Search results for " Plastic"

showing 10 items of 3463 documents

Bipedicled DIEAP flaps for reconstruction of limb soft tissue defects in male patients.

2016

Summary Background Extensive soft tissue deficiencies involving the limbs can be difficult to reconstruct and may require more than one microsurgical flap transfer to cover the defect. This can be particularly challenging in male patients, where the sacrifice of a donor muscle could result in considerable comorbidity. This paper describes the use of the bipedicled deep inferior epigastric artery perforator (DIEAP) flap to perform a one-stage reconstruction of extensive soft tissue defects in male patients. Methods By using preoperative multidetector computed tomographic (MDCT) angiography, the dominant perforators of the abdominal wall were identified and the bipedicled DIEAP flap was used …

MaleMicrosurgeryMale patientmedicine.medical_treatmentSettore MED/19 - Chirurgia Plastica030230 surgerySurgical FlapsAbdominal wall0302 clinical medicineRetrospective StudieArm InjurieArm Injuriesmedicine.diagnostic_testSoft tissueEpigastric ArterieMiddle AgedForearmmedicine.anatomical_structureTreatment Outcome030220 oncology & carcinogenesisVascular Surgical ProceduresHumanArteryAdultmedicine.medical_specialtySoft Tissue InjuriesLeg Injurie03 medical and health sciencesmedicineReconstructive Surgical ProcedureHumansVascular Surgical ProcedureRetrospective StudiesLegBipedicled DIEAP flapbusiness.industryDeep Inferior Epigastric ArteryAbdominal WallMicrosurgeryPlastic Surgery ProceduresEpigastric Arterieseye diseasesSurgerySurgical FlapMale patientAngiographyExtremity traumaAbdomenSurgerybusinessLeg InjuriesJournal of plastic, reconstructiveaesthetic surgery : JPRAS
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Long-term effects on motor cortical excitability induced by repeated muscle vibration during contraction in healthy subjects

2008

article i nfo Objective: The effects of a novel repeated muscle vibration intervention (rMV; 100 Hz, 90 min over 3 consecutive days) on corticomotor excitability were studied in healthy subjects. Methods: rMV was applied over the flexor carpi radialis (FCR) during voluntary contraction (experiment 1), during relaxation and during contraction without vibration (experiment 2). Focal transcranial magnetic stimulation (TMS) was applied before rMV and one hour, and one, two and three weeks after the last muscle vibration intervention. At each of these time points, we assessed the motor map area and volume in the FCR, extensor digitorum communis (EDC) and abductor digiti minimi (ADM). Short-inter…

MaleMotor disorderpaired-pulse tmsTime FactorsContraction (grammar)H-Reflex; Analysis of Variance; Humans; Electromyography; Neural Inhibition; Electric Stimulation; Muscle Skeletal; Motor Cortex; Brain Mapping; Vibration; Evoked Potentials Motor; Adult; Middle Aged; Muscle Contraction; Transcranial Magnetic Stimulation; Time Factors; Female; Malemedicine.medical_treatmentH-ReflextmsEvoked PotentialsBrain Mappingcortical plasticitySkeletalMiddle AgedTranscranial Magnetic Stimulationmedicine.anatomical_structureMotorNeurologyAnesthesiaMuscleFemaleSettore MED/26 - NeurologiaPrimary motor cortexmedicine.symptomPsychologyMuscle ContractionMotor cortexMuscle contractionAdultintracortical inhibitionVibrationNOmuscle vibrationmotor cortexNeuroplasticitymedicineHumansMuscle SkeletalAnalysis of VarianceneurorehabilitationElectromyographytms; muscle vibration; paired-pulse tms; neurorehabilitation; motor cortex; cortical plasticity; intracortical inhibitionNeural InhibitionEvoked Potentials Motormedicine.diseaseElectric Stimulationbody regionsTranscranial magnetic stimulationNeurology (clinical)NeuroscienceExtensor Digitorum Communis
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Cheek Volumization and the Nasolabial Fold.

2018

Sir: We have read the article by Mowlds and Lambros1 with great interest and enthusiasm. The findings of their study show that the nasolabial fold does not improve after cheek injection. By analyzing three-dimensional images of the face before and immediately after cheek injection of high- G′ hyaluronic acid, they demonstrate that the perceived nasolabial fold improvement, reported after cheek injections,2,3 is attributable to overall improvement in facial appearance rather than to actual nasolabial fold improvement. As a consequence, it might be ruled out that the nasolabial fold is a consequence of cheek deflating and it is likely attributable predominantly to change in the corner of the …

MaleNasolabial FoldInjections SubcutaneousSettore MED/19 - Chirurgia PlasticaFacial MusclesAdipose tissueCosmetic Techniques030230 surgery030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicineImaging Three-DimensionalDermal FillersmedicineImage Processing Computer-AssistedPhotographyHumansHyaluronic AcidInjections subcutaneousOrthodonticsbusiness.industryAnatomyCheekNasolabial foldFacial MuscleFacial Expressionstomatognathic diseasesFacial musclesmedicine.anatomical_structureCheekAdipose Tissue030220 oncology & carcinogenesisSurgeryFemalebusinessPlastic and reconstructive surgery
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The Polysialylated Form of the Neural Cell Adhesion Molecule (PSA-NCAM) Is Expressed in a Subpopulation of Mature Cortical Interneurons Characterized…

2010

Principal neurons in the adult cerebral cortex undergo synaptic, dendritic, and spine remodeling in response to different stimuli, and several reports have demonstrated that the polysialylated form of the neural cell adhesion molecule (PSA-NCAM) participates in these plastic processes. However, there is only limited information on the expression of this molecule on interneurons and on its role in the structural plasticity of these cells. We have found that PSA-NCAM is expressed in mature interneurons widely distributed in all the extension of the cerebral cortex and have excluded the expression of this molecule in most principal cells. Although PSA-NCAM expression is generally considered a …

MaleNeurogenesisCognitive NeuroscienceCellular differentiationNeural InhibitionNeural Cell Adhesion Molecule L1BiologyInhibitory postsynaptic potentialRats Sprague-DawleyCellular and Molecular NeuroscienceInterneuronsNeural PathwaysNeuroplasticitymedicineAnimalsCell ShapeCerebral CortexNeuronal PlasticityEmbryogenesisNeurogenesisCell DifferentiationNeural InhibitionRatsmedicine.anatomical_structurenervous systemCerebral cortexSialic AcidsNeural cell adhesion moleculeNeuroscienceCerebral Cortex
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D1/D5 modulation of synaptic NMDA receptor currents.

2009

Converging evidence suggests that salience-associated modulation of behavior is mediated by the release of monoamines and that monoaminergic activation of D1/D5receptors is required for normal hippocampal-dependent learning and memory. However, it is not understood how D1/D5modulation of hippocampal circuits can affect salience-associated learning and memory. We have observed in CA1 pyramidal neurons that D1/D5receptor activation elicits a bidirectional long-term plasticity of NMDA receptor-mediated synaptic currents with the polarity of plasticity determined by NMDA receptor, NR2A/B subunit composition. This plasticity results in a decrease in the NR2A/NR2B ratio of subunit composition. Sy…

MaleNeuronal PlasticityGeneral Neurosciencemusculoskeletal neural and ocular physiologyReceptors Dopamine D1Long-term potentiationAMPA receptorNeurotransmissionBiologyReceptors N-Methyl-D-AspartateSynaptic TransmissionArticleMice Inbred C57BLMicenervous systemSynaptic plasticitySynapsesNMDA receptorAnimalsReceptors Dopamine D5Nerve NetReceptorLong-term depressionNeuronal memory allocationNeuroscienceThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Effects of chronic fluoxetine treatment on the rat somatosensory cortex: Activation and induction of neuronal structural plasticity

2009

Recent hypotheses support the idea that disruption of normal neuronal plasticity mechanisms underlies depression and other psychiatric disorders, and that antidepressant treatment may counteract these changes. In a previous report we found that chronic fluoxetine treatment increases the expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule involved in neuronal structural plasticity, in the somatosensory cortex. In the present study we intended to find whether, in fact, cell activation and neuronal structural remodeling occur in parallel to changes in the expression of this molecule. Using immunohistochemistry, we found that chronic fluoxetine trea…

MaleNeuronsNeuronal PlasticityDose-Response Relationship DrugGeneral NeuroscienceCentral nervous systemHippocampusSomatosensory CortexBiologySomatosensory systemRatsRats Sprague-Dawleymedicine.anatomical_structurenervous systemFluoxetineApical dendriteNeuroplasticitymedicineAnimalsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeCell activationPrefrontal cortexNeuroscienceNeuroscience Letters
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PSA-NCAM expression in the rat medial prefrontal cortex

2005

The rat medial prefrontal cortex, an area considered homologous to the human prefrontal cortex, is a region in which neuronal structural plasticity has been described during adulthood. Some plastic processes such as neurite outgrowth and synaptogenesis are known to be regulated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). Since PSA-NCAM is present in regions of the adult CNS which are undergoing structural remodeling, such as the hypothalamus or the hippocampus, we have analyzed the expression of this molecule in the medial prefrontal cortex of adult rats using immunohistochemistry. PSA-NCAM immunoreactivity was found both in cell bodies and in the neuropil of…

MaleNeuropilNeuriteInterneuronAntimetabolitesCell SurvivalSynaptophysinSynaptogenesisPrefrontal CortexHippocampusNeural Cell Adhesion Molecule L1BiologyRats Sprague-DawleyNeuroplasticityNeuropilmedicineAnimalsFluorescent Antibody Technique IndirectPrefrontal cortexNeuronsNeuronal PlasticityGlutamate DecarboxylasePyramidal CellsGeneral NeuroscienceImmunohistochemistryRatsPhenotypemedicine.anatomical_structureBromodeoxyuridinenervous systemSialic AcidsNeural cell adhesion moleculeNeuroscienceNeuroscience
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Acute intermittent nicotine treatment induces fibroblast growth factor-2 in the subventricular zone of the adult rat brain and enhances neuronal prec…

2007

Abstract Over the past years, evidence has accumulated that stem cells are present in the adult brain, and generate neurons and/or glia from two active germinal zones: the subventricular zone (SVZ) of the lateral ventricles and the subgranular zone (SGZ) of the dentate gyrus of the hippocampus. This study shows that acute intermittent nicotine treatment significantly enhances neuronal precursor cell proliferation in the SVZ of adult rat brain, but not in the SGZ of the hippocampus, and pre-treatment with mecamylamine, a nonselective nAChR antagonist, blocks the enhanced precursor proliferation by nicotine. This effect is supported by up-regulation of fibroblast growth factor-2 (FGF-2) mRNA …

MaleNicotinemedicine.medical_specialtyBasic fibroblast growth factorSubventricular zoneNicotinic AntagonistsReceptors NicotinicBiologyFibroblast growth factorSettore BIO/09 - FisiologiaHippocampusSubgranular zonechemistry.chemical_compoundLateral VentriclesInternal medicinePrecursor cellmedicineAnimalsPyrrolesNicotinic AgonistsRNA MessengerReceptor Fibroblast Growth Factor Type 1Rats WistarCell ProliferationNeuronsNeuronal PlasticityStem CellsGeneral NeuroscienceFibroblast growth factor receptor 1Dentate gyrusNeurogenesisCell DifferentiationNerve RegenerationRatsUp-RegulationCell biologymedicine.anatomical_structureEndocrinologynervous systemchemistryneurogenesis FGF-2 FGFR-1 subventricular zone nicotineFibroblast Growth Factor 2Neuroscience
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Chronic fluoxetine treatment alters the structure, connectivity and plasticity of cortical interneurons

2014

Novel hypotheses suggest that antidepressants, such as the selective serotonin reuptake inhibitor fluoxetine, induce neuronal structural plasticity, resembling that of the juvenile brain, although the underlying mechanisms of this reopening of the critical periods still remain unclear. However, recent studies suggest that inhibitory networks play an important role in this structural plasticity induced by fluoxetine. For this reason we have analysed the effects of a chronic fluoxetine treatment in the hippocampus and medial prefrontal cortex (mPFC) of transgenic mice displaying eGFP labelled interneurons. We have found an increase in the expression of molecules related to critical period pla…

MalePERINEURONAL NET EXPRESSIONTime FactorsDendritic spinePSA-NCAMCritical period plasticityHippocampusCell CountADULT BRAIN PLASTICITYTREATMENT INCREASESHippocampusMice0302 clinical medicinePharmacology (medical)Prefrontal cortexCerebral Cortex0303 health sciencesNeuronal PlasticitybiologyGlutamate DecarboxylaseMEDIAL PREFRONTAL CORTEXPOLYSIALIC ACIDmusculoskeletal neural and ocular physiologyPerineuronal net3. Good healthPsychiatry and Mental healthParvalbuminsmedicine.anatomical_structureCerebral cortexCELL-ADHESION MOLECULEAntidepressive Agents Second-GenerationDendritic SpinesGreen Fluorescent ProteinseducationMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1Inhibitory postsynaptic potentialRAT HIPPOCAMPUS03 medical and health sciencesmedicineAnimalsPSA-NCAM EXPRESSION030304 developmental biologyPharmacologyperineuronal netsinterneuronsCENTRAL-NERVOUS-SYSTEMfluoxetine3112 NeurosciencesGene Expression Regulationnervous systemVesicular Glutamate Transport Protein 1Sialic Acidsbiology.proteinNeural cell adhesion moleculeNerve NetNeuroscience030217 neurology & neurosurgeryParvalbuminThe International Journal of Neuropsychopharmacology
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Membrane-Derived Phospholipids Control Synaptic Neurotransmission and Plasticity

2015

Synaptic communication is a dynamic process that is key to the regulation of neuronal excitability and information processing in the brain. To date, however, the molecular signals controlling synaptic dynamics have been poorly understood. Membrane-derived bioactive phospholipids are potential candidates to control short-term tuning of synaptic signaling, a plastic event essential for information processing at both the cellular and neuronal network levels in the brain. Here, we showed that phospholipids affect excitatory and inhibitory neurotransmission by different degrees, loci, and mechanisms of action. Signaling triggered by lysophosphatidic acid (LPA) evoked rapid and reversible depress…

MalePatch-Clamp TechniquesQH301-705.5NeurotransmissionBiologyInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyMicePregnancySynaptic augmentationMetaplasticityAnimalsRats WistarBiology (General)Motor Neuronsrho-Associated KinasesNeuronal PlasticityGeneral Immunology and MicrobiologyCalcineurinGeneral NeuroscienceReceptors GABA-ACell biologySynaptic fatigueBiochemistrySynapsesSynaptic plasticityExcitatory postsynaptic potentialFemalelipids (amino acids peptides and proteins)Synaptic signalingLysophospholipidsrhoA GTP-Binding ProteinGeneral Agricultural and Biological SciencesResearch Article
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