Search results for " Regulation"

showing 10 items of 3187 documents

Allosteric modulation of nicotinic receptors as a treatment strategy for Alzheimer's disease.

2000

Impairment of the central cholinergic system has a pivotal role in the cognitive decline observed in patients with Alzheimer’s disease (AD). One of the most prominent cholinergic deficits is the reduced number of nicotinic acetylcholine receptors (nAChR) in the brain. Since these receptors are important for memory and learning, enhancing nicotinic neurotransmission is a promising treatment strategy for AD. The two most common approaches to correcting these cholinergic deficits are to increase the synaptic availability of acetylcholine (ACh) by inhibiting acetylcholinesterase (AChE), or to mimic the effects of ACh (nicotinic agonists) by acting directly on nicotinic receptors. Clinical studi…

Agonistmedicine.drug_classGalantamineCognitive NeuroscienceAllosteric regulationCholinergic AgentsPharmacologyReceptors NicotinicAcetylcholinesteraseSynaptic TransmissionPsychiatry and Mental healthchemistry.chemical_compoundNicotinic agonistchemistryAlzheimer DiseaseGalantaminemedicineCholinergicHumansGeriatrics and GerontologyCognitive declineNeuroscienceAcetylcholinemedicine.drugDementia and geriatric cognitive disorders
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Postnatal alterations of the inhibitory synaptic responses recorded from cortical pyramidal neurons in the Lis1/sLis1 mutant mouse

2006

Mutations in the mouse Lis1 gene produce severe alterations in the developing cortex. We have examined some electrophysiological responses of cortical pyramidal neurons during the early postnatal development of Lis/sLis1 mutant mice. In P7 and P30 Lis1/sLis1 neurons we detected a lower frequency and slower decay phase of mIPSCs, and at P30 the mIPSCs amplitude and the action potential duration were reduced. Zolpidem (an agonist of GABAA receptors containing the alpha1 subunit) neither modified the amplitude nor the decay time of mIPSCs at P7 in Lis1/sLis1 neurons, whereas it increased the decay time at P30. The levels of GABAA receptor alpha1 subunit mRNA were reduced in the Lis1/sLis1 brai…

Agonistmedicine.medical_specialtyZolpidemPyridinesmedicine.drug_classAction PotentialsIn Vitro TechniquesBiologyInhibitory postsynaptic potentialMiceCellular and Molecular NeuroscienceInternal medicinemedicineAnimalsReceptorGABA AgonistsMolecular BiologyCerebral CortexReverse Transcriptase Polymerase Chain ReactionGABAA receptorPyramidal CellsAge FactorsGene Expression Regulation DevelopmentalCell BiologyElectric StimulationMice Mutant StrainsCortex (botany)ZolpidemElectrophysiologymedicine.anatomical_structureEndocrinologyAnimals NewbornInhibitory Postsynaptic Potentialsnervous systemCerebral cortex1-Alkyl-2-acetylglycerophosphocholine EsteraseMicrotubule-Associated Proteinsmedicine.drugMolecular and Cellular Neuroscience
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Galantamine is an allosterically potentiating ligand of neuronal nicotinic but not of muscarinic acetylcholine receptors.

2003

Galantamine (Reminyl), an approved treatment for Alzheimer's disease (AD), is a potent allosteric potentiating ligand (APL) of human alpha 3 beta 4, alpha 4 beta 2, and alpha 6 beta 4 nicotinic receptors (nAChRs), and of the chicken/mouse chimeric alpha 7/5-hydroxytryptamine3 receptor, as was shown by whole-cell patch-clamp studies of human embryonic kidney-293 cells stably expressing a single nAChR subtype. Galantamine potentiates agonist responses of the four nAChR subtypes studied in the same window of concentrations (i.e., 0.1-1 microM), which correlates with the cerebrospinal fluid concentration of the drug at the recommended daily dosage of 16 to 24 mg. At concentrations10 microM, gal…

Agonistmedicine.medical_specialtymedicine.drug_classRecombinant Fusion ProteinsAllosteric regulationPhenylcarbamatesRivastigminePharmacologyReceptors NicotinicMiceAllosteric RegulationPiperidinesInternal medicineMuscarinic acetylcholine receptormedicineGalantamineAnimalsHumansDonepezilReceptorTrichlorfonCells CulturedPharmacologyNeuronsChemistryGalantamineLigand (biochemistry)Receptors MuscarinicEndocrinologyNicotinic agonistIndansTacrineMolecular MedicineCholinergicCarbamatesCholinesterase Inhibitorsmedicine.drugThe Journal of pharmacology and experimental therapeutics
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Prostaglandin D2 regulates joint inflammation and destruction in murine collagen-induced arthritis.

2012

Item does not contain fulltext OBJECTIVE: Prostaglandin D2 (PGD2) may exert proinflammatory or antiinflammatory effects in different biologic systems. Although this prostanoid and the enzymes responsible for its synthesis are up-regulated by interleukin-1beta (IL-1beta) in human chondrocytes in vitro, the role of PGD2 in arthritis remains unclear. This study was undertaken to investigate the role of PGD2 in the inflammatory response and in joint destruction during the development of collagen-induced arthritis (CIA) in mice. METHODS: PGD2 and cytokine levels in mice with CIA were determined by enzyme-linked immunosorbent assay. Expression of hematopoietic PGD synthase (h-PGDS), lipocalin-typ…

Agonistmusculoskeletal diseasesmedicine.medical_specialtyIndolesmedicine.drug_classmedicine.medical_treatmentChemokine CXCL1ImmunologyInterleukin-1betaReceptors ProstaglandinArthritisInflammationProinflammatory cytokinechemistry.chemical_compoundMiceRheumatologyBone MarrowInternal medicinemedicineImmunology and AllergyAnimalsPharmacology (medical)Receptors ImmunologicReceptorintegumentary systembusiness.industryProstaglandin D2Hydantoinsmedicine.diseaseArthritis ExperimentalLipocalinsHindlimbInterleukin-10Up-RegulationIntramolecular OxidoreductasesInterleukin 10CytokineEndocrinologychemistryMice Inbred DBACytokinesJointslipids (amino acids peptides and proteins)Prostaglandin D2Immune Regulation Auto-immunity transplantation and immunotherapy [NCMLS 2]medicine.symptombusiness
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LXR antagonists induce ABCD2 expression

2014

X-linked adrenoleukodystrophy (X-ALD) is a rare neurodegenerative disorder characterized by the accumulation of very-long-chain fatty acids resulting from a beta-oxidation defect. Oxidative stress and inflammation are also key components of the pathogenesis. X-ALD is caused by mutations in the ABCDI gene, which encodes for a peroxisomal half ABC transporter predicted to participate in the entry of VLCFA-CoA into the peroxisome, the unique site of their beta-oxidation. Two homologous peroxisomal ABC transporters, ABCD2 and ABCD3 have been proven to compensate for ABCD1 deficiency when overexpressed. Pharmacological induction of these target genes could therefore represent an alternative ther…

Agonistx-ald;very-long-chain fatty acid;lxr;hydroxycholesterol;abcd2medicine.medical_specialtymedicine.drug_classx-aldEndogenyContext (language use)ATP-binding cassette transporterBiologyATP Binding Cassette Transporter Subfamily DInternal medicinemedicineHumanslxr[ SDV.BDD ] Life Sciences [q-bio]/Development BiologyhydroxycholesterolLiver X receptorAdrenoleukodystrophyMolecular Biology[SDV.BDD]Life Sciences [q-bio]/Development BiologyLiver X ReceptorsFatty AcidsBiologie du développementNeurosciencesCell BiologyHep G2 CellsPeroxisomemedicine.diseaseOrphan Nuclear ReceptorsDevelopment BiologyHydroxycholesterolsvery-long-chain fatty acidOxidative StressEndocrinologyGene Expression RegulationCell cultureabcd2Neurons and Cognition[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Cancer researchlipids (amino acids peptides and proteins)AdrenoleukodystrophyATP-Binding Cassette Transporters[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]
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Estimating economic severity of Air Traffic Flow Management regulations

2021

The development of trajectory-based operations and the rolling network operations plan in European air traffic management network implies a move towards more collaborative, strategic flight planning. This opens up the possibility for inclusion of additional information in the collaborative decision-making process. With that in mind, we define the indicator for the economic risk of network elements (e.g., sectors or airports) as the expected costs that the elements impose on airspace users due to Air Traffic Flow Management (ATFM) regulations. The definition of the indicator is based on the analysis of historical ATFM regulations data, that provides an indication of the risk of accruing dela…

Air traffic flow managementGeneral Economics (econ.GN)ATFM regulationProcess (engineering)ATFM regulations; Cost of delay; Economic risk; Economic severity; Strategic flight planningAir traffic managementTransportationComputerApplications_COMPUTERSINOTHERSYSTEMSPlan (drawing)Management Science and Operations ResearchEconomic riskNetwork operations centerCost of delayFOS: Economics and businessNetwork elementFlight planningATFM regulationsRisk analysis (engineering)Automotive EngineeringBusinessMetric (unit)Economic severityStrategic flight planningEconomics - General EconomicsCivil and Structural Engineering
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Ajuste psicológico en cuidadores familiares de adolescentes: un análisis longitudinal durante el confinamiento por la COVID-19 en España

2021

Debido al confinamiento por la COVID-19, las familias españolas tuvieron que adaptarse a nuevas situaciones y rutinas. El objetivo de esta investigación ha sido conocer qué variables explicaban el ajuste psicológico de cuidadores familiares de adolescentes ante la COVID-19, mediante un estudio longitudinal (evaluando primero a las 2-3 semanas del inicio del estado de alarma y después tras 8 semanas de su instauración en España). Participaron 32 progenitores de adolescentes. Tras aceptar participar, se evaluó: clima familiar, satisfacción vital, regulación emocional, resiliencia y psicopatología. Se realizaron modelos de análisis comparativo cualitativo de conjuntos difusos (fsQCA). Nuestros…

Alarm stateCoronavirus disease 2019 (COVID-19)Qualitative comparative analysismedia_common.quotation_subjectEmotional regulationLife satisfactionGeneral Medicinemedicine.diseasemedicinePsychological resiliencePsychologySomatizationmedia_commonPsychopathologyClinical psychologyAnálisis y Modificación de Conducta
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Regulation of microRNAs and their role in liver development, regeneration and disease

2013

Abstract Since their discovery more than a decade ago microRNAs have been demonstrated to have profound effects on almost every aspect of biology. Numerous studies in recent years have shown that microRNAs have important roles in development and in the etiology and progression of disease. This review is focused on microRNAs and the roles they play in liver development, regeneration and liver disease; particularly chronic liver diseases such as alcoholic liver disease, non-alcoholic fatty liver disease, non-alcoholic steatohepatitis, viral hepatitis and primary liver cancer. The key microRNAs identified in liver development and chronic liver disease will be discussed together with, where pos…

Alcoholic liver diseaseLiver DiseasesFatty liverCell BiologyDiseaseBiologyBioinformaticsmedicine.diseaseChronic liver diseaseBiochemistryLiver RegenerationMicroRNAsLiver diseaseGene Expression RegulationLivermicroRNAmedicineAnimalsHumansSteatohepatitisViral hepatitisThe International Journal of Biochemistry & Cell Biology
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Aldehyde dehydrogenase and HSP90 co-localize in human glioblastoma biopsy cells.

2013

The concept of a stem cell subpopulation as understood from normal epithelial tissue or bone marrow function has been extended to our understanding of cancer tissue and is now the target of treatment efforts specifically directed to this subpopulation. In glioblastoma, as well as in other cancers, increased expression of aldehyde dehydrogenase (ALDH) has been found localized within a minority sub-population of tumor cells which demonstrate stem cell properties. A separate body of research associated increased expression of heat-shock protein-90 (HSP90) with stem cell attributes. We present here results from our initial immunohistochemistry study of human glioblastoma biopsy tissue where bot…

Aldehyde dehydrogenasePharmacologyBiochemistryAldehyde dehydrogenase; Disulfiram; Glioblastoma; HSP90; Ritonavir; Stem cell; TemozolomideCancer stem cellBiopsyDisulfirammedicineTemozolomideHSP90HumansHSP90 Heat-Shock ProteinsTemozolomideRitonavirStem cellmedicine.diagnostic_testbiologyCancerGeneral MedicineAldehyde Dehydrogenasemedicine.diseaseGene Expression Regulation NeoplasticProtein Transportmedicine.anatomical_structurebiology.proteinCancer researchImmunohistochemistryBone marrowStem cellGlioblastomamedicine.drugBiochimie
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Alterations in the Emotional Regulation Process in Gambling Addiction: The Role of Anger and Alexithymia

2017

This study aims at the assessment of alexithymia and anger levels in 100 treatment-seeking pathological gamblers compared with controls, who were matched for age, gender and education. Furthermore a positive correlation between alexithymia, anger and severity of gambling disorder and a relationship between gambling behaviour and anger after controlling for alexithymia, are investigated. Finally the role that gender plays in anger in pathological gamblers was also evaluated. Psychological assessment includes the South Oaks Gambling Screen, State-Trait Anger Expression Inventory-2 and the twenty-item Toronto Alexithymia Scale. Statistical analysis of the results shows a higher level of anger …

AlexithymiaAdultMalemedicine.medical_specialtyPsychology (all)Personality InventorySociology and Political Sciencemedia_common.quotation_subjectEmotionsBehavioural addictionAngerAngerGambling disorderbehavioral disciplines and activities03 medical and health sciencesToronto Alexithymia Scale0302 clinical medicineAlexithymiaSettore M-PSI/08 - Psicologia Clinicamental disordersmedicineHumansPsychological testingAffective SymptomsPsychiatryPathologicalGeneral Psychologymedia_commonEmotionPathological gamblingmedicine.diagnostic_testAffective SymptomAddictionEmotional regulationmedicine.disease030227 psychiatryBehavior AddictiveDisordered GamblingGamblingbehavior and behavior mechanismsGambling disorderFemalePsychology030217 neurology & neurosurgerypsychological phenomena and processesHuman
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