Search results for " Obstructive"

showing 10 items of 477 documents

Effectiveness and safety of concurrent beta-blockers and inhaled bronchodilators in COPD with cardiovascular comorbidities

2017

Chronic obstructive pulmonary disease (COPD) is the most common chronic respiratory disease and its prevalence is increasing worldwide, in both industrialised and developing countries. Its prevalence is ∼5% in the general population and it is the fourth leading cause of death worldwide. COPD is strongly associated with cardiovascular diseases; in fact, ∼64% of people suffering from COPD are treated for a concomitant cardiovascular disease and approximately one in three COPD patients die as a consequence of cardiovascular diseases.Inhaled bronchodilators might have adverse cardiovascular effects, including ischaemic events and arrhythmias, and beta-blockers might adversely influence the resp…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyAdrenergic beta-AntagonistsPopulationComorbidityDiseaseRisk AssessmentPulmonary Disease Chronic Obstructive03 medical and health sciences0302 clinical medicineRisk FactorsAdministration InhalationEpidemiologymedicineHumans030212 general & internal medicineeducationIntensive care medicineLungCause of deathlcsh:RC705-779education.field_of_studyCOPDbusiness.industryRespiratory diseaselcsh:Diseases of the respiratory systemmedicine.diseaseComorbidityBronchodilator AgentsTreatment Outcome030228 respiratory systemCardiovascular DiseasesbusinessRisk assessmentEuropean Respiratory Review
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Circulating haemopoietic and endothelial progenitor cells are decreased in COPD

2006

Circulating CD34+ cells are haemopoietic progenitors that may play a role in tissue repair. No data are available on circulating progenitors in chronic obstructive pulmonary disease (COPD). Circulating CD34+ cells were studied in 18 patients with moderate-to-severe COPD (age: mean+/-sd 68+/-8 yrs; forced expiratory volume in one second: 48+/-12% predicted) and 12 controls, at rest and after endurance exercise. Plasma concentrations of haematopoietic growth factors (FMS-like tyrosine kinase 3 (Flt3) ligand, kit ligand), markers of hypoxia (vascular endothelial growth factor (VEGF)) and stimulators of angiogenesis (VEGF, hepatocyte growth factor (HGF)) and markers of systemic inflammation (tu…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyAngiogenesisCD34Antigens CD34cd34Settore MED/10 - Malattie Dell'Apparato RespiratorioSettore BIO/09 - Fisiologiachronic obstructive pulmonary diseaseimmunologyPulmonary Disease Chronic Obstructivecd34+ cellscd34+cellschemistry.chemical_compoundantigensbloodstem cellsInternal medicinegrowth factorsmiddle agedMedicineProgenitor cellhumansCD34+ cells chronic obstructive pulmonary disease exercise growth factors hypoxiacell countpulmonary diseaseCOPDchronic obstructiveexercisehypoxiabusiness.industryaged; antigens; blood; cd34; cd34+ cells; cd34+cells; cell count; chronic obstructive; chronic obstructive pulmonary disease; endothelial cells; exercise; growth factors; hematopoietic stem cells; humans; hypoxia; immunology; middle aged; pulmonary disease; stem cellsHypoxia (medical)medicine.diseaseendothelial cellshematopoietic stem cellsEndothelial stem cellVascular endothelial growth factoragedEndocrinologychemistryHepatocyte growth factormedicine.symptombusinessmedicine.drugEuropean Respiratory Journal
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European Respiratory Society statement on sleep apnoea, sleepiness and driving risk

2021

Obstructive sleep apnoea (OSA) is highly prevalent and is a recognised risk factor for motor vehicle accidents (MVA). Effective treatment with continuous positive airway pressure has been associated with a normalisation of this increased accident risk. Thus, many jurisdictions have introduced regulations restricting the ability of OSA patients from driving until effectively treated. However, uncertainty prevails regarding the relative importance of OSA severity determined by the apnoea–hypopnoea frequency per hour and the degree of sleepiness in determining accident risk. Furthermore, the identification of subjects at risk of OSA and/or accident risk remains elusive. The introduction of off…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyAutomobile DrivingSleepinessmedia_common.quotation_subjectmedicine.medical_treatmentPoison controlSettore MED/10 - Malattie Dell'Apparato RespiratorioSleep ApnoeaSuicide preventionOccupational safety and healthDriving Simulators03 medical and health sciences0302 clinical medicineRisk FactorsEpidemiologyInjury preventionSleep Apnoea Driving Accident Risk Sleepiness Screening Driving Simulators Treatment RegulationsmedicineHumansContinuous positive airway pressureIntensive care medicineRegulationsmedia_commonSleep Apnea ObstructiveContinuous Positive Airway Pressurebusiness.industryAccidents TrafficHuman factors and ergonomicsnervous system diseasesrespiratory tract diseasesTreatment030228 respiratory systemAccident RiskScreeningHuman medicinebusiness030217 neurology & neurosurgeryDrivingVigilance (psychology)
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Aclidinium inhibits human lung fibroblast to myofibroblast transition

2011

Background Fibroblast to myofibroblast transition is believed to contribute to airway remodelling in lung diseases such as asthma and chronic obstructive pulmonary disease. This study examines the role of aclidinium, a new long-acting muscarinic antagonist, on human fibroblast to myofibroblast transition. Methods Human bronchial fibroblasts were stimulated with carbachol (10 −8 to 10 −5  M) or transforming growth factor-β1 (TGF-β1; 2 ng/ml) in the presence or absence of aclidinium (10 −9 to 10 −7  M) or different drug modulators for 48 h. Characterisation of myofibroblasts was performed by analysis of collagen type I and α-smooth muscle actin (α-SMA) mRNA and protein expression as well as α…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyCarbacholChronic Obstructive Pulmonary DiseaseBronchiMuscarinic AntagonistsBiologyCholinergic AgonistsCollagen Type ITransforming Growth Factor beta1Downregulation and upregulationWestern blotanticholinergicCell MovementInternal medicinemedicineCOPDHumans1506RNA MessengerAutocrine signallingFibroblastMyofibroblastsCells CulturedCell Proliferationmedicine.diagnostic_testDose-Response Relationship Drugairway epitheliumCell Differentiationasthmainterstitial fibrosisFibroblastsAdenosineMolecular biologymyofibroblastActinsUp-RegulationEndocrinologymedicine.anatomical_structurePhosphorylationFibroblastCarbacholMyofibroblastmedicine.drugTropanesThorax
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Protocol for the EARCO Registry

2020

Rationale and objectives Alpha-1 antitrypsin deficiency (AATD) is a genetic condition that leads to an increased risk of emphysema and liver disease. Despite extensive investigation, there remain unanswered questions concerning the natural history, pathophysiology, genetics and the prognosis of the lung disease in association with AATD. The European Alpha-1 Clinical Research Collaboration (EARCO) is designed to bring together researchers from European countries and to create a standardised database for the follow-up of patients with AATD. Study design and population The EARCO Registry is a non-interventional, multicentre, pan-European, longitudinal observational cohort study enrolling patie…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyChronic Obstructive Pulmonary DiseasePopulation1MEDLINElcsh:Medicine61032 Biomedical and Clinical Sciences610 Medicine & health[SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract03 medical and health sciencesLiver diseaseStudy Protocol0302 clinical medicineRare DiseasesClinical ResearchmedicineGenetics030212 general & internal medicineIntensive care medicineeducation3202 Clinical SciencesLungProtocol (science)Emphysemaeducation.field_of_studybusiness.industryPreventionlcsh:Rmedicine.disease3. Good healthNatural historyClinical research030228 respiratory systemObservational study10178 Clinic for PneumologybusinessCohort study
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Which factors affect the choice of the inhaler in chronic obstructive respiratory diseases?

2015

Inhalation is the preferred route of drug administration in chronic respiratory diseases because it optimises delivery of the active compounds to the targeted site and minimises side effects from systemic distribution. The choice of a device should be made after careful evaluation of the patient's clinical condition (degree of airway obstruction, comorbidities), as well as their ability to coordinate the inhalation manoeuvre and to generate sufficient inspiratory flow. These patient factors must be aligned with the specific advantages and limitations of each inhaler when making this important choice. Finally, adherence to treatment is not the responsibility of the patient alone, but should …

Pulmonary and Respiratory Medicinemedicine.medical_specialtyChronic ObstructiveSocio-culturaleSettore MED/10 - Malattie Dell'Apparato RespiratorioAffect (psychology)Anti-asthmatic AgentMedication AdherencePulmonary DiseasePulmonary Disease Chronic ObstructiveSuspensionsAdministration InhalationmedicineCOPDHumansPharmacology (medical)Anti-Asthmatic AgentsMetered Dose InhalersRespiratory systemParticle SizeIntensive care medicineAsthmaAdherence; Asthma; COPD; Inhaler; Administration Inhalation; Anti-Asthmatic Agents; Dry Powder Inhalers; Humans; Medication Adherence; Metered Dose Inhalers; Particle Size; Pulmonary Disease Chronic Obstructive; Solutions; Suspensions; Nebulizers and Vaporizers; Pulmonary and Respiratory Medicine; Pharmacology (medical); Biochemistry (medical); Medicine (all)COPDInhalationbusiness.industryMedicine (all)InhalerNebulizers and VaporizersBiochemistry (medical)Adherence; Asthma; COPD; Inhaler; Administration Inhalation; Anti-Asthmatic Agents; Dry Powder Inhalers; Humans; Medication Adherence; Metered Dose Inhalers; Particle Size; Pulmonary Disease Chronic Obstructive; Solutions; Suspensions; Nebulizers and Vaporizers; Pulmonary and Respiratory Medicine; Biochemistry (medical); Pharmacology (medical)InhalerDry Powder InhalersAirway obstructionmedicine.diseaseAsthmaSolutionsInhalationAdherenceAdministrationAdherence; Asthma; COPD; InhalerbusinessPulmonary pharmacologytherapeutics
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Sleep apnoea as an independent risk factor for cardiovascular disease: current evidence, basic mechanisms and research priorities

2007

Considerable evidence is available in support of an independent association between obstructive sleep apnoea syndrome (OSAS) and cardiovascular disease, which is particularly strong for systemic arterial hypertension and growing for ischaemic heart disease, stroke, heart failure, atrial fibrillation and cardiac sudden death. The pathogenesis of cardiovascular disease in OSAS is not completely understood but likely to be multifactorial, involving a diverse range of mechanisms including sympathetic nervous system overactivity, selective activation of inflammatory molecular pathways, endothelial dysfunction, abnormal coagulation and metabolic dysregulation, the latter particularly involving in…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyDiseaseSettore MED/10 - Malattie Dell'Apparato RespiratorioCardiovascular disease intermittent hypoxia mechanisms obstructive sleep apnoeaMetabolic DiseasesRisk FactorsDiabetes mellitusInternal medicinemedicineHumansmedia_common.cataloged_instanceEuropean unionIntensive care medicineStrokemedia_commonSleep Apnea ObstructiveMechanism (biology)business.industryResearchSleep apneaAtrial fibrillationmedicine.diseaseEndocrinologyCardiovascular DiseasesHeart failurebusinessEuropean Respiratory Journal
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Effects of inhaled corticosteroids in stable chronic obstructive pulmonary disease.

2011

Chronic obstructive pulmonary disease (COPD) has been described as a heterogeneous multifactorial disorder associated with an abnormal inflammatory response of the peripheral airways and with variable morphologic, physiologic and clinical phenotypes. This notion of the disease is actually poorly supported by data, and there are substantial discrepancies and a weak correlation between inflammation, structural damage, functional impairment and degree of clinical symptoms. This problem is compounded by a poor understanding of the complexity and intricacies on the inflammatory pathways in COPD. Despite the evidence for efficacy of inhaled corticosteroids (ICS) on selected clinical endpoints in …

Pulmonary and Respiratory Medicinemedicine.medical_specialtyDrug ResistancePulmonary diseaseInhaled corticosteroidsInflammationDiseasePulmonary Disease Chronic ObstructiveQuality of lifeAdrenal Cortex HormonesAdministration InhalationmedicineClinical endpointHumansPharmacology (medical)Intensive care medicineCOPDbusiness.industryBiochemistry (medical)medicine.diseaserespiratory tract diseasesWeak correlationPhysical therapyDisease ProgressionQuality of Lifemedicine.symptombusinessPulmonary pharmacologytherapeutics
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Mild obstructive sleep apnea increases hypertension risk, challenging traditional severity classification

2020

STUDY OBJECTIVES: The association of mild obstructive sleep apnea (OSA) with important clinical outcomes remains unclear. We aimed to investigate the association between mild OSA and systemic arterial hypertension (SAH) in the European Sleep Apnea Database cohort. METHODS: In a multicenter sample of 4,732 participants, we analyzed the risk of mild OSA (subclassified into 2 groups: mild(AHI 5-<11/h) (apnea-hypopnea index [AHI], 5 to <11 events/h) and mild(AHI 11-<15/h) (AHI, ≥11 to <15 events/h) compared with nonapneic snorers for prevalent SAH after adjustment for relevant confounding factors including sex, age, smoking, obesity, daytime sleepiness, dyslipidemia, chronic obstructive pulmona…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyEuropean Sleep Apnea Database Mild obstructive sleep apnea Systemic arterial hypertensionPolysomnographyDisorders of Excessive SomnolenceSettore MED/10 - Malattie Dell'Apparato RespiratorioHypertension risk03 medical and health sciences0302 clinical medicinestomatognathic systemRisk FactorsInternal medicinesystemic arterial hypertensionmedicineHumansEuropean Sleep Apnea DatabaseSleep Apnea ObstructiveSystemic arterial hypertensionbusiness.industrymedicine.diseaseScientific InvestigationsClinical neurologyrespiratory tract diseasesnervous system diseasesObstructive sleep apneamild obstructive sleep apneaDiabetes Mellitus Type 2NeurologyHypertensionNeurology (clinical)Human medicinebusiness030217 neurology & neurosurgery
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Current and future pharmacologic therapy of exacerbations in chronic obstructive pulmonary disease and asthma.

2005

Exacerbations are an important cause of the morbidity and mortality associated with asthma and chronic obstructive pulmonary disease. Newer therapies include long-acting beta(2)-agonists, which are more effective than short-acting bronchodilators. Inhaled corticosteroids and, in asthma, leukotriene receptor antagonists may have roles in the early phase of exacerbation as an alternative to or added to oral prednisolone. In the future, combinations of long-acting beta(2)-agonists and anticholinergic bronchodilators may offer additive clinical benefits. However, although the treatment and prevention of exacerbations of chronic obstructive pulmonary disease and asthma have been improved by usin…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyExacerbationPulmonary diseaseInhaled corticosteroidsPulmonary Disease Chronic ObstructivePharmacotherapyAdrenal Cortex HormonesmedicineHumansPharmacologic therapyAnti-Asthmatic AgentsIntensive care medicineAsthmabusiness.industryAdrenergic beta-Agonistsmedicine.diseaseAsthmaAnti-Bacterial AgentsBronchodilator AgentsDrug CombinationsEtiologyDisease ProgressionLeukotriene AntagonistsDrug Therapy CombinationEarly phasebusinessForecastingProceedings of the American Thoracic Society
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