Search results for " Pathologic"

showing 10 items of 285 documents

Hypoxia in Breast Cancer: Pathogenesis, Characterization and Biological/Therapeutic Implications

2002

Nearly 50% of locally advanced breast cancers exhibit hypoxic and/or anoxic tissue areas that are heterogeneously distributed within the tumour mass. Hypoxia is predominantly caused by structural and functional abnormalities of the newly formed tumour vessels arising from neovascularization, by a disturbed microcirculation, by enlarged diffusion distances, and by tumour-associated or therapy-induced anaemia. The extent of pretherapeutically measured hypoxic tissue areas is independent of clinical tumour size and stage, and histological type and grade. Anaemia can substantially worsen tumour O2 depletion. Hypoxia is known to directly or indirectly confer resistance to irradiation and some ch…

Pathologymedicine.medical_specialtyBreast NeoplasmsDisease-Free SurvivalTreatment failureMicrocirculationPathogenesisNeovascularizationBreast cancerHumansMedicineTreatment FailureNeovascularization Pathologicbusiness.industryMicrocirculationGeneral MedicineHypoxia (medical)Prognosismedicine.diseaseCell HypoxiaClinical trialCancer researchFemalemedicine.symptomMalignant progressionbusinessWiener Medizinische Wochenschrift
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Ductal Carcinoma In Situ of the Breast: A Comparative Analysis of Histology, Nuclear Area, Ploidy, and Neovascularization Provides Differentiation Be…

2002

Ductal carcinoma in situ (DCIS) is a heterogeneous group of lesions that has been subdivided into three types: well differentiated (grade I), moderately differentiated (grade II), and poorly differentiated (grade III). Forty-five cases of DCIS were analyzed for image analysis: nuclear area, DNA ploidy, and vascularization in order to establish a more precise correlation between the histologic grade and these morphometric parameters. Our results confirm that the mean nuclear area, DNA ploidy, and microvessel density (MVD) progressively increased from DCIS grade I to DCIS grade III. The analysis of the nuclear area in relationship to DCIS grading demonstrated a progressive increase of values …

Pathologymedicine.medical_specialtyBreast NeoplasmsNeovascularizationStatistical significanceImage Processing Computer-AssistedInternal MedicineHumansMedicineNeoplasmskin and connective tissue diseasesneoplasmsGrading (tumors)Cell NucleusPloidiesNeovascularization Pathologicbusiness.industryCarcinoma in situCarcinoma Ductal BreastHistologyDNA NeoplasmDuctal carcinomamedicine.diseasebody regionsOncologyTumor progressionFemaleSurgerymedicine.symptombusinessCarcinoma in SituThe Breast Journal
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Effect of antiangiogenic treatment on peritoneal endometriosis-associated nerve fibers

2012

Objective To investigate the effect of antiangiogenic treatment on experimental endometriotic lesion nerve fibers. Design Heterologous mouse model of endometriosis. Setting University Institute IVI, University Hospital La Fe. Animal(s) Ovariectomized nude mice (n = 16) receiving human endometrial fragments from oocyte donors (n = 4). Intervention(s) Endometrium fragments stuck in the peritoneum of 5-week-old female nude mice treated with vehicle (n = 8) and antiangiogenic agent cabergoline (n = 8; Cb 2, 0.05 mg/kg/day) for 14 days. Main Outcome Measure(s) Immunofluorescence analysis of von-Willebrand factor (vWF) and vascular smooth muscle cells (αSMA) for evaluating the number of immature …

Pathologymedicine.medical_specialtyCabergolineTime FactorsAngiogenesisOvariectomyEndometriosisEndometriosisFluorescent Antibody TechniqueMice NudeAngiogenesis InhibitorsNerve fiberPeritoneal DiseasesEndometriumEndometriumMicechemistry.chemical_compoundNerve FibersPeritoneumvon Willebrand FactorAnimalsHumansMedicineMast CellsErgolinesNeovascularization Pathologicbusiness.industryMacrophagesObstetrics and GynecologyMast cellmedicine.diseaseImmunohistochemistryActinsVascular endothelial growth factorDisease Models Animalmedicine.anatomical_structureReproductive MedicinechemistryMicrovesselsImmunologyFemalebusinessBiomarkersBlood vesselFertility and Sterility
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18F-fluorodeoxyglucose hypometabolism in cerebellar tonsil and flocculus in downbeat nystagmus.

2006

A patient with downbeat nystagmus was examined by F-fluorodeoxyglucose-positron emission tomography once while off and twice while on successful treatment with 4-aminopyridine. All positron emission tomography scans of the patient showed a reduced cerebral glucose metabolism bilaterally in the region of the cerebellar tonsil and flocculus/paraflocculus when compared with a normal database of the whole brain. An additional region-of-interest analysis revealed that 4-aminopyridine treatment lessened the hypometabolism. This finding supports the hypothesis that the cerebellar tonsil and (para-) flocculus play a crucial role in downbeat nystagmus. The hypometabolism might reflect reduced inhibi…

Pathologymedicine.medical_specialtyCerebellumgenetic structuresEye MovementsNystagmusFlocculusNystagmus PathologicDownbeat nystagmusImaging Three-DimensionalVestibular nucleiFluorodeoxyglucose F18CerebellummedicinePotassium Channel BlockersHumans4-AminopyridineAgedFluorodeoxyglucosemedicine.diagnostic_testbusiness.industryGeneral Neurosciencemedicine.anatomical_structurePositron emission tomographyPositron-Emission TomographyCerebellar tonsilFemalemedicine.symptombusinessNeurosciencemedicine.drugNeuroreport
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Endothelial capillaries chemotactically attract tumour cells.

2001

Directional migration of capillaries towards tumour implants is generally assumed to be regulated by chemotaxis. Preliminary evidence has also been presented for the existence of a reverse chemotactic signalling pathway, with capillaries attracting tumour cells via paracrine factors. By using a variety of endothelial cell types and tumour cell lines, this study has systematically investigated chemotaxis between endothelial cells and tumour cells in two- and three-dimensional systems. Checkerboard analysis revealed faint attraction of human umbilical vein endothelial cells (HUVECs), but not porcine aortic endothelial cells (PAECs), by tumour cells. In reverse, both PAECs and HUVECs potently …

Pathologymedicine.medical_specialtyEndotheliumAngiogenesisSwineCell Culture TechniquesCell CommunicationBiologyPathology and Forensic MedicineParacrine signallingVasculogenesisNeoplasmsmedicineTumor Cells CulturedAnimalsHumansMicroscopy Phase-ContrastMelanomaFibrinNeovascularization PathologicChemotaxisMicrocarrierChemotaxisCell biologyCapillariesEndothelial stem cellmedicine.anatomical_structureCell cultureCulture Media Conditionedcardiovascular systemEndothelium VascularGlioblastomaThe Journal of pathology
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Dextran sulfate sodium leads to chronic colitis and pathological angiogenesis in Endoglin heterozygous mice

2010

Pathological angiogenesis is an intrinsic component of chronic intestinal inflammation, which results in remodeling and expansion of the gut microvascular bed. Endoglin is essential for endothelial cell function and physiological angiogenesis. In this study we investigated its potential role in the regulation of inflammation by testing the response of Endoglin heterozygous (Eng(+/-)) mice to experimental colitis.C57BL/6 Eng(+/-) and littermate control mice drank water supplemented with 3% dextran sulfate sodium (DSS) for 5 days and were monitored for up to 26 days for clinical signs of colitis. Inflammation, crypt damage, and angiogenic index were scored on histological sections of distal c…

Pathologymedicine.medical_specialtyHeterozygoteAngiogenesisColonVascular permeabilityInflammatory bowel diseaseArticleNeovascularizationCapillary Permeabilitychemistry.chemical_compoundMicemedicineImmunology and AllergyAnimalsColitisAcute colitisNeovascularization Pathologicbusiness.industryVascular Endothelial Growth FactorsDextran SulfateGastroenterologyEndoglinIntracellular Signaling Peptides and ProteinsEndoglinmedicine.diseaseColitisVascular endothelial growth factorMice Inbred C57BLDisease Models AnimalchemistryAcute Diseasemedicine.symptombusinessAngiopoietins
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Inflammation-Induced Intussusceptive Angiogenesis in Murine Colitis

2010

Intussusceptive angiogenesis is a morphogenetic process that forms new blood vessels by the division of a single blood vessel into two lumens. Here, we show that this process of intraluminal division participates in the inflammation-induced neovascularization associated with chemically induced murine colitis. In studies of both acute (4-7 days) and chronic (28-31 days) colitis, intravital microscopy of intravascular tracers demonstrated a twofold reduction in blood flow velocity. In the acute colitis model, the decreased velocity was associated with marked dilatation of the mucosal plexus. In contrast, chronic inflammation was associated with normal caliber vessels and duplication (and trip…

Pathologymedicine.medical_specialtyHistologyColonAngiogenesisBiologyCorrosion CastingArticleMicrocirculationNeovascularizationMicemedicineAnimalsColitisIntussusceptive angiogenesisEcology Evolution Behavior and SystematicsAcute colitisFluorescent DyesSprouting angiogenesisMice Inbred BALB CMucous MembraneNeovascularization PathologicStaining and LabelingEndothelial CellsColitismedicine.diseaseAdaptation PhysiologicalCapillariesMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureRegional Blood FlowMicroscopy Electron ScanningNanoparticlesInflammation MediatorsAnatomymedicine.symptomIntussusceptionBiotechnologyBlood vesselThe Anatomical Record: Advances in Integrative Anatomy and Evolutionary Biology
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C-reactive protein and efficacy of antiplatelet therapy in (intracranial) atherosclerosis

2018

C-reactive protein (CRP) and other inflammatory biomarkers can indicate both the severity and extent of atherosclerosis, reflecting the inflammatory nature of the disease process.1 Atherogenesis begins with an inflammatory response to vascular injury with cells and mediators initiating the healing response and later inducing growth of atherosclerotic plaques. Inflammation then increases plaque instability, promoting rupture, fissuring, or erosion—the pathogenetic milieu of thrombosis in atherothrombotic ischemic strokes.

Pathologymedicine.medical_specialtyInflammationConstriction PathologicDisease030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicinemedicineHumansbiologyC-reactive protein intracranial atherosclerosisbusiness.industryIschemic strokesC-reactive proteinAtherosclerosismedicine.diseaseThrombosisInflammatory biomarkersC-Reactive Proteinbiology.proteinPlatelet aggregation inhibitorNeurology (clinical)Intracranial Atherosclerosismedicine.symptombusinessPlatelet Aggregation Inhibitors030217 neurology & neurosurgeryNeurology
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Comprehensive three‐dimensional morphology of neoangiogenesis in pulmonary veno‐occlusive disease and pulmonary capillary hemangiomatosis

2019

Abstract Pulmonary veno‐occlusive disease (PVOD) is a rare lung disease characterized by fibrotic narrowing of pulmonary veins leading to pulmonary hypertension (PH) and finally to death by right heart failure. PVOD is often accompanied by pulmonary capillary hemangiomatosis (PCH), a marked abnormal proliferation of pulmonary capillaries. Both morphological patterns often occur together and are thought to be distinct manifestations of the same disease process and accordingly are classified together in group 1′ of the Nice classification of PH. The underlying mechanisms of these aberrant remodeling processes remain poorly understood. In this study, we investigated the three‐dimensional struc…

Pathologymedicine.medical_specialtyLung NeoplasmsHypertension Pulmonarypulmonary veno‐occlusive diseasePulmonary capillary hemangiomatosis030204 cardiovascular system & hematologypulmonary capillary hemangiomatosisPathology and Forensic Medicine03 medical and health sciencesThree dimensional morphology0302 clinical medicineRight heart failurepulmonary hypertensionmedicinelcsh:PathologyHumansHemangioma CapillaryLungNeovascularization Pathologicbusiness.industryBrief Definitive Reportintussusceptive neoangiogenesismedicine.diseasePulmonary hypertension3. Good healthmedicine.anatomical_structurePulmonary VeinsLung disease030220 oncology & carcinogenesisPulmonary Veno-Occlusive DiseaseImmunohistochemistryPulmonary Veno-Occlusive Diseasebusinesspulmonary vascular remodelinglcsh:RB1-214The Journal of Pathology: Clinical Research
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Automated Measurement of Microcirculatory Blood Flow Velocity in Pulmonary Metastases of Rats

2014

Because the lung is a major target organ of metastatic disease, animal models to study the physiology of pulmonary metastases are of great importance. However, very few methods exist to date to investigate lung metastases in a dynamic fashion at the microcirculatory level, due to the difficulty to access the lung with a microscope. Here, an intravital microscopy method is presented to functionally image and quantify the microcirculation of superficial pulmonary metastases in rats, using a closed-chest pulmonary window and automated analysis of blood flow velocity and direction. The utility of this method is demonstrated to measure increases in blood flow velocity in response to pharmacologi…

Pathologymedicine.medical_specialtyLung NeoplasmsIntravital MicroscopyGeneral Chemical EngineeringBreast NeoplasmsGeneral Biochemistry Genetics and Molecular BiologyMicrocirculationNeovascularizationRats NudeCell Line TumormedicineAnimalsHumansCancer BiologyLungNeovascularization PathologicGeneral Immunology and Microbiologybusiness.industryMicrocirculationGeneral NeuroscienceBlood flowmedicine.diseaseRatsmedicine.anatomical_structureHeterograftsFemaleSarcoma ExperimentalSarcomamedicine.symptombusinessPulmonary tumorBlood Flow VelocityTarget organIntravital microscopyJournal of Visualized Experiments
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