Search results for " Regulator"

showing 10 items of 728 documents

Excessive CpG 1668 stimulation triggers IL-10 production by cDC that inhibits IFN-alpha responses by pDC.

2008

Upon stimulation with a wide range of concentrations of CpG oligodeoxynucleotide 2216 (CpG 2216), plasmacytoid DC are induced to produce type I IFN (IFN-alpha/beta). In contrast, CpG 1668 shows a bell-shaped dose-response correlation, i.e. only intermediate but not high doses of CpG 1668 induce IFN-alpha/beta. Interestingly, high-dose CpG 1668 completely inhibited IFN-alpha responses induced by CpG 2216. Experiments using supernatant of high-dose CpG-1668-treated cells indicated that secreted inhibitor(s) mediated the IFN-alpha shut-off. Among modulating cytokines, IL-10 turned out to be one important negative regulator. In line with this, supernatants of IL-10-deficient DC cultures stimula…

MuromegalovirusCpG OligodeoxynucleotideImmunologyStimulationmedicine.disease_causeNegative regulatorAutoimmunityMiceAdjuvants ImmunologicmedicineImmunology and AllergyAnimalsCells CulturedbiologyTLR9Interferon-alphaDendritic Cellsbiology.organism_classificationMolecular biologyInterleukin-10Interleukin 10CpG siteOligodeoxyribonucleotidesVesicular stomatitis virusToll-Like Receptor 9ImmunologyCytokinesEuropean journal of immunology
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Investigation of the roles of AgrA and σB regulators in Listeria monocytogenes adaptation to roots and soil

2020

ABSTRACT Little is known about the regulatory mechanisms that ensure the survival of the food-borne bacterial pathogen Listeria monocytogenes in the telluric environment and on roots. Earlier studies have suggested a regulatory overlap between the Agr cell–cell communication system and the general stress response regulator σB. Here, we investigated the contribution of these two systems to root colonisation and survival in sterilised and biotic soil. The ability to colonise the roots of the grass Festuca arundinacea was significantly compromised in the double mutant (∆agrA∆sigB). In sterile soil at 25°C, a significant defect was observed in the double mutant, suggesting some synergy between …

MutantPopulationSoil survivalRoots colonizationSigma Factor[SDV.SA.SDS]Life Sciences [q-bio]/Agricultural sciences/Soil studymedicine.disease_causeMicrobiologyPlant RootsAgrA σBMicrobiology03 medical and health sciencesListeria monocytogenesBacterial Proteinstranscription regulatorsGeneticsmedicineeducationMolecular BiologyGenePathogenSoil Microbiology030304 developmental biology2. Zero hunger0303 health scienceseducation.field_of_studybiology030306 microbiology15. Life on landbiology.organism_classificationAdaptation PhysiologicalListeria monocytogenesColonisation[SDV.MP]Life Sciences [q-bio]/Microbiology and Parasitology13. Climate actionAdaptationFestuca arundinacea
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Functional analysis of endo-1,4-β-glucanases in response to Botrytis cinerea and Pseudomonas syringae reveals their involvement in plant-pathogen int…

2013

Plant cell wall modification is a critical component in stress responses. Endo-1,4-β-glucanases (EGs) take part in cell wall editing processes, e.g. elongation, ripening and abscission. Here we studied the infection response of Solanum lycopersicum and Arabidopsis thaliana with impaired EGs. Transgenic TomCel1 and TomCel2 tomato antisense plants challenged with Pseudomonas syringae showed higher susceptibility, callose priming and increased jasmonic acid pathway marker gene expression. These two EGs could be resistance factors and may act as negative regulators of callose deposition, probably by interfering with the defence-signalling network. A study of a set of Arabidopsis EG T-DNA insert…

Mutantendo-glucanasesArabidopsisGene ExpressionPseudomonas syringaePlant ScienceCyclopentanestomatoGenes PlantMarker genechemistry.chemical_compoundBotrytis cinereaCellulaseSolanum lycopersicumPlant Growth RegulatorsCell WallGene Expression Regulation PlantArabidopsisBotanyPseudomonas syringaeArabidopsis thalianaOxylipinsGlucansEcology Evolution Behavior and SystematicsBotrytis cinereaDisease ResistancePlant DiseasesPlant ProteinsbiologyJasmonic acidCallosefungifood and beveragesGeneral Medicinebiology.organism_classificationdefence responseCell biologychemistryHost-Pathogen Interactionscell wallBotrytisSignal TransductionPlant biology (Stuttgart, Germany)
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Friend retrovirus infection of myeloid dendritic cells impairs maturation, prolongs contact to naïve T cells, and favors expansion of regulatory T ce…

2007

AbstractRetroviruses have developed immunmodulatory mechanisms to avoid being attacked by the immune system. The mechanisms of this retrovirus-associated immune suppression are far from clarified. Dendritic cells (DCs) have been attributed a decisive role in these pathogenic processes. We have used the Friend retrovirus (FV) mouse model in order to acquire further knowledge about the role of infection of DCs in virus-induced immunosuppression. About 20% of the myeloid DCs that were generated from the bone marrow of FV-infected mice carried FV proteins. The infection was productive, and infected DCs transmitted the virus in cell culture and in vivo. FV infection of DCs led to a defect in DC …

MyeloidImmunologyPopulationMedizinBone Marrow CellsMice Transgenicchemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationT-Lymphocytes RegulatoryBiochemistryMiceImmune systemAntigenImmune TolerancemedicineAnimalsCytotoxic T cellMyeloid CellseducationCell ProliferationAntigen PresentationMice Inbred BALB Ceducation.field_of_studyFollicular dendritic cellsModels ImmunologicalFOXP3hemic and immune systemsDendritic CellsCell BiologyHematologyFriend murine leukemia virusCell biologymedicine.anatomical_structureImmunologyBone marrowRetroviridae InfectionsBlood
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Effect of an Insect Growth Regulator (Halofenozide) on the Cuticular Hydrocarbons ofCulex pipiensLarvae

2013

The cuticular hydrocarbons of the fourth-instar larvae of Culex pipiens aged one and six days were analysed by gas chromatography and gas chromatography-mass spectrometry. The effect of an insect growth regulator (halofenozide = RH-0345) on the cuticular hydrocarbons was evaluated using newly moulted fourth-instar larvae of C. pipiens, at the lethal concentrations (LC50 = 12.58 and LC90 = 28.58 µ/l). A previous study revealed that RH-0345 is toxic to larvae of C. pipiens by induction of precocious larval moulting. Several cuticular hydrocarbons (CHCs) were identified and many of the other compounds remained unknown. Our data show that the CHC classes consisted of n-alkanes (C23-C29) and dif…

N alkanesLarvaanimal structuresCuticlefungiBiologybiology.organism_classificationInsect ScienceBotanyCulex pipiensInsect growth regulatorGas chromatographyHalofenozideAgronomy and Crop ScienceMoultingEcology Evolution Behavior and SystematicsAfrican Entomology
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Increased immunosuppressive function of CD4(+)CD25(+)Foxp3(+)GITR+ T regulatory cells from NFATc2((-/-)) mice controls allergen-induced experimental …

2012

The expansion of effector T cells is tightly controlled by transcription factors like nuclear factor of activated T cells (NFAT) family members that mediate early intracellular responses to T cell receptor-mediated signals. In this study we show that, after allergen challenge, NFATc2((-/-)) mice had augmented number of functionally intact CD4(+)CD25(++)GITR(++) T regulatory (T regs) cells in the lung. Anti-GITR antibody treatment inhibited T regulatory cell function and enhanced the number of activated lung CD4(+) T cells associated with increased IL-2 and pSTAT-5 in the airways of NFATc2((-/-)) mice in experimental allergic asthma. This agonistic treatment led to increased inflammation in …

NFATC2T cellImmunologyInflammationBiologyT-Lymphocytes RegulatoryInterleukin 21MiceTh2 CellsGlucocorticoid-Induced TNFR-Related ProteinmedicineImmunology and AllergyAnimalsIL-2 receptorLungImmunosuppression TherapyMice KnockoutNFATC Transcription FactorsInterleukin-2 Receptor alpha SubunitFOXP3NFATForkhead Transcription FactorsHematologyAllergensAsthmarespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureImmunologyCD4 Antigensbiology.proteinCytokinesTh17 CellsFemalemedicine.symptomAntibodySpleenImmunobiology
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IL-10 Controls Ultraviolet-Induced Carcinogenesis in Mice

2007

Abstract UV radiation-induced immunosuppression contributes significantly to the development of UV-induced skin cancer by inhibiting protective immune responses. IL-10 has been shown to be a key mediator of UV-induced immunosuppression. To investigate the role of IL-10 during photocarcinogenesis, groups of IL-10+/+, IL-10+/−, and IL-10−/− mice were chronically irradiated with UV. IL-10+/+ and IL-10+/− mice developed skin cancer to similar extents, whereas IL-10−/− mice were protected against the induction of skin malignancies by UV. Because UV is able to induce regulatory T cells, which play a role in the suppression of protective immunity, UV-induced regulatory T cell function was analyzed…

Neoplasms Radiation-InducedSkin NeoplasmsUltraviolet RaysRegulatory T cellImmunologyMice NudeBiologymedicine.disease_causeT-Lymphocytes RegulatoryMiceImmune systemImmunityImmune TolerancemedicineAnimalsImmunology and AllergyIL-2 receptorMice KnockoutMolecular biologyInterleukin-10Mice Inbred C57BLInterleukin 10medicine.anatomical_structureImmunologyGranzyme ACytokinesCarcinogenesisCD8The Journal of Immunology
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Sorafenib, but not sunitinib, affects function of dendritic cells and induction of primary immune responses

2008

AbstractThe tyrosine kinase inhibitors sorafenib and sunitinib are approved for the treatment of patients with malignant diseases. To analyze the possible use of these compounds in combination with immunotherapeutic approaches, we analyzed the effects of both inhibitors on the immunostimulatory capacity of human dendritic cells (DCs) and the induction of primary immune responses in vivo. Sorafenib, but not sunitinib, inhibits function of DCs, characterized by reduced secretion of cytokines and expression of CD1a, major histocompatibility complex, and costimulatory molecules in response to TLR ligands as well as by their impaired ability to migrate and stimulate T-cell responses. These inhib…

NiacinamideSorafenibIndolesPyridinesImmunologyAntineoplastic AgentsApoptosisCD8-Positive T-LymphocytesPharmacologyBiologyurologic and male genital diseasesMajor histocompatibility complexT-Lymphocytes RegulatoryBiochemistryPeripheral blood mononuclear cellMiceImmune systemCell MovementIn vivoSunitinibmedicineAnimalsHumansCytotoxic T cellPyrrolesCells CulturedSunitinibPhenylurea CompoundsBenzenesulfonatesGranulocyte-Macrophage Colony-Stimulating FactorDextransDendritic CellsCell BiologyHematologySorafenibEndocytosisfemale genital diseases and pregnancy complicationsMice Inbred C57BLToll-Like Receptor 4biology.proteinCytokinesFemaleInterleukin-4Lymphocyte Culture Test MixedTyrosine kinaseCell DivisionSignal Transductionmedicine.drugBlood
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The Endothelial Transcription Factor ERG Mediates a Differential Role in the Aneurysmatic Ascending Aorta with Bicuspid or Tricuspid Aorta Valve: A P…

2022

The pathobiology of ascending aorta aneurysms (AAA) onset and progression is not well understood and only partially characterized. AAA are also complicated in case of bicuspid aorta valve (BAV) anatomy. There is emerging evidence about the crucial role of endothelium-related pathways, which show in AAA an altered expression and function. Here, we examined the involvement of ERG-related pathways in the differential progression of disease in aortic tissues from patients having a BAV or tricuspid aorta valve (TAV) with or without AAA. Our findings identified ERG as a novel endothelial-specific regulator of TGF-β-SMAD, Notch, and NO pathways, by modulating a differential fibrotic or calcified A…

NotchHeart Valve DiseasesCatalysisInorganic ChemistryBicuspid Aortic Valve DiseaseTranscriptional Regulator ERGascending aorta aneurysm; bicuspid aorta valve; tricuspid aorta valve; ERG transcriptional factor pathway; TGF-β-SMAD; Notch; NO pathways modulationTransforming Growth Factor betaSettore MED/05 - Patologia ClinicaHumansTGF-β-SMADEndotheliumPhysical and Theoretical ChemistryMolecular BiologySpectroscopyAortabicuspid aorta valveOrganic ChemistryERG transcriptional factor pathwayGeneral Medicineascending aorta aneurysmComputer Science ApplicationsSettore MED/23Aortic Valvetricuspid aorta valveNO pathways modulationBiomarkersTranscription FactorsInternational Journal of Molecular Sciences; Volume 23; Issue 18; Pages: 10848
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Gatekeeper of pluripotency: A common Oct4 transcriptional network operates in mouse eggs and embryonic stem cells

2011

Abstract Background Oct4 is a key factor of an expanded transcriptional network (Oct4-TN) that governs pluripotency and self-renewal in embryonic stem cells (ESCs) and in the inner cell mass from which ESCs are derived. A pending question is whether the establishment of the Oct4-TN initiates during oogenesis or after fertilisation. To this regard, recent evidence has shown that Oct4 controls a poorly known Oct4-TN central to the acquisition of the mouse egg developmental competence. The aim of this study was to investigate the identity and extension of this maternal Oct4-TN, as much as whether its presence is circumscribed to the egg or maintained beyond fertilisation. Results By comparing …

Octamer Transcription Factor-3lcsh:QH426-470lcsh:BiotechnologycellsGene regulatory networkDown-RegulationBiologyTranscriptomeMicelcsh:TP248.13-248.65GeneticsInner cell massAnimalsGene Regulatory NetworksEmbryonic Stem Cellsreproductive and urinary physiologyOligonucleotide Array Sequence AnalysisGeneticsGene Expression ProfilingfungiEmbryoEmbryonic stem cellGene expression profilinglcsh:GeneticsMultigene FamilyCancer cellembryonic structuresOocytesFemalebiological phenomena cell phenomena and immunityFunction and Dysfunction of the Nervous SystemOctamer Transcription Factor-3Research ArticleBiotechnologyBMC Genomics
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