Search results for " Pathologic"

showing 10 items of 285 documents

Identification and validation of novel ERBB2 (HER2, NEU) targets including genes involved in angiogenesis.

2005

V-erb-b2 erythroblastic leukemia viral oncogene homolog 2 (ERBB2; synonyms HER2, NEU) encodes a transmembrane glycoprotein with tyrosine kinase-specific activity that acts as a major switch in different signal-transduction processes. ERBB2 amplification and overexpression have been found in a number of human cancers, including breast, ovary and kidney carcinoma. Our aim was to detect ERBB2-regulated target genes that contribute to its tumorigenic effect on a genomewide scale. The differential gene expression profile of ERBB2-transfected and wild-type mouse fibroblasts was monitored employing DNA microarrays. Regulated expression of selected genes was verified by RT-PCR and validated by West…

Cancer ResearchReceptor ErbB-2Blotting WesternViral OncogeneDown-RegulationComputational biologyBiologymedicine.disease_causeTransfectionGenomeMiceGene expressionmedicineAnimalsHumansskin and connective tissue diseasesGeneDNA PrimersGlycoproteinsOligonucleotide Array Sequence AnalysisGeneticsRegulation of gene expressionGenomeNeovascularization PathologicReverse Transcriptase Polymerase Chain ReactionFibroblastsGenes erbB-2Up-RegulationGene expression profilingGene Expression Regulation NeoplasticCell Transformation NeoplasticOncologyNIH 3T3 CellsDNA microarrayCarcinogenesisSignal TransductionInternational journal of cancer
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Tumor and its microenvironment: a synergistic interplay.

2013

The mutual and interdependent interaction between tumor and its microenvironment is a crucial topic in cancer research. Recently, it was reported that targeting stromal events could improve efficacies of current therapeutics and prevent metastatic spreading. Tumor microenvironment is a "complex network" of different cell types, soluble factors, signaling molecules and extracellular matrix components, which orchestrate the fate of tumor progression. As by definition, cancer stem cells (CSCs) are proposed to be the unique cell type able to maintain tumor mass and survive outside the primary tumor at metastatic sites. Being exposed to environmental stressors, including reactive oxygen species …

Cancer ResearchStromal cellEpithelial-Mesenchymal TransitionAngiogenesisCell SurvivalBiologyCancer stem cellCell MovementNeoplasmsmedicineTumor MicroenvironmentAnimalsHumansEpithelial–mesenchymal transitionNeoplasm MetastasisStem Cell NicheHypoxiaTumor microenvironmentNeovascularization Pathologicmedicine.diseaseAngiogenesis CAFs CAMs CRC CSCs ECM EMT GSH HIF Hypoxia MMPs ROS Tumor microenvironment VEGF cancer stem cells cancer-associated fibroblasts cancer-associated macrophages colorectal cancer epithelial mesenchymal transition extracellular matrix hypoxia-inducible factor matrix metalloproteinase reactive oxygen species reduced glutathione vascular endothelial growth factorPrimary tumorTumor progressionImmunologyCancer researchNeoplastic Stem CellsCancer-Associated FibroblastsOxidation-ReductionSignal Transduction
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Tumor-intrinsic and -extrinsic roles of c-Kit: mast cells as the primary off-target of tyrosine kinase inhibitors.

2011

c-Kit tyrosine kinase receptor and its ligand stem cell factor have multiple functions during development, whereas in adulthood they are mostly needed for stem cell (SC) maintenance and mast cell (MC) biology. c-Kit plays an essential tumor-cell-intrinsic role in many types of cancer, either providing the tumorigenic force when aberrantly activated or conferring stem-like features characterizing the most aggressive variants. A tumor-cell-extrinsic role occurs through c-Kit-dependent accessory cells (such as MCs) that infiltrate tumors and deeply influence their progression. c-Kit-targeted therapy with tyrosine kinase inhibitors (TKIs) may ideally work against both tumor and stromal cells. H…

Cancer ResearchStromal cellStem cell factorAntineoplastic AgentsBiologyc-kit; mast cells; mouse mutants; off-target; tyrosine kinase inhibitorsReceptor tyrosine kinaseMicec-KitNeoplasmstyrosine kinase inhibitorsGeneticsmedicineAnimalsHumansNeoplasm InvasivenessMast CellsMolecular BiologyProtein Kinase InhibitorsStem Cell Factormouse mutantsNeovascularization PathologicMast cellRatsProto-Oncogene Proteins c-kitmedicine.anatomical_structureTumor progressionmast cells.biology.proteinCancer researchStem cellTyrosine kinasePlatelet-derived growth factor receptoroff-targetMastocytosisOncogene
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Induction of apoptosis and inhibition of cell growth in human hepatocellular carcinoma cells by COX-2 inhibitors

2005

The aim of the present study was to examine the effects of nonselective (indomethacin) and selective cyclooxygenase-2 (COX-2) inhibitors (NS-398, nimesulide, and CAY10404) on cell growth, cell cycle distribution, and apoptosis in three human hepatocellular carcinoma cell lines (HepG2, HuH-6, and HA22T/VGH) with different characteristics of differentiation and biological behavior. The four COX inhibitors showed a dose-dependent growth-inhibitory effect in all the cell lines. No substantial arrests in the progression of the cells through the cell cycle were observed after treatment of HuH-6 or HA22T/VGH for 48 h with the various inhibitors. On the other hand, there were significant increases …

Carcinoma HepatocellularTime FactorsApoptosisPharmacologyBiologyGeneral Biochemistry Genetics and Molecular BiologyFlow cytometryInhibitory Concentration 50History and Philosophy of ScienceCell Line TumorCarcinomamedicineHumansProtein IsoformsCyclooxygenase InhibitorsEnzyme InhibitorsCell ProliferationCyclooxygenase 2 InhibitorsDose-Response Relationship DrugNeovascularization Pathologicmedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionCell growthGeneral NeuroscienceAnti-Inflammatory Agents Non-SteroidalCell CycleMembrane Proteinsantineoplastic activity apoptosis cancer cell cultureCell cycleFlow Cytometrymedicine.diseaseCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesCell cultureApoptosisHepatocellular carcinomaNimesulidemedicine.drug
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Local transient myocardial liposomal gene transfer of inducible nitric oxide synthase does not aggravate myocardial function and fibrosis and leads t…

2010

Microcirculation (2010) 17, 69–78. doi: 10.1111/j.1549-8719.2010.00002.x Abstract Background:  This study was designed to explore the effect of transient inducible nitric oxide synthase (iNOS) overexpression via cationic liposome-mediated gene transfer on cardiac function, fibrosis, and microvascular perfusion in a porcine model of chronic ischemia. Methods and Results:  Chronic myocardial ischemia was induced using a minimally invasive model in 23 landrace pigs. Upon demonstration of heart failure, 10 animals were treated with liposome-mediated iNOS-gene-transfer by local intramyocardial injection and 13 animals received a sham procedure to serve as control. The efficacy of this iNOS-gene-…

Cardiac function curveMalemedicine.medical_specialtyPhysiologySus scrofaIschemiaMyocardial IschemiaGene ExpressionNitric Oxide Synthase Type IINitric OxideVentricular Function LeftNeovascularizationFibrosisPhysiology (medical)Internal medicinemedicineAnimalsHumansMolecular BiologyEjection fractionbiologyNeovascularization Pathologicbusiness.industryMyocardiumGene Transfer Techniquesmedicine.diseaseFibrosisMagnetic Resonance ImagingRecombinant ProteinsNitric oxide synthaseArteriolesHeart failureLiposomesCardiologybiology.proteinDobutamineFemalemedicine.symptomCardiology and Cardiovascular Medicinebusinessmedicine.drugMicrocirculation (New York, N.Y. : 1994)
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“The importance of being external”: review of the literature of the rare phenomenon of common carotid occlusion with bulb reverse-crossed stenosis an…

2022

Common carotid artery occlusion (CCAO) is a rare phenomenon associated with the development of neurological symptoms. In CCAO, diversion of blood flow from the external carotid artery (ECA) to the internal carotid artery (ICA) via the carotid bulb (CB) may occur. This pathway activation has been called "carotid steal". Starting from a particular case we describe the ECD finding of a complete occlusion of CCA with patency of ICA and ECA. In case of occlusion of CCA, inversion of the ECA flow may occur, towards the ICA, and it can be damped by a significant stenosis crossed in retrograde direction that may concur to maintain the pressure balance between the two circulations. Usually, this par…

Carotid Artery DiseasesCarotid steal Collateral pathway Common carotid occlusion External carotid arteryCarotid Artery ExternalInternal MedicineHumansCarotid StenosisRadiology Nuclear Medicine and imagingConstriction PathologicGeneral MedicineCarotid Artery Internal
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Delayed filling of retinal and ciliary circulation after central retinal artery occlusion

1988

The filling times of the retinal and choroidal circulation were evaluated in 17 patients with central retinal artery occlusion by means of fluorescein angiography. A correlation between the filling times of the two circulations was found. However, two filling patterns were seen: first, both fillings delayed; second, both fillings normal. It is probable that in the former case an obstructive disease of the carotid arteries exists, while in the latter case an important pathology of the carotid arteries is lacking, favouring a rapid restoration of the retinal circulation. This hypothesis seems to be confirmed by the results of the Doppler velocimetry of the carotid arteries.

Carotid Artery DiseasesMalemedicine.medical_specialtyRetinal Artery OcclusionConstriction PathologicRetinachemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineHumansFluorescein AngiographyAgedUltrasonographyRetinamedicine.diagnostic_testbusiness.industryCiliary BodyRetinalAnatomyMiddle AgedLaser Doppler velocimetrymedicine.diseaseFluorescein angiographySensory SystemsOphthalmologymedicine.anatomical_structurechemistryRegional Blood FlowAngiographyCardiologyCentral retinal artery occlusionFemaleChoroidbusinessRetinopathyDocumenta Ophthalmologica
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External stenting with a new polyester mesh reduces neointimal hyperplasia of vein grafts in a sheep model.

2007

Objective External stents placed around vein grafts have demonstrated effectiveness in reducing neointimal hyperplasia by preventing distension of the thin-walled vein grafts when exposed to arterial pressure. However, the ideal stent material has yet to be defined. The following study investigates the short- and long-term effects of an innovative polyester mesh stent designed with optimized adaptation of circumferential compliance. Methods Following in vitro definition of the ideal macro-porous polyester stent material, a total of 12 sheep underwent implantation of bilateral carotid artery vein graft bypasses. In six sheep, the short-term outcome (four weeks of implantation) was investigat…

Carotid Artery Diseasesmedicine.medical_specialtyCarotid Artery CommonCarotid arteriesmedicine.medical_treatmentPolyesters030232 urology & nephrologyBiomedical EngineeringMedicine (miscellaneous)BioengineeringVein graft030204 cardiovascular system & hematologyDistensionBiomaterials03 medical and health sciencesBlood Vessel Prosthesis Implantation0302 clinical medicineMedicineAnimalsSaphenous VeinNeointimal hyperplasiaPolyester meshHyperplasiaSheepbusiness.industryGraft Occlusion VascularStentGeneral MedicineSurgical Meshequipment and suppliesmedicine.diseaseSurgeryCompliance (physiology)Disease Models Animalsurgical procedures operativeSurgical meshStentsbusinessTunica IntimaDilatation PathologicThe International journal of artificial organs
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Brain and Cancer: The Protective Role of Erythropoietin

2005

Erythropoietin (Epo) is a pleiotropic agent, that is to say, it can act on several cell types in different ways. An independent system Epo/Epo receptor (EpoR) was detected in brain, leading to the hypothesis that this hormone could be involved in cerebral functions. Epo/EpoR expression changes during ontogenesis, thus indicating the importance of this system in neurodevelopment. Moreover, the hypoxia-induced production of Epo in the adult brain suggests that it could exert a neurotrophic and neuroprotective effect in case of brain injury. Epo could also influence neuro- transmission, inducing neurotransmitters (NT) release. Epo therapy in anemic cancer patients is still a controversial issu…

Cell typeCentral nervous systemPharmacologyModels BiologicalNeuroprotectionNeoplasmshemic and lymphatic diseasesDrug DiscoveryReceptors ErythropoietinmedicineAnimalsHumanscancerReceptorPleiotropyPharmacologyNeurotransmitter AgentsNeovascularization Pathologicbiologyhypoxiabusiness.industryMedicine (all)Organic ChemistryBrainangiogenesiGeneral MedicineNeuroprotectionneuroprotective effectErythropoietin receptorErythropoietin (Epo); brain; central nervous system (CNS) diseases; neuroprotective effectmedicine.anatomical_structureErythropoietin (Epo)Erythropoietinbiology.proteinMolecular MedicineerythropoietinSignal transductionbusinessNeurosciencecentral nervous system (CNS) diseasesmedicine.drugNeurotrophinChemInform
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MicroRNAs: Promising New Antiangiogenic Targets in Cancer

2014

[EN] MicroRNAs are one class of small, endogenous, non-coding RNAs that are approximately 22 nucleotides in length; they are very numerous, have been phylogenetically conserved, and involved in biological processes such as development, differentiation, cell proliferation, and apoptosis. MicroRNAs contribute to modulating the expression levels of specific proteins based on sequence complementarity with their target mRNA molecules and so they play a key role in both health and disease. Angiogenesis is the process of new blood vessel formation from preexisting ones, which is particularly relevant to cancer and its progression. Over the last few years, microRNAs have emerged as critical regulat…

Cell typeDOWN-REGULATIONArticle SubjectAngiogenesisHUMAN BREAST-CANCERMIR-200 FAMILYlcsh:MedicineAngiogenesis InhibitorsReview ArticleBiologyBioinformaticsGeneral Biochemistry Genetics and Molecular BiologyNUCLEAR EXPORTTUMOR ANGIOGENESISNeovascularizationMicroprocessor complexSMALL RNASDownregulation and upregulationNeoplasmsmicroRNAGene expressionmedicineAnimalsHumansMolecular Targeted TherapyPrecision MedicineIN-VIVOGENE-EXPRESSIONGeneral Immunology and MicrobiologyNeovascularization PathologicCell growthlcsh:RMICROBIOLOGIAGeneral MedicineMICROPROCESSOR COMPLEXMicroRNAsENDOTHELIAL GROWTH-FACTORCancer researchmedicine.symptom
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