Search results for " Polymorphism."

showing 10 items of 1006 documents

Association Between Platelet Endothelial Cellular Adhesion Molecule-1 Polymorphisms and Atherosclerosis: Results of a Study on Patients from Northern…

2010

Adhesion of circulating cells to the arterial surface is among the first detectable events in atherogenesis. Cellular adhesion molecules, expressed by the vascular endothelium and by circulating leukocytes, mediate cell recruitment and their transendothelial migration. Platelet endothelial cellular adhesion molecule-1 (PECAM-1), involved in this migration, has been associated with the development of atherosclerosis. Studies have investigated an association between coronary artery disease (CAD) and single-nucleotide polymorphisms (SNPs) located in functionally important domains of the PECAM-1 gene with inconsistent results. Thus, we have analyzed the distribution of V125L, N563S, and G670R S…

MaleUntranslated regionAgingGenotypeSingle-nucleotide polymorphismCoronary Artery DiseaseBiologyPolymorphism Single NucleotideCoronary artery diseaseGene FrequencymedicineHumansGenetic Predisposition to DiseasePlateletCell adhesionGeneAgedCell adhesion moleculeAdhesionMiddle AgedAtherosclerosismedicine.diseasePlatelet Endothelial Cell Adhesion Molecule-1ItalyCase-Control StudiesImmunologycardiovascular systemFemaleGeriatrics and GerontologyRejuvenation Research
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Association Between Single Nucleotide Polymorphisms in the Cyclooxygenase-2, Tumor Necrosis Factor-α, and Vascular Endothelial Growth Factor-A Genes,…

2011

Cyclooxygenase-2 (COX-2), vascular endothelial growth factor-A (VEGF-A), and tumor necrosis factor-α (TNF-α) are mediators of inflammation and angiogenesis; all of them are produced in liver cirrhosis (LC) and in hepatocellular carcinoma (HCC). It was proposed that there is an association between single nucleotide polymorphisms (SNPs) and HCC. These allelic variants influence the transcriptional activity of these genes, and therefore the proteins levels. The VEGF-A pathway is a potential therapeutic target in HCC, and several antiangiogenic agents have entered clinical trials in HCC. We evaluated the frequency of SNPs of COX-2, TNF-α, and VEGF-A genes in patients with HCC versus LC patients…

MaleVascular Endothelial Growth Factor AHeterozygoteCarcinoma HepatocellularCirrhosisAngiogenesisSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideBiochemistryCOX-2 TNFa VEGF Hepatocellular Carcinoma SNPsGeneticsmedicineHumansneoplasmsMolecular BiologyTumor Necrosis Factor-alphaHomozygoteLiver Neoplasmsnucleotide polymorphisms cyclooxygenase-2 tumor necrosis factor-α vascular endothelial growth factor-A geneshepatocellular carcinoma.Heterozygote advantagemedicine.diseasedigestive system diseasesVascular endothelial growth factor ACyclooxygenase 2Hepatocellular carcinomaImmunologyCancer researchMolecular MedicineFemaleTumor necrosis factor alphaRestriction fragment length polymorphismBiotechnologyOMICS: A Journal of Integrative Biology
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Deletion of the Hunter gene and both DXS466 and DXS304 in a patient with mucopolysaccharidosis type II.

1992

Hunter syndrome is an X-linked mucopoly-saccharidosis due to deficiency of the lysosomal enzyme iduronate-2-sulfatase (IDS). A cDNA clone containing the entire coding region of the human IDS gene, mapped in Xq28, has been used as molecular probe to study a patient with Hunter syndrome. A submicroscopic deletion has been detected that spans the IDS gene as well as DXS466 and DXS304, 2 loci mapped probably not more than 900 kb from the IDS locus. A detailed clinical description of the patient is provided and his phenotype is compared to that of other patients with IDS deletion described recently. By following the segregation of a restriction fragment length polymorphism at the IDS locus in th…

MaleX ChromosomeLocus (genetics)Iduronate SulfataseBiologyGene mappingmedicineHumansMucopolysaccharidosis type IIChildGenetics (clinical)X chromosomeMucopolysaccharidosis IIGeneticsIduronate-2-sulfataseChromosome MappingHunter syndromeDNAmedicine.diseaseXq28PedigreeBlotting SouthernFemaleRestriction fragment length polymorphismChromosome DeletionPolymorphism Restriction Fragment LengthAmerican journal of medical genetics
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Molecular analysis in patients with mucopolysaccharidosis type II suggests that DXS466 maps within the Hunter gene

1993

Hunter disease is an X-linked mucopolysaccharidosis caused by deficiency of the lysosomal enzyme iduronate-2-sulfatase (IDS). Using the IDS cDNA and DNA probes corresponding to loci flanking the IDS locus, we performed molecular genetic studies in two patients with Hunter syndrome. An interstitial deletion spanning the middle part of the IDS gene was found in the first patient. The second patient carries a gross gene rearrangement that can be detected after HindIII or EcoRI digestion of genomic DNA, and is similar to that found recently in seven unrelated Hunter patients. Our data suggest that the structural aberration observed is a partial intragenic inversion. As the same altered hybridiz…

MaleX ChromosomeRestriction MappingLocus (genetics)Iduronate SulfataseHindIIIDeoxyribonuclease EcoRIGeneticsmedicineHumansMucopolysaccharidosis type IIChildDeoxyribonucleases Type II Site-SpecificGenetics (clinical)Mucopolysaccharidosis IIGeneticsbiologyHybridization probeHunter syndromeGene rearrangementmedicine.diseaseMolecular biologyBlotting Southerngenomic DNAChild Preschoolbiology.proteinRestriction fragment length polymorphismDNA ProbesPolymorphism Restriction Fragment LengthHuman Genetics
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Common variation in PHACTR1 is associated with susceptibility to cervical artery dissection

2014

Item does not contain fulltext Cervical artery dissection (CeAD), a mural hematoma in a carotid or vertebral artery, is a major cause of ischemic stroke in young adults although relatively uncommon in the general population (incidence of 2.6/100,000 per year). Minor cervical traumas, infection, migraine and hypertension are putative risk factors, and inverse associations with obesity and hypercholesterolemia are described. No confirmed genetic susceptibility factors have been identified using candidate gene approaches. We performed genome-wide association studies (GWAS) in 1,393 CeAD cases and 14,416 controls. The rs9349379[G] allele (PHACTR1) was associated with lower CeAD risk (odds ratio…

Male[SDV.IB.IMA]Life Sciences [q-bio]/Bioengineering/Imaging[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyMyocardial InfarctionGenome-wide association studyCarotid Artery Internal DissectionGastroenterologyepidemiology [Carotid Artery Internal Dissection]Brain Ischemia0302 clinical medicineMigraine DisorderOdds RatioFinlandVertebral Artery Dissection0303 health scienceseducation.field_of_studyepidemiology [Hypercholesterolemia]MESH: Middle AgedMESH: Polymorphism Single NucleotidePhactr-1 protein humanMESH: Brain IschemiaMESH: Follow-Up Studies3. Good healthMESH: Myocardial InfarctionHumanmedicine.medical_specialtyMigraine DisordersHypercholesterolemiaMESH: Vertebral Artery DissectionLower riskgenetics [Brain Ischemia]ArticleFollow-Up StudieMESH: Carotid Artery Internal Dissection03 medical and health sciencesGeneticSDG 3 - Good Health and Well-beinggenetics [Carotid Artery Internal Dissection]GeneticsGenetic predispositionepidemiology [Brain Ischemia]Humansepidemiology [Vertebral Artery Dissection]PolymorphismeducationAllelesMESH: Humansgenetics [Vertebral Artery Dissection]MESH: AdultOdds ratioMicrofilament Proteinmedicine.diseaseAdult; Brain Ischemia; Carotid Artery Internal Dissection; Female; Finland; Follow-Up Studies; Genetic Pleiotropy; Genetic Predisposition to Disease; Genome-Wide Association Study; Humans; Hypercholesterolemia; Hypertension; Male; Microfilament Proteins; Middle Aged; Migraine Disorders; Myocardial Infarction; Obesity; Odds Ratio; Risk Factors; Vertebral Artery Dissection; Alleles; Polymorphism Single NucleotideMESH: Genome-Wide Association StudyCarotid ArteryMESH: Female030217 neurology & neurosurgeryepidemiology [Finland]Cervical ArteryVertebral artery dissectionepidemiology [Hypertension]MESH: HypertensionRisk FactorsMESH: Risk FactorsMESH: ObesityStrokeAlleleGeneticsDissectionMESH: FinlandMicrofilament ProteinsMESH: Genetic Predisposition to DiseaseMESH: HypercholesterolemiaGenetic PleiotropySingle NucleotideMiddle AgedMESH: Migraine DisordersDisorders of movement Donders Center for Medical Neuroscience [Radboudumc 3]epidemiology [Myocardial Infarction][INFO.INFO-TI]Computer Science [cs]/Image Processing [eess.IV]HypertensionFemale[SPI.SIGNAL]Engineering Sciences [physics]/Signal and Image processingAdultPopulationMESH: Genetic Pleiotropyphysiology [Microfilament Proteins]BiologyPolymorphism Single NucleotideMESH: Microfilament ProteinsInternal medicineddc:570medicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingGenetic Predisposition to DiseaseObesity030304 developmental biologyepidemiology [Obesity]Risk FactorMESH: Alleles[INFO.INFO-CV]Computer Science [cs]/Computer Vision and Pattern Recognition [cs.CV]InternalMESH: Odds RatioMESH: Maleepidemiology [Migraine Disorders]genetics [Microfilament Proteins]Follow-Up StudiesGenome-Wide Association Study
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Associations of the MCM6-rs3754686 proxy for milk intake in Mediterranean and American populations with cardiovascular biomarkers, disease and mortal…

2016

Controversy persists on the association between dairy products, especially milk, and cardiovascular diseases (CVD). Genetic proxies may improve dairy intake estimations, and clarify diet-disease relationships through Mendelian randomization. We meta-analytically (n T (nonpersistence>persistence), dairy intake, and CVD biomarkers in American (Hispanics, African-American and Whites) and Mediterranean populations. Moreover, we analyzed longitudinal associations with milk, CVD and mortality in PREDIMED), a randomized Mediterranean diet (MedDiet) intervention trial (n = 7185). The MCM6-rs3754686/MCM6-rs309180 (as proxy), LP-allele (T) was strongly associated with higher milk intake, but inconsis…

MalealelosMediterranean dietMETABOLIC RISK-FACTORShumanosSINGLE-NUCLEOTIDE POLYMORPHISMComponente 6 del complejo de mantenimiento de minicromosomasDisease030204 cardiovascular system & hematologyMediterraneanAssociationsDiet MediterraneanMediterranean populationMCM6-rs37546860302 clinical medicineMedicineDisease030212 general & internal medicinemediana edadDAIRY CONSUMPTION2. Zero hungerancianoeducation.field_of_studyBioquímica y tecnologíaMultidisciplinarydietaMediterranean RegionHàbits alimentarisMendelian Randomization AnalysisMiddle AgedLlet3. Good healthBiochemistry and technologyBODY-WEIGHTCardiovascular diseasesMilkAmerican populationCardiovascular DiseasesMeta-analysisMarcadors bioquímicsFOOD-FREQUENCY QUESTIONNAIREFemaleLlet -- Aspectes nutricionalsGenetic MarkersFood habitsPopulationenfermedades cardiovascularesUNITED-STATESSistema cardiovascular malaltiesBioquímica i biotecnologiaArticleDIETleche03 medical and health sciencesSex Factors:Ciencias de la Salud::Medicina preventiva [Materias Investigacion]Sistema cardiovascular -- Malalties -- Aspectes genèticsEnvironmental healthMendelian randomizationMortalitatSNPAnimalsHumansPUERTO-RICANMortalityeducationMilk intakeLACTASE-PERSISTENCE PHENOTYPEAllelescardiovascular biomarkersAgedbusiness.industryMalalties cardiovascularsCardiovascular biomarkersmarcadores genéticosMendelian Randomization AnalysisMinichromosome Maintenance Complex Component 6United StatesDietBiotechnologyCardiovascular diseases (CVD)Lactase persistence2045-2322animalesanálisis de la aleatorización mendelianaDOSE-RESPONSE METAANALYSIS:Ciencias de la Salud::Endocrinología [Materias Investigacion]business
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Association of SUMO4 M55V polymorphism with autoimmune diabetes in Latvian patients.

2006

Small ubiquitin-related modifier (SUMO4), located in IDDM5, has been identified as a potential susceptibility gene for type 1 diabetes mellitus (T1DM). The novel polymorphism M55V, causing an amino acid change in the evolutionarily conserved met55 residue has been shown to activate the nuclear factor kappaB (NF-kappaB), hence the suspected role of SUMO4 in the pathogenicity of T1DM. The M55V polymorphism has been shown to be associated with susceptibility to T1DM in Asians, but not in Caucasians. Latent autoimmune diabetes in adults (LADA) is a slowly progressive form of T1DM and SUMO4 M55V has not been studied in LADA to date. The current study aims to test whether Latvians are similar to …

Maleendocrine system diseasesAdolescentHuman leukocyte antigenBiologyPolymerase Chain ReactionPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular Biologylaw.inventionAutoimmune DiseasesMethionineHistory and Philosophy of ScienceGene Frequencyimmune system diseaseslawmedicineHumansGenetic Predisposition to DiseaseAlleleChildPolymerase chain reactionAllelesGeneticsType 1 diabetesGeneral NeuroscienceInfantmedicine.diseasePathogenicityLatviaDiabetes Mellitus Type 1Autoimmune diabetesCase-Control StudiesChild PreschoolSmall Ubiquitin-Related Modifier ProteinsAmino acid changeFemaleRestriction fragment length polymorphismPolymorphism Restriction Fragment LengthAnnals of the New York Academy of Sciences
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Stronger Association of Common Variants in TCF7L2 Gene with Nonobese Type 2 Diabetes in the Latvian Population

2012

Polymorphisms in the gene coding for transcription factor 7 like 2 (TCF7L2) are recognized as the strongest common genetic risk factors for type 2 diabetes (T2D) across multiple ethnicities. This study was conducted to evaluate an association between TCF7L2 variants and diabetes susceptibility in the population of Latvia. We genotyped 4 single nucleotide polymorphisms (SNP) rs7901695, rs7903146, rs11196205 and rs12255372 in 1 093 controls and 1 043 diabetic subjects. Association with T2D was found for 3 SNPs rs7901695, rs7903146 and rs12255372 in the whole sample (under an additive genetic model, the adjusted odds ratios (OR) were 1.26, 95% CI [1.08-1.48], P=0.003; OR=1.32, 95% CI [1.12-1.5…

Maleendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismPopulationSingle-nucleotide polymorphismType 2 diabetesBiologyPolymorphism Single NucleotideBody Mass IndexEndocrinologyPolymorphism (computer science)Databases GeneticGenetic modelInternal MedicinemedicineHumansSNPGenetic Predisposition to DiseaseObesityeducationGenetic Association StudiesGeneticseducation.field_of_studynutritional and metabolic diseasesGeneral MedicineOdds ratioMiddle Agedmedicine.diseaseLatviaDiabetes Mellitus Type 2Case-Control StudiesFemaleTranscription Factor 7-Like 2 ProteinTCF7L2Experimental and Clinical Endocrinology & Diabetes
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Low Levels of Mitochondrial DNA and Symbiont Diversity in the Worldwide Agricultural Pest, the Greenhouse Whitefly Trialeurodes vaporariorum (Hemipte…

2015

Trialeurodes vaporariorum, the greenhouse whitefly, is a cosmopolitan agricultural pest. Little is known about the genetic diversity of T. vaporariorum and the bacterial symbionts associated with this species. Here, we undertook a large phylogeographic study by investigating both the mitochondrial (mt) diversity and the infection status of 38 T. vaporariorum collections from 18 countries around the world. Genetic diversity of T. vaporariorum was studied by analyzing sequence data from the mt cytochrome oxidase I, cytochrome b, and NADH dehydrogenase subunit 5 genes. Maximum-likelihood (ML) phylogeny reconstruction delineated 2 clades characterized by limited sequence divergence: one clade c…

Malefood.ingredientArsenophonusMolecular Sequence DataZoologyTrialeurodesDNA MitochondrialHemipterafoodSpecies SpecificityGenetic variationBotanyGeneticsAnimalssymbiont communitiesSymbiosisMolecular BiologyPhylogenyGenetics (clinical)ta415Likelihood FunctionsGenetic diversityBacteriaBase SequenceModels GeneticbiologyCytochrome bta1184mtDNA polymorphismGenetic VariationGreenhouse whiteflygenetic diversitySequence Analysis DNAbiology.organism_classificationPhylogeographyta1181Multilocus sequence typingFemaleWolbachiaArsenophonusAnimal DistributionBiotechnologyJournal of Heredity
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Homoarginine Levels are Regulated by L-Arginine: Glycine Amidinotransferase and Affect Stroke Outcome: Results from Human and Murine Studies

2013

Background— Endogenous arginine homologues, including homoarginine, have been identified as novel biomarkers for cardiovascular disease and outcomes. Our studies of human cohorts and a confirmatory murine model associated the arginine homologue homoarginine and its metabolism with stroke pathology and outcome. Methods and Results— Increasing homoarginine levels were independently associated with a reduction in all-cause mortality in patients with ischemic stroke (7.4 years of follow-up; hazard ratio for 1-SD homoarginine, 0.79 [95% confidence interval, 0.64–0.96]; P =0.019; n=389). Homoarginine was also independently associated with the National Institutes of Health Stroke Scale+age score …

Malegenetics [Homoarginine]AmidinotransferasesArginineGenome-wide association studyCohort StudiesMicesingle nucleotide polymorphismMedicinehomoarginineProspective StudiesStrokegenetics [Arginine]CARDIOVASCULAR RISKHazard ratioMOUSE MODELMiddle Ageddiagnosis [Stroke]strokeDEFICIENCYTreatment OutcomeISCHEMIC-STROKEgenetics [Stroke]genetics [Amidinotransferases]genetics [Polymorphism Single Nucleotide]FemaleCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtySingle-nucleotide polymorphismMASS-SPECTROMETRIC DETERMINATIONArginineGUANIDINO COMPOUNDSPhysiology (medical)Internal medicineglycine amidinotransferaseAnimalsHumansCREATINEddc:610Translational research Energy and redox metabolism [ONCOL 3]AgedNITRIC-OXIDEBLOOD-FLOWbusiness.industryVascular diseasemedicine.diseaseHomoarginineCEREBRAL-ARTERY OCCLUSIONL-arginine:glycine amidinotransferaseMice Inbred C57BLDisease Models AnimalEndocrinologyHEK293 CellsGlycinegenome-wide association studiesHuman medicineArginine:glycine amidinotransferasebusinessGenome-Wide Association StudyCirculation
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