Search results for " Pathologic"

showing 10 items of 285 documents

Three-dimensional helical CT of the tracheobronchial tree: evaluation of imaging protocols and assessment of suspected stenoses with bronchoscopic co…

1996

To assess the accuracy of three-dimensional (3D) helical CT of normal airways, we evaluated different imaging protocols in test objects and patients. The clinical value of 3D helical CT was composed with bronchoscopy in patients with suspected stenoses, especially before and after endobronchial procedures.Solid test objects--one of central airways and one of peripheral airways--were scanned and assessed for volume defects and stairstep artifacts. Fifty helical studies were performed in 36 patients. We evaluated these images for visualization of segmental bronchi; frequency of artifacts; and presence, localization, and degree of stenoses. Bronchoscopic correlation was available for 40 CT exa…

Pulmonary and Respiratory MedicineAdultMalemedicine.medical_specialtygenetic structuresBronchiConstriction PathologicBronchoscopyImage Processing Computer-AssistedMedicineHumansRadiology Nuclear Medicine and imagingIn patientProspective StudiesAgedAged 80 and overBronchographymedicine.diagnostic_testbusiness.industryGeneral MedicineBronchographyMiddle Agedmedicine.diseaseHelical ctEndoscopyTracheaStenosisClinical valueFemaleTomographyRadiologyNuclear medicinebusinessArtifactsTomography X-Ray ComputedTracheal StenosisAJR. American journal of roentgenology
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Symptomatic atherosclerotic occlusive disease of all supra-aortic arch vessels treated with total aortic arch rerouting.

2011

We present an uncommon case of symptomatic atherosclerotic occlusive disease of all supra-aortic arch vessels and its surgical treatment by total aortic arch rerouting after endarteriectomy of all target vessels.

Pulmonary and Respiratory MedicineAortic archmedicine.medical_specialtyAortographymedicine.medical_treatmentAortic DiseasesHemodynamicsAorta ThoracicConstriction PathologicEndarterectomyAortographySeverity of Illness IndexMagnetic resonance angiographyBlood Vessel Prosthesis Implantationmedicine.arteryInternal medicinemedicineThoracic aortaHumansArchEndarterectomyAgedAortamedicine.diagnostic_testbusiness.industryHemodynamicsAtherosclerosisTreatment OutcomeRegional Blood Flowcardiovascular systemCardiologySurgeryFemaleCardiology and Cardiovascular MedicinebusinessTomography X-Ray ComputedMagnetic Resonance AngiographyInteractive cardiovascular and thoracic surgery
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Aortic valve-sparing root replacement from inside the aorta using three Dacron skirts preserving the native Valsalva sinuses geometry and stabilizing…

2009

We present an alternative idea for valve-sparing technique combining the advantages of the reimplantation and the benefits of the remodeling techniques. We replace the sinuses of Valsalva using three Dacron skirts from inside of the aorta. The physiological anatomy of the Valsalva sinuses and their dynamic properties are preserved and the aortic commissures displacement avoided. The distal rim of each Dacron skirt was anchored to the aorto-ventricular junction using Ticron (2-0) U stitches placed from the ventricular side to the aortic side. We stabilize the base of the aortic annulus to prevent future dilation.

Pulmonary and Respiratory MedicineAortic valveAortic root aneurysm; Valve-sparing surgery; Easy procedurePolyestersAortic root aneurysmAortic DiseasesProsthesis DesignBlood Vessel Prosthesis ImplantationEasy proceduremedicine.arteryMedicineHumanscardiovascular diseasesCardiac skeletonSurgical ReplantationAortaCardiopulmonary BypassSuturesbusiness.industryPolyethylene TerephthalatesSettore MED/23 - Chirurgia CardiacaAnatomySinus of ValsalvaAortic AneurysmBlood Vessel ProsthesisValve-sparing surgeryParanasal sinusesmedicine.anatomical_structureTreatment OutcomeAortic Valvecardiovascular systemSurgeryCardiology and Cardiovascular MedicinebusinessTomography X-Ray Computedcirculatory and respiratory physiologyDilatation PathologicInteractive cardiovascular and thoracic surgery
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Establishment and comparative characterization of novel squamous cell non-small cell lung cancer cell lines and their corresponding tumor tissue.

2010

Abstract Background Cell lines play an important role for studying tumor biology and novel therapeutic agents. Particularly in pulmonary squamous cell carcinoma (SCC) the availability of cell lines is limited and knowledge about their representativeness for corresponding tumor tissue is scanty. Materials and methods We established three novel SCC cell lines from fresh tumor tissue of 28 donors, including 8 SCC. Two cell lines were derived from different localizations of the same donor, i.e. primary tumor and lymph node metastasis. This represents a so far unique combination in lung cancer. The genotypes, gene expression profiles and mutational status of epidermal growth factor receptor ( EG…

Pulmonary and Respiratory MedicineCancer ResearchLung NeoplasmsAngiogenesisCarcinogenicity TestsCellIn situ hybridizationCell Growth ProcessesBiologymedicine.disease_causeMiceCell MovementCarcinoma Non-Small-Cell LungCell Line TumormedicineCell AdhesionAnimalsHumansCell LineageIn Situ Hybridization FluorescenceMutationComparative Genomic Hybridizationmedicine.diagnostic_testNeovascularization PathologicGene Expression ProfilingCell Differentiationmedicine.diseasePrimary tumorMolecular biologyDNA FingerprintingGene expression profilingErbB Receptorsmedicine.anatomical_structureGenes rasOncologyCell cultureTandem Repeat SequencesLymphatic MetastasisMutationCarcinoma Squamous CellFluorescence in situ hybridizationLung cancer (Amsterdam, Netherlands)
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Effect of ketotifen on the bronchodilation induced by salbutamol.

1988

Twelve subjects with stable asthma each inhaled two puffs (200 micrograms) of salbutamol on 2 separate days 3 h after double-blind oral administration of ketotifen (two 1-mg capsules) or identical placebo. FEV1 was recorded before and at intervals for 4 h after inhalation of salbutamol. Overall, the FEV1 was significantly greater during the 4-hour period after premedication with ketotifen (p less than 0.02) and the difference between the effect of placebo and ketotifen was statistically significant at 120, 180 and 240 min after salbutamol (p less than 0.05).

Pulmonary and Respiratory MedicineKetotifenAdultMaleBronchiHistamine H1 receptorPlaceboOral administrationForced Expiratory VolumemedicineHumansAlbuterolKetotifenAsthmaInhalationbusiness.industryMiddle Agedmedicine.diseaseAsthmarespiratory tract diseasesBronchodilatationAnesthesiaSalbutamolFemalebusinessmedicine.drugDilatation PathologicRespiration; international review of thoracic diseases
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Nintedanib in non-small cell lung cancer: from preclinical to approval

2015

Angiogenesis is a driving force of a tumor’s development. Targeting this process is an attractive option, as this is a feature shared by most of the solid tumors. A lot of antiangiogenic drugs have been developed following this path, including bevacizumab, sorafenib, sunitinib, vandetanib, ramucirumab, motesanib and many others. The latest drug of this class to be approved for patients with non-small cell lung cancer (NSCLC) was nintedanib, a triple angiokinase inhibitor. This molecule targets vascular endothelial growth factor (VEGF), platelet derived growth factor (PDGF) and fibroblast growth factor (FGF) pathways, avoiding the tumor’s switch to normal escape mechanisms. The pharmacokine…

Pulmonary and Respiratory MedicineSorafenibIndolesLung NeoplasmsBevacizumabBIBF1120Settore MED/06 - Oncologia Medicamedicine.drug_classDrug Evaluation PreclinicalAngiogenesis InhibitorsAntineoplastic AgentsAdenocarcinomaPharmacologyNSCLCVandetanibTyrosine-kinase inhibitorRamucirumabchemistry.chemical_compoundtyrosine kinase inhibitorCarcinoma Non-Small-Cell LungnintedanibmedicineMotesanibAnimalsHumansPharmacology (medical)Drug Approvalnon-small cell lung cancerlcsh:RC705-779Neovascularization PathologicSunitinibbusiness.industrylcsh:Diseases of the respiratory systemchemistryCancer researchNintedanibHuman medicinebusinessantiangiogenic drugmedicine.drugTherapeutic Advances in Respiratory Disease
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Successful surgery of neuroendocrine carcinoma infiltrating right ventricle and pulmonary artery.

2020

We present the clinical case of a 60-year-old woman complained of dyspnea on exertion. Echocardiogram showed a giant mass in the right ventricle (RV) with obstruction to the outflow tract. Thorax computed tomography confirmed a mass of greater than 60 mm infiltrating RV and causing severe stenosis in the pulmonary artery, with severe pericardial effusion. Cardiac surgery was performed for tumor resection and pulmonary root replacement with a biological valved conduit. Histological analysis diagnosed a poorly differentiated large-cell neuroendocrine carcinoma. The patient had no immediate postoperative complications and has completed radiotherapy at a 9-month follow-up.

Pulmonary and Respiratory MedicineThoraxmedicine.medical_specialtymedicine.medical_treatmentHeart VentriclesConstriction Pathologic030204 cardiovascular system & hematologyPulmonary ArteryPericardial effusionSeverity of Illness IndexPericardial EffusionHeart Neoplasms03 medical and health sciencesBlood Vessel Prosthesis Implantation0302 clinical medicinemedicine.arterymedicineHumansNeuroendocrine carcinomaExertionCardiac Surgical Proceduresbusiness.industryMiddle Agedmedicine.diseaseSurgeryCardiac surgeryCarcinoma NeuroendocrineRadiation therapymedicine.anatomical_structureTreatment Outcome030228 respiratory systemVentricleEchocardiographyPulmonary arterySurgeryFemaleCardiology and Cardiovascular MedicinebusinessTomography X-Ray ComputedJournal of cardiac surgeryREFERENCES
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A new pyrazolo pyrimidine derivative inhibitor of cyclooxygenase-2 with anti-angiogenic activity

2003

In a previous study, we reported a new pyrazolo pyrimidine derivative, N(4)-benzyl-N(6),N(6)-dimethyl-1-1(tert-butyl)-1H-pyrazolo[3,4-d]pyrimidine-6,4-diamine (DPP), which inhibited potently cyclooxygenase-2 activity in intact cell assays with minor activity against cyclooxygenase-1 (IC(50)=0.9 nM for cyclooxygenase-2 versus IC(50)=59.6 nM for cyclooxygenase-1). In the present work, this behaviour was confirmed in vivo by using the 24-h zymosan-injected mouse air pouch model (ID(50)=1.36 nM/pouch for prostaglandin E(2) level). We also studied the possible beneficial effect of DPP in the angiogenesis-dependent murine air pouch granuloma and rat paw carrageenan-induced hyperalgesia models. DP…

Pyrimidinemedicine.medical_treatmentAngiogenesis InhibitorsPharmacologyCarrageenanDinoprostoneMicechemistry.chemical_compoundIn vivomedicineAnimalsEdemaCyclooxygenase InhibitorsRats WistarProstaglandin E2IC50NitrobenzenesPharmacologySulfonamidesGranulomaCyclooxygenase 2 InhibitorsNeovascularization PathologicbiologyTumor Necrosis Factor-alphaZymosanRatsIsoenzymesPyrimidinesEicosanoidchemistryBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesHyperalgesiabiology.proteinPyrazolesFemaleCyclooxygenasemedicine.symptomInterleukin-1Prostaglandin Emedicine.drugEuropean Journal of Pharmacology
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Endothelial cells and normal breast epithelial cells enhance invasion of breast carcinoma cells by CXCR-4-dependent up-regulation of urokinase-type p…

2008

Here we show the increase of invasion of three breast cancer cell lines (8701-BC, MDA-MB-231 and SKBR3) upon long-term co-incubation with culture medium of normal microvascular endothelial cells (MVEC) and normal breast epithelial cells (HB2). The enhancement of invasion relied on the interaction of microvascular endothelial cell and normal breast epithelial cell CXCL12 (SDF1) chemokine, whose expression by breast cancer cells was very low, with the cognate CXCR4 receptor of malignant cells, which resulted in over-expression of the urokinase-type plasminogen activator receptor (uPAR) on their surfaces. uPAR over-expression, showed by RT-PCR and Western blotting, was paralleled by increased …

Receptors CXCR4MAP Kinase Kinase 4AngiogenesisCellBreast NeoplasmsReceptors Cell SurfaceCell CommunicationBiologyCell LineReceptors Urokinase Plasminogen ActivatorPathology and Forensic MedicineMetastasisangiogenesisbreast cancerTumor Cells CulturedmedicineHumansNeoplasm InvasivenessBreastSettore BIO/06 - Anatomia Comparata E CitologiaPhosphorylationskin and connective tissue diseasesCXCR4Settore MED/04 - Patologia GeneraleNeovascularization PathologicReverse Transcriptase Polymerase Chain ReactionFibrinolysisEpithelial CellsCXCL12invasionmedicine.diseasemicroenvironmentChemokine CXCL12Neoplasm ProteinsUp-RegulationEndothelial stem cellUrokinase receptormedicine.anatomical_structureCulture Media ConditionedCancer cellCancer researchFemaleJNKEndothelium VascularBreast diseaseSDF1uPARPlasminogen activatorThe Journal of Pathology
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Pathogenesis and molecular mechanisms of anderson–fabry disease and possible new molecular addressed therapeutic strategies

2021

Anderson–Fabry disease (AFD) is a rare disease with an incidenceof approximately 1:117,000 male births. Lysosomal accumulation of globotriaosylceramide (Gb3) is the element characterizing Fabry disease due to a hereditary deficiency α-galactosidase A (GLA) enzyme. The accumulation of Gb3 causes lysosomal dysfunction that compromises cell signaling pathways. Deposition of sphingolipids occurs in the autonomic nervous system, dorsal root ganglia, kidney epithelial cells, vascular system cells, and myocardial cells, resulting in organ failure. This manuscript will review the molecular pathogenetic pathways involved in Anderson–Fabry disease and in its organ damage. Some studies reported that i…

ReviewConstriction Pathologicendothelial dysfunctionPathogenesisMicechemistry.chemical_compoundKCa3.1 activitypodocyturiaProtein IsoformsEndothelial dysfunctionBiology (General)SpectroscopyglobotriaosylceramideGlobosidesMicrogliabiologyTOR Serine-Threonine KinasesTrihexosylceramidesmiR-26a-5pGeneral MedicineMitochondriaComputer Science ApplicationsCell biologymiR-152-5pChemistrymedicine.anatomical_structureCerebrovascular CirculationAnderson–Fabry disease Endothelial dysfunction Globotriaosylceramide KCa3.1 activity MiR-1307-5p MiR-152-5p MiR-21-5p MiR-26a-5p Podocyturia Valvular dysfunctionmiR-21-5pSignal TransductionQH301-705.5GlobotriaosylceramideCatalysisInorganic ChemistryAutophagymedicineAnimalsHumansEnzyme Replacement TherapyPhysical and Theoretical ChemistryMolecular BiologyMechanistic target of rapamycinQD1-999PI3K/AKT/mTOR pathwaySphingolipidsAnderson–Fabry diseasebusiness.industryMicrocirculationOrganic ChemistryEndothelial Cellsmedicine.diseaseFabry diseaseSphingolipidMicroRNAschemistrymiR-1307-5palpha-Galactosidasebiology.proteinFabry DiseaseGlycolipidsvalvular dysfunctionLysosomesbusiness
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