunity

2018

Significance In spite of TNF involvement in the pathogenesis of multiple sclerosis (MS), systemic TNF neutralization in MS patients was not successful. One of the possible reasons is that TNF possesses both pathogenic and protective features that may be related to TNFR1 versus TNFR2 receptor engagement. This study uncovers one of such protective functions of TNF mediated by intrinsic TNFR2 signaling in Treg cells. In mice bearing humanized TNF and TNFR2 genetic loci, TNFR2 ablation restricted to Treg cells led to reduced capacity to control Th17 cell responses, exacerbated experimental autoimmune encephalomyelitis (EAE) development, and affected the maintenance of Treg cells. These findings…

Central Nervous System0301 basic medicineEncephalomyelitis Autoimmune ExperimentalT regulatory cellsmedicine.medical_treatmentAutoimmunitychemical and pharmacologic phenomenaBiologymedicine.disease_causeT-Lymphocytes RegulatoryneuroinflammationAutoimmunityMice03 medical and health sciencesImmunology and Inflammation0302 clinical medicineImmune systemmedicineAnimalsHumansReceptors Tumor Necrosis Factor Type IIIL-2 receptorCells CulturedNeuroinflammationMice KnockoutAutoimmune diseaseMultidisciplinaryEAETumor Necrosis Factor-alphaExperimental autoimmune encephalomyelitisFOXP3hemic and immune systemsBiological Sciencesmedicine.diseaseTNF/TNFR2Mice Inbred C57BLDisease Models Animalhumanized mice030104 developmental biologyCytokineGene Expression RegulationImmunology030215 immunologyProceedings of the National Academy of Sciences
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Neurons of the dentate molecular layer in the rabbit hippocampus.

2012

The molecular layer of the dentate gyrus appears as the main entrance gate for information into the hippocampus, i.e., where the perforant path axons from the entorhinal cortex synapse onto the spines and dendrites of granule cells. A few dispersed neuronal somata appear intermingled in between and probably control the flow of information in this area. In rabbits, the number of neurons in the molecular layer increases in the first week of postnatal life and then stabilizes to appear permanent and heterogeneous over the individuals' life span, including old animals. By means of Golgi impregnations, NADPH histochemistry, immunocytochemical stainings and intracellular labelings (lucifer yellow…

Central Nervous SystemAnatomy and PhysiologyCell Countchemistry.chemical_compoundMolecular Cell BiologyComparative AnatomyNeuronsMultidisciplinaryNeuronal MorphologyPyramidal CellsQRAnimal ModelsAnatomyElectrophysiologymedicine.anatomical_structureNissl BodiesNissl bodysymbolsMedicineFemaleRabbitsCellular TypesResearch Articlemedicine.drugHistologyScienceNeurophysiologyBiologygamma-Aminobutyric acidsymbols.namesakeModel OrganismsDevelopmental NeuroscienceBiocytinmedicineAnimalsBiologyCell ShapeLucifer yellowStaining and LabelingDentate gyrusPerforant pathEntorhinal cortexElectrophysiological PhenomenaNeuroanatomyElectrophysiologychemistrynervous systemCellular NeuroscienceDentate GyrusBiophysicsNeural Circuit FormationNeurosciencePLoS ONE
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Pharmacological Suppression of CNS Scarring by Deferoxamine Reduces Lesion Volume and Increases Regeneration in an In Vitro Model for Astroglial-Fibr…

2015

Lesion-induced scarring is a major impediment for regeneration of injured axons in the central nervous system (CNS). The collagen-rich glial-fibrous scar contains numerous axon growth inhibitory factors forming a regeneration-barrier for axons. We demonstrated previously that the combination of the iron chelator 2,2'-bipyridine-5,5'-decarboxylic acid (BPY-DCA) and 8-Br-cyclic AMP (cAMP) inhibits scar formation and collagen deposition, leading to enhanced axon regeneration and partial functional recovery after spinal cord injury. While BPY-DCA is not a clinical drug, the clinically approved iron chelator deferoxamine mesylate (DFO) may be a suitable alternative for anti-scarring treatment (A…

Central Nervous SystemCollagen Type IVmedicine.medical_specialtyNeuriteCentral nervous systemlcsh:MedicineBiologyPharmacologyDeferoxamineIn Vitro TechniquesIron Chelating AgentsCicatrixIn vivoTransforming Growth Factor betamedicineCyclic AMPNeuritesAnimalsHumansRNA MessengerAxonRats Wistarlcsh:ScienceSpinal cord injurySpinal Cord InjuriesMultidisciplinaryDeferoxamine mesylatelcsh:RFibroblastsSpinal cordmedicine.diseaseAxonsSurgeryNerve RegenerationRatsDeferoxamineDisease Models Animalmedicine.anatomical_structureAstrocyteslcsh:QFemalemedicine.drugResearch ArticlePloS one
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Parallelized TCSPC for dynamic intravital fluorescence lifetime imaging : quantifying neuronal dysfunction in neuroinflammation

2013

Two-photon laser-scanning microscopy has revolutionized our view on vital processes by revealing motility and interaction patterns of various cell subsets in hardly accessible organs (e.g. brain) in living animals. However, current technology is still insufficient to elucidate the mechanisms of organ dysfunction as a prerequisite for developing new therapeutic strategies, since it renders only sparse information about the molecular basis of cellular response within tissues in health and disease. In the context of imaging, Forster resonant energy transfer (FRET) is one of the most adequate tools to probe molecular mechanisms of cell function. As a calibration-free technique, fluorescence lif…

Central Nervous SystemDiagnostic ImagingFluorescence-lifetime imaging microscopyPathologymedicine.medical_specialtyMouseScienceBiophysicsMedizinNeurophysiologyContext (language use)NeuroimagingBiosensing TechniquesBiologyIn Vitro TechniquesMiceCalcium imagingModel OrganismsMicroscopyMolecular Cell BiologyNeurobiology of Disease and RegenerationMedical imagingmedicineFluorescence Resonance Energy TransferAnimalsBiologyNeuroinflammationMultidisciplinaryPhysicsQRBrainAnimal ModelsIntravital ImagingCalcium ImagingFörster resonance energy transferMedicineCalciumFunction and Dysfunction of the Nervous SystemNeuroscienceResearch ArticleNeuroscience
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Single administration of tripeptide α-MSH(11-13) attenuates brain damage by reduced inflammation and apoptosis after experimental traumatic brain inj…

2013

Following traumatic brain injury (TBI) neuroinflammatory processes promote neuronal cell loss. Alpha-melanocyte-stimulating hormone (α-MSH) is a neuropeptide with immunomodulatory properties, which may offer neuroprotection. Due to short half-life and pigmentary side-effects of α-MSH, the C-terminal tripeptide α-MSH(11-13) may be an anti-inflammatory alternative. The present study investigated the mRNA concentrations of the precursor hormone proopiomelanocortin (POMC) and of melanocortin receptors 1 and 4 (MC1R/MC4R) in naive mice and 15 min, 6, 12, 24, and 48 h after controlled cortical impact (CCI). Regulation of POMC and MC4R expression did not change after trauma, while MC1R levels incr…

Central Nervous SystemMaleendocrine systemAnatomy and PhysiologyPro-OpiomelanocortinMouseScienceAnti-Inflammatory AgentsGene ExpressionApoptosisNeurological SystemImmunomodulationMiceModel OrganismsNeurorehabilitation and TraumaAnimalsMelanocyte-Stimulating HormonesBiologyCalcium-Binding ProteinsMicrofilament ProteinsQRBrainAnimal ModelsPeptide FragmentsMice Inbred C57BLHead InjuryNeurologyImmune SystemBrain InjuriesNervous System ComponentsCytokinesReceptor Melanocortin Type 4MedicineClinical ImmunologyMicrogliaInflammation MediatorsReceptor Melanocortin Type 1hormones hormone substitutes and hormone antagonistsResearch ArticleNervous System PhysiologyPLoS ONE
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