Search results for " Repair"

showing 10 items of 721 documents

Influence of DNA Repair on Nonlinear Dose-Responses for Mutation

2013

Recent evidence has challenged the default assumption that all DNA-reactive alkylating agents exhibit a linear dose-response. Emerging evidence suggests that the model alkylating agents methyl- and ethylmethanesulfonate and methylnitrosourea (MNU) and ethylnitrosourea observe a nonlinear dose-response with a no observed genotoxic effect level (NOGEL). Follow-up mechanistic studies are essential to understand the mechanism of cellular tolerance and biological relevance of such NOGELs. MNU is one of the most mutagenic simple alkylators. Therefore, understanding the mechanism of mutation induction, following low-dose MNU treatment, sets precedence for weaker mutagenic alkylating agents. Here, …

Hypoxanthine PhosphoribosyltransferaseMethyltransferaseDNA RepairDNA repairBiologyToxicologymedicine.disease_causePolymerase Chain ReactionCell Linechemistry.chemical_compoundalkylating agentsmedicineHumansnon-linearDNA Modification Methylasesgenetic toxicologyHypoxanthineDNA Primersdose-responsemutagenBase SequenceDose-Response Relationship DrugTumor Suppressor ProteinsgenotoxicityMutagenesisrisk assessmentDNA adductsO-6-methylguanine-DNA methyltransferaseMolecular biologyDNA Repair EnzymeschemistryMutationNOGELGenotoxicityMutagensResearch ArticleHypoxanthine PhosphoribosyltransferaseEthylnitrosoureaToxicological Sciences
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Styrene Metabolism, Genotoxicity, and Potential Carcinogenicity

2006

This report reviews styrene biotransformation, including minor metabolic routes, and relates metabolism to the genotoxic effects and possible styrene-related carcinogenicity. Styrene is shown to require metabolic activation in order to become notably genotoxic and styrene 7,8-oxide is shown to contribute quantitatively by far the most (in humans more than 95%) to the genotoxicity of styrene, while minor ring oxidation products are also shown to contribute to local toxicities, especially in the respiratory system. Individual susceptibility depending on metabolism polymorphisms and individual DNA repair capacity as well as the dependence of the nonlinearity of the dose-response relationships …

Individual susceptibilityDNA repairStyrene metabolismDNAMetabolismBiologymedicine.disease_causeStyrenesStyreneDNA Adductschemistry.chemical_compoundBiochemistrychemistryBiotransformationCarcinogensmedicineAnimalsHumansPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsBiotransformationGenotoxicityCarcinogenDNA DamageMutagensDrug Metabolism Reviews
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Portrait of inflammatory response to ionizing radiation treatment

2015

Ionizing radiation (IR) activates both pro-and anti-proliferative signal pathways producing an imbalance in cell fate decision. IR is able to regulate several genes and factors involved in cell-cycle progression, survival and/or cell death, DNA repair and inflammation modulating an intracellular radiation-dependent response. Radiation therapy can modulate anti-tumour immune responses, modifying tumour and its microenvironment. In this review, we report how IR could stimulate inflammatory factors to affect cell fate via multiple pathways, describing their roles on gene expression regulation, fibrosis and invasive processes. Understanding the complex relationship between IR, inflammation and …

Ionizing radiationDNA repairFibrosimedicine.medical_treatmentClinical BiochemistryInflammationReviewCell fate determinationBioinformaticsImmune systemMedicineCytokineRegulation of gene expressionInflammationInvasivenebusiness.industryCancerIonizing radiation Inflammation Cytokine Fibrosis InvasivenessCell Biologymedicine.diseaseFibrosisInvasivenessRadiation therapyCytokineCancer researchmedicine.symptombusinessJournal of Inflammation
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Combined inguinal hernia in the elderly. Portraying the progression of hernia disease

2016

Abstract Introduction Identification of a combined hernia is a common occurrence in the course of inguinal hernia repair. This type of protrusion disease seems to affect the elderly, in particular. Very few investigations have been carried out to ascertain the structural changes that occur in the groin affected by this clinical condition. Method Analysis of intraoperative findings of combined inguinal hernias evidenced in the elderly, from the most recent 100 groin hernia repair procedures carried out by a single operator, represents the basis of the article. Protrusions that presumably represent the forerunner of this type of hernia were also analyzed: double ipsilateral inguinal hernias c…

Ipsilateral herniaAdultMalemedicine.medical_specialtyHerniamedicine.medical_treatmentHernia InguinalDisease030230 surgery03 medical and health sciencesYoung Adult0302 clinical medicinemedicineHumansHerniaIn patientAge FactorHerniorrhaphyAgedAged 80 and overGroinbusiness.industryCombined herniaInguinal herniaAge FactorsGeneral MedicineMiddle Agedmedicine.diseaseHernia repairSurgeryInguinal herniaCombined hernia; Hernia; Inguinal hernia; Ipsilateral hernia; Adult; Age Factors; Aged; Aged 80 and over; Disease Progression; Female; Hernia Inguinal; Herniorrhaphy; Humans; Male; Middle Aged; Young Adult; SurgeryCombined hernia; Hernia; Inguinal hernia; Ipsilateral hernia; Surgerysurgical procedures operativemedicine.anatomical_structure030220 oncology & carcinogenesisDisease ProgressionFemaleSurgerybusinessInferior epigastric vesselsProgressive diseaseHuman
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The elemental role of iron in DNA synthesis and repair

2017

Iron is an essential redox element that functions as a cofactor in many metabolic pathways. Critical enzymes in DNA metabolism, including multiple DNA repair enzymes (helicases, nucleases, glycosylases, demethylases) and ribonucleotide reductase, use iron as an indispensable cofactor to function. Recent striking results have revealed that the catalytic subunit of DNA polymerases also contains conserved cysteine-rich motifs that bind iron–sulfur (Fe/S) clusters that are essential for the formation of stable and active complexes. In line with this, mitochondrial and cytoplasmic defects in Fe/S cluster biogenesis and insertion into the nuclear iron-requiring enzymes involved in DNA synthesis a…

Iron-Sulfur Proteins0301 basic medicineDNA RepairDNA polymeraseDNA damageDNA repairIronBiophysicsDNA repairEukaryotic DNA replicationSaccharomyces cerevisiaeBiochemistryDNA GlycosylasesBiomaterials03 medical and health sciencesRibonucleotide ReductasesHumansProtein–DNA interactionRibonucleotide reductaseReplication protein Achemistry.chemical_classificationDNA ligaseDeoxyribonucleasesDNA synthesis030102 biochemistry & molecular biologybiologyIron deficiencyDNA HelicasesMetals and AlloysHelicaseDNAYeast030104 developmental biologyIron cofactorBiochemistrychemistryChemistry (miscellaneous)biology.proteinIron-sulfur clusterMetallomics
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The Friedreich's Ataxia protein frataxin modulates DNA base excision repair in prokaryotes and mammals

2010

DNA-repair mechanisms enable cells to maintain their genetic information by protecting it from mutations that may cause malignant growth. Recent evidence suggests that specific DNA-repair enzymes contain ISCs (iron–sulfur clusters). The nuclearencoded protein frataxin is essential for the mitochondrial biosynthesis of ISCs. Frataxin deficiency causes a neurodegenerative disorder named Friedreich's ataxia in humans. Various types of cancer occurring at young age are associated with this disease, and hence with frataxin deficiency. Mice carrying a hepatocyte-specific disruption of the frataxin gene develop multiple liver tumours for unresolved reasons. In the present study, we show that frata…

Iron-Sulfur ProteinsDNA Repairmedicine.disease_causeBiochemistryDNA Glycosylases8-oxoG 78-dihydro-8-oxoguanineMice0302 clinical medicineIron-Binding Proteinsoxidative stressBER base excision repairCells CulturedMammalsMice Knockout0303 health sciencesfrataxinDMEM Dulbecco's modified Eagle's mediumbiologyLiver NeoplasmsSalmonella entericairon–sulfur clusterLife SciencesIron-binding proteinsTransfection3. Good healthLB Luria–BertaniOGG1 8-oxoguanine DNA glycosylase 1ISC iron–sulfur clusterFpg formamido-pyrimidine DNA glycosylaseHPRT hypoxanthine phosphoribosyltransferaseResearch ArticleDNA damageDNA repairSSB DNA single-strand breakTransfectionCell Line03 medical and health sciencesFRDA Friedreich's ataxiaROS reactive oxygen speciesmedicineAnimalsHumansMUTYH human mutY homologue (Escherichia coli)Molecular BiologyGene030304 developmental biologyFriedreich's ataxiaCell BiologyFibroblastsMolecular biologytumorigenesisProkaryotic CellsFriedreich AtaxiaDNA base excision repairDNA glycosylaseMutationHepatocytesFrataxinbiology.proteinInstitut für ErnährungswissenschaftCarcinogenesisMAPK mitogen-activated protein kinase030217 neurology & neurosurgeryDNA Damage
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Do nonmelanoma skin cancers develop from extra-cutaneous stem cells?

2008

A hypothesis is presented that nonmelanoma skin cancers can develop from extra-cutaneous stem cells, and not exclusively from skin keratinocytes. This idea is supported by recent findings regarding the initiation of cancers in the digestive tract, and by a cancer stem cell model of a neoplasia. It is known that multipotent adult progenitor cells can trans-differentiate into very diverse cellular lineages and can be recruited to areas of profound tissue injury. In these settings, they might also initiate malignant transformation. Some epidemiological data and recent findings regarding mechanisms of wound healing indicate that skin cancers could also originate from bone marrow-derived or othe…

KeratinocytesCancer ResearchPathologymedicine.medical_specialtySkin NeoplasmsBone Marrow CellsCancer stem cellepidermisAnimalsHumansMedicineProgenitor cellSkin repairintegumentary systembusiness.industryStem Cellsmedicine.diseasehematopoietic stem cellsCell Transformation Neoplasticmedicine.anatomical_structureOncologyBone marrowSkin cancerStem cellbusinessKeratinocyteWound healingInternational Journal of Cancer
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Genetic ablation of mast cells redefines the role of mast cells in skin wound healing and bleomycin-induced fibrosis.

2014

Conclusive evidence for the impact of mast cells (MCs) in skin repair is still lacking. Studies in mice examining the role of MC function in the physiology and pathology of skin regenerative processes have obtained contradictory results. To clarify the specific role of MCs in regenerative conditions, here we used a recently developed genetic mouse model that allows conditional MC ablation to examine MC-specific functions in skin. This mouse model is based on the cell type–specific expression of Cre recombinase in connective tissue–type MCs under control of the Mcpt5 promoter and the Cre-inducible diphtheria toxin receptor–mediated cell lineage ablation by diphtheria toxin. In response to ex…

KeratinocytesPathologymedicine.medical_specialtymedicine.medical_treatmentCellCre recombinaseMice TransgenicDermatologyBiologyBleomycinBiochemistrySkin Diseaseschemistry.chemical_compoundBleomycinMiceFibrosismedicineLeukocytesAnimalsMast CellsMolecular BiologyDiphtheria toxinSkin repairWound HealingAntibiotics AntineoplasticGranulation tissueCell BiologyAblationmedicine.diseaseFibrosisDisease Models Animalmedicine.anatomical_structurechemistryGranulation TissueThe Journal of investigative dermatology
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Minimum Leading Edge Protection Application Length to Combat Rain-Induced Erosion of Wind Turbine Blades

2021

Leading edge erosion (LEE) repairs of wind turbine blades (WTBs) involve infield application of leading edge protection (LEP) solutions. The industry is currently aiming to use factory based LEP coatings that can applied to the WTBs before they are shipped out for installation. However, one of the main challenges related to these solutions is the choice of a minimum LEP application length to be applied in the spanwise direction of the WTBs. Generally, coating suppliers apply 10–20 m of LEP onto the blades starting from the tip of the blade using the “rule of thumb”, and no studies in the literature exist that stipulate how these LEP lengths can be calculated. In this study, we extend the sc…

Leading edgeControl and OptimizationTurbine blade020209 energyEnergy Engineering and Power Technology020101 civil engineeringcoatings02 engineering and technologylcsh:TechnologyTurbine0201 civil engineeringlaw.inventionWind turbine bladesCoatingslawleading edge erosionwind energy0202 electrical engineering electronic engineering information engineeringSensitivity (control systems)Electrical and Electronic EngineeringWind energyEngineering (miscellaneous)Parametric statisticsWind powerlcsh:TRenewable Energy Sustainability and the Environmentbusiness.industrywind turbine blades; leading edge erosion; wind energy; repair; coatingswind turbine bladesRule of thumbPower (physics)VDP::Teknologi: 500repairEnvironmental sciencebusinessRepairLeading edge erosionEnergy (miscellaneous)Marine engineeringEnergies
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Theories and Mechanisms of Aging

2013

The more one learns about single processes and genes known to be involved in aging, the more it becomes evident that these are connected and there is no unifying theory of aging. The individual theories put individual factors and processes in focus and for each theory there are direct links to life span or to age-related disorders. In the following chapter, the key theories of aging focusing on telomeres, DNA damage, oxidative stress as well as possible roles of nutrition, the interplay between genes and environment (epigenetics) and cellular protein homeostasis are presented. In animal models the life span can be altered by targeting specific genes, proteins and signalling pathways. After …

Life spanDNA repairDNA damageProcess (engineering)EpigeneticsBiologyNeuroscienceSignalling pathwaysCellular proteinTelomere
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