Search results for " Purpura"

showing 10 items of 66 documents

PANCREATITIS IN HENOCH-SCHONLEIN PURPURA. A SINGLE CENTRE OBSERVATIONAL STUDY

2018

Introduction: Henoch-Schönlein purpura (HSP) is the most frequent vasculitis in children. Typically, it is characterized by palpable purpura, joints swelling, arthralgia, abdominal pain with possible intestinal bleeding. In more severe cases, the patients show acute abdomen. Acute pancreatitis is a rare dramatically evolutive, life-treating manifestation of SHS and it can be associated with a fulminant course. Persistent abdominal pain, need to be investigated by the dosage of serum pancreatic amylase, lipase and by abdominal MRI. In these patients, corticosteroid treatment is recommended and must be associated with parenteral feeding. Objectives: We analysed the full series of children wit…

Settore MED/38 - Pediatria Generale E SpecialisticaPANCREATITISHENOCH-SCHONLEIN PURPURA
researchProduct

Seidlmayer’s purpura: five cases and review of the literature

2014

Settore MED/38 - Pediatria Generale E SpecialisticaSeidlmayer’s purpura review
researchProduct

A critical evaluation of caplacizumab for the treatment of acquired thrombotic thrombocytopenic purpura

2020

Introduction: Acquired thrombotic thrombocytopenic purpura (aTTP) is a thrombotic microangiopathy caused by inhibitory autoantibodies against ADAMTS13 protein. Until recently, the combination of plasma exchange (PEX) and immunosuppression has been the standard front-line treatment in this disorder. However, aTTP-related mortality, refractoriness, and relapse are still a matter of concern. Areas covered: The better understanding of the pathophysiological mechanisms of aTTP has allowed substantial improvements in the diagnosis and treatment of this disease. Recently, the novel anti-VWF nanobody caplacizumab has been approved for acute episodes of aTTP. Caplacizumab is capable to block the adh…

Thrombotic microangiopathyExacerbationvirusesmedicine.medical_treatmentADAMTS13 ProteinDiseaseBioinformaticsAutoantigens03 medical and health sciencesPlatelet Adhesiveness0302 clinical medicineFibrinolytic AgentsProtein DomainsCrotalid Venomsvon Willebrand FactormedicineHumansImmunologic FactorsMulticenter Studies as TopicLectins C-TypeMolecular Targeted TherapyDrug ApprovalClinical Trials as TopicAcquired Thrombotic Thrombocytopenic PurpuraPlasma ExchangePurpura Thrombotic Thrombocytopenicbusiness.industryStandard treatmentfungiImmunosuppressionDrugs InvestigationalHematologyAptamers NucleotideSingle-Domain Antibodiesbiochemical phenomena metabolism and nutritionmedicine.diseaseCombined Modality TherapyRecombinant ProteinsADAMTS13AcetylcysteineTreatment Outcome030220 oncology & carcinogenesisDrug Therapy CombinationCaplacizumabbusinessImmunosuppressive Agents030215 immunologyExpert Review of Hematology
researchProduct

COVID-19 as a Potential Trigger for Immune Thrombotic Thrombocytopenic Purpura and Reason for an Unusual Treatment: A Case Report

2021

AbstractImmune thrombotic thrombocytopenic purpura (iTTP) is a rare autoimmune disorder characterized by severely reduced activity of the von Willebrand factor (VWF)-cleaving protease ADAMTS13 (a disintegrin and metalloproteinase with a thrombospondin type 1 motif, member 13) due to autoantibodies. This leads to the development of pathogenic multimers of VWF, causing a thrombotic microangiopathy with decreased number of platelets, hemolysis, and life-threatening tissue ischemia of mostly brain, heart, and kidneys. Standard treatment of iTTP involves daily plasma exchange to remove ultra large multimers of VWF, inhibitors, substituting ADAMTS13, and the accompaniment of an immunosuppressive …

Thrombotic microangiopathybiologybusiness.industryThrombotic thrombocytopenic purpuraAutoantibodyHematology030204 cardiovascular system & hematologymedicine.diseaseADAMTS1303 medical and health sciences0302 clinical medicineVon Willebrand factorhemic and lymphatic diseases030220 oncology & carcinogenesisImmunologybiology.proteinmedicineRituximabPlateletCaplacizumabbusinessmedicine.drugHämostaseologie
researchProduct

Coelomocytes and post-traumatic response in the common sea star Asterias rubens.

2008

Coelomocytes are recognized as the main cellular component of the echinoderm immune system. They are the first line of defense and their number and type can vary dramatically during infections or following injury. Sea stars have been used as a model system to study the regeneration process after autotomy or predation. In the present study we examined the cellular and biochemical responses of coelomocytes from the European sea star Asterias rubens to traumatic stress using immunochemical and biochemical approaches. In terms of trauma and post-traumatic stress period, here we consider the experimental arm amputation and the repair phase involved in the first 24 hours post-amputation, which mi…

Time FactorsImmunocytochemistryPopulationCell CountBiochemistryAndrologymedicineAnimalsHSP70 Heat-Shock ProteinsPseudopodiaeducationGlycoproteinseducation.field_of_studyPhagocytesbiologyAsteriasAntibodies MonoclonalCell BiologyOriginal Articlesbiology.organism_classificationStrongylocentrotus purpuratusCoelomic epitheliummedicine.anatomical_structureEchinodermAsteriasImmunologyCoelomAutotomyCell stresschaperones
researchProduct

Anti‐laminin auto antibodies in ANCA‐associated vasculitis

2000

Background. Endothelial cell damage occurs during vasculitic processes in vivo. With the alteration of the endothelium, exposure to basement membrane components may occur with induction of humoral immunity. Methods. In the present study, we evaluated the prevalence of antibodies against the basement membrane antigen laminin (LMN) in patients with ANCA-associated systemic vasculitis (AASV), pathologic controls (systemic lupus erythematosus, mixed cryoglobulinaemia, Henoch Schonlein purpura, primary glomerulonephritis) and normal individuals. Results. By ELISA, 21.6% of AASV (16/74) and 10% of pathologic controls (3/30), but only one of the normal controls (2.8%) had these antibodies (P = 0.0…

VasculitisPathologymedicine.medical_specialtyHenoch-Schonlein purpuraMyeloblastinEnzyme-Linked Immunosorbent AssayAntibodies Antineutrophil CytoplasmicEpitopesAntigenReference Valuesimmune system diseasesmedicineHumansReference Valuecardiovascular diseasesAutoantibodiesPeroxidaseAnti-neutrophil cytoplasmic antibodyTransplantationbusiness.industrySerine EndopeptidasesGranulomatosis with PolyangiitisGlomerulonephritismedicine.diseaseAutoantibodieSerine EndopeptidaseNephrologyImmunologyEpitopeLamininGranulomatosis with PolyangiitiGranulomatosis with polyangiitisVasculitisbusinessMicroscopic polyangiitisHumanSystemic vasculitisNephrology Dialysis Transplantation
researchProduct

Idiopathic Seidlmayer's Purpura: A Case Report

2014

Acute hemorrhagic edema of infancy (AHEI) was considered a rare form of Henoch-Schönlein purpura; however, it is now regarded as an independent disease typically involving patients aged 4-24 months. The authors describe the clinical case of a toddler aged 8 months, with skin erythematous pomphoid<b> </b>lesions, treated at home with topical steroids without benefits. The appearance of new lesions and the worsening of the previous skin signs induced the parents to drive the child to the hospital. The medical history revealed the administration of a vaccine dose 2 months before.

Vasculitismedicine.medical_specialtySeidlmayer's purpuraDermatologyDiseaseAcute hemorrhagic edema of infancyPublished online: May 2014Settore MED/38 - Pediatria Generale E Specialisticalcsh:DermatologymedicineCorticosteroidsCorticosteroidMedical historyToddlerbusiness.industryAcute hemorrhagic edema of infancylcsh:RL1-803medicine.diseaseDermatologySurgeryPurpuraClinical casemedicine.symptombusinessVasculitisSeidlmayer’s purpura
researchProduct

Immunogenic hotspots in the spacer domain of ADAMTS13 in immune‐mediated thrombotic thrombocytopenic purpura

2021

International audience; Background Immune-mediated thrombotic thrombocytopenic purpura (iTTP) is caused by anti-ADAMTS13 autoantibodies inducing a severe deficiency of ADAMTS13. Epitope mapping studies on samples obtained during acute iTTP episodes have shown that the iTTP immune response is polyclonal, with almost all patients having autoantibodies targeting the spacer domain of ADAMTS13.Objectives To identify the immunogenic hotspots in the spacer domain of ADAMTS13.Patients/methods A library of 11 full-length ADAMTS13 spacer hybrids was created in which amino acid regions of the spacer domain of ADAMTS13 were exchanged by the corresponding region of the spacer domain of ADAMTS1. Next, th…

autoantibodiesADAMTS13 Protein030204 cardiovascular system & hematologyEpitope03 medical and health sciencesEpitopes0302 clinical medicineVon Willebrand factorimmunophenotypinghemic and lymphatic diseasesHumansthrombotic thrombocytopenic purpurachemistry.chemical_classificationbiologyPurpura Thrombotic ThrombocytopenicAutoantibodyHematologyMolecular biologyADAMTS13ADAMTS133. Good healthAmino acidepitope mappingEpitope mappingchemistryPolyclonal antibodiesImmunoglobulin Gbiology.proteinDNA IntergenicAntibody[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
researchProduct

Heat Shock Proteins in Sea Urchin Embryos. (heat shock proteins/sea urchin embryos)

1989

biologyEcologyHeat shock proteinEmbryoCell BiologySea urchin embryobiology.organism_classificationStrongylocentrotus purpuratusParacentrotus lividusDevelopmental BiologyCell biologyDevelopment, Growth and Differentiation
researchProduct

Prolonged Response of Chronic Immune Thrombocytopenic Purpura (ITP) to Extracorporeal Immunoadsorption

2000

business.industry030232 urology & nephrologyBiomedical EngineeringMedicine (miscellaneous)BioengineeringGeneral MedicineExtracorporeal immunoadsorption030204 cardiovascular system & hematologymedicine.diseaseThrombocytopenic purpuraBiomaterials03 medical and health sciences0302 clinical medicineImmune systemImmunologymedicinebusinessThe International Journal of Artificial Organs
researchProduct