Search results for " Muscle"

showing 10 items of 1495 documents

Study with surface electromyography, borg scale and rom upper trapezius, deltoid of the three portions (clavicular, acromial, spinal) and the latissi…

2017

The pathologies located at shoulder can be caused by misuse or continued implementation of a sporting practice or physical activity. These pathologies are second position in the ranking of Traumatologics and Orthopedic Clinicals (Windt, Koes, De Jong & Bouter, 1995). Shoulder’s muscle injuries can occur when the arm is in a position that exceeds normal, this fact is repeated in sport, may be the repetition of the hand signal of the inductor problem (Kapandji, 2006). Given the importance of muscle function of the shoulder joint, along the existing boom of new techniques in the specific muscle activation has been carried out an experimental study using a " Test neuro proprioceptive response "…

Upper trapeziusmusculoskeletal diseasesmedicine.medical_specialtyShoulderDeltoid curvePhysical activityPhysical Therapy Sports Therapy and RehabilitationElectromyographyNeuropropioceptionPhysical medicine and rehabilitationEMGEducación Física y DeportivaMedicinelcsh:Sports medicineForcemedicine.diagnostic_testbusiness.industryLatissimus dorsi muscleMuscle activationAnatomymedicine.anatomical_structureMATMatOrthopedic surgeryShoulder jointbusinesslcsh:RC1200-1245
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Fibronectin urothelial gene expression as a new reliable biomarker for early detection of local toxicity secondary to adjuvant intravesical therapy f…

2020

Background: A marker of urothelial damage could be helpful for early detection and monitoring of local toxicity due to intravesical therapy for non-muscle invasive bladder cancer (NMIBC). The aim of the study was to investigate the correlation between fibronectin (FN) gene expression in bladder washings and local toxicity secondary to adjuvant intravesical therapy. Materials and methods: Patients undergoing adjuvant intravesical therapy for NMIBC and age-matched healthy patients were enrolled. Real time polymerase chain reaction was performed to analyze FN expression in bladder washings. Local toxicity was classified as: 0–1 mild (no medical therapy), 2 moderate (medical therapy and/or inst…

Urologymedicine.medical_treatment030232 urology & nephrologyEarly detectionBacillus Calmette–Guerinlcsh:RC870-92303 medical and health sciences0302 clinical medicinefibronectinnon-muscle invasive bladder cancerGene expressionbladder washingMedicineBladder cancerbiologybusiness.industrytoxicitylcsh:Diseases of the genitourinary system. Urologymedicine.diseaseFibronectin030220 oncology & carcinogenesisToxicitybiology.proteinCancer researchBiomarker (medicine)biomarkerOriginal ArticlebusinessNon muscle invasiveAdjuvantTherapeutic advances in urology
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Ovine Carotid Artery-Derived Cells as an Optimized Supportive Cell Layer in 2-D Capillary Network Assays

2014

PLoS one 9(3), e91664 (2014). doi:10.1371/journal.pone.0091664

Vascular Endothelial Growth Factor APathologyCellBecaplerminlcsh:MedicineCardiovascularUmbilical veinUmbilical CordDrug DiscoveryMolecular Cell BiologyBiological Systems EngineeringMyocyteCardiovascular Imaginglcsh:ScienceMultidisciplinaryProto-Oncogene Proteins c-sisAnimal ModelsFlow CytometryEndothelial stem cellBevacizumabmedicine.anatomical_structureCarotid ArteriesMonoclonalMedicineImmunohistochemical AnalysisResearch ArticleBiotechnologymedicine.medical_specialtyCell typeDrugs and DevicesDrug Research and DevelopmentMyocytes Smooth MuscleImmunologyBiomedical EngineeringBioengineeringBiologyAntibodies Monoclonal HumanizedCell LineModel OrganismsVascular Biologymedicine.arterymedicineAnimalsHumansBiologySheeplcsh:REndothelial CellsFeeder CellsUmbilical arteryMolecular biologyVascular Endothelial Growth Factor Receptor-2Coculture TechniquesCapillariesCell cultureImmunologic Techniqueslcsh:QCytometry
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Microphthalmia, persistent hyperplastic hyaloid vasculature and lens anomalies following overexpression of VEGF-A188 from the αA-crystallin promoter

2007

Purpose During growth of the embryonic eye, dose- and site-specific expression of heparin-binding growth factors is critical for the formation of an appropriate vascular supply. Overexpression of vascular endothelial growth factor-A188 (VEGF-A188), a strongly heparin-binding, endothelial-specific mitogen, leads to severe disturbance of vascular and overall ocular morphology. This study aimed to evaluate the effects of VEGF-A188 overexpression on growth of ocular tissue components. Methods Stereological and immunohistochemical methods were employed to identify the vascular profiles, ocular tissue proportions, and cell types in VEGF-A188 transgenic mice and compare them with wild-type mice. R…

Vascular Endothelial Growth Factor Agenetic structuresMyocytes Smooth MuscleCell CountMice TransgenicEyealpha-Crystallin A ChainCongenital AbnormalitiesCorneaMiceLens CrystallineAnimalsMicrophthalmosVascular DiseasesPromoter Regions GeneticHyperplasiaEndothelial CellsHypertrophyEmbryo MammalianAntigens DifferentiationImmunohistochemistryeye diseasesActinsDisease Models AnimalAnimals NewbornBlood Vesselssense organsPericytesHeparan Sulfate ProteoglycansResearch ArticleMolecular Vision
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Characterization of Membrane-Bound Cyclic Nucleotide Phosphodiesterases from Bovine Aortic Smooth Muscle

1992

This study reports the isolation and characterization of cyclic nucleotide phosphodiesterases (PDEs) associated with membrane fraction in comparison to cytosolic forms from bovine aorta. DEAE-Sephacel chromatography of a solubilized membrane fraction from a homogenate, prepared under isotonic conditions in the presence of protease inhibitors, yielded one major peak of PDE activity that specifically hydrolyzed cAMP and was not stimulated by calmodulin: It appeared to contain two subtypes of PDE. The first subtype belonged to the cyclic GMP (cGMP)-inhibited PDE family, (PDE III): It had an apparent Km value of 0.4 microM and was potently inhibited by cGMP, LY186126, and cilostamide. The secon…

Vascular smooth muscleCalmodulinPhosphodiesterase InhibitorsMuscle Smooth Vascularchemistry.chemical_compoundCytosolCalmodulinCyclic AMPmedicineAnimalsheterocyclic compoundsCyclic GMPRolipramPharmacologyCilostamidebiologyCyclic nucleotide phosphodiesterasePhosphoric Diester HydrolasesHydrolysisCell MembraneBiological membranemusculoskeletal systemenzymes and coenzymes (carbohydrates)Mechanism of actionBiochemistrychemistryEnzyme inhibitorbiology.proteinCattleChromatography Thin Layersense organsmedicine.symptomCardiology and Cardiovascular Medicinecirculatory and respiratory physiologymedicine.drugJournal of Cardiovascular Pharmacology
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Oxidative stress in vascular disease: causes, defense mechanisms and potential therapies

2007

Endothelial cells control vascular homeostasis by generating paracrine factors that regulate vascular tone, inhibit platelet function, prevent adhesion of leukocytes, and limit proliferation of vascular smooth muscle. The dominant factor responsible for many of those effects is endothelium-derived nitric oxide (NO). Endothelial dysfunction characterized by enhanced inactivation or reduced synthesis of NO, alone or in combination, is seen in conjunction with risk factors for cardiovascular disease. Endothelial dysfunction can promote vasospasm, thrombosis, vascular inflammation, and proliferation of the intima. Vascular oxidative stress and increased production of reactive oxygen species con…

Vascular smooth muscleEndotheliumArteriosclerosisPharmacologyNitric Oxidemedicine.disease_causeAntioxidantsReceptor Angiotensin Type 1Superoxide dismutaseRisk FactorsmedicineHumansEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologybusiness.industryAnticholesteremic AgentsGeneral Medicinemedicine.diseaseOxidative Stressmedicine.anatomical_structureMitochondrial respiratory chainchemistryImmunologybiology.proteinEndothelium VascularHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessOxidative stressNature Clinical Practice Cardiovascular Medicine
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Staphylococcus aureus alpha toxin mediates polymorphonuclear leukocyte-induced vasocontraction and endothelial dysfunction.

2002

The effect of Staphylococcus aureus alpha toxin (alpha-toxin) on selectin-mediated neutrophil adhesion was investigated in polymorphonuclear leukocyte- (PMN) induced vasocontraction and endothelial dysfunction. Adherence of human PMNs to rat aortic endothelium increased significantly following stimulation of the endothelium with alpha-toxin (0.1, 0.5, and 1 microg/mL). This effect could be significantly attenuated by monoclonal antibodies directed against P-selectin or fucoidin, a carbohydrate known to block selectins. Unstimulated human PMNs (10(6)cells/mL) were added to organ chambers containing rat aortic rings stimulated with alpha-toxin (0.5 microg/mL). PMNs elicited a significant vaso…

Vascular smooth muscleEndotheliumNeutrophilsBacterial ToxinsPharmacologyBiologyIn Vitro TechniquesCritical Care and Intensive Care MedicineMicrocirculationHemolysin ProteinsFibrinolytic AgentsmedicineCell AdhesionAnimalsHumansEndothelial dysfunctionStaphylococcus aureus alpha toxinAortaThrombinAzepinesTriazolesmedicine.diseaseRatsmedicine.anatomical_structureVasoconstrictionImmunologyEmergency MedicineEndothelium Vascularmedicine.symptomVasoconstrictionSelectinBlood vesselShock (Augusta, Ga.)
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Transmembrane signalling mechanisms regulating expression of cationic amino acid transporters and inducible nitric oxide synthase in rat vascular smo…

1999

The signalling mechanisms involved in the induction of nitric oxide synthase and l-arginine transport were investigated in bacterial lipopolysaccharide (LPS)- and interferon-gamma (IFN-gamma)-stimulated rat cultured aortic smooth muscle cells (RASMCs). The expression profile of transcripts for cationic amino acid transporters (CATs) and their regulation by LPS and IFN-gamma were also examined. Control RASMCs expressed mRNA for CAT-1, CAT-2A and CAT-2B. Levels of all three transcripts were significantly elevated in activated cells. Stimulated CAT mRNA expression and l-arginine transport occurred independently of protein kinase C (PKC), protein tyrosine kinase (PTK) and p44/42 mitogen-activat…

Vascular smooth muscleKinasep38 mitogen-activated protein kinasesCell BiologyBiologyBiochemistryNitric oxideCell biologyNitric oxide synthasechemistry.chemical_compoundchemistrybiology.proteinSignal transductionMolecular BiologyTyrosine kinaseProtein kinase CBiochemical Journal
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Vascular Contraction Model Based on Multi-agent Systems

2017

This paper presents a first approximation to the simulation of vascular smooth muscle cell following an agent-based simulation approach. This simulation incorporates mathematical models that describe the behaviour of these cells, which are used by the agents in order to emulate vascular contraction. A first tool, implemented in Netlogo, is provided to allow the performance of the proposed simulation.

Vascular smooth muscleMathematical modelNetLogoControl theoryComputer scienceMulti-agent systemcomputercomputer.programming_languageVascular contraction
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Regulation of NOS expression in vascular diseases

2020

Nitric oxide synthases (NOS) are the major sources of nitric oxide (NO), a small bioactive molecule involved in the regulation of many cellular processes. One of the most prominent functions of NO is regulation of vasodilatation and thereby control of blood pressure. Most important for vascular tone is NOS3. Endothelial NOS3-generated NO diffuses into the vascular smooth muscle cells, activates the soluble guanylate cyclase resulting in enhanced cGMP concentrations and smooth muscle cell relaxation. However, more and more evidence exist that also NOS1 and NOS2 contribute to vascular function. We summarize the current knowledge about the regulation of NOS expression in the vasculature by tra…

Vascular smooth muscleNitric Oxide Synthase Type IIINOS1CellNitric Oxide Synthase Type IIBlood PressureVasodilationInflammationNitric Oxide Synthase Type INitric OxideMuscle Smooth VascularNitric oxidechemistry.chemical_compoundmedicineAnimalsHumansProtein IsoformsVascular DiseasesRNA Processing Post-TranscriptionalInflammationRegulation of gene expressionInnate immune systemAtherosclerosisImmunity InnateCell biologyGene Expression Regulation Neoplasticmedicine.anatomical_structurechemistryNitric Oxide Synthasemedicine.symptomProtein Processing Post-TranslationalFrontiers in Bioscience-Landmark
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